Thematische Bibliographien / Neurosecretion

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile

Auswahl der wissenschaftlichen Literatur zum Thema „Neurosecretion“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 9. November 2024

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Zeitschriftenartikel zum Thema "Neurosecretion"

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Grundschober, Christophe, Maria Luisa Malosio, Laura Astolfi, Tiziana Giordano, Patrick Nef und Jacopo Meldolesi. „Neurosecretion Competence“. Journal of Biological Chemistry 277, Nr. 39 (17.06.2002): 36715–24. http://dx.doi.org/10.1074/jbc.m203777200.

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Robinson, Linda J., und Thomas FJ Martin. „Docking and fusion in neurosecretion“. Current Opinion in Cell Biology 10, Nr. 4 (August 1998): 483–92. http://dx.doi.org/10.1016/s0955-0674(98)80063-x.

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Predel, R., und Manfred Eckert. „Neurosecretion: peptidergic systems in insects“. Naturwissenschaften 87, Nr. 8 (25.08.2000): 343–50. http://dx.doi.org/10.1007/s001140050737.

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Brosius, D. C., J. T. Hackett und J. B. Tuttle. „Ca(2+)-independent and Ca(2+)-dependent stimulation of quantal neurosecretion in avian ciliary ganglion neurons“. Journal of Neurophysiology 68, Nr. 4 (01.10.1992): 1229–34. http://dx.doi.org/10.1152/jn.1992.68.4.1229.

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1. Although it is generally agreed that Ca2+ couples depolarization to the release of neurotransmitters, hypertonic saline and ethanol (ETOH) evoke neurosecretion independent of extracellular Ca2+. One possible explanation is that these agents release Ca2+ from an intracellular store that then stimulates Ca(2+)-dependent neurosecretion. An alternative explanation is that these agents act independently of Ca2+. 2. This work extends previous observations on the action of ETOH and hypertonic solutions (HOSM) on neurons to include effects on [Ca2+]i. We have looked for Ca(2+)-independent or -dependent neurosecretion evoked by these agents in parasympathetic postganglionic neurons dissociated from chick ciliary ganglia and maintained in tissue culture. The change in concentration of free Ca2+ in the micromolar range inside neurons ([Ca2+]i) was measured with indo-1 with the use of a Meridian ACAS 470 laser scanning microspectrophotometer. 3. Elevated concentration of extracellular KCl increased [Ca2+]i and the frequency of quantal events. Also, a twofold increase in osmotic pressure (HOSM) produced a similar increase in quantal release and a significant rise in [Ca2+]i; however, the Ca2+ appeared to come from intracellular stores. 4. In contrast, ETOH stimulated quantal neurosecretion without a measurable change in [Ca2+]i. It appears the alcohol exerts its influence on some stage in the process of exocytosis that is distal to or independent of the site of Ca2+ action. 5. The effects of high [KCl]o and osmotic pressure were occlusive. This is explained in part by the observation that hypertonicity reduced Ca2+ current, but an action on Ca2+ stores is also likely.(ABSTRACT TRUNCATED AT 250 WORDS)
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Mishra, Nirmal Kumar. „Neurosecretion in reproductive behaviour of leeches“. Journal of Biosciences 35, Nr. 3 (07.08.2010): 327–28. http://dx.doi.org/10.1007/s12038-010-0036-0.

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Galoyan, A. „Neurosecretory Hypothalamus-Endocrine Heart as a Functional System“. Physiology 7, Nr. 6 (01.12.1992): 279–83. http://dx.doi.org/10.1152/physiologyonline.1992.7.6.279.

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Cardiac atrial neurosecretion is precisely regulated by cardiotropic protein-hormonal complexes, formed by neurosecretory nuclei of the hypothalamus. There is a close neurohumoral interrelation between the neuroendocrine heart and the hypothalamus.
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Dunlap, Karthleen, Jennifer I. Luebke und Timothy J. Turner. „Identification of Calcium Channels That Control Neurosecretion“. Science 266, Nr. 5186 (04.11.1994): 828–30. http://dx.doi.org/10.1126/science.266.5186.828.b.

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Westerink, R. H. S., und A. G. Ewing. „The PC12 cell as model for neurosecretion“. Acta Physiologica 192, Nr. 2 (15.11.2007): 273–85. http://dx.doi.org/10.1111/j.1748-1716.2007.01805.x.

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Dunlap, K., J. Luebke und T. Turner. „Identification of calcium channels that control neurosecretion“. Science 266, Nr. 5186 (04.11.1994): 828. http://dx.doi.org/10.1126/science.7973643.

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Dunlap, K., J. I. Luebke und T. J. Turner. „Identification of Calcium Channels That Control Neurosecretion“. Science 266, Nr. 5186 (04.11.1994): 828–30. http://dx.doi.org/10.1126/science.266.5186.828-a.

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Dissertationen zum Thema "Neurosecretion"

1

Pupier, Sandrine. „Role des cysteine string proteins dans la neurosecretion“. Aix-Marseille 2, 1997. http://www.theses.fr/1997AIX20668.

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Moore, S. E. H. „Biochemical aspects of neurosecretion in the posterior pituitary gland“. Thesis, University of Oxford, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375290.

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Richards, S. J. „Hypothalamic neurosecretion with special reference to the Brattleboro rat“. Thesis, Open University, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.484408.

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Leoni, Chiara. „Molecular interactions between neurosecretion and neurite extension in PC12 subclones“. Thesis, Open University, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310217.

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Dumpala, Bindya. „Design, construction, optimization, and characterization of a temperature control system for studying the effects of a rapid and reversible changes in temperature on neurosecretion“. abstract and full text PDF (free order & download UNR users only), 2006. http://0-gateway.proquest.com.innopac.library.unr.edu/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1438916.

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Guldenaar, S. E. F. „Questions about the localization of oxytocin and vasopressin in gonads and brain : Some answers fron immunocytochemistry“. Thesis, University of Bristol, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375018.

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Huang, Chunwei. „Influence of dopamine receptor activation on neurosecretion from perfused rat hypothalamo-neurohypophysial explants“. Thesis, McGill University, 1992. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=56990.

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The hypothalamic supraoptic nucleus (SON) contains both vasopressin-secreting (AVP) and oxytocin-secreting (OT) magnocellular neurons which release their respective hormones from the posterior pituitary gland into the systemic circulation. A direct relationship has been demonstrated in earlier physiological studies between the excitability of SON neurons and hormone release from the neurohypophysis. Catecholamines have been shown to activate SON neurons, thereby increasing the action potential frequency to the neural lobe. The present study examined the ability of catecholamines to evoke release of vasopressin and/or oxytocin from the neural lobe of an intraarterially perfused preparation of rat hypothalamo-neurohypophysis.
Experiments were carried out on male Long-Evans rats. Drugs (noradrenaline, dopamine, D1 and D2 dopamine receptor agonists) were delivered both into the perfusion medium and directly over the SON. Fractions of effluent medium were collected from the area of the neurohypophysis and tested for hormone release using a selective radioimmunoassay. Noradrenaline (NA) in concentrations of 10$ sp{-5}$ to 10$ sp{-4}$ M stimulated AVP release from neural lobe. The $ alpha sb1$-adrenergic receptor antagonist, prazosin (50 nM in perfusion experiments; 1$ mu$M in local injection experiments) blocked the noradrenaline-stimulated release. Dopamine (10$ sp{-4}$ to 10$ sp{-3}$ M), D1 (SKF 38393, 0.5 mM) and D2 (QNP 0.5 mM) dopamine receptor agonists also increased the release of AVP. In the presence of prazosin, DA, QNP and SKF 38393 still evoked AVP release. Therefore, it is proposed that both noradrenaline and dopamine stimulate the release of AVP in this preparation; the responses to dopamine appear to be mediated by both D1 and D2 dopamine receptors, presumably located at the level of the cell somata/dendrites, possibly also on axon terminals in the neural lobe.
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Wolf, Alexander. „A molecular toolbox to study the pleiotropic functions of phosphatidic acid in neurosecretion“. Electronic Thesis or Diss., Strasbourg, 2024. http://www.theses.fr/2024STRAJ033.

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La neurosecrétion nécessite une coopération entre différents acteurs protéiques pour orchestrer le transport, la fusion et le recyclage de vésicules sécrétrices. Cependant les lipides, composants majeurs des membranes cellulaires, ont été moins étudiées lors de ce processus, principalement à cause d’un manque d’outils. Ainsi lors de ma thèse, j’ai implémenté deux approches pour étudier les fonction pléïotropiques d’un lipide de signalisation : l’acide phosphatidique (PA). L’utilisation d’analogues du PA modifié chimiquement m’a permis d’établir leur dynamique ainsi que leurs interactomes directement dans des cellules neurosécrétrices. Une seconde approche optogénétique permet de moduler les niveaux intracellulaires de PA avec une résolution spatiale et temporelle sans précédent pour établir les fonctions non-redondantes de différents pools de PA subcellulaires. Ainsi, grâce à cette nouvelle boite à outil moléculaire, j’ai pu approfondir notre compréhension des rôles du PA dans la neurosécrétion
Neurosecretion requires multiple proteins acting together to orchestrate transport, fusion and recycling of secretory vesicles. However, contribution of lipids, main components of cellular membranes, have been much less studied in these processes, primarily because of a lack of adequate tools. Hence, during my thesis, I implemented two novel approaches to study the pleiotropic functions of a peculiar signaling lipid: phosphatidic acid (PA). The use of chemically modified PA click-analogues enabled me to establish their dynamics as well as to characterize their interactome directly within neurosecretory cells. A second approach based on the optogenetic targeting of enzymes involved in the PA metabolism modulated its cellular levels with an unprecedented spatial and temporal resolution. This work allowed to unravel the non-redundant functions of various subcellular PA pools. Our novel molecular toolbox for PA, represents a breakthrough in lipid biology that will greatly improve our understanding of the roles of PA in the neurosecretory process
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Cimino, Irène. „Study of new molecular factors regulating GnRH migration, axonal targeting and neurosecretion : insights into the acquisition of reproductive competence“. Thesis, Lille 2, 2014. http://www.theses.fr/2014LIL2S044/document.

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Chez les mammifères, la reproduction est regulée par des neurones spécifiques qui sécrètent le neuropeptide GnRH (Gonadotropin Releasing Hormone). Ces cellules naissent au stade prénatal dans la placode nasale et migrent dans l'hypothalamus, le long des nerfs olfactifs voméro-nasaux, pour devenir des membres à part entière de l'axe hypothalamo-hypophyso-gonadique. Un certain nombre de pathologies de la reproduction humaine sont associées à la perturbation soit de la migration neuronale des cellules à GnRH ou soit de la sécrétion de la GnRH.L'objectif général de ma thèse était d'identifier de nouveaux facteurs moléculaires régulant la migration des cellules à GnRH, leur ciblage axonale à l'éminence médiane, mais aussi leur neurosécrétion au cours de la vie reproductive.Les événements complexes du développement correcte du système à GnRH sont strictement régulés par l'expression spatio-temporelles des molecules de guidage et des molécules de la matrice extracellulaire, dont les fonctions, sont en partie médiées par leur liaison avec la β1-intégrine (Itgb1). La première partie de mon travail a été d’étudier le rôle biologique de ces protéines de surface dans la reproduction. La technologie Cre/loxP a été utilisée pour générer des souris conditionnelles GnRH spécifiques KO pour la β1-intégrine (GnRH-Itgb1-/-). La perte d’activité de la β1-intégrine altére la migration des neurones à GnRH, leur extension axonale à l’eminence mediane et la fertilité de ces souris. Ces résultats mettent en évidence que la β1-intégrine joue un rôle important dans le développement normal du système GnRH et dans l'acquisition de compétences reproductives normales chez les rongeurs.Dans la deuxième partie de ma thèse de doctorat, j'ai identifié de nouveaux facteurs moléculaires qui pourraient être responsables de l'apparition du syndrôme des ovaires polykystiques (SOPK). Cette maladie est présente chez près de 10 % des femmes. Il s’agit d’une hyperandrogénie associée à une oligo-anovulation chronique, une morphologie ovarienne polykystique et d'autres situations cliniques de transition d'un état endocrinien à un autre. Chez les patientes atteintes du SOPK, le niveau d'hormone antimüllérienne (AMH) est élevé et indique clairement que l'AMH pourrait est un marqueur possible dans le diagnostic et le traitement du SOPK. Une autre manifestation du syndrome est une élévation des sécrétions du GnRH provoquant une augmentation des taux de LH et un rapport LH/FSH élevés, qui stimulent la production d'androgènes ovariens. Toutefois, jusqu'à présent cette maladie a été considérée principalement comme une pathologie gonadique et des régulations possibles plus élevées au niveau du système nerveux central ou des interactions avec ce dernier n'ont pas été étudiées. En particulier, des informations concernant les effets extra-ovariens possibles de l'AMH sur l'axe hypothalamo-hypophyso-gonadique manquent actuellement. Mon projet de recherche a été d’étudier le rôle encore méconnu de l'AMH dans la régulation de la physiologie du système à GnRH. Mes études ont permis d’identifier un nouveau rôle extra-ovarien pour l'AMH, et notamment comme un puissant activateur de la neurosécrétion de la GnRH
During aging, oxidative stress occurs characterized by an imbalance between the production reactive oxygen species and antioxidant capacity. It’s well recognized that acute exercise induces oxidative stress which response may be affected by aging. Only few studies have focused on the response of oxidative stress parameters to an acute exercise in relation with aging and they were not made in humans. The first aim of this work was to investigate the response of oxidative stress parameters to acute exercise in young and older subjects. Our results showed that aging has no effects on oxidative stress parameters at rest. However, in response to an acute physical exercise, our results showed that aging is characterized by an antioxidant defenses deficiency and an increase in free radical damage markers. On the other hand, regular physical activity is considered as an effective way to reduce free radical attacks and enhance the antioxidant defense. These adaptations to regular physical activity are often related to the level of physical activity and this has been shown in young subjects. The second aim of this work was to study oxidative stress parameters in elderly subjects with different physical activity levels. Our results showed a positive correlation between physical activity level and antioxidant potential. However, physical activity at high level increases free radical damage in older adults. In view of changes in oxidative stress parameters with aging, adaptations of those to regular physical activity could also be affected by aging phenomena. The aim of the third study of this work was to investigate the effects of aging and physical activity level on oxidative stress parameters responses to an acute exercise. To do so, we compared these parameters in two young subjects groups (active and sedentary) and two older adults groups (active and sedentary) before and after an acute exercise. Our results showed that, benefits of regular physical activity on the oxidative parameters stress were more pronounced in younger age groups compared to older groups. On the other hand, the effects of aging on oxidative stress parameters in the active groups were lower than those noted in the sedentary groups
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Scobie, David Roger. „Short term effects of stress hormones on cell division rate in wool follicles : a thesis submitted in fulfilment of the requirements for the degree of Doctor of Philosophy“. Title page, abstract and contents only, 1992. http://web4.library.adelaide.edu.au/theses/09PH/09phs421.pdf.

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Includes bibliographical references (leaves 183-207) A local intradermal technique using colchicine to estimate cell division rate in wool follicles is refined and used throughout the thesis. Statistical methods used to analyse data obtained with this method are described and discussed. The implications of the findings are of great significance to research into the influence of physiological changes on wool production, and suggest experiments should be conducted under controlled environmental conditions, with a minimum of stress imposed on the animals.
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Bücher zum Thema "Neurosecretion"

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Pickering, Brian T., Jonathan B. Wakerley und Alastair J. S. Summerlee, Hrsg. Neurosecretion. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1.

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Lemos, José R., und Govindan Dayanithi, Hrsg. Neurosecretion: Secretory Mechanisms. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-22989-4.

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International Symposium on Neurosecretion (9th Susono-shi, Japan). Neurosecretion and the biology of neuropeptides: Proceedings of the Ninth International Symposium on Neurosecretion. Tokyo: Japan Scientific Societies Press, 1985.

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Elissa, Romano, und De Luca Sara, Hrsg. New research on neurosecretory systems. New York: Nova Science Publishers, 2008.

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International Symposium on Neurosecretion (10th 1987 Bristol, England). Neurosecretion: Cellular aspects of the production and release of neuropeptides. New York: Plenum Press, 1988.

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Kenkyūjo, Gunma Daigaku Naibunpi, und Gunma Symposium on Endocrinology (26th : 1988 : Maebashi-shi, Japan), Hrsg. Cell biology of secretion. Tokyo: Center for Academic Publications Japan, 1989.

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Fink, George, Donald W. Pfaff und Jon E. Levine. Handbook of neuroendocrinology. Amsterdam: Academic Press/Elsevier, 2012.

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S, Penkava Nejc, und Haight Logan R, Hrsg. Neuroendocrinology research developments. Hauppauge, N.Y: Nova Science, 2009.

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Marcella, Motta, Hrsg. Brain endocrinology. 2. Aufl. New York: Raven Press, 1991.

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Van de Kar, Louis D., Hrsg. Methods in neuroendocrinology. Boca Raton: CRC Press, 1998.

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Buchteile zum Thema "Neurosecretion"

1

Kovács, L., und B. Lichardus. „Neurosecretion“. In Vasopressin, 19–29. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0449-1_3.

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Sachs, Howard. „Neurosecretion“. In Advances in Enzymology - and Related Areas of Molecular Biology, 327–72. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2006. http://dx.doi.org/10.1002/9780470122778.ch8.

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Arch, S., und H. Gainer. „Neurosecretion“. In Neurochemical Systems, 281–307. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-7018-5_12.

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Gainer, Harold, Miriam Altstein und Yoshinobu Hara. „Oxytocin and Vasopressin: After the Genes, What Next?“ In Neurosecretion, 1–10. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1_1.

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Tribollet, E., C. Barberis, S. Jard, J. Elands, M. Dubois-Dauphin, A. Marguerat und J. J. Dreifuss. „Mapping and Analysis of Receptors for Neurohypophyseal Peptides Present in the Brain“. In Neurosecretion, 81–87. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1_10.

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Chan-Palay, Victoria. „Somatostatin and Neuropeptide Y: Coexistence in the Hippocampus and Alterations in Alzheimer’s Disease“. In Neurosecretion, 89–97. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1_11.

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O’Shea, Michael. „Bioactive Peptides at the Neuromuscular Junction of Insects“. In Neurosecretion, 99–105. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1_12.

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Bilbe, Graeme, und H. Chica Schaller. „The Role of Head Activator in Cell Growth and Control Processes“. In Neurosecretion, 107–12. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1_13.

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Morris, John, David Pow und Fraser Shaw. „Release of Neuropeptides from Magnocellular Neurones: Does Anatomical Compartmentation Have a Functional Significance?“ In Neurosecretion, 113–22. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1_14.

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Roubos, E. W. „Biosynthesis and Release of Multiple Peptides by the Caudodorsal Cells of Lymnaea Stagnalis“. In Neurosecretion, 123–35. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5502-1_15.

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