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Thematische Bibliographien / Mitotic spindle orientations

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Zeitschriftenartikel

Auswahl der wissenschaftlichen Literatur zum Thema „Mitotic spindle orientations“

Autor: Grafiati

Veröffentlicht am 25. Mai 2024

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Zeitschriftenartikel zum Thema "Mitotic spindle orientations"

1

Wang, S. W., F. J. Griffin, and W. H. Clark. "Cell-cell association directed mitotic spindle orientation in the early development of the marine shrimp Sicyonia ingentis." Development 124, no. 4 (1997): 773–80. http://dx.doi.org/10.1242/dev.124.4.773.

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During early cleavages of Sicyonia ingentis embryos, mitotic spindle orientations differ between blastomeres and change in a predictable manner with each successive mitosis. From 2nd through 7th cleavages, spindles orient at a 90 degrees angle with respect to the spindle of the parent blastomere. Thus, spindle orientation is parallel to the cleavage plane that formed the blastomere. To determine if specific spindle orientations were intrinsic properties of individual blastomeres, we altered blastomere associations and asked how mitotic spindle orientation was affected in successive cleavages u

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2

Juschke, C., Y. Xie, M. P. Postiglione, and J. A. Knoblich. "Analysis and modeling of mitotic spindle orientations in three dimensions." Proceedings of the National Academy of Sciences 111, no. 3 (2013): 1014–19. http://dx.doi.org/10.1073/pnas.1314984111.

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3

Chan, Derek C. H., Joshua Xu, Ana Vujovic, et al. "Arhgef2 regulates mitotic spindle orientation in hematopoietic stem cells and is essential for productive hematopoiesis." Blood Advances 5, no. 16 (2021): 3120–33. http://dx.doi.org/10.1182/bloodadvances.2020002539.

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Abstract How hematopoietic stem cells (HSCs) coordinate their divisional axis and whether this orientation is important for stem cell–driven hematopoiesis is poorly understood. Single-cell RNA sequencing data from patients with Shwachman-Diamond syndrome (SDS), an inherited bone marrow failure syndrome, show that ARHGEF2, a RhoA-specific guanine nucleotide exchange factor and determinant of mitotic spindle orientation, is specifically downregulated in SDS hematopoietic stem and progenitor cells (HSPCs). We demonstrate that transplanted Arhgef2−/− fetal liver and bone marrow cells yield impaire

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4

Sei, Yoshitatsu, Jianying Feng, Carson C. Chow, and Stephen A. Wank. "Asymmetric cell division-dominant neutral drift model for normal intestinal stem cell homeostasis." American Journal of Physiology-Gastrointestinal and Liver Physiology 316, no. 1 (2019): G64—G74. http://dx.doi.org/10.1152/ajpgi.00242.2018.

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The normal intestinal epithelium is continuously regenerated at a rapid rate from actively cycling Lgr5-expressing intestinal stem cells (ISCs) that reside at the crypt base. Recent mathematical modeling based on several lineage-tracing studies in mice shows that the symmetric cell division-dominant neutral drift model fits well with the observed in vivo growth of ISC clones and suggests that symmetric divisions are central to ISC homeostasis. However, other studies suggest a critical role for asymmetric cell division in the maintenance of ISC homeostasis in vivo. Here, we show that the stocha

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5

Copp, A. J., F. A. Brook, and H. J. Roberts. "A cell-type-specific abnormality of cell proliferation in mutant (curly tail) mouse embryos developing spinal neural tube defects." Development 104, no. 2 (1988): 285–95. http://dx.doi.org/10.1242/dev.104.2.285.

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The mouse mutant curly tail (ct) provides a model system for studies of neurulation mechanisms. 60% of ct/ct embryos develop spinal neural tube defects (NTD) as a result of delayed neurulation at the posterior neuropore whereas the remaining 40% of embryos develop normally. In order to investigate the role of cell proliferation during mouse neurulation, cell cycle parameters were studied in curly tail embryos developing spinal NTD and in their normally developing litter-mates. Measurements were made of mitotic index, median length of S-phase and percent reduction of labelling index during a [3

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6

Li, Jie, Hiroki Shima, Hironari Nishizawa, et al. "Phosphorylation of BACH1 switches its function from transcription factor to mitotic chromosome regulator and promotes its interaction with HMMR." Biochemical Journal 475, no. 5 (2018): 981–1002. http://dx.doi.org/10.1042/bcj20170520.

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The transcription repressor BACH1 performs mutually independent dual roles in transcription regulation and chromosome alignment during mitosis by supporting polar ejection force of mitotic spindle. We now found that the mitotic spindles became oblique relative to the adhesion surface following endogenous BACH1 depletion in HeLa cells. This spindle orientation rearrangement was rescued by re-expression of BACH1 depending on its interactions with HMMR and CRM1, both of which are required for the positioning of mitotic spindle, but independently of its DNA-binding activity. A mass spectrometry an

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7

Kapoor, Tarun M., Thomas U. Mayer, Margaret L. Coughlin, and Timothy J. Mitchison. "Probing Spindle Assembly Mechanisms with Monastrol, a Small Molecule Inhibitor of the Mitotic Kinesin, Eg5." Journal of Cell Biology 150, no. 5 (2000): 975–88. http://dx.doi.org/10.1083/jcb.150.5.975.

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Monastrol, a cell-permeable small molecule inhibitor of the mitotic kinesin, Eg5, arrests cells in mitosis with monoastral spindles. Here, we use monastrol to probe mitotic mechanisms. We find that monastrol does not inhibit progression through S and G2 phases of the cell cycle or centrosome duplication. The mitotic arrest due to monastrol is also rapidly reversible. Chromosomes in monastrol-treated cells frequently have both sister kinetochores attached to microtubules extending to the center of the monoaster (syntelic orientation). Mitotic arrest–deficient protein 2 (Mad2) localizes to a sub

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Woodard, Geoffrey E., Ning-Na Huang, Hyeseon Cho, Toru Miki, Gregory G. Tall та John H. Kehrl. "Ric-8A and Giα Recruit LGN, NuMA, and Dynein to the Cell Cortex To Help Orient the Mitotic Spindle". Molecular and Cellular Biology 30, № 14 (2010): 3519–30. http://dx.doi.org/10.1128/mcb.00394-10.

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ABSTRACT In model organisms, resistance to inhibitors of cholinesterase 8 (Ric-8), a G protein α (Gα) subunit guanine nucleotide exchange factor (GEF), functions to orient mitotic spindles during asymmetric cell divisions; however, whether Ric-8A has any role in mammalian cell division is unknown. We show here that Ric-8A and Gαi function to orient the metaphase mitotic spindle of mammalian adherent cells. During mitosis, Ric-8A localized at the cell cortex, spindle poles, centromeres, central spindle, and midbody. Pertussis toxin proved to be a useful tool in these studies since it blocked th

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Siletti, Kimberly, Basile Tarchini, and A. J. Hudspeth. "Daple coordinates organ-wide and cell-intrinsic polarity to pattern inner-ear hair bundles." Proceedings of the National Academy of Sciences 114, no. 52 (2017): E11170—E11179. http://dx.doi.org/10.1073/pnas.1716522115.

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The establishment of planar polarization by mammalian cells necessitates the integration of diverse signaling pathways. In the inner ear, at least two systems regulate the planar polarity of sensory hair bundles. The core planar cell polarity (PCP) proteins coordinate the orientations of hair cells across the epithelial plane. The cell-intrinsic patterning of hair bundles is implemented independently by the G protein complex classically known for orienting the mitotic spindle. Although the primary cilium also participates in each of these pathways, its role and the integration of the two syste

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10

Giansanti, M. G., M. Gatti, and S. Bonaccorsi. "The role of centrosomes and astral microtubules during asymmetric division of Drosophila neuroblasts." Development 128, no. 7 (2001): 1137–45. http://dx.doi.org/10.1242/dev.128.7.1137.

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Drosophila neuroblasts are stem cells that divide asymmetrically to produce another large neuroblast and a smaller ganglion mother cell (GMC). During neuroblast division, several cell fate determinants, such as Miranda, Prospero and Numb, are preferentially segregated into the GMC, ensuring its correct developmental fate. The accurate segregation of these determinants relies on proper orientation of the mitotic spindle within the dividing neuroblast, and on the correct positioning of the cleavage plane. In this study we have analyzed the role of centrosomes and astral microtubules in neuroblas

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