Auswahl der wissenschaftlichen Literatur zum Thema „Mitochondrial apoptosis“
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Zeitschriftenartikel zum Thema "Mitochondrial apoptosis"
Adhihetty, Peter J., Vladimir Ljubicic und David A. Hood. „Effect of chronic contractile activity on SS and IMF mitochondrial apoptotic susceptibility in skeletal muscle“. American Journal of Physiology-Endocrinology and Metabolism 292, Nr. 3 (März 2007): E748—E755. http://dx.doi.org/10.1152/ajpendo.00311.2006.
Der volle Inhalt der QuelleParsons, Melissa J., und Douglas R. Green. „Mitochondria in cell death“. Essays in Biochemistry 47 (14.06.2010): 99–114. http://dx.doi.org/10.1042/bse0470099.
Der volle Inhalt der QuelleHeikaus, Sebastian, Linda van den Berg, Tobias Kempf, Csaba Mahotka, Helmut Erich Gabbert und Uwe Ramp. „HA14-1 is Able to Reconstitute the Impaired Mitochondrial Pathway of Apoptosis in Renal Cell Carcinoma Cell Lines“. Analytical Cellular Pathology 30, Nr. 5 (01.01.2008): 419–33. http://dx.doi.org/10.1155/2008/693095.
Der volle Inhalt der QuelleZamzami, N., S. A. Susin, P. Marchetti, T. Hirsch, I. Gómez-Monterrey, M. Castedo und G. Kroemer. „Mitochondrial control of nuclear apoptosis.“ Journal of Experimental Medicine 183, Nr. 4 (01.04.1996): 1533–44. http://dx.doi.org/10.1084/jem.183.4.1533.
Der volle Inhalt der QuelleMajewski, Nathan, Veronique Nogueira, R. Brooks Robey und Nissim Hay. „Akt Inhibits Apoptosis Downstream of BID Cleavage via a Glucose-Dependent Mechanism Involving Mitochondrial Hexokinases“. Molecular and Cellular Biology 24, Nr. 2 (15.01.2004): 730–40. http://dx.doi.org/10.1128/mcb.24.2.730-740.2004.
Der volle Inhalt der QuelleSeo, Young Ah, Veronica Lopez und Shannon L. Kelleher. „A histidine-rich motif mediates mitochondrial localization of ZnT2 to modulate mitochondrial function“. American Journal of Physiology-Cell Physiology 300, Nr. 6 (Juni 2011): C1479—C1489. http://dx.doi.org/10.1152/ajpcell.00420.2010.
Der volle Inhalt der QuelleTang, Ho Lam, Anh-Huy Phan Le und Hong Lok Lung. „The increase in mitochondrial association with actin precedes Bax translocation in apoptosis“. Biochemical Journal 396, Nr. 1 (26.04.2006): 1–5. http://dx.doi.org/10.1042/bj20060241.
Der volle Inhalt der QuelleMayer, Bernd, und Rainer Oberbauer. „Mitochondrial Regulation of Apoptosis“. Physiology 18, Nr. 3 (Juni 2003): 89–94. http://dx.doi.org/10.1152/nips.01433.2002.
Der volle Inhalt der QuelleSugioka, Rie, Shigeomi Shimizu und Yoshihide Tsujimoto. „Fzo1, a Protein Involved in Mitochondrial Fusion, Inhibits Apoptosis“. Journal of Biological Chemistry 279, Nr. 50 (30.09.2004): 52726–34. http://dx.doi.org/10.1074/jbc.m408910200.
Der volle Inhalt der QuelleSu, Ching-Chieh, Jia-Ying Yang, Hsin-Ban Leu, Yumay Chen und Ping H. Wang. „Mitochondrial Akt-regulated mitochondrial apoptosis signaling in cardiac muscle cells“. American Journal of Physiology-Heart and Circulatory Physiology 302, Nr. 3 (Februar 2012): H716—H723. http://dx.doi.org/10.1152/ajpheart.00455.2011.
Der volle Inhalt der QuelleDissertationen zum Thema "Mitochondrial apoptosis"
Sun, Mei Guo. „Mitochondrial structure during apoptosis“. Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3273480.
Der volle Inhalt der QuelleTitle from first page of PDF file (viewed August 31, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 129-140).
Joza, Nicholas. „Differential requirement for the mitochondrial apoptosis-inducing factor in apoptotic pathways“. Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/MQ63071.pdf.
Der volle Inhalt der QuelleSani, Marc-Antoine. „Apoptosis Regulation via the Mitochondrial Pathway : Membrane Response upon Apoptotic Stimuli“. Doctoral thesis, Umeå universitet, Kemiska institutionen, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1883.
Der volle Inhalt der QuelleSani, Marc Antoine. „Apoptosis regulation via the mitochondrial pathway : membrane response upon apoptotic stimuli“. Thesis, Bordeaux 1, 2008. http://www.theses.fr/2008BOR13651/document.
Der volle Inhalt der QuelleThe aim of this thesis was the investigation of the mitochondrial response mechanisms upon apoptotic stimuli. The specific objectives were the biophysical characterization of membrane dynamics and the specific roles of lipids in the context of apoptotic regulation occurring at the mitochondrion and its complex membrane systems. The BH4 domain is an anti-apoptotic specific domain of the Bcl-2 protein. Solid phase peptide synthesis was used to produce large amount of the peptide for biophysical studies. A protocol has been established and optimized, guarantying the required purity for biophysical studies. In detail the purification by high performance liquid chromatography and the characterisation via mass spectroscopy are described. The secondary structure of BH4 changes significantly in the presence of lipid vesicles as observed by infrared spectroscopy and circular dichroism. The BH4 peptide aggregates at the membrane surface and inserts slightly into the hydrophobic part of the membrane. Using nuclear magnetic resonance (NMR) and calorimetry techniques, it could even be shown that the BH4 domain modifies the dynamic and organization of the liposomes which mimic a mitochondrial surface. The second study was on the first helix of the pro-apoptotic protein Bax. This sequence called Bax-a1 has the function to address the cytosolic Bax protein to the mitochondrial membrane upon activation. Once again a protocol has been established for the synthesis and purification of this peptide. The aim was to elucidate the key role of cardiolipin, a mitochondria-specific phospholipid, in the interaction of Bax-a1 with the mitochondrial membrane system. The NMR and circular dichroism studies showed that Bax-a1 interacts with the membrane models only if they contain the cardiolipin, producing a strong electrostatic lock effect which is located at the membrane surface. Finally, a new NMR approach was developed which allows the investigation of the lipid response of isolated active mitochondria upon the presence of apoptotic stimuli. The goal was there to directly monitor lipid specific the occurring changes during these physiological activities
Zhao, Ming. „The lysosomal-mitochondrial axis theory of apoptosis /“. Linköping : Univ, 2002. http://www.bibl.liu.se/liupubl/disp/disp2002/med747s.pdf.
Der volle Inhalt der QuelleBender, Cheryl E. „The mitochondrial pathway of apoptosis in invertebrates“. Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC IP addresses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3288845.
Der volle Inhalt der QuelleTitle from first page of PDF file (viewed June 2, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 190-218).
Wang, Jianming. „Life without mitochondrial DNA : studies of transgenic mice /“. Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4491-1/.
Der volle Inhalt der QuelleCipolat, Sara. „From mitochondrial morphology to apoptosis: genetic analysis of OPA1 function and regulation“. Doctoral thesis, Università degli studi di Padova, 2008. http://hdl.handle.net/11577/3425557.
Der volle Inhalt der QuelleKatz, Elad. „Mitochondrial regulation of apoptosis during B cell selection“. Thesis, University of Glasgow, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327569.
Der volle Inhalt der QuelleOkaro, Madubuchi Chiedu. „Mitochondrial directed apoptosis sensitising studies in cholangiocarcinoma cells“. Thesis, University College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412629.
Der volle Inhalt der QuelleBücher zum Thema "Mitochondrial apoptosis"
Joza, Nicholas. Differential requirement for the mitochondrial apoptosis-inducing factor in apoptotic pathways. Ottawa: National Library of Canada, 2001.
Den vollen Inhalt der Quelle findenWadia, Jehangir S. R(-)-deprenyl treatment blocks apoptosis in PC12 cells by affecting mitochondrial membrane potential, mitochondrial calcium and superoxide radical generation. Ottawa: National Library of Canada, 1996.
Den vollen Inhalt der Quelle findenSaavedra-Molina, Alfredo. Mitochondrial dysfunctions related to oxidative stress. Hauppauge, N.Y: Nova Science Publishers, 2010.
Den vollen Inhalt der Quelle findenWadia, J. S. Changes in mitochondrial protein import during apoptosis. 2002.
Den vollen Inhalt der Quelle findenLee, Hong Kyu, Salvatore DiMauro, Masashi Tanaka und Yau-Huei Wei. Mitochondrial Pathogenesis: From Genes and Apoptosis to Aging and Disease. Springer London, Limited, 2014.
Den vollen Inhalt der Quelle findenMitochondrial Pathogenesis: From Genes and Apoptosis to Aging and Disease (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2004.
Den vollen Inhalt der Quelle findenSeth, Rohit. Zinc deficiency induces apoptosis via mitochondrial p53- and caspase-dependent pathways in human neuronal precursor cells. Elseveir, 2014.
Den vollen Inhalt der Quelle findenJoza, Nicholas. Genetic elucidation of the roles of apoptosis-inducing factor (AIF) in mitochondrial respiration and programmed cell death. 2005.
Den vollen Inhalt der Quelle findenKorea) Asian Society for Mitochondrial Research and Medicine. Scientific Meeting (1st : 2003 : Seoul und Hong Kyu Lee. Mitochondrial Pathogenesis: From Genes and Apoptosis to Aging and Disease (Annals of the New York Academy of Sciences, V. 1011). New York Academy of Sciences, 2004.
Den vollen Inhalt der Quelle findenLestienne, Patrick. Mitochondrial Diseases: Models and Methods. Springer, 2011.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Mitochondrial apoptosis"
Mignotte, B., und G. Kroemer. „Roles of Mitochondria in Apoptosis“. In Mitochondrial Diseases, 239–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_18.
Der volle Inhalt der QuelleAntonsson, Bruno. „The Mitochondrial Apoptosis Pathway“. In Essentials of Apoptosis, 85–99. Totowa, NJ: Humana Press, 2003. http://dx.doi.org/10.1007/978-1-59259-361-3_6.
Der volle Inhalt der QuellePeluso, G., O. Petillo, S. Margarucci, A. Calarco und M. Calvani. „Deregulation of Mitochondrial Apoptosis in Cancer“. In Mitochondrial Disorders, 71–87. Paris: Springer Paris, 2002. http://dx.doi.org/10.1007/978-2-8178-0929-8_7.
Der volle Inhalt der QuelleLee, Myung-Shik, Ja-Young Kim und Sun Young Park. „Resistance of ρ0 Cells against Apoptosis“. In Mitochondrial Pathogenesis, 146–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-41088-2_15.
Der volle Inhalt der QuelleCleland, Megan M., und Richard J. Youle. „Mitochondrial Dynamics and Apoptosis“. In Mitochondrial Dynamics and Neurodegeneration, 109–38. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1291-1_4.
Der volle Inhalt der QuelleServidei, S., S. Di Giovanni, A. Broccolini, A. D’amico, M. Mirabella und G. Silvestri. „Apoptosis and Oxidative Stress in Mitochondrial Disorders“. In Mitochondrial Disorders, 37–45. Paris: Springer Paris, 2002. http://dx.doi.org/10.1007/978-2-8178-0929-8_4.
Der volle Inhalt der QuellePetit, Patrice X., und Guido Kroemer. „Mitochondrial Regulation of Apoptosis“. In Mitochondrial DNA Mutations in Aging, Disease and Cancer, 147–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-12509-0_8.
Der volle Inhalt der QuelleEl-Osta, Hazem, und Magdalena L. Circu. „Mitochondrial ROS and Apoptosis“. In Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease, 1–23. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42139-1_1.
Der volle Inhalt der QuellePetit, Patrice Xavier, Naoufal Zamzami, Jean-Luc Vayssière, Bernard Mignotte, Guido Kroemer und Maria Castedo. „Implication of mitochondria in apoptosis“. In Detection of Mitochondrial Diseases, 185–88. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-6111-8_28.
Der volle Inhalt der QuelleCampello, Silvia, und Luca Scorrano. „The Mitochondrial Pathway: Focus on Shape Changes“. In Essentials of Apoptosis, 151–75. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-381-7_6.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Mitochondrial apoptosis"
Luo, Yu, Zhuoyan Zhang und David Kessel. „Role of mitochondrial photodamage in PDT-induced apoptosis“. In BiOS '98 International Biomedical Optics Symposium, herausgegeben von Thomas J. Dougherty. SPIE, 1998. http://dx.doi.org/10.1117/12.308138.
Der volle Inhalt der QuellePourzia, Alexandra, Michael Olson, Stefanie Bailey, Aditi Aryal, Jeremy Ryan, Marcela Maus und Anthony Letai MD. „269 Mitochondrial apoptosis mediates CAR T cell cytotoxicity“. In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0269.
Der volle Inhalt der QuelleZebo, Tang, Liu Yanbo, Li Chun, Wen Na und Gai Xiaodong. „pLXSN-Tum-5 inducing HUVEC apoptosis through mitochondrial pathway“. In 2011 International Conference on Human Health and Biomedical Engineering (HHBE). IEEE, 2011. http://dx.doi.org/10.1109/hhbe.2011.6027904.
Der volle Inhalt der QuelleLee, Yuan-Hao, Exing Wang, Neeru Kumar und Randolph D. Glickman. „Ursolic acid mediates photosensitization by initiating mitochondrial-dependent apoptosis“. In SPIE BiOS, herausgegeben von E. Duco Jansen und Robert J. Thomas. SPIE, 2013. http://dx.doi.org/10.1117/12.2000225.
Der volle Inhalt der QuelleHamacher-Brady, Anne, Verena Lang und Nathan R. Brady. „Abstract 3324: FATE1 promotes mitochondrial hyperfusion and supports maintenance of mitochondrial networks following apoptosis stimulation“. In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3324.
Der volle Inhalt der QuelleZhuang, Cai-ping, Qian Liang, Xiao-ping Wang und Tong-sheng Chen. „Hydrogen peroxide induces apoptosis via a mitochondrial pathway in chondrocytes“. In SPIE BiOS, herausgegeben von Daniel L. Farkas, Dan V. Nicolau und Robert C. Leif. SPIE, 2012. http://dx.doi.org/10.1117/12.905786.
Der volle Inhalt der QuelleSui, Cliff, und Nada Boustany. „Potential application of the FDTD technique to study mitochondrial apoptosis“. In Biomedical Optics (BiOS) 2007, herausgegeben von Adam Wax und Vadim Backman. SPIE, 2007. http://dx.doi.org/10.1117/12.702151.
Der volle Inhalt der QuelleKichkina, D. O., S. S. Patrushev, A. D. Moralev, E. E. Shults, M. A. Zenkova und A. V. Markov. „NEW SEMISYNTHETIC SESQUITERPENE LACTONES AS INDUCERS OF OXIDATIVE STRESS IN TUMOR CELLS AND BLOCKERS OF THE AGGRESSIVE PHENOTYPE OF GLIOBLASTOMA MULTIFORME CELLS“. In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-331.
Der volle Inhalt der QuelleDasari, Venkata Ramesh, Swapna Asuthkar, Arun Kumar Nalla und Jasti S. Rao. „Abstract 1731: Decreased cyclin B1 expression contributes to mitochondrial apoptosis in medulloblastoma.“ In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1731.
Der volle Inhalt der QuelleHambright, Heather G., Addanki P. Kumar und Rita Ghosh. „Abstract 5466: Mitochondrial superoxide inhibits autophagy and induces apoptosis through SQSTM1-mediated mechanism“. In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5466.
Der volle Inhalt der QuelleBerichte der Organisationen zum Thema "Mitochondrial apoptosis"
Myers, Charles. Mitochondrial Apoptosis: A New Foundation for Combining Agents in Prostate Cancer Treatment. Fort Belvoir, VA: Defense Technical Information Center, März 2001. http://dx.doi.org/10.21236/ada402436.
Der volle Inhalt der QuelleTweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality,Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, Dezember 2013. http://dx.doi.org/10.21236/ada612817.
Der volle Inhalt der QuelleTweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, Dezember 2012. http://dx.doi.org/10.21236/ada612818.
Der volle Inhalt der QuelleMyers, Charles E. Mitochondrial Apoptosis: A New Foundation for Combing Agents in Prostate Cancer Treatment. Fort Belvoir, VA: Defense Technical Information Center, März 2000. http://dx.doi.org/10.21236/ada392324.
Der volle Inhalt der QuelleTweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction Using IL-6 as a Resuscitation Adjuvant. Fort Belvoir, VA: Defense Technical Information Center, Dezember 2011. http://dx.doi.org/10.21236/ada612819.
Der volle Inhalt der QuelleMarassi, Francesca M. Structural Basis for Bc12-Regulated Mitochondrion-Dependent Apoptosis. Fort Belvoir, VA: Defense Technical Information Center, April 2005. http://dx.doi.org/10.21236/ada437659.
Der volle Inhalt der QuelleMarassi, Francesca M. Structural Basis for Bcl-2-Regulated Mitochondrion-Dependent Apoptosis. Fort Belvoir, VA: Defense Technical Information Center, April 2004. http://dx.doi.org/10.21236/ada429719.
Der volle Inhalt der QuelleSick, Thomas J. Pro-Apoptotic Changes in Brain Mitochondria After Toxic Exposure. Fort Belvoir, VA: Defense Technical Information Center, Juli 2001. http://dx.doi.org/10.21236/ada397717.
Der volle Inhalt der QuelleSick, Thomas J. Pro-Apoptotic Changes in Brain Mitochondria After Toxin Exposure. Fort Belvoir, VA: Defense Technical Information Center, Oktober 2004. http://dx.doi.org/10.21236/ada434079.
Der volle Inhalt der QuelleChandra, Dhyan. A Novel Mitochondria-Dependent Apoptotic Pathway (MAP) in Prostate Cancer (PCa) Cells. Fort Belvoir, VA: Defense Technical Information Center, Januar 2003. http://dx.doi.org/10.21236/ada415386.
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