Thematische Bibliographien / Microencephaly

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Buchteile

Auswahl der wissenschaftlichen Literatur zum Thema „Microencephaly“

Autor: Grafiati
Veröffentlicht am 25. Juni 2021
Zuletzt aktualisiert am 17. Februar 2022

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Zeitschriftenartikel zum Thema "Microencephaly"

1

Balduini, Walter, Lucio G. Costa und Flaminio Cattabeni. „Molecular mechanisms involved in experimental microencephaly“. Pharmacological Research 22 (September 1990): 26. http://dx.doi.org/10.1016/s1043-6618(09)80082-0.

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2

Garbossa, Diego, und Alessandro Vercelli. „Experimentally-induced microencephaly: effects on cortical neurons“. Brain Research Bulletin 60, Nr. 4 (Mai 2003): 329–38. http://dx.doi.org/10.1016/s0361-9230(03)00053-4.

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3

Furukawa, Satoshi, Koji Usuda, Masayoshi Abe, Seigo Hayashi und Izumi Ogawa. „Indole-3-acetic acid induces microencephaly in mouse fetuses“. Experimental and Toxicologic Pathology 59, Nr. 1 (September 2007): 43–52. http://dx.doi.org/10.1016/j.etp.2006.12.001.

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4

Furukawa, Satoshi, Masayoshi Abe, Koji Usuda und Izumi Ogawa. „Indole-3-Acetic Acid Induces Microencephaly in Rat Fetuses“. Toxicologic Pathology 32, Nr. 6 (Oktober 2004): 659–67. http://dx.doi.org/10.1080/01926230490520269.

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5

Shapira Zaltsberg, G., H. McMillan und E. Miller. „P.067 Phosphoserine aminotransferase (PSAT) deficiency: Imaging findings in a child with congenital microcephaly“. Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 45, s2 (Juni 2018): S33. http://dx.doi.org/10.1017/cjn.2018.169.

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Background: Serine deficiency disorders can result from deficiency in one of three enzymes. Deficiency of the second enzyme in the serine biosynthesis pathway, 3-phosphoserine aminotransferase (PSAT), has been reported in two siblings when the eldest was investigated for acquired microcephaly, progressive spasticity and intractable epilepsy. Methods: Our patient had neurological symptoms apparent at birth. Fetal magnetic resonance imaging (MRI) at 35 weeks gestation demonstrated microencephaly and simplification of the the gyration (anterior>posterior) which was confirmed upon subsequent post-natal MRI. Congenital microcephaly was apparent at birth. Results: PSAT deficiency was confirmed when exome sequencing identified biallelic mutations in PSAT1; c.44C>T, p.Ala15Val and; c.432delA, p.Pro144fs and biochemical testing noted low plasma serine 22 mcmol/L (normal 83-212 mcmol/L) and low CSF serine 10 mcmol/L (normal 22-61 mcmol/L). Despite oral serine and glycine supplementation at 4 months old the patient showed little neurodevelopmental progress and developed epileptic spasms at 10 months old. Serological testing for TORCH infections was negative. Conclusions: PSAT deficiency should be considered for patients with congenital microcephaly. Although further characterization of MRI findings in other patients is required, microencephaly with simplified gyral pattern could provide imaging clues for this rare metabolic disorder.
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Cattabeni, F., M. P. Abbracchio, M. Cimino, D. Cocchi, M. Di Luca, L. Mennuni, F. Rosi und P. Zaratin. „Methylazoxymethanol-induced microencephaly: persistent increase of cortical somatostatin-like immunoreactivity“. Developmental Brain Research 47, Nr. 1 (Mai 1989): 156–59. http://dx.doi.org/10.1016/0165-3806(89)90120-x.

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7

Wichgers Schreur, P. J., L. van Keulen, D. Anjema, J. Kant und J. Kortekaas. „Microencephaly in fetal piglets following in utero inoculation of Zika virus“. Emerging Microbes & Infections 7, Nr. 1 (29.03.2018): 1–11. http://dx.doi.org/10.1038/s41426-018-0044-y.

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8

Chandra, P. S., N. Salamon, S. T. Nguyen, J. W. Chang, M. N. Huynh, C. Cepeda, J. P. Leite et al. „Infantile spasm-associated microencephaly in tuberous sclerosis complex and cortical dysplasia“. Neurology 68, Nr. 6 (05.02.2007): 438–45. http://dx.doi.org/10.1212/01.wnl.0000252952.62543.20.

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9

Naus, C. C. G., M. Cimino, G. R. Wood, M. Di Luca und F. Cattabeni. „Cellular expression of somatostatin in MAM-induced microencephaly in the rat“. Developmental Brain Research 70, Nr. 1 (November 1992): 39–46. http://dx.doi.org/10.1016/0165-3806(92)90101-2.

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10

Tamaru, Masao, Yukio Yoneda, Kiyokazu Ogita, Jun Shimizu, Tenhoshimaru Matsutani und Yutaka Nagata. „Excitatory amino acid receptors in brains of rats with methylazoxymethanol-induced microencephaly“. Neuroscience Research 14, Nr. 1 (Juni 1992): 13–25. http://dx.doi.org/10.1016/s0168-0102(05)80003-3.

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Dissertationen zum Thema "Microencephaly"

1

Mittelstadt, Brent. „Transforming the Brute : On the Ethical Acceptability of Creating Painless Animals“. Thesis, Linköping University, Centre for Applied Ethics, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-19447.

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Transforming the Brute addresses the ethical acceptability of creating painless animals for usage in biomedical experimentation.  In recent decades the possibility of creating genetically decerebrate animals or AMLs for human ends has been discussed in scientific, academic, and corporate communities.  While the ability to create animals that cannot feel, experience, and are more plant than animal remains science fiction, biomedicine may now be able to eliminate or significantly reduce the capacity to feel pain and nociception through genetic engineering.  With this new technology comes the opportunity to vastly increase the welfare of animals used in biomedical experimentation, yet this possibility has largely been ignored by the scientific and academic community.  This work seeks to reveal the moral necessity of creating painless animals for usage in biomedical experimentation for animal welfare ends.  Intrinsic objections relating to animal integrity, rights, companionship, the alteration of telos, humility and virtue are considered.  The benefit of eliminating nociceptive pain in experimental animals is addressed, and differences are examined between biomedical experimentation and other usage of animals for human ends which makes the proposed creation of painless animals ethically unique.  Finally, an argument is presented for the moral necessity of replacing normal animals with painless animals in biomedical experimentation with consideration given to genetically decerebrate animals.

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2

Shelton, Samantha. „Effects of Zika virus on neural precursor cell types and microencephaly in a model of direct embryonic murine brain infection“. Thesis, 2021. https://hdl.handle.net/2144/42703.

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Prenatal exposure to Zika virus (ZIKV) can result in microencephaly and congenital Zika syndrome but why some brain cells and structures are initially spared by the virus is unknown. Here, a novel murine model of ZIKV infection incorporating in utero electroporation with cell type specific promotors was used to identify the time course of ZIKV infection and to determine which neural precursor cells are initially infected or spared. In vivo time course studies revealed early presence of ZIKV in apical radial glial cells (aRGCs) while infection of basal intermediate progenitor cells climbed after three days of virus exposure. ZIKV-exposed fetal brains exhibited microencephaly as early as 1 day post injection, caused by apoptosis and reduced proliferation, and this change in brain size persisted until birth regardless of developmental age at infection. During infection, 60% of aRGC basal fibers were perturbed while 40% retained normal morphology, indicating that aRGCs are not uniformly vulnerable to ZIKV infection. To evaluate this heterogeneous vulnerability, we generated cell type-specific fate mapping plasmid probes using a previously published single cell RNA-Seq dataset on the E15.5 mouse neocortical wall. The results indicate that one class of aRGC preferentially expresses the putative ZIKV entry receptor AXL, and that these cells are more vulnerable to ZIKV infection than the other aRGC subtypes with low AXL expression. Together, these data highlight important temporal and cellular details of ZIKV fetal brain infection and may be important for prevention strategies and for management of congenital Zika syndrome.
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Buchteile zum Thema "Microencephaly"

1

„Microencephaly“. In Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 1207. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_10346.

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2

„Microencephaly“. In Encyclopedia of Clinical Neuropsychology, 1596. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_4820.

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3

Loupe, Pippa S., Stephen R. Schroeder und Richard E. Tessel. „The Behavior and Neurochemistry of the Methylazoxymethanol-Induced Microencephalic Rat“. In International Review of Research in Mental Retardation, 187–220. Elsevier, 1997. http://dx.doi.org/10.1016/s0074-7750(08)60280-3.

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