Auswahl der wissenschaftlichen Literatur zum Thema „Medial artery calcification“
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Zeitschriftenartikel zum Thema "Medial artery calcification"
Lehto, Seppo, Leo Niskanen, Matti Suhonen, Tapani Rönnemaa und Markku Laakso. „Medial Artery Calcification“. Arteriosclerosis, Thrombosis, and Vascular Biology 16, Nr. 8 (August 1996): 978–83. http://dx.doi.org/10.1161/01.atv.16.8.978.
Der volle Inhalt der QuelleArbustini, Eloisa, Antonio Bozzani und Francesco Prati. „Medial Artery Calcification“. JACC: Advances 2, Nr. 9 (November 2023): 100652. http://dx.doi.org/10.1016/j.jacadv.2023.100652.
Der volle Inhalt der QuelleMandalapu, Aniruddh, und Kathryn J. Stevens. „Imaging findings of arterial calcification due to deficiency of CD73: A case study“. Journal of Radiology Case Reports 17, Nr. 12 (09.02.2024): 27–33. http://dx.doi.org/10.3941/jrcr.v17i12.5175.
Der volle Inhalt der QuelleMeer, Romain, Anna G. Hoek, Emma J. Bouman, Teddo Doesburg, Petra J. M. Elders, Pim A. de Jong, Joline Beulens und UCC-SMART Study Group. „Association between lower extremity arterial calcification and coronary arterial calcification in a population at increased risk of cardiovascular disease“. BMJ Open Diabetes Research & Care 12, Nr. 1 (Februar 2024): e003811. http://dx.doi.org/10.1136/bmjdrc-2023-003811.
Der volle Inhalt der QuelleBobryshev, Yuri V., Reginald S. A. Lord und Dinh Tran. „Chlamydia pneumoniaein foci of “early” calcification of the tunica media in arteriosclerotic arteries: an incidental presence?“ American Journal of Physiology-Heart and Circulatory Physiology 290, Nr. 4 (April 2006): H1510—H1519. http://dx.doi.org/10.1152/ajpheart.01055.2005.
Der volle Inhalt der QuelleLiu, En-Shao, Nai-Ching Chen, Tzu-Ming Jao und Chien-Liang Chen. „Dextromethorphan Reduces Oxidative Stress and Inhibits Uremic Artery Calcification“. International Journal of Molecular Sciences 22, Nr. 22 (13.11.2021): 12277. http://dx.doi.org/10.3390/ijms222212277.
Der volle Inhalt der QuelleDogan, Omer Faruk, Musturay Karcaaltincaba, Umit Duman, Deniz Akata, Aytekin Besim und Erkmen Boke. „Assessment of the Radial Artery and Hand Circulation by Computed Tomography Angiography: A Pilot Study“. Heart Surgery Forum 8, Nr. 1 (16.02.2005): 28. http://dx.doi.org/10.1532/hsf98.20041042.
Der volle Inhalt der QuelleLiu, Huan, Ning-Yu Ru, Yue Cai, Qiang Lyu, Chi-Hua Guo, Ying Zhou, Shao-Hua Li et al. „The OPG/RANKL/RANK system modulates calcification of common carotid artery in simulated microgravity rats by regulating NF-κB pathway“. Canadian Journal of Physiology and Pharmacology 100, Nr. 4 (April 2022): 324–33. http://dx.doi.org/10.1139/cjpp-2021-0329.
Der volle Inhalt der QuelleHappy, Happy, S. M. Ear E-Mahabub, Nazir Uddin Mollah, Md Emran Hossain, Dr Halima Khatun und Md Magfur Rahman. „Breast Arterial Calcification on Mammography and Risk of Coronary Artery Disease“. Scholars Journal of Applied Medical Sciences 10, Nr. 5 (24.05.2022): 810–15. http://dx.doi.org/10.36347/sjams.2022.v10i05.023.
Der volle Inhalt der QuelleNiskanen, L., O. Siitonen, M. Suhonen und M. I. Uusitupa. „Medial Artery Calcification Predicts Cardiovascular Mortality in Patients With NIDDM“. Diabetes Care 17, Nr. 11 (01.11.1994): 1252–56. http://dx.doi.org/10.2337/diacare.17.11.1252.
Der volle Inhalt der QuelleDissertationen zum Thema "Medial artery calcification"
Teyssières, Salle Laurence. „Artériopathie des membres inférieurs chez le patient vivant avec un diabète de type 2 : marqueurs du risque et impact des thérapeutiques“. Electronic Thesis or Diss., Limoges, 2024. http://www.theses.fr/2024LIMO0031.
Der volle Inhalt der QuelleDespite diagnostic and therapeutic advances in the management of cardiovascular disease, it remains the leading cause of mortality in people living with type 2 diabetes (T2D). While coronary artery disease is increasingly screened for and managed optimally, peripheral artery disease remains underdiagnosed and undertreated, despite diabetes predisposes to more atypical, earlier, and severe forms. In individuals with T2D, the measurement of the ankle-brachial index (ABI), a validated screening tool, may be biased due to the presence of medial arterial calcification (MAC). In a population of 1119 asymptomatic patients with T2D, we demonstrated that the presence of ultrasound-detected MAC was independently associated with the occurrence of Major Adverse Cardiovascular Events (MACE) and Major Adverse Lower limb Events (MALE), regardless of the ABI. In the entire cohort, MAC appears as a better risk predictor than high ABI, often considered as a proxy for MAC. Beyond screening and risk estimation, we investigated the impact of different therapies on the peripheral arterial risk of patients living with T2D. The introduction of sodium-glucose cotransporter-2 (SGLT2) inhibitors, with proven cardio-renal protective effects, re-launched the debate on the link between diuretics and peripheral arterial risk. Our group's meta-analysis highlighted the need to expand literature. Among the 5 included studies, we showed a significant increase in the risk of amputation associated with diuretic use (OR=1.73, p<0.001). Subsequently, we conducted a retrospective observational study involving 1309 patients with T2D. In multivariable analysis, diuretics were significantly associated with the occurrence of MALE after adjustment for propensity score (aHR=1.66; 1.08-2.56, p=0.020) and inverse probability of treatment weighting analysis (aHR=1.76; 1.67-1.84, p<0.001). We identified at-risk profiles with diuretics treatment. In subgroup analysis, the risk of MALE with diuretics was significantly increased with abnormal ABI (<0.90 or 1.40) (aHR=2.29, interaction p=0.02). Loop or thiazide diuretics were associated with an increased risk, but not potassium-sparing diuretics. The third part of the work focused on the XATOA registry in patients with diabetes. The COMPASS trial demonstrated the benefit of double antithrombotic therapy (aspirin and rivaroxaban 2.5mg twice daily) on MACE and MALE in patients with stable cardiovascular disease. The results of this randomized controlled trial were confirmed by real-life data from the XATOA registry. The decisions that drive to double antithrombotic therapy prescription werecomparable between diabetic and non diabetic patients. The benefit on MACE and MALE was similar to that observed in the COMPASS trial.Our work highlights the need to assess peripheral arterial risk in people with T2D, use appropriate assessment tools and define therapies suited to the level of risk
Zhou, Bo. „DETECTION AND QUANTIFICATION OF CORONARY CALCIUM FROMDUAL ENERGY CHEST X-RAYS: PHANTOM FEASIBILITY STUDY“. Case Western Reserve University School of Graduate Studies / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=case1481216785398625.
Der volle Inhalt der QuelleLaurinavicius, Antonio Gabriele. „Avaliação da aterosclerose subclínica em portadores de HDL-colesterol marcadamente elevado“. Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-06062016-103124/.
Der volle Inhalt der QuelleHDL-c is a negative cardiovascular risk factor and its plasma concentration is inversely related to the incidence of cardiovascular events. However, evidence of benefit among subjects with HDL-c levels above the 95th percentile of the general population is still scarce and the impact of hyperalphalipoproteinemia (HALP) on cardiovascular risk continues to represent matter of debate in the medical literature. Some studies with specific populations indicated an increased cardiovascular risk associated with HALP. In addition, other reports identified groups of patients with marked hypoalphalipoproteinemia and longevity. Hence, there could be a dissociation between HDL-c levels and cardiovascular risk in certain populations, possibly due to dysfunctional HDL particles. The aim of this study was to investigate the role of HALP phenotype in determining cardiovascular risk; to compare the prevalence of subclinical cardiovascular disease, assessed by ultrasound measurement of Carotid Intima-Media Thickness (CIMT) among patients with HDL-c >= 90mg/dL (HALP group) and patients with HDL-c currently considered normal (40-50mg/dL for men and 50-60mg/dL for women); and to evaluate HDL functionality in patients with HALP through the study of its composition, its cholesterol efflux capacity, and its anti-inflammatory and antioxidant activity; correlating those characteristics with the presence of subclinical cardiovascular disease assessed by CIMT, Pulse Wave Velocity (PWV) and Coronary Artery Calcification (CAC). To answer these questions, the present study was articulated into two arms: Arm 1: ELSA-Brasil study cohort analysis in order to assess HALP prevalence in a general population, defining demographic, anthropometric and metabolic profile of HALP individuals; and comparing the prevalence of subclinical vascular disease among HALP subjects with controls with normal HDL-c. Arm 2: Recruitment of 80 healthy volunteers with HALP to study the correlation between subclinical vascular disease and HDL functionality in this group. Our study led to four main conclusions: 1) markedly elevated HDL-c is associated with lower CIMT compared to the control group with normal HDL-c levels. Although individuals with HALP display a more favorable metabolic profile than subjects with normal HDL-c, the association between CIMT and HALP was independent of traditional risk factors, indicating that the lower prevalence of subclinical vascular disease in this group is only partially justified by the lower prevalence of cardiovascular risk factors; 2) Although HALP can be regarded as an atheroprotective phenotype, there are individuals with markedly elevated levels of HDL-c who develop cardiovascular disease. Our results indicate good correlation of the three methods here adopted to study subclinical vascular disease among HALP patients: CIMT, VOP and CAC; 3) Traditional risk factors continue to exert their weight in determining cardiovascular risk in patients with HALP. Age, smoking, hypertension, hypertriglyceridemia and high levels of LDL-c were significantly associated with the presence of subclinical vascular disease among HALP individuals; 4) the assessment of the HDL composition and functionality in patients with HALP may allow to identify individuals specifically more susceptible to atherosclerosis. Our results indicate that, in particular, cholesterol efflux capacity, the anti-inflammatory activity of HDL, and triglyceride transfer capacity were independently associated with lower CIMT in HALP individuals, while higher levels of Apo A-IV were associated with a greater burden of subclinical cardiovascular disease
Miranda, Érique José Peixoto de. „Associação entre doença tireoidiana subclínica, aterosclerose coronariana, índice de espessura de médio-íntima carotídea e rigidez arterial aórtica em análise transversal do Estudo Longitudinal de Saúde do Adulto (ELSA-Brasil)“. Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5169/tde-19062017-154141/.
Der volle Inhalt der QuelleIntroduction: Subclinical thyroid disease includes subclinical hypothyroidism and subclinical hyperthyroidism. Association between subclinical thyroid disease and cardiovascular morbidity and mortality is controversial and data about the relationship between those clinical conditions and subclinical atherosclerosis is scarce. Objectives: This study aims to evaluate the association between subclinical thyroid disease, coronary artery calcification (CAC), coronary artery disease (CAD), mean common carotid intima-media thickness (IMT) and carotid-femoral pulse wave velocity (cf-PWV) in the Brazilian Longitudinal Study of Adult Health (ELSA-Brasil). Methods: We included euthyroid subjects, defined as TSH between 0.4 and 4.0 mIU/l and FT4 between 0.8 and 1.9 ng/dL, and individuals with subclinical hypothyroidism, defined as TSH > 4.0 mIU/l and normal FT4, and subclinical hyperthyroidism, defined as TSH < 0.4 mIU/L and normal FT4. We excluded individuals with other thyroid disorders, subjects who used medication that altered thyroid function, subjects with past of cardiovascular disease. In computed angiotomography analysis, we have excluded subjects with subclinical hyperthyroidism because of the small sample, and in cf-PWV analysis, we have excluded individuals with chronic kidney disease, use of anti-hypertensive and diuretics. The association between TSH quintiles was evaluated in logistic regression models for CAC and CAD, and the association between IMT, cf-PWV (as continuous variables or as factor, categorized at 75th sample\'s percentile) and TSH levels or subclinical thyroid diseases was evaluated by multivariate logistic and linear regression models. All models were adjusted for demographic variables and cardiovascular risk factors. Results: CAC analysis included 3,836 subjects, median of age 49 years (IQR=44-56), 1,999 (52.1%) women. CAC > 100 was independently associated with first quintile of TSH, using the third quintile as the reference (adjusted OR=1.57, 95% CI=1.05-2.35, P=0.027). Computed angiotomography analysis included 796 subjects, median of age 55 years (IQR=48-60), 406 (51%) women. CAD and CAC > 0 was independently associated with first quintile in comparison with third quintile (adjusted OR=1.73, 95% CI=1.08-2.79, P=0.023 and adjusted OR=1.76, 95% CI=1.09-2.82, P= 0.02, respectively), but not with burden of disease. In IMT analysis, 8,623 subjects were included, median of age 50 years (IQR=45-57 years), 4,624 (53.6%) women in the subclinical hypothyroidism subanalysis, and 8,193, median age 50 years (IQR = 44-57 years), 4,382 (53.5%) women, in the subclinical hyperthyroidism subanalysis. Subclinical hypothyroidism, but not subclinical hyperthyroidism, was independently associated with IMT as continuous variable (beta=0.010, IC 95%=0.0004-0.019, P=0.041) or as factor categorized at 75th percentile adjusted for age, sex and race (adjusted OR=1.30, 95% CI=1.07-1.61, P=0.010). In cf-PWV analysis, 8,341 subjects were included, median of age 50 years (IQR=44-56 years), 4,383 (52.5%) women in the subclinical hypothyroidism subanalysis, and 7,790, median age 50 years (IQR = 44-57 years), 4,191 (53.8%) women in subclinical hyperthyroidism subanalysis. Cf- PWV was not associated with subclinical thyroid disease. Conclusion: In separated analysis, CAC and CAD was independently associated with first quintile of TSH using the third as the reference; IMT was independently associated with subclinical hypothyroidism, and cf-PWV was not associated with subclinical thyroid diseases
Bücher zum Thema "Medial artery calcification"
Holbrook, Anna I. Vascular Calcifications. Herausgegeben von Christoph I. Lee, Constance D. Lehman und Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0034.
Der volle Inhalt der QuelleCovic, Adrian, Mugurel Apetrii, Luminita Voroneanu und David J. Goldsmith. Vascular calcification. Herausgegeben von David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.
Der volle Inhalt der QuelleLevy, David. Macrovascular complications, hypertension, and lipids. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198766452.003.0008.
Der volle Inhalt der QuelleBuchteile zum Thema "Medial artery calcification"
Kumar PK, Mithun, und Mohammad Motiur Rahman. „Calcifications Attenuation in Left Coronary Artery CT Images Using FDA Domain“. In Medical Imaging, 1258–80. IGI Global, 2017. http://dx.doi.org/10.4018/978-1-5225-0571-6.ch051.
Der volle Inhalt der QuelleMesserli, Michael. „Thoracic great vessels“. In EACVI Handbook of Cardiovascular CT, herausgegeben von Oliver Gaemperli, Pal Maurovich-Horvat, Koen Nieman, Gianluca Pontone und Francesca Pugliese, 255–68. Oxford University PressOxford, 2022. http://dx.doi.org/10.1093/med/9780192884459.003.0030.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Medial artery calcification"
Delgado, Ana, Pengfei Dong, Mahyar Sameti, Vladislav N. Zimin, Juhwan Lee, Yazan Gharaibeh, Hiram G. Bezerra, David Wilson, Christopher Bashur und Linxia Gu. „Mechanical Characterization of Calcificaiton in Diseased Coronary Artery with Atomic Force Microscope“. In 2022 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2022. http://dx.doi.org/10.1115/dmd2022-1055.
Der volle Inhalt der QuelleSubramaniam, Dhananjay Radhakrishnan, und Mary Beth Kossuth. „Influence of Tissue Prestress and Calcification Levels on Iliac Balloon Angioplasty Biomechanics“. In 2024 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2024. http://dx.doi.org/10.1115/dmd2024-1040.
Der volle Inhalt der QuelleLiu, Xiuming, Shice Wang, Yufeng Deng und Kuan Chen. „Coronary artery calcification (CAC) classification with deep convolutional neural networks“. In SPIE Medical Imaging, herausgegeben von Samuel G. Armato und Nicholas A. Petrick. SPIE, 2017. http://dx.doi.org/10.1117/12.2253974.
Der volle Inhalt der QuelleXie, Yiting, Matthew D. Cham, Claudia Henschke, David Yankelevitz und Anthony P. Reeves. „Automated coronary artery calcification detection on low-dose chest CT images“. In SPIE Medical Imaging, herausgegeben von Stephen Aylward und Lubomir M. Hadjiiski. SPIE, 2014. http://dx.doi.org/10.1117/12.2043840.
Der volle Inhalt der QuelleLiu, Jiamin, Le Lu, Jianhua Yao, Mohammadhadi Bagheri und Ronald M. Summers. „Pelvic artery calcification detection on CT scans using convolutional neural networks“. In SPIE Medical Imaging, herausgegeben von Samuel G. Armato und Nicholas A. Petrick. SPIE, 2017. http://dx.doi.org/10.1117/12.2255247.
Der volle Inhalt der QuelleLazebnik, R. S., P. B. Sachs und R. C. Gilkeson. „Improved detection of coronary artery calcifications using dual energy subtraction radiography“. In Medical Imaging, herausgegeben von Yulei Jiang und Berkman Sahiner. SPIE, 2007. http://dx.doi.org/10.1117/12.709041.
Der volle Inhalt der QuelleXie, Yiting, Shuang Liu, Albert Miller, Jeffrey A. Miller, Steven Markowitz, Ali Akhund und Anthony P. Reeves. „Coronary artery calcification identification and labeling in low-dose chest CT images“. In SPIE Medical Imaging, herausgegeben von Samuel G. Armato und Nicholas A. Petrick. SPIE, 2017. http://dx.doi.org/10.1117/12.2254125.
Der volle Inhalt der QuelleDeVos, Amanda N., und Paul A. Iaizzo. „ANALYSES OF THE DEVICE-TISSUE INTERFACES OF PREVIOUSLY IMPLANTED STENTS WITHIN PERFUSION-FIXED HUMAN HEARTS UTIILIZING MICRO COMPUTED TOMOGRAPHY“. In 2023 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2023. http://dx.doi.org/10.1115/dmd2023-4061.
Der volle Inhalt der QuelleSvane, Jakob, Tomasz Wiktorski, Trygve Eftestøl und Stein Ørn. „Machine Learning Methods For Classification of Individuals With Coronary Artery Calcification“. In 2024 IEEE 37th International Symposium on Computer-Based Medical Systems (CBMS). IEEE, 2024. http://dx.doi.org/10.1109/cbms61543.2024.00013.
Der volle Inhalt der QuelleDehlin, M. I., P. Drivelegka, V. Sigurdardottir, O. Angerås, L. T. Jacobsson und H. Forsblad d’Elia. „FRI0228 Urate correlates to coronary artery calcification but not to intima media thickness“. In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.1973.
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