Auswahl der wissenschaftlichen Literatur zum Thema „MCP-Counter“
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Zeitschriftenartikel zum Thema "MCP-Counter"
Inami, K. „MCP-PMT development for Belle-II TOP counter“. Physics Procedia 37 (2012): 683–90. http://dx.doi.org/10.1016/j.phpro.2012.02.417.
Der volle Inhalt der QuelleInami, K., N. Kishimoto, Y. Enari, M. Nagamine und T. Ohshima. „A 5 ps TOF-counter with an MCP–PMT“. Nuclear Instruments and Methods in Physics Research Section A: Accelerators, Spectrometers, Detectors and Associated Equipment 560, Nr. 2 (Mai 2006): 303–8. http://dx.doi.org/10.1016/j.nima.2006.01.027.
Der volle Inhalt der QuelleJung, Seok-Kyu, und Jung-Myung Lee. „(32) Effects of 1-MCP Treatments on Several Major Apple Cultivars Grown in Korea“. HortScience 40, Nr. 4 (Juli 2005): 1004D—1004. http://dx.doi.org/10.21273/hortsci.40.4.1004d.
Der volle Inhalt der QuelleGruss, H. J., M. A. Brach, R. R. Schumann und F. Herrmann. „Regulation of MCP-1/JE gene expression during monocytic differentiation.“ Journal of Immunology 153, Nr. 11 (01.12.1994): 4907–14. http://dx.doi.org/10.4049/jimmunol.153.11.4907.
Der volle Inhalt der QuelleCalvi, M., S. Capelli, P. Carniti, C. Gotti und G. Pessina. „Single photon counting performance of the Auratek-Square MCP-PMT“. Journal of Instrumentation 17, Nr. 11 (01.11.2022): P11009. http://dx.doi.org/10.1088/1748-0221/17/11/p11009.
Der volle Inhalt der QuelleWierzbicki, Jarosław, Artur Lipiński, Iwona Bednarz-Misa, Łukasz Lewandowski, Katarzyna Neubauer, Paulina Lewandowska und Małgorzata Krzystek-Korpacka. „Monocyte Chemotactic Proteins (MCP) in Colorectal Adenomas Are Differently Expressed at the Transcriptional and Protein Levels: Implications for Colorectal Cancer Prevention“. Journal of Clinical Medicine 10, Nr. 23 (26.11.2021): 5559. http://dx.doi.org/10.3390/jcm10235559.
Der volle Inhalt der QuelleNagaraju, Raju, Apurva Kumar R. Joshi, Sowmya Giriyapura Vamadeva und Rajini Padmanabhan Sharda. „Plasma paraoxonase1 activity in rats treated with monocrotophos: a study of the effect of duration of exposure“. Interdisciplinary Toxicology 12, Nr. 3 (01.11.2019): 129–35. http://dx.doi.org/10.2478/intox-2019-0015.
Der volle Inhalt der QuelleAkiyoshi, Takashi, Zhe Wang, Tomoko Kaneyasu, Osamu Gotoh, Norio Tanaka, Sayuri Amino, Noriko Yamamoto et al. „Transcriptomic Analyses of Pretreatment Tumor Biopsy Samples, Response to Neoadjuvant Chemoradiotherapy, and Survival in Patients With Advanced Rectal Cancer“. JAMA Network Open 6, Nr. 1 (20.01.2023): e2252140. http://dx.doi.org/10.1001/jamanetworkopen.2022.52140.
Der volle Inhalt der QuelleMatsuoka, K. „Development and production of the MCP-PMT for the Belle II TOP counter“. Nuclear Instruments and Methods in Physics Research Section A: Accelerators, Spectrometers, Detectors and Associated Equipment 766 (Dezember 2014): 148–51. http://dx.doi.org/10.1016/j.nima.2014.05.003.
Der volle Inhalt der QuelleWhite, Kieron, Maxime Meylan, Antoine Bougoüin, Kate Connor, Manuela Salvucci, Franck Bielle, Jochen H. M. Prehn et al. „TAMI-51. IDENTIFYING NEW TUMOR MICROENVIRONMENT (TME) CONTEXTS OF VULNERABILITY IN GLIOBLASTOMA“. Neuro-Oncology 22, Supplement_2 (November 2020): ii224. http://dx.doi.org/10.1093/neuonc/noaa215.938.
Der volle Inhalt der QuelleDissertationen zum Thema "MCP-Counter"
De, Vries-Brilland Manon. „Caractérisation du microenvironnement immunitaire des carcinomes papillaires du rein“. Electronic Thesis or Diss., Angers, 2023. http://www.theses.fr/2023ANGE0017.
Der volle Inhalt der QuelleArticle 1: Checkpoint inhibitors in metastatic papillary renal cell carcinoma : papillary Renal Cell Carcinoma (pRCC) is the most common non-clear cell RCC (nccRCC) and a distinct entity, although heterogenous, associated with poor outcomes. The treatment landscape of metastatic pRCC (mpRCC) relied so far on targeted therapies, mimicking previous developments in metastatic clear-cell renal cell carcinoma. However, antiangiogenics as well as mTOR inhibitors retain only limited activity in mpRCC. As development of immune checkpoint inhibitors (ICI) is now underway in patients with mpRCC, we aimed at discussing early activity data and potential for future therapeutic strategies in monotherapy or combination. Expression of immune checkpoints such as PD-L1 and infiltrative immune cells in pRCC could provide insights into their potential immunogenicity, although this is currently poorly described. Based on retrospective and prospective data, efficacy of ICI as single agent remains limited. Combinations with tyrosine-kinase inhibitors, notably with anti-MET inhibitors, harbor promising response rates and may enter the standard of care in untreated patients. Collaborative work is needed to refine the molecular and immune landscape of pRCC, and pursue efforts to set up predictive biomarker-driven clinical trials in these rare tumors. Article 2 : Comprehensive analyses of immune tumor microenvironment in papillary renal cell carcinoma. Background : papillary Renal CellCarcinoma (pRCC) is the most common non-clear cell RCC (nccRCC), and associated with poor outcomes in the metastatic setting. In this study, we aimed to comprehensively evaluate the immune tumor microenvironment (TME) ,largely unknown, of patients with metastatic pRCC and identify potential therapeutic targets. Methods : we performed quantitative gene expression analysis of TME using MCP-counter methodology, on 2 independent cohorts of localized pRCC (n=271 and n=98). We then characterized the TME, using immunohistochemistry (n=38) and RNA-sequencing (RNA-seq) (n=30) on metastatic pRCC from the prospective AXIPAP trial cohort. Results: unsupervised clustering identified 2 "TME subtypes", in each of the cohorts : the “immune-enriched” and the “immune-low”.Within AXIPAP trial cohort, the “immune-enriched” cluster was significantly associated with a worse prognosis according to the median overall survival to 8 months (95%CI, 6-29) versus 37 months (95%CI, 20-NA,p=0.001).The 2 immune signatures, Teff and JAVELIN Renal 101 Immuno signature, predictive of response to immune checkpoint inhibitors (CPI) in ccRCC, were significantly higher in the “immune-enriched” group (adjusted p<0.05). Finally, 5 differentially overexpressed genes were identified, corresponding mainly to B lymphocyte populations. Conclusion : for the first time, using RNA-seqand IHC, we have highlighted a specific immune TME subtype of metastatic pRCC, significantly more infiltrated with T and Bimmune population. This “immune-enriched” group appears to have a worse prognosis and could have a potential predictive value for response to immunotherapy, justifying the confirmation of these results in a cohort of metastatic pRCC treated with CPI and incombination with targeted therapies
Konferenzberichte zum Thema "MCP-Counter"
Matsuoka, Kodai, Shigeki Hirose, Toru Iijima, Kenji Inami, Yuji Kato, Yosuke Maeda, Ryo Mizuno, Yutaro Sato und Kazuhito Suzuki. „Performance of the MCP-PMT for the Belle II TOP counter“. In International Conference on New Photo-detectors. Trieste, Italy: Sissa Medialab, 2016. http://dx.doi.org/10.22323/1.252.0028.
Der volle Inhalt der QuelleCho, Doo Jin, und G. Michael Morris. „Dead-time effects in curved-channel microchannel plates“. In OSA Annual Meeting. Washington, D.C.: Optica Publishing Group, 1989. http://dx.doi.org/10.1364/oam.1989.wq4.
Der volle Inhalt der QuelleYONEKURA, Takuya. „Performance of the MCP-PMTs for the TOP counter in the Belle II experiment“. In Technology and Instrumentation in Particle Physics 2014. Trieste, Italy: Sissa Medialab, 2015. http://dx.doi.org/10.22323/1.213.0082.
Der volle Inhalt der QuelleMatsuoka, Kodai. „Performance of the MCP-PMTs of the TOP Counter in the First Beam Operation of the Belle II Experiment“. In Proceedings of the 5th International Workshop on New Photon-Detectors (PD18). Journal of the Physical Society of Japan, 2019. http://dx.doi.org/10.7566/jpscp.27.011020.
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