Auswahl der wissenschaftlichen Literatur zum Thema „Marqueurs moteurs“
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Zeitschriftenartikel zum Thema "Marqueurs moteurs"
Capdevielle, D. „Coordinations motrices interpersonnelles dans la schizophrénie : un marqueur phénotypique ?“ European Psychiatry 29, S3 (November 2014): 581. http://dx.doi.org/10.1016/j.eurpsy.2014.09.283.
Der volle Inhalt der QuelleKrebs, M. O., O. Gay, G. Martinez und I. Amado. „Signes neurologiques mineurs et contrôle moteur : ce qu’ils nous apprennent sur la schizophrénie, ses mécanismes et ses frontières“. European Psychiatry 29, S3 (November 2014): 580–81. http://dx.doi.org/10.1016/j.eurpsy.2014.09.282.
Der volle Inhalt der QuelleLimosin, F. „Maladie de Parkinson : la psychiatrie en première ligne ?“ European Psychiatry 29, S3 (November 2014): 577. http://dx.doi.org/10.1016/j.eurpsy.2014.09.273.
Der volle Inhalt der QuelleSeassau, M., T. Weiss, R. Carcangiu und F. Duval. „Les mouvements oculomoteurs : un marqueur d’efficacité du méthylphénidate (MPH) dans le TDA/H“. European Psychiatry 29, S3 (November 2014): 597. http://dx.doi.org/10.1016/j.eurpsy.2014.09.191.
Der volle Inhalt der QuelleCoulibaly, Daouda. „Congruence de deux théories polyphoniques dans l’analyse stylistique des textes romanesques : intertextualité et dialogisme dans L’état z’héros ou la guerre des gaous de Maurice Bandaman“. e-Scripta Romanica 10 (13.10.2022): 1–12. http://dx.doi.org/10.18778/2392-0718.10.01.
Der volle Inhalt der QuelleVeronese, Alexandre, und Marcelo Barros da Cunha. „A utilização das marcas alheias nos algoritmos de geração de palavras-chaves: uma análise sobre a jurisprudência do tribunal de justiça da união europeia para pensar sobre o caso brasileiro / l’utilisation de la marque d’autrui dans les générateurs de mots clés: une analyse des décisions de la cour de justice de l´union européenne pour reflechir sur le cas brésilien / The use of other parties trademarks in algorithms to generate keywords: an analysis of the Court of Justice of the European Union to shed light over the Brazilian case“. Revista Brasileira de Direito 13, Nr. 2 (18.08.2017): 232. http://dx.doi.org/10.18256/2238-0604/revistadedireito.v13n2p232-255.
Der volle Inhalt der QuelleValette, Jean-Francois, Hugues Pécout und France Guérin-Pace. „Caracterizar las desigualdades territoriales en la Zona Metropolitana del Valle de México a través de la movilidad residencial y cotidiana / Understanding territorial inequalities in the Mexico City Metropolitan Area through residential and daily mobility“. Revista Trace, Nr. 82 (31.07.2022): 113. http://dx.doi.org/10.22134/trace.82.2022.813.
Der volle Inhalt der QuelleMallet, J., N. Ramoz, P. Gorwood und C. Dubertret. „Cannabis et schizophrénie : approche clinique, cognitive et neurologique dans la définition d’un nouveau phénotype“. European Psychiatry 28, S2 (November 2013): 7–8. http://dx.doi.org/10.1016/j.eurpsy.2013.09.018.
Der volle Inhalt der QuellePelletier, Réjean. „Le militant du R.I.N. et son parti“. Articles 13, Nr. 1 (12.04.2005): 41–72. http://dx.doi.org/10.7202/055558ar.
Der volle Inhalt der QuelleAndrade, Inácio de Carvalho Dias de. „UMA MODERNIDADE ALTERNATIVA?“ Revista Educação e Ciências Sociais 3, Nr. 5 (22.10.2020). http://dx.doi.org/10.38090/recs.2595-9980.2020.v3.n5.40-58.
Der volle Inhalt der QuelleDissertationen zum Thema "Marqueurs moteurs"
Dessaud, Eric. „Marqueurs de sous-populations neuronales et analyse du développement des neurones moteurs et sensoriels chez la souris“. Aix-Marseille 2, 2005. http://www.theses.fr/2005AIX22006.
Der volle Inhalt der QuelleChini, Catherine. „Recherche d'un ou plusieurs marqueurs de la pollution de l'air par l'aérosol diesel“. Paris 12, 1997. http://www.theses.fr/1997PA120021.
Der volle Inhalt der QuelleDi, Scala Franck. „Analysis of skeletal muscle in amyotrophic lateral sclerosis : Etiological, diagnostic and therapeutic aspects“. Université Louis Pasteur (Strasbourg) (1971-2008), 2005. http://www.theses.fr/2005STR13188.
Der volle Inhalt der QuelleAmyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disease that attacks nerve cells in the brain and spinal cord, and produces muscle weakness and atrophy. ALS is emerging as a “multi-system” disease in which the alteration in muscle, motor neuron and glia cells may act synergistically to induce the disease. Although skeletal muscle is quite an untested tissue, many findings establish muscle as a promising primary target in ALS research. The aim of the present PhD work was to explore gene expression modifications in ALS skeletal muscle by conducting two independent approaches. First, our gene expression studies targeted Nogo-A and uncoupling protein 3 (UCP3). We showed that Nogo-A exhibits a specific pattern of expression in ALS skeletal muscle. This result, together with the observation that Nogo-A immunoreactivity correlates with the severity of motor impairment in ALS patients, suggest that Nogo-A expression in muscle could be used as a marker of ALS functional disability. As far as UCP3 is concerned, we showed that UCP3 is increased in ALS skeletal muscle, and stable in other ALS-like syndromes, suggesting that UCP3 is a potential marker for ALS diagnosis. Second, we extensively explored the transcriptome of human ALS muscle using a DNA microarray approach. This strategy enabled us to identify two groups of genes of particular interest from a clinical point of view. One group contain interesting candidates to measure disease progression and the other group contain interesting candidates for early diagnosis of ALS. In addition, several hits emerging from our data analysis are linked to retinoic acid (RA) metabolism and we currently explore the role of these alterations in ALS pathogenesis. Finally, we generated a molecular identity card of skeletal muscle affected by ALS, concretized by a database that might be a source to fill the gap in molecular targets for efficient therapeutic strategies
Mathieu, Julie. „Traversée d'intersection et évitement de collision en conduite automobile : identification du support perceptif et des marqueurs du couplage information-mouvement“. Thesis, Aix-Marseille, 2017. http://www.theses.fr/2017AIXM0602/document.
Der volle Inhalt der QuelleThis Ph.D. project aims at studying the perceptual-motor mechanisms implicated in intersection crossing task. The first part of this manuscript explains the human, economic and societal challenges associated with the successfully complete the intersection crossing task. The main results of the former studies that have examined the identification of risks associated with this type of maneuver at an intersection are developed. These former studies mainly focused on high-level processes (e.g., discrete judgment or decision tasks), we have decided to focus our attention on low-level processes (e.g., visual guidance) during this Ph.D. project. For this to happen, the Ecological Approach to Perception and Action seems to be a relevant theoretical framework for studying this complex task. Indeed, it preserves the natural link between information and movement. The second part of this manuscript is devoted to the experiments completed during this research. In order to carry out our three experiments, virtual reality fixed-base driving simulator was used. This work contributes to better understand the perceptual substrate involved in intersection crossing task. Also, this work identifies different markers of control based on information-movement coupling. Firstly, our results revealed that the perceptual substrate underlying judgments of arrival time of a vehicle moving towards an intersection is distinct from the perceptual substrate underlying the active control of one’s own approach to the same intersection. Secondly, the results have also strengthened the assumption that the control of approach and intersection crossing task is based on information-movement coupling
Plonka, Alexandra. „Diagnostic précoce et différentiel des Aphasies Primaires Progressives : apport d'analyses motrices et langagières“. Electronic Thesis or Diss., Université Côte d'Azur, 2023. http://www.theses.fr/2023COAZ6015.
Der volle Inhalt der QuellePrimary Progressive Aphasia (PPA) is a neurodegenerative syndrome initially characterized by an isolated language disorder and which diagnosis is mainly clinical. Three main PPA subtypes have been described in the literature: logopenic PPA (lvPPA), semantic PPA (svPPA) and non-fluent/agrammatic PPA (nfavPPA). The latest data in the literature have highlighted a late diagnosis, sometimes erroneous, associated with the need for an early medical-care management. The creation of new tools for the diagnosis and classification of PPAs seems to be crucial for an early and adapted patients' care.The objective of this thesis is to verify the interest of the analysis of markers using new technologies such as motor activity parameters (graphic and gestural) and language activity parameters (linguistic and acoustic) within the speech therapy evaluation, to improve the early and differential diagnosis of PPAs.Initially, this thesis work made it possible to analyze the specificities of the PPA diagnosis among the 167,191 diagnoses recorded in the French National Alzheimer Bank between 2010 and 2016. We highlighted diagnostic wavering and misdiagnosis of patients with PPA. These results underline the importance of developing tools that can be integrated into speech therapy practice and that can allow a greater accuracy in diagnosis.Following these observations, we have demonstrated the interest of using new technologies that can be easily integrated into a diagnostic approach. By using touch tablets, we have demonstrated the interest of the analysis of graphic parameters, such as writing pressure and strokes, for the differential diagnosis of PPA patients in comparison with patients with Alzheimer's Disease (AD) or patient with Posterior Cortical Atrophy. These parameters have also proven to be useful in the classification of the three main subtypes of PPA.To complete the evaluation of strokes for which the gestural behaviors during the pencil lifting times cannot be directly recovered on an electronic tablet, we were interested in the evaluation of the writing gesture in the context of a nfavPPA type of impairment. The video analysis showed a complementary interest for the more specific diagnosis of this non-fluent variant.In a third step, and in a perspective of multimodal analysis, we verified the interest of the contribution of language and acoustic analyses of patients with PPA.The ‘sentence span' test was used to compare the memory span of PPA and AD patients. Our results showed a significantly different verbal span.The vocal analysis allowed us, beyond the differential diagnosis PPA/AD and via temporal and prosodic acoustic markers, to differentiate the physiopathological profiles of lvPPA patients.Finally, in a post-Covid-19 pandemic context and more specifically because of the neurological disorders observed in post-acute Covid-19 syndrome, we questioned, through a systematic review and an experts' consensus, the relevance of using these motor and language markers for the early diagnosis of AD. Indeed, cognitive impairments in post-acute Covid-19 syndrome may, in the future, complicate the early diagnosis of neurodegenerative diseases due to common disorders
Truchon, Dany. „Nouveaux marqueurs pour l'observation du moteur flagellaire bactérien“. Thesis, Université Laval, 2011. http://www.theses.ulaval.ca/2011/28827/28827.pdf.
Der volle Inhalt der QuelleLongo, Laurence. „Vers des moteurs de recherche "intelligents" : un outil de détection automatique de thèmes : méthode basée sur l'identification automatique des chaînes de référence“. Phd thesis, Université de Strasbourg, 2013. http://tel.archives-ouvertes.fr/tel-00939243.
Der volle Inhalt der QuelleCalbiac, Hortense de. „Mechanisms of C9ORF72 pathogenicity and related autophagy impairment in amyotrophic lateral sclerosis Sqstm1 knockdown causes a locomotor phenotype ameliorated by rapamycin in a zebrafish model of ALS/FTLD Depdc5 knockdown causes mTOR-dependent motor hyperactivity in zebrafish“. Thesis, Sorbonne université, 2019. http://www.theses.fr/2019SORUS561.
Der volle Inhalt der QuelleTo investigate the pathogenic mechanisms induced by SQSTM1 mutations in ALS, we developed a zebrafish model of sqstm1 haploinsufficiency. We observed that loss of function of sqstm1 leads to a specific motor phenotype. To elucidate the common cellular mechanisms underlying motor neuron degeneration in ALS, we analyzedc9orf72 and sqstm1 epistatic interactions inzebrafish. C9orf72 and sqstm1 partial inhibitions have an additive effect and C9ORF72 rescues the phenotype induced by sqstm1 knockdown. Thus, both proteins belong to the same pathway and c9orf72 acts downstream of sqstm1. Also, we observed that depletion of these genes in mouse motor neurons primary cultures leads to the early death of motor neurons associated with autophagy impairment. To develop a vertebrate model that recapitulates the different mechanisms associated withthe C9ORF72 HRE pathogenicity in ALS, we combined the partial inhibition of c9orf72 with the expression of the DPRs in zebrafish. This induces a robust motor phenotype characterized by locomotor defects and paralysis. Focusing on GP repeats, we observed that the loss of function of c9orf72 is essential to inhibitpoly(GP) clearance.This is associated with SQSTM1/p62 accumulation, severe motor neurons abnormalities and loss. These phenotypes are rescued by the inhibition of caspase 9, a regulator of apoptosis. Also, rapamycinis able to improve the clearance of poly(GP) and p62, with restored swim and motor neurons features, thus confirming the role of C9ORF72 in autophagy.These results show that DPR toxicity is related to lowered expression of C9ORF72, suggesting that both gain and loss of function synergize in the C9ORF72 HRE pathogenicity
Rey-robert, Benoit. „Étude des marqueurs communs du vieillissement cognitif et sensori-moteur : l'exemple du ralentissement du traitement de l'information“. Thesis, Aix-Marseille, 2012. http://www.theses.fr/2012AIXM4038/document.
Der volle Inhalt der QuelleThe starting point of this work was to realize that, when looking at aging research, studies are split in a multitude of domains, paradigms and theories resulting in a vast puzzle of knowledge. However, at the behavioral level, despite obvious differences between field of studies, there are empirical results showing common evolution between functional systems during the aging process. It is notably the case for cognitive and sensorimotor tasks. For example, when speed is the performance criterion, elderly are slower than their younger counterparts; there are also more variable and less adaptable as difficulty increases. These phenomenons can be observed in both cognitive and sensorimotor domain. More generally, correlation between performance in different functional domain increases through aging, and most notably between the cognitive and sensorimotor domain (Schäfer et al., 2006 ; Lindenberger & Ghisletta, 2009). We thus wanted to understand how and why those common behavioral phenomenon were observed. A considered hypothesis is that there would come from structural and functional modifications of the central nervous system which would become less and less differentiated through the course of aging (Reuter-Lorenz & Park, 2010 ; Heuninckx et al., 2005). We wanted to explore this hypothesis. At the start of our work, to our knowledge there was no theoretical framework explaining the compression of the cognitive and sensorimotor system in one less differentiated
Jokic, Natasa. „Etude du rôle de nogo dans la sclérose latérale amyotrophique : Validation d'un marqueur moléculaire“. Université Louis Pasteur (Strasbourg) (1971-2008), 2005. http://www.theses.fr/2005STR13052.
Der volle Inhalt der QuelleAmyotrophic lateral sclerosis (ALS) is a neurodegenerative disease characterized by the loss of motor neurons in the spinal cord, brain stem and motor cortex. The objective of my PhD was to analyse the cellular and molecular mechanisms implicated in the loss of motor neurons. In this context, we carried out the studies conducted in our laboratory on the neurite outgrowth inhibitor Nogo identified as potential marker of ALS disease. Different isoforms of Nogo exhibit a specific pattern of expression in the muscle biopsies of ALS patients. Our studies showed that the protein levels of Nogo-A and Nogo-B correlated with the severity of clinical disability and with the mean area of oxidative fibres. In addition, the Nogo-A immunoreactivity was observed selectively in atrophic oxidative muscle fibres of ALS patients. These results suggest that Nogo-A and Nogo-B are the markers of disease severity. In order to characterise the function of Nogo-A in the pathology we crossbred the G86R ALS transgenic mice with the Nogo-A (Nogo-A-/-) knock-out mice. The life span G86R/ Nogo-A-/-was significantly increased. In order to determine if the muscular expression of Nogo-A as we observed in the muscle biopsies of ALS patients and in the muscle of G86R mice was sufficient to provoke destabilisation of neuromuscular junction (NMJ), we performed electrotransfer of a plasmid carrying Nogo-A gene into the soleus muscle of wild type mice. We observed that the morphology of the NMJ was affected by ectopic expression of Nogo-A. In conclusion, our results suggest that the muscle participates to the development of ALS
Bücher zum Thema "Marqueurs moteurs"
Edition, Biker's Club. Carnet d'entretien Voiture : Pour la Maintenance de Votre Véhicule à Moteur Dimensions: 20,96 Cm X 15,24 Cm, 100 Pages Pages à Remplir Pour Garder une Trace des Interventions Valable Pour Toutes les Marques et Types de Véhicule Motorisé. Independently Published, 2020.
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