Auswahl der wissenschaftlichen Literatur zum Thema „Macrophages“
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Zeitschriftenartikel zum Thema "Macrophages":
Rodriguez, Eric, Frederic Boudard, Michele Mallié, Jean-Marie Bastide und Madeleine Bastide. „Murine macrophage elastolytic activity induced by Aspergillus fumigatus strains in vitro: evidence of the expression of two macrophage-induced protease genes“. Canadian Journal of Microbiology 43, Nr. 7 (01.07.1997): 649–57. http://dx.doi.org/10.1139/m97-092.
Lu, Yufei, Leiming Guo und Gaofeng Ding. „PD1+ tumor associated macrophages predict poor prognosis of locally advanced esophageal squamous cell carcinoma“. Future Oncology 15, Nr. 35 (Dezember 2019): 4019–30. http://dx.doi.org/10.2217/fon-2019-0519.
Hargarten, Jessica C., Tyler C. Moore, Thomas M. Petro, Kenneth W. Nickerson und Audrey L. Atkin. „Candida albicans Quorum Sensing Molecules Stimulate Mouse Macrophage Migration“. Infection and Immunity 83, Nr. 10 (20.07.2015): 3857–64. http://dx.doi.org/10.1128/iai.00886-15.
Yadav, Mahesh, und Jeffrey S. Schorey. „The β-glucan receptor dectin-1 functions together with TLR2 to mediate macrophage activation by mycobacteria“. Blood 108, Nr. 9 (01.11.2006): 3168–75. http://dx.doi.org/10.1182/blood-2006-05-024406.
Gallego, Carolina, Douglas Golenbock, Maria Adelaida Gomez und Nancy Gore Saravia. „Toll-Like Receptors Participate in Macrophage Activation and Intracellular Control of Leishmania (Viannia) panamensis“. Infection and Immunity 79, Nr. 7 (25.04.2011): 2871–79. http://dx.doi.org/10.1128/iai.01388-10.
McKenzie, C. G. J., U. Koser, L. E. Lewis, J. M. Bain, H. M. Mora-Montes, R. N. Barker, N. A. R. Gow und L. P. Erwig. „Contribution of Candida albicans Cell Wall Components to Recognition by and Escape from Murine Macrophages“. Infection and Immunity 78, Nr. 4 (01.02.2010): 1650–58. http://dx.doi.org/10.1128/iai.00001-10.
Wilson, Justin E., Bhuvana Katkere und James R. Drake. „Francisella tularensis Induces Ubiquitin-Dependent Major Histocompatibility Complex Class II Degradation in Activated Macrophages“. Infection and Immunity 77, Nr. 11 (24.08.2009): 4953–65. http://dx.doi.org/10.1128/iai.00844-09.
Careau, Éric, Léa-Isabelle Proulx, Philippe Pouliot, Annie Spahr, Véronique Turmel und Élyse Y. Bissonnette. „Antigen sensitization modulates alveolar macrophage functions in an asthma model“. American Journal of Physiology-Lung Cellular and Molecular Physiology 290, Nr. 5 (Mai 2006): L871—L879. http://dx.doi.org/10.1152/ajplung.00219.2005.
Shinonaga, Masamichi, Cha Cheng Chang, Noriyuki Suzuki, Masazumi Sato und Takeo Kuwabara. „Immunohistological evaluation of macrophage infiltrates in brain tumors“. Journal of Neurosurgery 68, Nr. 2 (Februar 1988): 259–65. http://dx.doi.org/10.3171/jns.1988.68.2.0259.
Fedorov, A. A., N. A. Ermak, T. S. Gerashchenko, E. B. Topolnitskii, N. A. Shefer, E. O. Rodionov und M. N. Stakheyeva. „Polarization of macrophages: mechanisms, markers and factors of induction“. Siberian journal of oncology 21, Nr. 4 (03.09.2022): 124–36. http://dx.doi.org/10.21294/1814-4861-2022-21-4-124-136.
Dissertationen zum Thema "Macrophages":
Svensson, Ulf. „Macrophage activation by bacteria signalling to prostaglandin and cytokine responses /“. Lund : Dept. of Medical & Physiological Chemistry, Lund University, 1994. http://books.google.com/books?id=sAhrAAAAMAAJ.
Higuera, González Laura 1993. „Novel transcription regulators of tissue macrophages and alternative macrophage polarization“. Doctoral thesis, TDX (Tesis Doctorals en Xarxa), 2021. http://hdl.handle.net/10803/672702.
Los macrófagos juegan un papel muy importante en la defensa del organismo frente a una amplia variedad de patógenos. Los macrófagos se adaptan rápidamente a las perturbaciones en el microambiente gracias a que existe una compleja red de factores de transcripción que modulan sus respuestas. En los últimos años se han identificado factores de transcripción que regulan la identidad de los macrófagos, sin embargo, apenas se está comenzando a conocer la importancia de otros factores de transcripción que permiten adaptar la respuesta de los macrófagos, tanto en condiciones homeostáticas como frente a infecciones. Anteriormente nuestro grupo identificó reguladores transcripcionales de las respuestas pro-inflamatorias de los macrófagos, y en este trabajo hemos explorado la función de nuevos mecanismos reguladores que participan en la regulación de la distribución de los macrófagos en homeostasis, así como en las respuestas anti-inflamatorias de los macrófagos. Hemos estudiado poblaciones de macrófagos con diferentes ontogenias que habitan dentro de los tejidos y hemos caracterizado su regulación transcripcional. Además, hemos comparado la respuesta anti-inflamatoria de los diferentes macrófagos tisulares y así hemos identificado que existe un mecanismo transcripcional específico que controla la expresión de genes anti-inflamatorios según el origen del macrófago.
Tabata, Yasuhiko. „Macrophage phagocytosis of polymer microspheres and antitumor activation of macrophages“. Kyoto University, 1987. http://hdl.handle.net/2433/74704.
Raborn, Erinn Shenee. „Cannabinoid Modulation of Chemotaxis of Macrophages and Macrophage-like Cells“. VCU Scholars Compass, 2007. http://hdl.handle.net/10156/1333.
Grand-Perret, Thierry A. R. „Induction d'une activité anti-tumorale chez les macrophages péritonéaux murins“. Paris 11, 1986. http://www.theses.fr/1986PA112301.
Bouchareychas, Laura. „Implication des phagocytes mononuclées dans l'évolution de la plaque d'athérosclérose et relation avec l'homéostasie du cholestérol et des lipoprotéines“. Thesis, Paris 6, 2014. http://www.theses.fr/2014PA066282/document.
Atherosclerosis represents a chronic pathophysiological process implicated in the majority of cardiovascular diseases. The development of atherosclerotic lesions is characterized by an accumulation of extra and intracellular lipids in the arterial wall at the origin of a strong inflammatory response involving macrophages.Macrophages are considered key actors in the development of atherosclerotic plaques. Indeed, because of their ability to metabolize cholesterol (capture, storage, efflux), to regulate inflammation and to phagocyte apoptotic cells, they exert pro and/or anti-atherogenic functions that may be modulated therapeutically. In this context, we evaluated the therapeutic potential of macrophages protected against apoptosis, on the progression of established atherosclerotic lesions.We have demonstrated that increased macrophage survival can slow down the progression of established lesions, stabilize lesion and reduce cholesterol levels. These athero-protective effects are attributed to the increase in Kupffer cells and Ly-6Clow monocytes partly due to their ability to produce apolipoprotein E. We also show that Kupffer cells are involved in the clearance of pro-atherogenic lipoproteins. The increase in ApoE pool and in Kupffer cells reduces cholesterol levels and thus lesion progression
Di, Maggio Paula. „Dietary lipids and inflammation : chylomicron remnants suppress pro-inflammatory pathways and activate antioxidant defence mechanisms in human macrophages“. Thesis, Royal Veterinary College (University of London), 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618287.
Georges, George Tharwat. „Novel Characteristics of Murine Bone Marrow-Derived Macrophages and Human Macrophage-Like Cells“. VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/932.
Awomoyi, Agnes Abiola Oluwatoyin. „Genetics of susceptibility to tuberculosis“. Thesis, Open University, 2000. http://oro.open.ac.uk/58012/.
Suñer, Navarro Clara. „CPEB4 function in macrophages“. Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/663483.
Como células del sistema inmune innato, los macrófagos detectan señales de peligro endógenas y exógenas y responden desencadenando procesos inflamatorios. Estas respuestas inflamatorias tienen que ser inducidas rápidamente pero a su vez, deben ser eficientemente resueltas. Para ello, los macrófagos inducen la expresión de mediadores pro- y anti- inflamatorios que controlan la expresión unos de otros mediante complejos circuitos regulatorios. Estos procesos requieren un estricto control de la expresión génica a distintos niveles. En los últimos años, se ha descrito que la regulación de los mRNAs por deadenilación es un elemento crucial para regular intensidad y sobretodo la duración de las respuestas inflamatorias. La família de proteínas de unión al RNA CPEBs (Cytoplasmic Polyadenylation Element Binding, CPEB1-4), unen mRNAs que contienen CPEs (Cytoplasmic Polyadenylation Elements) en su 3’UTR. Las CPEBs pueden reclutar dos tipos de complejos en los mRNAs que unen. Estos complejos modulan la longitud de la cola poly(A) y, por tanto, pueden reprimir o estimular su traducción. Los mRNAs de múltiples mediadores inflamatorios y son susceptibles de ser regulados por las CPEBs ya que contienen CPEs en sus 3’UTRs. Por tanto, las CPEBs podrían ser un nuevo mecanismo regulador del desarrollo de las respuestas inflamatorias. En este trabajo hemos descubierto que CPEB4 participa en la respuesta de los macrófagos frente a LPS. El tratamiento con LPS provoca un incremento en los niveles de CPEB4 y promueve que su función sea de polyadenylación. Este proceso es mediado por las MAPK p38α y ERK1/2 y dos proteínas que regulan mRNAs mediante la unión a AREs. El patrón de expresión de CPEB4 sugiere que esta proteína participa en la fase tardía de la respuesta a LPS, cuándo la respuesta inflamatoria es resuelta. Apoyando esta hipótesis, ratones KO para CPEB4 en las células mieloides son más sensibles al shock séptico inducido por LPS. Identificando los mRNAs que CPEB4 regula en este contexto, hemos descrito que CPEB4 regula la expresión de inhibidores de la señalización de la vía MAPK. De este modo, CPEB4 es necesaria para la resolución de la inflamación en respuesta a LPS. Además, hemos descrito como la regulación de mRNAs por CPEB4, HuR y TTP define diferentes patrones temporales de expresión durante el desarrollo de respuestas inflamatorias.
Bücher zum Thema "Macrophages":
Kloc, Malgorzata, Hrsg. Macrophages. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54090-0.
Rousselet, Germain, Hrsg. Macrophages. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3.
Michna, Horst. The human macrophage system: Activity and functional morphology. Basel: Karger, 1988.
Lawrence, Toby, und Thorsten Hagemann, Hrsg. Tumour-Associated Macrophages. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-0662-4.
H, Heppner Gloria, und Fulton Amy M. 1950-, Hrsg. Macrophages and cancer. Boca Raton, Fla: CRC Press, 1988.
David, Evered, Nugent Jonathan, O'Connor Maeve, Ciba Foundation und Symposium on Biochemistry of Microphages (1985 : Ciba Foundation), Hrsg. Biochemistry of macrophages. Chichester: John Wiley, 1986.
Lawrence, Toby. Tumour-Associated Macrophages. New York, NY: Springer Science+Business Media, LLC, 2012.
Mass, Elvira, Hrsg. Tissue-Resident Macrophages. New York, NY: Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3437-0.
Reiner, Neil E., Hrsg. Macrophages and Dendritic Cells. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-396-7.
Horton, Michael A., Hrsg. Macrophages and Related Cells. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9534-9.
Buchteile zum Thema "Macrophages":
Kelly, Aoife, Aleksander M. Grabiec und Mark A. Travis. „Culture of Human Monocyte-Derived Macrophages“. In Macrophages, 1–11. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_1.
Ian Cumming, R., und Yen-Rei A. Yu. „Phenotyping Tumor-Associated Macrophages“. In Macrophages, 99–109. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_10.
Dalby, Elizabeth. „Activating Murine Macrophages In Vitro“. In Macrophages, 111–17. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_11.
Huang, Xuan, Yong Li, Mingui Fu und Hong-Bo Xin. „Polarizing Macrophages In Vitro“. In Macrophages, 119–26. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_12.
Roback, Linda, und Lisa P. Daley-Bauer. „Viral Replication Assay in Bone Marrow-Derived Macrophages“. In Macrophages, 127–34. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_13.
Aribi, Mourad. „Macrophage Bactericidal Assays“. In Macrophages, 135–49. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_14.
Montaño, Fernando, Sergio Grinstein und Roni Levin. „Quantitative Phagocytosis Assays in Primary and Cultured Macrophages“. In Macrophages, 151–63. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_15.
Mularski, Anna, Florence Marie-Anaïs, Julie Mazzolini und Florence Niedergang. „Observing Frustrated Phagocytosis and Phagosome Formation and Closure Using Total Internal Reflection Fluorescence Microscopy (TIRFM)“. In Macrophages, 165–75. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_16.
Rousselet, Germain. „Chromatin Immunoprecipitation in Macrophages“. In Macrophages, 177–86. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_17.
Keller, Andrea-Anneliese, Marten B. Maeß, Michael Schnoor, Berith Scheiding und Stefan Lorkowski. „Transfecting Macrophages“. In Macrophages, 187–95. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_18.
Konferenzberichte zum Thema "Macrophages":
Mahgoub, Yasmine, Rida Arif und Susu Zughaier. „Pyocyanin pigment from Pseudomonas aeruginosa modulates innate immune defenses in macrophages“. In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0137.
van Dam-Mieras, M. C. E., A. D. Muller und G. Hornstra. „DIETARY LIPIDS, INFECTION AND MACROPHAGE PROCOAGULANT ACTIVITY“. In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643398.
McGee, Maria, und Henry Rothberger. „MECHANISMS OF PROCOAGULANT GENERATION BY ALVEOLAR MACROPHAGES DURING MATURATION“. In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643168.
Reinhard, Björn M., Hongyun Wang und Linxi Wu. „Monitoring Cellular Trafficking of Nanoparticle Cargo in Murine Macrophages Through Plasmon Coupling Microscopy“. In ASME 2013 2nd Global Congress on NanoEngineering for Medicine and Biology. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/nemb2013-93078.
Adany, R., A. Kiss, J. Kappelmayer, R. J. Ablin und L. Muszbek. „EXPRESSION OF FACTOR XIII SUBUNIT A IN DIFFERENT TYPES OF HUMAN MACROPHAGES“. In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644651.
Muszbek, L., und R. Adány. „CELLULAR DISTIBUTION OF FACTOR XIII IN HUMAN UTERUS AND PLACENTA“. In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644648.
Belchamber, K., und E. Sapey. „S51 Hungry hungry macrophages: how multiple prey affects macrophage phagocytosis“. In British Thoracic Society Winter Meeting, Wednesday 17 to Friday 19 February 2021, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2021. http://dx.doi.org/10.1136/thorax-2020-btsabstracts.56.
Gijsen, Frank, Anna Ten Have, Jolanda Wentzel und Antonius Van Der Steen. „Temperature Measurement of Advanced Murine Atherosclerotic Plaques“. In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176307.
Alahmadi, Turki, Nurlan Dauletbaev, Kassey Herscovitch, Moishe Liberman und Larry Lands. „The Role Of P38 Mitogen Activated Protein Kinase In Macrophage Inflammatory Responses: Comparison Between Airway Macrophages And Monocytes-Derived Macrophages“. In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1377.
Hughes, Jody M., und Keith M. Wille. „When Macrophages Attack“. In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5362.
Berichte der Organisationen zum Thema "Macrophages":
Havell, Edward A. Actions of Interferons on Macrophages. Fort Belvoir, VA: Defense Technical Information Center, Juni 1985. http://dx.doi.org/10.21236/ada157006.
Naftolin, Frederick. Macrophages, Estrogen and the Microenvironment in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 1999. http://dx.doi.org/10.21236/ada383077.
Benson, J. M., K. J. Nikula und R. A. Guilmette. Evidence for particle transport between alveolar macrophages in vivo. Office of Scientific and Technical Information (OSTI), Dezember 1995. http://dx.doi.org/10.2172/381362.
Shpigel, Nahum, Raul Barletta, Ilan Rosenshine und Marcelo Chaffer. Identification and characterization of Mycobacterium paratuberculosis virulence genes expressed in vivo by negative selection. United States Department of Agriculture, Januar 2004. http://dx.doi.org/10.32747/2004.7696510.bard.
Yull, Fiona. NF-kappaB Activity in Macrophages Determines Metastatic Potential of Breast Tumor Cells. Fort Belvoir, VA: Defense Technical Information Center, August 2010. http://dx.doi.org/10.21236/ada541379.
Nelson, Corwin, Donald C. Beitz, Tim Reinhardt und John Lippolis. Toll-Like Receptor Signaling in Bovine Macrophages Increases 1,25-Dihydroxyvitamin D3 Production. Ames (Iowa): Iowa State University, Januar 2008. http://dx.doi.org/10.31274/ans_air-180814-482.
Yull, Fiona. NF-kappaB Activity in Macrophages Determines Metastatic Potential of Breast Tumor Cells. Fort Belvoir, VA: Defense Technical Information Center, August 2011. http://dx.doi.org/10.21236/ada554014.
Adiga, Umesh, Brian Bell, Larissa Ponomareva, Sandra Nelson, Stephen Kanzleman, Debbie Taylor, Ryan Kramer und Thomas Lamkin. Automated Analysis and Classification of Infected Macrophages Using Bright-Field Amplitude Contrast Data. Fort Belvoir, VA: Defense Technical Information Center, August 2012. http://dx.doi.org/10.21236/ada578711.
Kim, Isaac. Neuroendocrine Differentiation in Prostate Cancer: Role of Bone Morphogenetic Protein-6 and Macrophages. Fort Belvoir, VA: Defense Technical Information Center, Juli 2011. http://dx.doi.org/10.21236/ada555480.
Splitter, Gary, und Menachem Banai. Microarray Analysis of Brucella melitensis Pathogenesis. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7709884.bard.