Thematische Bibliographien / Lyst

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Auswahl der wissenschaftlichen Literatur zum Thema „Lyst“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 16. Februar 2022

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Zeitschriftenartikel zum Thema "Lyst"

Teves, Franco, Mónica Lamas-Maceiras, Carlos García-Estrada, Javier Casqueiro, Leopoldo Naranjo, Ricardo V. Ullán, José-Martín Scervino, Xiaobin Wu, Tania Velasco-Conde und Juan F. Martín. „Transcriptional upregulation of four genes of the lysine biosynthetic pathway by homocitrate accumulation in Penicillium chrysogenum: homocitrate as a sensor of lysine-pathway distress“. Microbiology 155, Nr. 12 (01.12.2009): 3881–92. http://dx.doi.org/10.1099/mic.0.031005-0.

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The lysine biosynthetic pathway has to supply large amounts of α-aminoadipic acid for penicillin biosynthesis in Penicillium chrysogenum. In this study, we have characterized the P. chrysogenum L2 mutant, a lysine auxotroph that shows highly increased expression of several lysine biosynthesis genes (lys1, lys2, lys3, lys7). The L2 mutant was found to be deficient in homoaconitase activity since it was complemented by the Aspergillus nidulans lysF gene. We have cloned a gene (named lys3) that complements the L2 mutation by transformation with a P. chrysogenum genomic library, constructed in an autonomous replicating plasmid. The lys3-encoded protein showed high identity to homoaconitases. In addition, we cloned the mutant lys3 allele from the L2 strain that showed a G1534 to A1534 point mutation resulting in a Gly495 to Asp495 substitution. This mutation is located in a highly conserved region adjacent to two of the three cysteine residues that act as ligands to bind the iron–sulfur cluster required for homoaconitase activity. The L2 mutant accumulates homocitrate. Deletion of the lys1 gene (homocitrate synthase) in the L2 strain prevented homocitrate accumulation and reverted expression levels of the four lysine biosynthesis genes tested to those of the parental prototrophic strain. Homocitrate accumulation seems to act as a sensor of lysine-pathway distress, triggering overexpression of four of the lysine biosynthesis genes.

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Westphal, Andreas, Weijia Cheng, Jinbo Yu, Guntram Grassl, Martina Krautkrämer, Otto Holst, Niko Föger und Kyeong-Hee Lee. „Lysosomal trafficking regulator Lyst links membrane trafficking to toll-like receptor–mediated inflammatory responses“. Journal of Experimental Medicine 214, Nr. 1 (23.11.2016): 227–44. http://dx.doi.org/10.1084/jem.20141461.

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Subcellular compartmentalization of receptor signaling is an emerging principle in innate immunity. However, the functional integration of receptor signaling pathways into membrane trafficking routes and its physiological relevance for immune responses is still largely unclear. In this study, using Lyst-mutant beige mice, we show that lysosomal trafficking regulator Lyst links endolysosomal organization to the selective control of toll-like receptor 3 (TLR3)– and TLR4-mediated proinflammatory responses. Consequently, Lyst-mutant mice showed increased susceptibility to bacterial infection and were largely resistant to endotoxin-induced septic shock. Mechanistic analysis revealed that Lyst specifically controls TLR3- and TLR4-induced endosomal TRIF (TIR domain–containing adapter-inducing interferon β) signaling pathways. Loss of functional Lyst leads to dysregulated phagosomal maturation, resulting in a failure to form an activation-induced Rab7+ endosomal/phagosomal compartment. This specific Rab7+ compartment was further demonstrated to serve as a major site for active TRIF signaling events, thus linking phagosomal maturation to specific TLR signaling pathways. The immunoregulatory role of Lyst on TLR signaling pathways was confirmed in human cells by CRISPR/Cas9-mediated gene inactivation. As mutations in LYST cause human Chédiak-Higashi syndrome, a severe immunodeficiency, our findings also contribute to a better understanding of human disease mechanisms.

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Morsing, Ole. „Det fælles er at brede lyst!“ Slagmark - Tidsskrift for idéhistorie, Nr. 11 (31.01.2018): 168–70. http://dx.doi.org/10.7146/sl.v0i11.103504.

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Thing, Morten. „Pornografica: lyst & frihed, profit & censur“. Magasin fra Det Kongelige Bibliotek 15, Nr. 1 (01.03.2002): 59–67. http://dx.doi.org/10.7146/mag.v15i1.66839.

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Grodal, Torben Kragh. „Arbejds-lyst eller Back to the Future“. K&K - Kultur og Klasse 15, Nr. 57 (28.01.1987): 41–75. http://dx.doi.org/10.7146/kok.v15i57.20750.

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Harris, Edward, Ning Wang, Wei-l. Wu, Alisha Weatherford, Arturo De Lozanne und James Cardelli. „DictyosteliumLvsB Mutants Model the Lysosomal Defects Associated with Chediak-Higashi Syndrome“. Molecular Biology of the Cell 13, Nr. 2 (Februar 2002): 656–69. http://dx.doi.org/10.1091/mbc.01-09-0454.

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Chediak-Higashi syndrome is a genetic disorder caused by mutations in a gene encoding a protein named LYST in humans (“lysosomal trafficking regulator”) or Beige in mice. A prominent feature of this disease is the accumulation of enlarged lysosome-related granules in a variety of cells. The genome of Dictyostelium discoideumcontains six genes encoding proteins that are related to LYST/Beige in amino acid sequence, and disruption of one of these genes,lvsA (large volumesphere), results in profound defects in cytokinesis. To better understand the function of this family of proteins in membrane trafficking, we have analyzed mutants disrupted in lvsA, lvsB, lvsC, lvsD, lvsE, and lvsF. Of all these, onlylvsA and lvsB mutants displayed interesting phenotypes in our assays. lvsA-null cells exhibited defects in phagocytosis and contained abnormal looking contractile vacuole membranes. Loss of LvsB, theDictyostelium protein most similar to LYST/Beige, resulted in the formation of enlarged vesicles that by multiple criteria appeared to be acidic lysosomes. The rates of endocytosis, phagocytosis, and fluid phase exocytosis were normal inlvsB-null cells. Also, the rates of processing and the efficiency of targeting of lysosomal α-mannosidase were normal, although lvsB mutants inefficiently retained α-mannosidase, as well as two other lysosomal cysteine proteinases. Finally, results of pulse-chase experiments indicated that an increase in fusion rates accounted for the enlarged lysosomes inlvsB-null cells, suggesting that LvsB acts as a negative regulator of fusion. Our results support the notion that LvsB/LYST/Beige function in a similar manner to regulate lysosome biogenesis.

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Seip, Ingebjørg. „Platon igjen: Kunst som sorgarbeid og filosofiens lyst“. Norsk filosofisk tidsskrift 43, Nr. 02 (04.06.2008): 100–102. http://dx.doi.org/10.18261/issn1504-2901-2008-02-01.

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Jorth, Peter, und Marvin Whiteley. „Characterization of a Novel Riboswitch-Regulated Lysine Transporter in Aggregatibacter actinomycetemcomitans“. Journal of Bacteriology 192, Nr. 23 (01.10.2010): 6240–50. http://dx.doi.org/10.1128/jb.00935-10.

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ABSTRACT Aggregatibacter actinomycetemcomitans is an opportunistic pathogen that resides primarily in the mammalian oral cavity. In this environment, A. actinomycetemcomitans faces numerous host- and microbe-derived stresses, including intense competition for nutrients and exposure to the host immune system. While it is clear that A. actinomycetemcomitans responds to precise cues that allow it to adapt and proliferate in the presence of these stresses, little is currently known about the regulatory mechanisms that underlie these responses. Many bacteria use noncoding regulatory RNAs (ncRNAs) to rapidly alter gene expression in response to environmental stresses. Although no ncRNAs have been reported in A. actinomycetemcomitans, we propose that they are likely important for colonization and persistence in the oral cavity. Using a bioinformatic and experimental approach, we identified three putative metabolite-sensing riboswitches and nine small regulatory RNAs (sRNAs) in A. actinomycetemcomitans during planktonic and biofilm growth. Molecular characterization of one of the riboswitches revealed that it is a lysine riboswitch and that its target gene, lysT, encodes a novel lysine-specific transporter. Finally, we demonstrated that lysT and the lysT lysine riboswitch are conserved in over 40 bacterial species, including the phylogenetically related pathogen Haemophilus influenzae.

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Petersen, Anders Klostergaard. „Religionshistorie på lyst og fromme eller hvad?: En review-artikel af Mikael Rothstein, Den kristne krop.“ Religionsvidenskabeligt Tidsskrift, Nr. 69 (05.03.2019): 236–46. http://dx.doi.org/10.7146/rt.v0i69.112775.

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Schepelern, Peter. „Mellem lyst og pligt: Filmkultur og filmkritik i Danmark“. MedieKultur: Journal of media and communication research 11, Nr. 23 (02.09.1995): 21. http://dx.doi.org/10.7146/mediekultur.v11i23.1046.

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Filmen står måske i dag for de fleste, som det af de audiovisuelle medier der er tættest på at repræsentere kunsten. Men sådan har det ikke altid været. Som alle nye medier måtte filmen først igennem en lang historisk fase, hvor den nærmest blev betragtet som en trussel mod kulturen, indtil den i dag er blevet en statsbeskyttet filmkultur. I denne artikel skriver Peter Schepelern oplagt og underholdende om den danske filmkulturs udvikling fra skadelig kultur til kunst, og samtidig fortæller han historien om kritikken og teorierne om filmen fra starten til i dag i lyset af de store ideologiske og kulturelle bevægelser og i relation til de andre kunstarter. Hans konklusion er, at filmen i dag har erhvervet respektabilitet i det etablerede kulturlivs øjne, men stadigvæk ikke har fået ligestilling.

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Dissertationen zum Thema "Lyst"

Trantow, Colleen. „Genetic pathways of Lyst and exfoliation syndrome“. Diss., University of Iowa, 2009. https://ir.uiowa.edu/etd/896.

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Human eyes with exfoliation syndrome (XFS) exhibit a distinctive pattern of iris transillumination defects that are recapitulated in Lyst mutant mice carrying the beige allele. Here I present the identification and characterization of the B6-Lystbg-J mouse model of XFS, modifiers of Lyst mediated ocular phenotypes, mechanisms of intraocular pressure (IOP) pathology related to circadian rhythms, and mechanisms of iris transillumination defects in the B6-Lystbg-J mice. Clinical and histological analysis shows that the B6-Lystbg-J mice have multiple similarities to human XFS including: iris transillumination defects, production of an exfoliative-like material, and pronounced pigment dispersion. Despite these insults, Lyst mutation does not cause increased IOP or optic nerve damage within the context of a C57BL/6J genetic background. However, defects in the circadian rhythm regulation of IOP were identified. Sequence analysis identifies that the beige mutation is predicted to delete a single isoleucine from the WD40 domain of the LYST protein. I identified CSNK2B as a binding partner of LYST and showed that LYSTbg-J completely disrupts the interaction. CSNK2B function in regulating E-cadherin and β-catenin binding is subsequently disrupted. These results lead to a working hypothesis that aspects of the XFS phenotype involve LYST and CSNK2B pathways, likely influencing cell-cell adherens junctions. Epistasis experiments were used to test for genetic modifiers of Lyst, which demonstrated that albino Lyst mutant mice exhibited complete rescue of Lyst-dependent iris phenotypes. In a genetic background-driven approach, a DBA/2J strain of congenic mice was created. The DBA/2J background, which harbors multiple mutations influencing melanosomal-proteins, enhanced Lyst dependent iris phenotypes. Thus, both experimental approaches implicated melanosomes, a potential source of oxidative stress, as mechanistically contributory. Supporting a contributory role of oxidative damage, Lyst mutation resulted in genetic context sensitive changes in iris lipid hydroperoxide levels, being lowest in albino and highest in DBA/2J mice. These results identified an association between oxidative damage to lipid membranes and severity of Lyst-mediated phenotypes, uncovering a new mechanism contributing to pathophysiology involving LYST. In conclusion these results demonstrate that mutation of the Lyst gene can produce ocular features of human XFS and suggests that LYST or LYST-interacting genes may contribute to XFS.

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Burgess, Agathe. „Fonction et interactions de la protéine lyst, responsable du syndrome de Chediak Higashi“. Paris 5, 2011. http://www.theses.fr/2011PA05T004.

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Le syndrome de Chediak-Higashi (CHS) est une maladie rare, de transmission autosomale récessive, qui associe un albinisme partiel et une pathologie immunitaire sévère, associée notamment à une activité cytotoxique des lymphocytes T déficiente. L’apparition d’une activation exagérée et incontrôlée des lymphocytes et des macrophages, connue sous le nom de « phase accélérée » de la maladie, détermine le pronostique vital du patient. La caractéristique la plus remarquable de cette maladie est la présence de granules cytoplasmiques géants dans la plupart des types cellulaires. Le gène responsable, identifié en 1996, code pour la protéine cytosolique LYST (pour Lysosomal Trafficking Regulator), à laquelle on attribue de manière générale un rôle dans la régulation du trafic vésiculaire, mais dont la compréhension moléculaire reste encore à ce jour très limitée. L’objectif des présents travaux doctoraux est de chercher à mieux appréhender le rôle de la protéine LYST, causative du syndrome de Chediak Higashi, dans la cellule et d’identifier de nouveaux partenaires de cette protéine. Dans ce but, deux axes principaux de recherche ont été développés. Le premier axe intéresse l’analyse structurale de la protéine. LYST est une protéine cytosolique de 425 kDa et appartient à la famille BEACH (Beige and Chediak Higashi), qui comprend un éventail de protéines de taille importante ayant en commun la même architecture C-terminale (un domaine homologue à la pleckstrine PH, suivi d’un domaine BEACH et enfin de motifs WD40 répétés). De manière surprenante, la connaissance de la séquence de LYST a peu apporté à la compréhension de la fonction de cette protéine. Les 2/3 N-terminaux de LYST forment une séquence orpheline, sans motifs canoniques reconnaissables qui permettraient de lui attribuer une fonction propre. Afin de progresser dans la caractérisation fonctionnelle de cette protéine, une étude structurale fine réalisée avec le groupe de Jean-Paul Mornon et d’Isabelle Callebaut (IMPMC, CNRS UMR 7590) nous a permis d’identifier un nouveau domaine lectine au sein de LYST qui ouvre de nouvelles perspectives quant au rôle de cette protéine dans la cellule. D'autre part, la recherche de mutations faux-sens chez des patients présentant une forme modérée du syndrome nous a permis de désigner plusieurs résidus fonctionnellement importants. Le second axe de recherche vise à caractériser plus directement les conséquences d’un défaut de LYST dans le trafic vésiculaire intracellulaire. Nous avons questionné la nature de la granulation géante présente dans les cellules cytotoxiques de patients CHS ; ce compartiment contient la perforine (protéine lytique spécifique des granules cytotoxiques), exprime la molécule LAMP (caractéristique des endosomes tardifs et des lysosomes) et sa génération semble intervenir au point terminal de la voie d’endocytose. De plus, nous mettons en évidence dans ces lymphocytes une fusion anormale de compartiments endosomaux impliqués dans la formation des « vésicules d’exocytoses », nécessaires à la maturation terminale des granules cytotoxiques. L’étude de certaines voies du trafic vésiculaire intracellulaire impliquant le réseau endosomal nous a permis de montrer une perturbation de la voie de dégradation des protéines ubiquitinilées médiée par les ESCRTs (Endosomal Sorting Complex Required for Transport) dans les fibroblastes de patients CHS. L’importance physiologique de cette voie des ESCRTs et son implication dans divers processus cellulaires commencent tout juste à être cernées. Nos résultats favorisent un rôle primaire de LYST dans la biogénèse du compartiment endosomal qui affecte la maturation terminale des lysosomes sécrétoires
The Chediak Higashi syndrome (CHS) is an autosomic recessive disorder, which associates partial albinism with severe immune dysfunctions including a defective cytotoxic activity of T lymphocytes and NK cells. The occurrence of an uncontrolled activation of lymphocytes and macrophages, known as « accelerated phase » of the disease, determines the vital prognosis of the patient. The hallmark of this disease is the presence of giant cytoplasmic granules or organelles in most cell types. The mutant gene, identified in 1996, codes for the cytosolic LYST protein (for Lysosomal Trafficking Regulator), which is defined as a vesicle-trafficking regulatory protein, but whose precise function remains unknown. The main goal of this doctoral work was to try to better understand the role of the LYST protein, responsible for the Chediak Higashi syndrome, in the cell and to identify new partners of this protein. In this purpose, two main research aims have been developped. The first aim is based on the structural analysis of the protein. LYST is a 425kDa cytosolic protein and belongs to the BEACH family (Beige and Chediak Higashi) which is composed of various large-sized proteins sharing the three same C-terminal domains (a pleckstrine homologous PH domain, followed by the BEACH domain and finally by WD40 repeats). Noticeably, the knowledge of the sequence of LYST was relatively unrevealing. Indeed, its N-terminal 2/3 part forms an orphan sequence within which no canonical motifs with assigned function could be found. To progress in the functionnal characterization of LYST, a fine structural study was performed with the group of Isabelle Callebaut and Jean-Paul Mornon (IMPMC, CNRS UMR 7590) which lead us to the identification of a new concanavalin-A lectin domain in the LYST protein. This data helps opening new perspectives regarding the potential role of LYST in the cellular machinery. On the other hand, the search for missense mutations in patients expressing a mild form of the disease allowed us to point out some functionnally critical residues of the protein. The second aim of our research has tried to better characterize the consequences of a LYST defect in the intracellular vesicular trafficking. We questionned the nature of the giant granulation in CHS cytotoxic cells ; this compartment contains the store of perforin (the lytic protein specific of cytotoxic granules), carries the LAMP molecule (characteristic for late endosomes and lysosomes) and its generation seems to occur at the very end point of the endocytosis pathway. Moreover, we highlighted in these lymphocytes an abnormal fusion of endosomal compartments involved in the formation of the « exocytic vesicles », which are necessary to the terminal maturation of cytotoxic granules. The study of three main intracellular vesicular trafficking pathways involving the endosomal network has allowed us to show an impairment of the degradative ESCRT-mediated pathway (Endosomal Sorting Complex Required for Transport) in CHS patient fibroblasts. The physiological importance of this pathway and its involvment in various cellular processes just begins to be figured out. Our results support a primary role of LYST in endosomal biogenesis which affects the terminal maturation of secretory lysosomes

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Schurek, Beate Bozena [Verfasser]. „Die funktionelle Rolle der Membrantransportregulatoren RIN3 und Lyst in der Mastzelldegranulation / Beate Bozena Schurek“. Lübeck : Zentrale Hochschulbibliothek Lübeck, 2012. http://d-nb.info/1028951418/34.

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Kuusela, Tommy. „"Hallen var lyst i helig frid" : Krig och fred mellan gudar och jättar i en fornnordisk hallmiljö“. Doctoral thesis, Stockholms universitet, Institutionen för etnologi, religionshistoria och genusvetenskap, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-147261.

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This thesis is the first study to examine the interaction between gods and giants in Old Norse mythology from the perspective of Iron Age halls. Its central aim is to contextualise Old Norse mythological narratives that describe the interactions between gods and giants in a hall environment, and to show how the mythological depictions can be compared to the norms and rules found in Iron Age hall culture, especially in connection with its warrior ideology. The relationships observed also apply to the Iron Age’s aristocratic sovereigns and their dynamic dealings – both peaceful and martial – found in the connection and rivalry between different halls and hall owners. The giants are related to the concept of “the Other”, and as hall-owners can thus be contextualised with real social relations in Iron Age society. The investigation centers arounds key topics from the perspective of a hall setting, departing from mythic traditions regarding Óðinn and Þórr as guests in the halls of giants. These topics include grið within the hall; the good and generous host; the dangerous and hostile guest; the hall as an arena for knowledge and mead; and finally the destruction of halls as an attack on the hall owner’s fame and honour. Similarities and differences between myths about Óðinn’s and Þórr’s interaction with hall-owning giants are examined in depth, and it is argued that Óðinn embodies wisdom and extracts knowledge or valuables from the giants by cunning tricks or manipulation, having (usually) travelled there alone and in disguise. Þórr, on the other hand, is argued to embody physical strength, honour, glory and courage, and his dealings with the giants revolve around these issues. He seldom seems to travel alone or under cover, and when his courage or honour is threatened, his response is to kill his host (and his retinue) and to destroy the giant’s hall. It is argued that the Old Norse conception of the world is to be understood as neither dualistic or monistic. Instead, it is proposed that the myths can be understood from a perspective of conflicts that are temporal and not permanent in nature.

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Best, Cameron A. „Improvement of the Tissue-Engineered Vascular Graft and Discovery of a Novel Immunomodulator“. The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1562760692281514.

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Cheng, Weijia [Verfasser], Kyeong-Hee [Akademischer Betreuer] Lee und Guntram [Akademischer Betreuer] Graßl. „The role of lysosomal trafficking regulator Lyst in Toll-like receptor-mediated signaling and inflammation / Weijia Cheng ; Akademische Betreuer: Kyeong-Hee Lee, Guntram Graßl ; Institut für Klinische Chemie, AG: Entzündungsforschung“. Hannover : Bibliothek der Medizinischen Hochschule Hannover, 2017. http://d-nb.info/1149159901/34.

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Cheng, Weijia [Verfasser], Kyeong-Hee [Akademischer Betreuer] Lee und Guntram Alexander [Akademischer Betreuer] Graßl. „The role of lysosomal trafficking regulator Lyst in Toll-like receptor-mediated signaling and inflammation / Weijia Cheng ; Akademische Betreuer: Kyeong-Hee Lee, Guntram Graßl ; Institut für Klinische Chemie, AG: Entzündungsforschung“. Hannover : Bibliothek der Medizinischen Hochschule Hannover, 2017. http://d-nb.info/1149159901/34.

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Rønning, Benedicte Stordal. „”Jeg har jo lyst til å vise han at jeg ser han” : En kvalitativ intervjustudie av hva tre lærere legger vekt på i sin relasjon med elever som viser innagerende atferd“. Thesis, Norges teknisk-naturvitenskapelige universitet, Pedagogisk institutt, 2014. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-26006.

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Innagerende atferd er nesten like vanlig som utagerende atferd (Sørlie & Nordahl, 1998), likevel får utagerende atferd langt større oppmerksomhet i skolen (Nordahl, Manger, Sørlie, & Tveit, 2005). I denne studien har jeg valgt å rette fokus mot lærer-elev-relasjonen og elever som viser innagerende atferd. Innagerende atferd kan defineres som en atferd ”der følelser, opplevelser og tanker vendes innover mot en selv. Uttrykk som kommuniseres kan være: sårbar, avvisende, deprimert, tilbaketrukket, angst og usikkerhet” (Lund, 2012, s. 27). Formålet med denne studien er å rette fokus mot denne gruppen elever i skolen, og videre prøve å få frem tre lærer sine opplevelser og erfaringer rundt det å undervise elever som viser innagerende atferd. Problemstillingen som belyses i denne studien er som følger: ”Hva legger tre lærer vekt på i sin relasjon med elever som viser innagerende atferd?” For å belyse problemstillingen er det valgt en kvalitativ metode, nærmere bestemt et kvalitativt forskningsintervju. Studiens empiri bygger på intervju med tre barneskolelærere som har erfaring med å undervise elever som viser innagerende atferd. På bakgrunn av analyse og tolkning av studiens empiri kom jeg frem til en hovedkategori; å se elevene som viser innagerende atferd. Denne kategorien utpekte seg når lærerne uttrykker seg om deres relasjon til elever som viser innagerende atferd. Studien er videre delt inn i fire kategorier som går på hva lærerne uttrykker at de legger vekt på for å se disse elevene. Den første kategorien er å ta seg tid, og omhandler hvordan lærerne uttrykker at de tar seg tid til å prate med og hilse på elevene. Kategorien å ikke gi seg går inn på lærerne sitt fokus på å ikke gi seg og alltid være der for elever som viser innagerende atferd, samt betydningen av dette. Kategorien å ha tro på elevene belyser hvordan lærerne gir uttrykk for at de har forventninger til elevene som viser innagerende atferd, samt hvordan de tilrettelegger for at elevene skal oppleve mestring. Studiens siste kategori er å legge til rette for trygghet i sosiale situasjoner, og belyser lærerne sine tanker rundt det å skape trygghet knyttet til sosiale situasjoner som plassering i klasserommet, gruppearbeid og friminuttet. Studien i sin helhet viser at lærerne hadde kunnskap og oppmerksomhet rettet mot elever som viser innagerende atferd i skolen.

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Gamonet, Franck. „Biosynthèse de la lysine chez la levure Saccharomyces Cerevisiae : rôle(s) des gènes LYS7 et LYS4“. Bordeaux 2, 1997. http://www.theses.fr/1997BOR28536.

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Wu, Chung-Pu. „Investigating histone H3 Lys4 and Lys9 methylation in conditionally immortalized olfactory placode by matrix-assisted laser desorption“. Master's thesis, University of Cape Town, 2002. http://hdl.handle.net/11427/4353.

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Includes bibliographical references.
Published studies have shown that distinctive site-specific histone H3 methylation patterns at lysine 9 and lysine 4 determine the formation of heterochromatin oreuchromatin, respectively. The biological significance of lysine 4 and lysine 9 methylation of histone N-terrriinal tails was investigated in this study with a new approach.

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Bücher zum Thema "Lyst"

Langholm, Siri. Av smertens lyst. Oslo: Solum, 2001.

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Langholm, Siri. Av smertens lyst. Oslo: Solum, 2001.

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Foster, Alan Dean. Cat-a-lyst. 2. Aufl. New York: Ace Books, 1991.

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Lyst do heroi︠a︡ pera--Mariï Matios: Lysty, statti, interv'i︠u︡ i take inshe. Lʹviv: BAK, 2013.

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Brønnum, Jakob. Verden ifølge U2: Lyst og længsel. Frederiksberg: Forlaget Alfa, 2012.

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Liv og lyst: Artikler og essays. [Copenhagen]: Gyldendal, 1987.

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Za ukraïnsʹku Ukraïnu: Vidkrytyĭ lyst ukraïnt︠s︡i︠a︡m. Kyïv: Prodi︠u︡sersʹko-vydavnychyĭ t︠s︡entr "ARATTA", 2005.

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Fielding, Joy. I lyst og np2sd. Kp2sbenhavn: Chr. Erichsen, 1991.

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Morhun, Volodymyr. Na zakhyst batʹka: Vidkrytyĭ lyst zemli͡a︡kam-poltavt͡s︡i͡a︡m. Shyshaky: Va "Psʹol", 1997.

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Thielst, Peter. Latterens lyst: Det frigørende og distancerende grin. [Copenhagen]: Tiderne skifter, 1988.

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Mehr Quellen

Buchteile zum Thema "Lyst"

Ji, Xiaojie, Bo Chang, Jürgen K. Naggert und Patsy M. Nishina. „Lysosomal Trafficking Regulator (LYST)“. In Retinal Degenerative Diseases, 745–50. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17121-0_99.

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Baum, Richard. „Lyot, Bernard“. In Biographical Encyclopedia of Astronomers, 1366–67. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4419-9917-7_878.

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Hohlbach, G. „Intraarterielle Lyse“. In Wandel der Chirurgie in unserer Zeit, 1020. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78145-2_280.

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Murara, Marco, Jeff Suzuki, Simone Dumont, Kim Plofker, Albert Bijaoui, Hartmut Frommert, Henry L. Giclas et al. „Lyot, Bernard“. In The Biographical Encyclopedia of Astronomers, 718–19. New York, NY: Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-30400-7_878.

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Martin, M., und B. J. O. Fiebach. „Arterielle Verschlußkrankheit: Lyse“. In Verhandlungen der Deutschen Gesellschaft für Innere Medizin, 371–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-84317-4_69.

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Spengel, F., und G. Küffer. „Lokale Lyse, Atherektomie, Stent“. In Aktuelle Therapieprinzipien bei der peripheren arteriellen Verschlußkrankheit, 127–39. Wiesbaden: Vieweg+Teubner Verlag, 1991. http://dx.doi.org/10.1007/978-3-322-84056-1_7.

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Klages, Matthias, und Edelgard Lindhoff-Last. „Hämostase, Hämotherapie und Lyse“. In Die Intensivmedizin, 479–509. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54953-3_35.

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Schomburg, Dietmar, und Dörte Stephan. „Peptidyl-Lys metalloendopeptidase“. In Enzyme Handbook 16, 237–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-58903-4_42.

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Brusò, Mayla, und Agostino Cortesi. „Non-repudiation Analysis with LySa“. In Emerging Challenges for Security, Privacy and Trust, 318–29. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-01244-0_28.

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Martin, M., und B. J. O. Fiebach. „Systemische Lyse bei subakuten Arterienverschlüssen“. In Deutsche Gesellschaft für Chirurgie, 417–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-48163-5_82.

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Konferenzberichte zum Thema "Lyst"

Donkor, Eric, Patrick D. Kumavor und Carlos Villa. „Tunable LYOT-filters“. In SPIE Defense, Security, and Sensing, herausgegeben von Michael J. Hayduk, Peter J. Delfyett, Jr., Andrew R. Pirich und Eric J. Donkor. SPIE, 2009. http://dx.doi.org/10.1117/12.819457.

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Matar, M., I. M. Bassett, B. Gordon, J. H. Haywood und A. Michiel. „An improved lyot fibre depolariser“. In Technical Digest - Symposium on Optical Fiber Measurements. IEEE, 2004. http://dx.doi.org/10.1109/sofm.2004.183491.

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Maadadi, S., S. Djabi, M. Rahmani, N. Mebarki und J. Mimouni. „Simulation of the Lyot Coronograph“. In THE THIRD ALGERIAN WORKSHOP ON ASTRONOMY AND ASTROPHYSICS. AIP, 2010. http://dx.doi.org/10.1063/1.3518341.

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Vezenkov, Lyubomir, und Lyubomir Milev. „Use of phenylacetyl- and phenylacetamidomethylprotecting groups in synthesis of [Lys8]vasopressin and [Mpa1,Lys8]vasopressin“. In VIIth Conference Biologically Active Peptides. Prague: Institute of Organic Chemistry and Biochemistry, Academy of Sciences of the Czech Republic, 2001. http://dx.doi.org/10.1135/css200104037.

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Saskevich, N. A., G. V. Sinitsyn und A. V. Kazberuk. „Transmission spectrum of the Lyot filter“. In International Conference on Lasers, Applications, and Technologies '07, herausgegeben von Valentin A. Orlovich, Vladislav Panchenko und Ivan A. Scherbakov. SPIE, 2007. http://dx.doi.org/10.1117/12.752848.

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Socker, Dennis G., Guenter E. Brueckner, Clarence M. Korendyke, D. N. Lilley, James H. Steenson, Preston M. Kohn, Gail M. Lyons et al. „LASCO spectrometric Lyot coronagraph tunable passband filter“. In SPIE's 1996 International Symposium on Optical Science, Engineering, and Instrumentation, herausgegeben von David M. Rust. SPIE, 1996. http://dx.doi.org/10.1117/12.259711.

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Vigan, A., M. N'Diaye und K. Dohlen. „Stop-less Lyot coronagraph for exoplanet characterization“. In SPIE Astronomical Telescopes + Instrumentation, herausgegeben von Ian S. McLean, Suzanne K. Ramsay und Hideki Takami. SPIE, 2012. http://dx.doi.org/10.1117/12.925294.

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Novotny, R. W., W. M. Doring, V. Dormenev, P. Drexler, M. Rost, M. Thiel und A. Thomas. „High-Energy Photon Detection With LYSO Crystals“. In 2006 IEEE Nuclear Science Symposium Conference Record. IEEE, 2006. http://dx.doi.org/10.1109/nssmic.2006.356042.

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Maillard, A., R. Maillartd, P. Villeval, M. Bourezzou, G. Aka, J. Lejay und P. Loiseau. „Crystal growth and optical properties of LYSB“. In Advances in Optical Materials. Washington, D.C.: OSA, 2011. http://dx.doi.org/10.1364/aiom.2011.aithe2.

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Matar, Mamdouh, Andrew Michie, Ian Bassett, John Canning, John Haywood und Justin Digweed. „2-Section PCF Hi-Bi Lyot Depolarisers“. In Optical Fiber Sensors. Washington, D.C.: OSA, 2006. http://dx.doi.org/10.1364/ofs.2006.the60.

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Berichte der Organisationen zum Thema "Lyst"

Atanov, N. Measurement of Time Resolution of the Mu2e LYSO Calorimeter Prototype. Office of Scientific and Technical Information (OSTI), September 2015. http://dx.doi.org/10.2172/1223256.

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Ren-Yuan Zhu. Cerium Doped LSO/LYSO Crystal Development for future High Energy Physics Experiments. Office of Scientific and Technical Information (OSTI), März 2012. http://dx.doi.org/10.2172/1036961.

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Madsen, Simon Ryberg, und Laura Louise Sarauw. Studerende i en fremdriftstid: Prioriteter, valg og dilemmaer set i lyset af fremdriftsreformen. Aarhus University Library, 2016. http://dx.doi.org/10.7146/aul.126.112.

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Piasecki, M. A. J. Possible Basement - Cover Relationships in the Fleur De Lys Terrane, western Newfoundland. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1987. http://dx.doi.org/10.4095/122367.

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Jensen, Bente. Kan daginstitutioner gøre en forskel? Fra forskning om daginstitutioner set i lyset af et kompetence- og eksklusionsperspektiv. Aarhus University, 2008. http://dx.doi.org/10.7146/aul.94.88.

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Coyle, M., und D. Oneschuk. Residual total magnetic field, Baie Verte aeromagnetic survey, Fleur de Lys, 12 I/01, Newfoundland and Labrador. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2008. http://dx.doi.org/10.4095/224693.

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Owen, J. V. Geology of the central part of the Long Range Inlier, Fleur de Lys, White Bay South District, Englee, western Newfoundland. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1987. http://dx.doi.org/10.4095/130277.

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Coyle, M., und D. Oneschuk. First vertical derivative of the magnetic field, Baie Verte aeromagnetic survey, Fleur de Lys, 12 I/01, Newfoundland and Labrador. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2008. http://dx.doi.org/10.4095/224687.

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Anderson, Camille O., Susanne Spiegelberg, John M. Prausnitz und Harvey W. Blanch. Effect of secondary structure on the interactions of peptide T4 LYS (11-36) in mixtures of aqueous sodium chloride and 2,2,2,-Trifluoroethanol. Office of Scientific and Technical Information (OSTI), Oktober 2001. http://dx.doi.org/10.2172/837230.

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Skulski, T., S. Castonguay, W. S. F. Kidd, V. J. McNicoll, C R van Staal und J. P. Hibbard. Geology, Baie Verte and parts of Fleur de Lys, Newfoundland and Labrador, NTS 12-H/16 and part of NTS 12-I/1. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2015. http://dx.doi.org/10.4095/295865.

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