Auswahl der wissenschaftlichen Literatur zum Thema „Lipophagie“
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Zeitschriftenartikel zum Thema "Lipophagie"
Zelickson, B. D., und R. K. Winkelmann. „Lipophagic panniculitis in re-excision specimens.“ Acta Dermato-Venereologica 71, Nr. 1 (01.01.1991): 59–61. http://dx.doi.org/10.2340/00015555715961.
Der volle Inhalt der QuelleSchott, Micah B., Shaun G. Weller, Ryan J. Schulze, Eugene W. Krueger, Kristina Drizyte-Miller, Carol A. Casey und Mark A. McNiven. „Lipid droplet size directs lipolysis and lipophagy catabolism in hepatocytes“. Journal of Cell Biology 218, Nr. 10 (07.08.2019): 3320–35. http://dx.doi.org/10.1083/jcb.201803153.
Der volle Inhalt der QuelleJonas, Wenke, Kristin Schwerbel, Lisa Zellner, Markus Jähnert, Pascal Gottmann und Annette Schürmann. „Alterations of Lipid Profile in Livers with Impaired Lipophagy“. International Journal of Molecular Sciences 23, Nr. 19 (06.10.2022): 11863. http://dx.doi.org/10.3390/ijms231911863.
Der volle Inhalt der QuelleAlexandrides, C. „Lipophagic Granuloma“. Acta Medica Scandinavica 154, S312 (24.04.2009): 449–60. http://dx.doi.org/10.1111/j.0954-6820.1956.tb17036.x.
Der volle Inhalt der QuellePeng, Peng, Wensheng Liu, Adam Utley, Colin Chavel, Louise Carlson, Scott H. Olejniczak und Kelvin P. Lee. „CD28 Induces Autophagy in Plasma Cells to Enhance Mitochondrial Respiration and Survival“. Journal of Immunology 204, Nr. 1_Supplement (01.05.2020): 71.2. http://dx.doi.org/10.4049/jimmunol.204.supp.71.2.
Der volle Inhalt der QuelleKumar, Ravinder, Muhammad Arifur Rahman und Taras Y. Nazarko. „Nitrogen Starvation and Stationary Phase Lipophagy Have Distinct Molecular Mechanisms“. International Journal of Molecular Sciences 21, Nr. 23 (29.11.2020): 9094. http://dx.doi.org/10.3390/ijms21239094.
Der volle Inhalt der QuelleLevy, Jack, Mark E. Burnett und Cynthia M. Magro. „Lipophagic Panniculitis of Childhood“. American Journal of Dermatopathology 39, Nr. 3 (März 2017): 217–24. http://dx.doi.org/10.1097/dad.0000000000000721.
Der volle Inhalt der QuelleUMBERT, I. J., und R. K. WINKELMANN. „Adult lipophagic atrophic panniculitis“. British Journal of Dermatology 124, Nr. 3 (März 1991): 291–95. http://dx.doi.org/10.1111/j.1365-2133.1991.tb00578.x.
Der volle Inhalt der QuelleWinkelmann, R. K., Marian T. McEvoy und Margot S. Peters. „Lipophagic panniculitis of childhood“. Journal of the American Academy of Dermatology 21, Nr. 5 (November 1989): 971–78. http://dx.doi.org/10.1016/s0190-9622(89)70285-1.
Der volle Inhalt der QuelleJuneja, Manish, Pankaj Raut, Milind Lohkare, Harshawardhan Ramteke, Vaishnavi Walke und Sakshi Bhatia. „Effects of Lipophagy on Atherosclerosis“. Central India Journal of Medical Research 2, Nr. 01 (15.05.2023): 17–25. http://dx.doi.org/10.58999/cijmr.v2i01.44.
Der volle Inhalt der QuelleDissertationen zum Thema "Lipophagie"
Chang, Yu-Chin, und 張毓秦. „Activation of lipophagy protects neurons from neurodegeneration caused by sphingolipid imbalance“. Thesis, 2016. http://ndltd.ncl.edu.tw/handle/59910094618794899139.
Der volle Inhalt der Quelle國立臺灣大學
生理學研究所
104
Sphingolipids are essential membrane components of the neuron; hence their levels need to be tightly regulated. Infertile crescent (Ifc) is an evolutionarily conserved dihydroceramide (DHC) desaturase which converts DHC to Ceramide (Cer) for the de novo synthesis of Cer in Drosophila. While the imbalance of Cer, a bioactive sphingolipid, has been associated with several neurodegenerative diseases, the neuronal function of its precursor DHC remains unknown. To investigate the role of ifc, we generated ifc knockout flies (ifc-KO). Sphingolipidomic analysis showed that loss of ifc resulted in increased DHC. Prolonged light stimuli to the ifc-KO eye led to activity-dependent degeneration of photoreceptors. Clonal analysis of ifc-KO photoreceptors revealed the accumulation of lipophagic structure and the increased H2DCF signals upon light stimuli, suggesting that DHC accumulation may activate lipophagy and induce the production of reactive oxygen species (ROS). However, it remains to be determined whether the degeneration is attributed to lipophagic cell death or the ROS insults. Reduction of ifc led to the increase of Atg8/LC3 puncta in the acidified compartment and elevation of lysosomal proteases, indicating the activated lipophagy can promote subsequent lysosomal function. ifc-dependent neurodegeneration can be partially rescued by an antioxidant AD4, indicating that ROS is at least partially responsible for the degeneration. In addition, both ROS elevation and lipid accumulation in ifc-KO was suppressed by treating with the autophagy inducer Rapamycin, suggesting that enhanced lipophagy plays a protective role in ifc-dependent neurodegeneration. Conversely, lipophagy can be downregulated by AD4, indicating ROS insults lead to the feedback upregulation of protective lipophagy. In summary, loss of ifc results in DHC accumulation and ROS generation, the latter of which subsequently activates lipophagy to protect against neurodegeneration. These findings support our hypothesis that DHC is bio-active and lipophagy can be protective, highlighting their potential as therapeutic targets for regulating sphingolipid homeostasis.
Christian, Patricia. „Investigating the Role of Autophagy in Intracellular Apolipoprotein B Traffic and Very-low-density-lipoprotein Assembly and Secretion“. Thesis, 2013. http://hdl.handle.net/1807/42743.
Der volle Inhalt der QuelleBuchteile zum Thema "Lipophagie"
Martinez-Lopez, Nuria. „Regulation of Lipophagy“. In Autophagy and Signaling, 147–72. Boca Raton, FL : CRC Press, 2018. | Series: Methods in signal transduction series: CRC Press, 2017. http://dx.doi.org/10.1201/9781315120638-10.
Der volle Inhalt der QuelleSteinberg, Christian E. W. „Lipid Homeostasis and Lipophagy—‘The Greasy Stuff Balanced’“. In Aquatic Animal Nutrition, 583–97. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-87227-4_24.
Der volle Inhalt der QuelleMagro, Cynthia M., und Josh H. Mo. „Lipophagic/Lipoatrophic Panniculitis: A TH1-Mediated Autoimmune Disorder of the Subcutaneous Fat“. In New and Emerging Entities in Dermatology and Dermatopathology, 277–86. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-80027-7_21.
Der volle Inhalt der QuelleSathyanarayan, Aishwarya. „A Coupled Approach Utilizing Immunohistochemistry and Immunocytochemistry to Visualize Cellular Lipophagy“. In Methods in Molecular Biology, 185–91. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6759-9_11.
Der volle Inhalt der QuelleSathyanarayan, Aishwarya. „Erratum to: A Coupled Approach Utilizing Immunohistochemistry and Immunocytochemistry to Visualize Cellular Lipophagy“. In Methods in Molecular Biology, E1. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6759-9_21.
Der volle Inhalt der Quelle„Lipophagic panniculitis of childhood“. In Dermatology Therapy, 358. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/3-540-29668-9_1645.
Der volle Inhalt der QuelleCristobal-Sarramian, A., M. Radulovic und S. D. Kohlwein. „Methods to Measure Lipophagy in Yeast“. In Methods in Enzymology, 395–412. Elsevier, 2017. http://dx.doi.org/10.1016/bs.mie.2016.09.087.
Der volle Inhalt der QuelleDutta, Shweta, Saraswati Prasad Mishra, Anil Kumar Sahu, Koushlesh Mishra, Pankaj Kashyap und Bhavna Sahu. „Hepatocytes and Its Role in Metabolism“. In Drug Metabolism [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99083.
Der volle Inhalt der QuelleRoccio, Federica, Aurore Claude-Taupin, Joëlle Botti, Etienne Morel, Patrice Codogno und Nicolas Dupont. „Monitoring lipophagy in kidney epithelial cells in response to shear stress“. In Methods in Cell Biology. Elsevier, 2021. http://dx.doi.org/10.1016/bs.mcb.2020.12.003.
Der volle Inhalt der QuelleEsmaeilian, Yashar, Sevgi Yusufoglu, Ece Iltumur, Gamze Bildik und Ozgur Oktem. „Visualizing Lipophagy as a New Mechanism of the Synthesis of Sex Steroids in Human Ovary and Testis Using Immunofluorescence Staining Method“. In Methods in Molecular Biology. New York, NY: Springer US, 2024. http://dx.doi.org/10.1007/7651_2024_520.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Lipophagie"
Irungbam, K., Y. Churin, M. Ocker, M. Roderfeld und E. Roeb. „CB1 knockout alleviates hepatic steatosis via lipophagy and lipolysis in HBs transgenic mice“. In 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677164.
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