Auswahl der wissenschaftlichen Literatur zum Thema „Lentivirusus“
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Zeitschriftenartikel zum Thema "Lentivirusus"
Clements, J. E., und M. C. Zink. „Molecular biology and pathogenesis of animal lentivirus infections.“ Clinical Microbiology Reviews 9, Nr. 1 (Januar 1996): 100–117. http://dx.doi.org/10.1128/cmr.9.1.100.
Der volle Inhalt der QuelleLairmore, M. D., S. T. Butera, G. N. Callahan und J. C. DeMartini. „Spontaneous interferon production by pulmonary leukocytes is associated with lentivirus-induced lymphoid interstitial pneumonia.“ Journal of Immunology 140, Nr. 3 (01.02.1988): 779–85. http://dx.doi.org/10.4049/jimmunol.140.3.779.
Der volle Inhalt der QuelleHötzel, Isidro, und William P. Cheevers. „Conservation of Human Immunodeficiency Virus Type 1 gp120 Inner-Domain Sequences in Lentivirus and Type A and B Retrovirus Envelope Surface Glycoproteins“. Journal of Virology 75, Nr. 4 (15.02.2001): 2014–18. http://dx.doi.org/10.1128/jvi.75.4.2014-2018.2001.
Der volle Inhalt der QuelleArmimi, Anastasia, Afina Firdaus Syuaib, Katherine Vanya, Marselina Irasonia Tan, Dessy Natalia, David Virya Chen, Chikako Ono, Yoshiharu Matsuura, Anita Artarini und Ernawati Arifin Giri-Rachman. „SARS-CoV-2 Neutralization Assay System using Pseudo-lentivirus“. Indonesian Biomedical Journal 15, Nr. 2 (18.04.2023): 179–86. http://dx.doi.org/10.18585/inabj.v15i2.2212.
Der volle Inhalt der QuelleCourgnaud, Valérie, Xavier Pourrut, Frédéric Bibollet-Ruche, Eitel Mpoudi-Ngole, Anke Bourgeois, Eric Delaporte und Martine Peeters. „Characterization of a Novel Simian Immunodeficiency Virus from Guereza Colobus Monkeys (Colobus guereza) in Cameroon: a New Lineage in the Nonhuman Primate Lentivirus Family“. Journal of Virology 75, Nr. 2 (15.01.2001): 857–66. http://dx.doi.org/10.1128/jvi.75.2.857-866.2001.
Der volle Inhalt der QuelleBrowning, Matthew T., Russell D. Schmidt, Kathy A. Lew und Tahir A. Rizvi. „Primate and Feline Lentivirus Vector RNA Packaging and Propagation by Heterologous Lentivirus Virions“. Journal of Virology 75, Nr. 11 (01.06.2001): 5129–40. http://dx.doi.org/10.1128/jvi.75.11.5129-5140.2001.
Der volle Inhalt der QuelleChen, Jianbo, Douglas Powell und Wei-Shau Hu. „High Frequency of Genetic Recombination Is a Common Feature of Primate Lentivirus Replication“. Journal of Virology 80, Nr. 19 (01.10.2006): 9651–58. http://dx.doi.org/10.1128/jvi.00936-06.
Der volle Inhalt der QuelleBaccam, Prasith, Robert J. Thompson, Yuxing Li, Wendy O. Sparks, Michael Belshan, Karin S. Dorman, Yvonne Wannemuehler, J. Lindsay Oaks, James L. Cornette und Susan Carpenter. „Subpopulations of Equine Infectious Anemia Virus Rev Coexist In Vivo and Differ in Phenotype“. Journal of Virology 77, Nr. 22 (15.11.2003): 12122–31. http://dx.doi.org/10.1128/jvi.77.22.12122-12131.2003.
Der volle Inhalt der QuelleNarayan, O., D. Sheffer, J. E. Clements und G. Tennekoon. „Restricted replication of lentiviruses. Visna viruses induce a unique interferon during interaction between lymphocytes and infected macrophages.“ Journal of Experimental Medicine 162, Nr. 6 (01.12.1985): 1954–69. http://dx.doi.org/10.1084/jem.162.6.1954.
Der volle Inhalt der QuelleKafri, Tal, Henriette van Praag, Ling Ouyang, Fred H. Gage und Inder M. Verma. „A Packaging Cell Line for Lentivirus Vectors“. Journal of Virology 73, Nr. 1 (01.01.1999): 576–84. http://dx.doi.org/10.1128/jvi.73.1.576-584.1999.
Der volle Inhalt der QuelleDissertationen zum Thema "Lentivirusus"
Robertson, David L. „Recombination in primate lentiviruses“. Thesis, University of Nottingham, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336866.
Der volle Inhalt der QuelleVödrös, Dalma. „Receptor use of primate lentiviruses /“. Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-497-6/.
Der volle Inhalt der QuelleBailes, Elizabeth. „Origins and evolution of primate lentiviruses“. Thesis, University of Nottingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246384.
Der volle Inhalt der QuelleCordeil, Stéphanie. „Etude de la différence de susceptibilité des lentivirus de primates aux interférons de type I“. Thesis, Lyon, École normale supérieure, 2012. http://www.theses.fr/2012ENSL0781.
Der volle Inhalt der QuelleType I Interferons (IFN-α/β, herein IFNs) provide an important mechanism of defense against pathogens and regulate in a paracrine and autocrine manner both intrinsic and adaptive immune responses. In the case of HIV-1 however, the relationship between IFNs and viral replication appears more complex. Indeed, if IFNs have been described to interfere with HIV-1 at basically all phases of its life cycle ex vivo, an IFN-induced state is linked to AIDS progression and to high viral loads in HIV-1 infected individuals. Similarly, a deregulated and prolonged IFN production/state seems one of the main distinguishing features between pathogenic and non-pathogenic SIV infection in primate animal models, suggesting that a deregulated IFN-state may be more detrimental to the host than to the virus itself in vivo.If this hypothesis is correct and if HIV-1 plays an active role in the perpetration of this antiviral state, it is possible that HIV-1 may have overall evolved to cope with this environment, remaining able to replicate despite it.To determine whether HIV-1 was better armed to replicate in the presence of an IFN-state environment than other primate lentiviruses, we compared HIV-1 to SIVmac and more importantly to HIV-2 that albeit capable of inducing AIDS in humans does so in a much less aggressive manner. In agreement with the initial hypothesis, our results indicate that HIV-1 is better fit to replicate in primary cells in the presence of amounts of IFN comparable to the ones measured in vivo, while the replication of HIV-2/SIVmac viruses is completely blocked even in the presence of low levels of IFN. By decorticating the effects of IFNs on the early and late phases of the viral life cycle in primary macrophages, we show here that the main target of the differential action of IFNs are the early phases of infection. More specifically, with time kinetics that we determine herein, IFNs induce cellular factor/s that differentially affect the stability of pre-reverse transcription complexes of HIV-2, but not of HIV-1. Our results could underlie a different evolutionary adaptation of primate lentiviruses to interferons that might be responsible for their different pathogenicity in vivo
Gelinas, Jean-Francois. „Enhancement of lentiviral vector production through alteration of virus-cell interactions“. Thesis, University of Oxford, 2016. https://ora.ox.ac.uk/objects/uuid:9921b8b4-e2b5-4eec-9efc-6036765c8d55.
Der volle Inhalt der QuelleKelly, Maureen C. „Parallels in tRNA primer acquisition by lentiviruses“. Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2009r/kelly.pdf.
Der volle Inhalt der QuelleMartin, Michaël. „Mécanisme moléculaire de l'antagonisme du complexe HUSH par les protéines lentivirales Vpx et Vpr“. Electronic Thesis or Diss., Université Paris Cité, 2021. http://www.theses.fr/2021UNIP5160.
Der volle Inhalt der QuelleHIV-1 and HIV-2, lentiviruses responsible for AIDS, appeared in humans after cross-species transmissions from simian viruses (SIV). In addition to their structural and regulatory proteins, lentiviruses encode auxiliary proteins that promote viral replication in the host cell by counteracting antiviral cellular factors, called restriction factors. The mechanism of action of these viral auxiliary proteins often relies on the hijacking of Ubiquitin-Ligase complexes, a mechanism widely used by various pathogens, to degrade host cell proteins. This mechanism is used by the Vpx protein, expressed only by HIV-2 (and not by HIV-1), which induces the degradation of SAMDH1, a restriction factor blocking the reverse transcription step. Thus, Vpx molecularly bridges the DCAF1 adaptor of the Cul4A-DDB1(DCAF1) Ubiquitin-Ligase complex with SAMHD1, resulting in ubiquitination and degradation of SAMHD1. In 2018, our team showed that Vpx induces the degradation of an additional cellular factor: the HUSH complex, composed of TASOR, MPP8 and Periphilin. This complex is involved in the epigenetic repression not only of many cellular genes, retro-transposable elements and endogenous retroviruses, but also of the HIV genome integrated into the infected cell. By degrading HUSH, Vpx promotes viral expression. In this context, the objectives of my thesis were to: (i) Determine whether HUSH degradation mechanism induced by HIV-2 Vpx was identical to SAMHD1 degradation mechanism. I was able to highlight important differences between the two mechanisms although Vpx uses, in both cases, the same Ubiquitin-Ligase adaptor, DCAF1 (main focus of the thesis work, submitted article). (ii) Characterize the molecular determinants involved in the antagonism of HUSH by other lentiviral proteins. First, we wanted to know if different Vpx-related viral proteins, in various simian virus species, had the same capacity to degrade the HUSH complex. This allowed us to reveal a lentiviral species-specificity of HUSH complex antagonism, a major characteristic of restriction factors (contribution to Chougui et al., Nature microbiology, 2018). Secondly, this led me to start studying the viral determinants of these Vpx-related proteins, such as the Vpr proteins from different strains of SIVagm (infecting the African green monkey) that present different phenotypes regarding both SAMHD1 or HUSH degradation (work in progress). All the results allowed us to better characterize the mechanism of HUSH antagonism by Vpx/Vpr lentiviral proteins, and to provide the first molecular tools to differentiate HUSH antagonism from SAMHD1 antagonism in primary cells. In the future, these data may help to better understand how various lentiviral proteins have adapted to their different cellular substrates (and vice versa) along evolution. Finally, targeting HUSH through the identification of interaction or degradation determinants could be interesting for the development of new therapeutic targets
Li, Li. „Short-term and long-term evolution of lentiviruses“. Thesis, University of Nottingham, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.575475.
Der volle Inhalt der QuelleLee, Wei-Cheng. „Studies on lentivirus infection of macrophages“. Thesis, University of Edinburgh, 1994. http://hdl.handle.net/1842/29845.
Der volle Inhalt der QuelleStarling, Isabella. „Mechanisms and specificity of lentivirus neurotoxicity“. Thesis, University of Edinburgh, 1998. http://webex.lib.ed.ac.uk/abstracts/starli01.pdf.
Der volle Inhalt der QuelleBücher zum Thema "Lentivirusus"
Maurizio, Federico, Hrsg. Lentivirus gene engineering protocols. Totowa, N.J: Humana Press, 2003.
Den vollen Inhalt der Quelle findenmissing], [name. Lentivirus gene engineering protocols. Totowa, NJ: Humana Press, 2003.
Den vollen Inhalt der Quelle findenMaurizio, Federico, Hrsg. Lentivirus gene engineering protocols. 2. Aufl. New York: Humana Press, 2010.
Den vollen Inhalt der Quelle findenFederico, Maurizio, Hrsg. Lentivirus Gene Engineering Protocols. Totowa, NJ: Humana Press, 2003. http://dx.doi.org/10.1385/1592593933.
Der volle Inhalt der QuelleFederico, Maurizio, Hrsg. Lentivirus Gene Engineering Protocols. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-533-0.
Der volle Inhalt der QuelleE, Wilcox G., Soeharsono S, Dharma D. M. N, Copland J. W, AustralianCentre for International Agricultural Research., Indonesia Direktorat Jenderal Petermakan und Bali Cattle Disease Investigation Unit., Hrsg. Jembrana disease and the bovine lentiviruses: Proceedings of a workshop 10-13 June 1996, Bali, Indonesia. Canberra: Australian Centre for International Agricultural Research in association with Direktorat Jenderal Petermakan and the Bali Cattle Disease Investigation Unit, 1997.
Den vollen Inhalt der Quelle findenMilne, Catherine E. Maedi visna: The disease, its potential impact on the UK sheep industry and a cost benefit appraisal ofcontrol strategies. [Aberdeen]: SAC, 1993.
Den vollen Inhalt der Quelle finden1949-, Morrow John, und Haigwood Nancy L, Hrsg. HIV molecular organization, pathogenicity, and treatment. Amsterdam: Elsevier, 1993.
Den vollen Inhalt der Quelle finden1963-, Sobo Elisa Janine, Hrsg. The endangered self: Managing the social risk of HIV. London: Routledge, 2000.
Den vollen Inhalt der Quelle findenNichols, Eve K. Expanding access to investigational therapies for HIV infection and AIDS: March 12-13, 1990, conference summary. Washington, D.C: National Academy Press, 1991.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Lentivirusus"
Federico, Maurizio. „From Lentiviruses to Lentivirus Vectors“. In Lentivirus Gene Engineering Protocols, 3–15. Totowa, NJ: Humana Press, 2003. http://dx.doi.org/10.1385/1-59259-393-3:3.
Der volle Inhalt der QuelleLu, Yichen, und Max Essex. „Lentivirus‡“. In The Springer Index of Viruses, 1693–702. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-95919-1_275.
Der volle Inhalt der QuelleGonda, Matthew A. „The Lentiviruses of Cattle“. In The Retroviridae, 83–109. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-1730-0_3.
Der volle Inhalt der QuellePlanelles, Vicente. „Hybrid Lentivirus Vectors“. In Lentivirus Gene Engineering Protocols, 273–84. Totowa, NJ: Humana Press, 2003. http://dx.doi.org/10.1385/1-59259-393-3:273.
Der volle Inhalt der QuelleSurdziel, Ewa, Matthias Eder und Michaela Scherr. „Lentivirus-Mediated Antagomir Expression“. In Methods in Molecular Biology, 237–48. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-811-9_16.
Der volle Inhalt der QuelleFultz, P. N. „SIVsmmPBj14: An Atypical Lentivirus“. In Current Topics in Microbiology and Immunology, 65–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78536-8_4.
Der volle Inhalt der QuelleZhao, Jing, und Andrew M. L. Lever. „Lentivirus-Mediated Gene Expression“. In Methods in Molecular Biology, 343–55. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-030-0_20.
Der volle Inhalt der QuelleBenskey, Matthew J., und Fredric P. Manfredsson. „Lentivirus Production and Purification“. In Gene Therapy for Neurological Disorders, 107–14. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3271-9_8.
Der volle Inhalt der QuelleDreyer, Jean-Luc. „Lentiviral Vector-Mediated Gene Transfer and RNA Silencing Technology in Neuronal Dysfunctions“. In Lentivirus Gene Engineering Protocols, 3–35. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-533-0_1.
Der volle Inhalt der QuelleCoppola, Valeria, Cesare Galli, Maria Musumeci und Désirée Bonci. „Manipulating the Cell Differentiation Through Lentiviral Vectors“. In Lentivirus Gene Engineering Protocols, 149–60. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-533-0_10.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Lentivirusus"
Xiao, Yun-Feng. „Label-free Detection of Single Nanoparticles and Lentiviruses Using an Optical Microcavity“. In Optical Sensors. Washington, D.C.: OSA, 2013. http://dx.doi.org/10.1364/sensors.2013.st2b.3.
Der volle Inhalt der QuelleHoffman, Robert M., Hiroyuki Kishimoto und Toshiyoshi Fujiwara. „Specific in vivo labeling with GFP retroviruses, lentiviruses, and adenoviruses for imaging“. In Biomedical Optics (BiOS) 2008, herausgegeben von Alexander P. Savitsky, Robert E. Campbell und Robert M. Hoffman. SPIE, 2008. http://dx.doi.org/10.1117/12.773308.
Der volle Inhalt der QuelleTulio, Robertha, und Rômulo Machado Balmant. „Dental care for HIV positive patients - care and importance - case report“. In II INTERNATIONAL SEVEN MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/homeinternationalanais-087.
Der volle Inhalt der QuelleTapanes-Castillo, Alexis, Derek Dykxhoorn, Leana Ramos, Milagros Mulero, Deliabell Hernandez und Vadym Trokhymchuk. „Culturing Human Neural Stem Cells and Quantifying Lentiviruses to Study Autism.“ In MOL2NET 2016, International Conference on Multidisciplinary Sciences, 2nd edition. Basel, Switzerland: MDPI, 2016. http://dx.doi.org/10.3390/mol2net-02-07008.
Der volle Inhalt der QuelleAlton, EWFW, AC Boyd, JC Davies, DR Gill, U. Griesenbach, TE Harman, SC Hyde und G. McLachlan. „S68 Towards a first-in-human trial with a pseudotyped lentivirus“. In British Thoracic Society Winter Meeting, Wednesday 17 to Friday 19 February 2021, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2021. http://dx.doi.org/10.1136/thorax-2020-btsabstracts.73.
Der volle Inhalt der QuelleLund-Palau, H., C. Meng, A. Pilou, N. Atsumi, A. Bhargava, M. Chan, A. Byrne et al. „T2 Lentivirus GM-CSF gene therapy ameliorates autoimmune pulmonary alveolar proteinosis“. In British Thoracic Society Winter Meeting 2018, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 5 to 7 December 2018, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2018. http://dx.doi.org/10.1136/thorax-2018-212555.2.
Der volle Inhalt der QuelleBurke, David, Kristine Drafahl, Clark Fjeld, Chad Galderisi und Cindy Spittle. „Abstract 896: Enhanced sensitivity detection of replication competent lentivirus by qPCR“. In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-896.
Der volle Inhalt der QuelleWilson, AA, GJ Murphy, H. Hamakawa, L. Kwok, S. Srinivasan, A. Hovav, RC Mulligan, S. Amar, B. Suki und DN Kotton. „Lentivirus-Based Expression of Human Alpha-1 Antitrypsin Ameliorates Emphysema in Mice.“ In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3509.
Der volle Inhalt der QuelleMarcotte, Richard, Azin Sayad, Maliha Haider, Kevin Brown, Troy Ketela, Jason Moffat und Benjamin G. Neel. „Abstract PR01: Functional characterization of breast cancer using pooled lentivirus shRNA screens“. In Abstracts: AACR Precision Medicine Series: Synthetic Lethal Approaches to Cancer Vulnerabilities - May 17-20, 2013; Bellevue, WA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.pms-pr01.
Der volle Inhalt der QuelleMarcotte, Richard, Kevin Brown, Azin Sayad, Maliha Haider, Troy Ketela, Jason Moffat und Benjamin G. Neel. „Abstract 5084: Functional genomic classification of breast cancer using pooled lentivirus shRNA screens“. In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5084.
Der volle Inhalt der QuelleBerichte der Organisationen zum Thema "Lentivirusus"
Lindner, Daniel. Complementation of Myelodysplastic Syndrome Clones with Lentivirus Expression Libraries. Fort Belvoir, VA: Defense Technical Information Center, Juli 2012. http://dx.doi.org/10.21236/ada566912.
Der volle Inhalt der QuelleLindner, Daniel J. Complementation of Myelodysplastic Syndrome Clones with Lentivirus Expression Libraries. Fort Belvoir, VA: Defense Technical Information Center, Januar 2013. http://dx.doi.org/10.21236/ada581503.
Der volle Inhalt der QuelleLindner, Daniel. Complementation of Myelodysplastic Syndrome Clones with Lentivirus Expression Libraries. Fort Belvoir, VA: Defense Technical Information Center, Juli 2011. http://dx.doi.org/10.21236/ada581646.
Der volle Inhalt der QuelleDeMartini, James C., Abraham Yaniv, Jonathan O. Carlson, Arnona Gazit, Leonard E. Pearson, Kalman Perk, J. K. Young, Noam Safran und A. Friedman. Evaluation of Naked Proviral DNA as a Vaccine for Ovine Lentivirus Infection. United States Department of Agriculture, September 1994. http://dx.doi.org/10.32747/1994.7570553.bard.
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