Thematische Bibliographien / Killer cell immunoglobulin live receptors

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Auswahl der wissenschaftlichen Literatur zum Thema „Killer cell immunoglobulin live receptors“

Autor: Grafiati

Veröffentlicht am 18. Mai 2024

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Zeitschriftenartikel zum Thema "Killer cell immunoglobulin live receptors"

Maslikova, U. V., E. G. Khamaganova, M. Yu Drokov, I. Yu Urybin, E. D. Mikhaltsova, L. A. Kuzmina und E. N. Parovichnikova. „Probability of allogeneic hematopoietic stem cell transplant failure depending on the recipient's killer immunoglobulin-like receptor genotype“. Transplantologiya. The Russian Journal of Transplantation 15, Nr. 1 (17.03.2023): 23–33. http://dx.doi.org/10.23873/2074-0506-2023-15-1-23-33.

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Introduction. Natural killers are the "first line" of antitumor and antiviral protection in the early stages after аllogeneic hematopoietic stem cell transplantation. Quantitative characteristics reach normal values already in the first month after the infusion of blood stem cells to the recipient. Self-tolerance of natural killers is achieved due to many receptors on their surface, but killer immunoglobulin-like receptors play a key role. Their role is to recognize "self" cells and block signals aimed at destroying their own cells. Knowledge of the functional activity of natural killers urged to studying the impact of mismatches between the inhibitory receptor gene and the ligand on the development of allogeneic hematopoietic stem cell transplant failure.The aim of research was to study the probability of the graft failure development in allogeneic hematopoietic stem cell transplantation depending on the recipient's killer immunoglobulin-like receptor genotype.Material and methods. Genotyping of killer-cell immunoglobulin-like receptors in 66 recipients of blood stem cells by the polymerase chain reaction method was performed in the study. Using an online calculator, receptors were classified as "best", "better" and "neutral" depending on the genotype. The end point of the assessment was the development of graft failure in the presence of different genotypes of immunoglobulin-like receptors in the recipient.Results. According to the data obtained, the presence of the “best” and "better" killer-cell immunoglobulin-like receptor genotype in the recipient significantly increased the risks of developing various forms of graft failure.Conclusion. The presence of the KIR2DL3 genotype in a recipient of hematopoietic stem cells significantly (by 3 times) reduces the likelihood of primary graft failure. This result is of great prognostic significance, although at present no ways of influencing it have been developed. The presence of the “best” killer immunoglobulin-like receptors genotype in the recipient increases the likelihood of developing graft failure by more than 3 times compared to the best and neutral genotype (44.4% vs. 13.4%).

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Parham, P. „Immunogenetics of killer-cell immunoglobulin-like receptors“. Tissue Antigens 62, Nr. 3 (September 2003): 194–200. http://dx.doi.org/10.1034/j.1399-0039.2003.00126.x.

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Parham, Peter. „Immunogenetics of killer cell immunoglobulin-like receptors“. Molecular Immunology 42, Nr. 4 (Februar 2005): 459–62. http://dx.doi.org/10.1016/j.molimm.2004.07.027.

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Fauriat, Cyril, Martin A. Ivarsson, Hans-Gustaf Ljunggren, Karl-Johan Malmberg und Jakob Michaëlsson. „Education of human natural killer cells by activating killer cell immunoglobulin-like receptors“. Blood 115, Nr. 6 (11.02.2010): 1166–74. http://dx.doi.org/10.1182/blood-2009-09-245746.

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Abstract Expression of inhibitory killer cell immunoglobulin-like receptors (KIRs) specific for self–major histocompatibility complex (MHC) class I molecules provides an educational signal that generates functional natural killer (NK) cells. However, the effects of activating KIRs specific for self-MHC class I on NK-cell education remain elusive. Here, we provide evidence that the activating receptor KIR2DS1 tunes down the responsiveness of freshly isolated human NK cells to target cell stimulation in donors homozygous for human leukocyte antigen (HLA)–C2, the ligand of KIR2DS1. The tuning was apparent in KIR2DS1+ NK cells lacking expression of inhibitory KIRs and CD94/NKG2A, as well as in KIR2DS1+ NK cells coexpressing the inhibitory MHC class I–specific receptors CD94/NKG2A and KIR2DL3, but not KIR2DL1. However, the tuning of responsiveness was restricted to target cell recognition because KIR2DS1+ NK cells responded well to stimulation with exogenous cytokines. Our results provide the first example of human NK-cell education by an activating KIR and suggest that the education of NK cells via activating KIRs is a mechanism to secure tolerance that complements education via inhibitory KIRs.

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Bimber, Benjamin N., und David T. Evans. „The killer-cell immunoglobulin-like receptors of macaques“. Immunological Reviews 267, Nr. 1 (18.08.2015): 246–58. http://dx.doi.org/10.1111/imr.12329.

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Rajalingam, Raja. „Diversity of Killer Cell Immunoglobulin-Like Receptors and Disease“. Clinics in Laboratory Medicine 38, Nr. 4 (Dezember 2018): 637–53. http://dx.doi.org/10.1016/j.cll.2018.08.001.

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Momot, T., S. Koch, N. Hunzelmann, T. Krieg, K. Ulbricht, R. E. Schmidt und T. Witte. „Association of killer cell immunoglobulin-like receptors with scleroderma“. Arthritis & Rheumatism 50, Nr. 5 (2004): 1561–65. http://dx.doi.org/10.1002/art.20216.

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Long, Eric O., Domingo F. Barber, Deborah N. Burshtyn, Mathias Faure, Mary Peterson, Sumati Rajagopalan, Valery Renard et al. „Inhibition of natural killer cell activation signals by killer cell immunoglobulin-like receptors (CD158)“. Immunological Reviews 181, Nr. 1 (Juli 2001): 223–33. http://dx.doi.org/10.1034/j.1600-065x.2001.1810119.x.

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Moretta, Lorenzo, Franco Locatelli, Daniela Pende, Emanuela Marcenaro, Maria Cristina Mingari und Alessandro Moretta. „Killer Ig–like receptor-mediated control of natural killer cell alloreactivity in haploidentical hematopoietic stem cell transplantation“. Blood 117, Nr. 3 (20.01.2011): 764–71. http://dx.doi.org/10.1182/blood-2010-08-264085.

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Abstract Natural killer (NK) cells are key members of the innate immune system. In a self-environment, they sense and kill target cells lacking major histocompatibility complex class I molecules and release various cytokines on activation. The discovery of human leukocyte antigen (HLA) class I specific inhibitory receptors (including the allotype-specific killer immunoglobulin-like receptors), and of various activating receptors and their ligands, provided the basis for understanding the molecular mechanism of NK-cell activation and function, mainly resulting from the balance between activating and inhibitory signals. In an allogeneic setting, such as T cell–depleted haploidentical hematopoietic stem cell transplantation, NK cells may express inhibitory killer immunoglobulin-like receptors that are not engaged by any of the HLA class I alleles present on allogeneic cells. Such “alloreactive” NK cells greatly contribute both to eradication of leukemia blasts escaping the preparative regimen and to clearance of residual host dendritic cells and T lymphocytes (thus preventing graft-versus-host disease and graft rejection, respectively). Improved prevention of graft-versus-host disease might be achieved by redirecting to lymph nodes adoptively transferred, alloreactive NK cells by inducing CCR7-uptake in vitro. Recent studies suggested that, after immune-suppressive therapy, alloreactive NK cells from an HLA-haploidentical donor may prevent leukemia recurrence also in patients who have not received allogeneic hematopoietic stem cell transplantation.

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Rajalingam, Raja. „Human diversity of killer cell immunoglobulin-like receptors and disease“. Korean Journal of Hematology 46, Nr. 4 (2011): 216. http://dx.doi.org/10.5045/kjh.2011.46.4.216.

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Dissertationen zum Thema "Killer cell immunoglobulin live receptors"

Abalos, Andrew T. „KILLER-CELL IMMUNOGLOBULIN-LIKE RECEPTORS AND HPV PREVALENCE AND INCIDENCE“. Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/145440.

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Introduction: Human papillomavirus (HPV) is the most commonly occurring sexually transmitted infection and is a necessary cause of cervical cancer. The progression from HPV infection to cervical cancer is incompletely understood. Innate immune response to HPV infection has recently been identified as a potential cofactor in this progression. This study examined potential association(s) between killer cell immunoglobulin-like receptors (KIR) and HPV infection. HPV concordance was estimated among heterosexual couples demonstrating the complexity of HPV infection.Methods: HPV concordance was cross-sectionally estimated in 29 heterosexual couples. A polymerase chain reaction based assay for KIR genotyping was developed and validated. 283 women from the Young Women's Health Study and 259 men from the HPV Infection in Men: A Prospective Cohort Study had HPV infection data and samples available for KIR genotyping. Associations between KIR genotype and haplotype with HPV prevalence, incidence and clearance were assessed.Results: Among 29 couples, prevalence for any HPV type was comparable between women 86.2% and men, 75.9%. Partial concordance was observed in 66% of the couples. Forty-one percent (41%) of couples had perfect concordance. A high degree of concordance was observed, however HPV type distributions differed in men and women. In women from the YWHS, KIR2DS5 was significantly associated with oncogenic HPV prevalence (Odds ratio [OR]: 0.56, 95% Confidence Interval [CI]: 0.31-0.99). Any HPV incidence was significantly associated with KIR2DL2 (Hazards Ratio [HR]: 2.11, 95% CI: 1.0-4.44), KIR2DS2 (HR: 2.44, 95% CI: 1.13-5.24), KIR2DS3 (HR: 2.36, 95% CI: 1.16-4.81), and KIR haplotype B (HR: 2.48, 95% CI: 1.02-6.02). Women lacking KIR2DS5 had an increased risk of any HPV acquisition in the presence of KIR2DL2 (HR: 2.95, 95% CI: 1.28-6.86), KIR2DS2 (HR: 3.33, 1.39-7.99), or KIR2DS3 (2.77, 95% CI: 1.24-6.19). In Men, KIR2DS3 was significantly associated with increased probability of any HPV clearance (HR: 1.91, 95% CI: 1.04-3.49).Conclusions: This research contributes to our understanding of HPV infection dynamics through the assessment HPV type concordance in sexual partners. Additionally, through the development of an assay for KIR genotyping, we were able to identify associations with KIR gene positivity and HPV prevalence, incidence, and clearance in men and women.

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Foley, Bree Amanda. „The immunogenetics of natural killer cell alloreactivity“. University of Western Australia. School of Pathology and Laboratory Medicine, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0242.

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[Truncated abstract] Natural killer (NK) cell alloreactivity can be exploited in haploidentical haematopoietic stem cell transplantation (HSCT) to improve graft survival, reduce graft versus host disease and decrease leukaemic relapse. NK cells lyse cells that have reduced expression of class I HLA molecules. In an allogeneic setting, donor NK cells may be activated by the absence of donor (self) class I HLA molecules on recipient cells; the absence of self-epitopes being detected by inhibitory KIR receptors on donor NK cells. The way in which genetic polymorphism of the receptors and ligands affects NK allorecognition of missing self, has not been fully elucidated. HLA-C molecules are divided into two groups, C1 and C2, with KIR2DL1 recognising cells expressing C2 and KIR2DL2 and KIR2DL3 recognising cells expressing C1. Donor NK cells expressing KIR2DL2 or KIR2DL3 can be alloreactive towards a recipient if they lack the C1 epitope and donor NK cells expressing KIR2DL1 can be alloreactive towards a recipient if they lack the C2 epitope. KIR3DL1 recognises the Bw4 epitope present on one-third of HLA-B alleles and certain HLA-A alleles. NK cells from donors expressing KIR3DL1 can be alloreactive towards recipients whose cells lack Bw4. Mismatches of KIR related HLA epitopes does not always results in NK alloreactivity. Therefore it is not possible to reliably predict NK alloreactivity based solely on the donor's HLA type and KIR repertoire and the recipient's HLA type. ... All Bw4-positive HLA-B alleles, with the exception of HLA-B*1301 and B*1302, protected targets from lysis. HLA-A*2402 and HLA-A*3201 unequivocally protected target cells from lysis whereas HLA-A*2501 and HLA-A*2301 provided only weak protection from lysis. KIR3DL1-dependent alloreactive NK clones were identified in donors whose only Bw4 positive allele was HLA-A*2402 but not in donors whose only Bw4 positive HLA allele was HLA-B*1301 or B*1302. Finally this thesis demonstrated that an activating KIR can control NK cell alloreactivity. Donors who are C2 negative and KIR2DS1 positive had NK cells that expressed the activating receptor KIR2DS1 and were capable of lysing cells expressing the C2 epitope. More so, KIR2DS1 dependent NK clones were shown to override inhibitory signals generated by NKG2A interacting with its ligand, HLA-E. The identification of these NK clones has important implications for haploidentical HSCT in that recipient expressing all three NK epitopes, C1, C2 and Bw4 were previously thought to be resistant to alloreactive NK cells controlled by inhibitory receptors. Such patients may be amenable to haploidentical HSCT from C2 negative, KIR2DS1 positive donors. These results will improve the ability to predict NK cell alloreactivity based on a donor's HLA type and KIR repertoire and the recipient?s HLA type.

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Guha, Pokhraj. „Study of the genetic diversity of killer cell immunoglobulin live receptors (KIRs) in some human populations of sub-himalayan region“. Thesis, University of North Bengal, 2017. http://ir.nbu.ac.in/handle/123456789/2586.

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Moesta, Achim Klaus. „Functional specificities of killer cell immunoglobulin-like receptors for MHC-C in humans and chimpanzees /“. May be available electronically:, 2008. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.

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Older, Aguilar Anastazia Magdalena. „Comparison of genomic structure and MHC specificities of killer cell immunoglobulin-like receptors in humans and orangutans /“. May be available electronically:, 2009. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.

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Suck, Garnet, Yeh Ching Linn und Torsten Tonn. „Natural Killer Cells for Therapy of Leukemia“. Karger, 2016. https://tud.qucosa.de/id/qucosa%3A71644.

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Clinical application of natural killer (NK) cells against leukemia is an area of intense investigation. In human leukocyte antigen-mismatched allogeneic hematopoietic stem cell transplantations (HSCT), alloreactive NK cells exert powerful anti-leukemic activity in preventing relapse in the absence of graft-versus-host disease, particularly in acute myeloid leukemia patients. Adoptive transfer of donor NK cells post-HSCT or in non-transplant scenarios may be superior to the currently widely used unmanipulated donor lymphocyte infusion. This concept could be further improved through transfusion of activated NK cells. Significant progress has been made in good manufacturing practice (GMP)-compliant large-scale production of stimulated effectors. However, inherent limitations remain. These include differing yields and compositions of the end-product due to donor variability and inefficient means for cryopreservation. Moreover, the impact of the various novel activation strategies on NK cell biology and in vivo behavior are barely understood. In contrast, reproduction of the thirdparty NK-92 drug from a cryostored GMP-compliant master cell bank is straightforward and efficient. Safety for the application of this highly cytotoxic cell line was demonstrated in first clinical trials. This novel ‘off-theshelf’ product could become a treatment option for a broad patient population. For specific tumor targeting chimeric-antigen-receptor-engineered NK-92 cells have been designed.

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Kruse, Philip Hermann Verfasser], Lutz [Akademischer Betreuer] Walter, Jürgen [Akademischer Betreuer] [Wienands und Wolfgang [Akademischer Betreuer] Engel. „Genetic and functional characterisation of killer cell immunoglobulin like receptors (KIR) of rhesus macaques (Macaca mulatta) / Philip Hermann Kruse. Gutachter: Lutz Walter ; Jürgen Wienands ; Wolfgang Engel. Betreuer: Lutz Walter“. Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2011. http://d-nb.info/1042640025/34.

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Jiang, Wei. „Killer-cell immunoglobulin-like receptor gene copy number, haplotypes and disease association“. Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607691.

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Sepulveda, Christian Alberto Garcia. „Killer cell Immunoglobulin-like Receptor (KIR) polymorphism : functional implications and clinical relevance“. Thesis, University College London (University of London), 2005. http://discovery.ucl.ac.uk/1444690/.

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NK cell function is regulated by Killer-cell Immunoglobulin-like Receptors (KIR) some of which recognise class I Major Histocompatibility Complex molecules. KIRs have been shown to exhibit a high degree of functional diversity which is generated at several levels. However, the functional relevance of this diversity remains largely unknown. This thesis describes our approach towards elucidating the functional relevance of KIR diversity. To study this we first compiled all known KIR sequences into a database. We developed bioinformatics tools to facilitate the study of these sequences and have made both the tools and database publicly accessible online. Subsequent efforts were directed towards investigating the structural impact of KIR polymorphism by means of molecular modelling software. The results that were generated by this approach have provided information with regards to the ligand binding properties of most activating KIR proteins. In addition, we have also developed a KIR gene typing system capable of detecting all known KIR genes as well as the alleles of five of the KIR proteins for which a ligand has been described. We have implemented this KIR typing system to three different sample panels: a reference panel of more than 100 B-lymphoblastoid cell lines (BLCL), a family based KIR haplotype segregation study and a cohort of 141 unrelated donor (UD) haematopoietic stem cell transplant pairs. Our investigations have allowed us to generate the largest KIR typing reference panel, to characterise the KIR profile of a Mexican Mestizo population and to investigate the clinical relevance of KIRs in UD-Haematopoietic Stem Cell Transplantation (HSCT). Our results demonstrate that the beneficial effect of NK alloreactivity in the Graft-versus-Host direction as predicted by Ruggeri's algorithm cannot be applied to the UD-HSCT setting. In addition, I describe our findings relating to the clinical role of KIR genes and alleles in the UD-HSCT cohort.

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Maxwell, L. D. „Investigation of the genetics and expression of killer cell immunoglobulin-like receptor genes“. Thesis, Queen's University Belfast, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426759.

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Buchteile zum Thema "Killer cell immunoglobulin live receptors"

Long, Eric O., Deborah N. Burshtyn, Christopher C. Stebbins und Carsten Watzl. „How do killer cell Ig-like receptors inhibit natural killer cells?“ In Activating and Inhibitory Immunoglobulin-like Receptors, 235–41. Tokyo: Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-53940-7_29.

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Rajalingam, Raja, Sarah Cooley und Jeroen van Bergen. „Killer Cell Immunoglobulin-Like Receptors in Clinical Transplantation“. In Manual of Molecular and Clinical Laboratory Immunology, 1150–60. Washington, DC, USA: ASM Press, 2016. http://dx.doi.org/10.1128/9781555818722.ch119.

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Ruggeri, Loredana, Shuhong Zhang und Sherif S. Farag. „Natural Killer Cell Activity and Killer Immunoglobulin-Like Receptors in Hematopoietic Stem Cell Transplantation“. In Cancer Treatment and Research, 47–69. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-78580-6_3.

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Rajalingam, Raja, und Elham Ashouri. „Gene-Specific PCR Typing of Killer Cell Immunoglobulin-Like Receptors“. In Methods in Molecular Biology, 239–55. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-493-7_12.

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Hou, Lihua, Minghua Chen, Noriko Steiner, Kanthi Kariyawasam, Jennifer Ng und Carolyn K. Hurley. „Killer Cell Immunoglobulin-Like Receptors (KIR) Typing by DNA Sequencing“. In Methods in Molecular Biology™, 431–68. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-842-9_25.

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Wang, Lawrence L., und Wayne M. Yokoyama. „Regulated expression of non-polymorphic gp49 molecules on mouse natural killer cells“. In Activating and Inhibitory Immunoglobulin-like Receptors, 25–31. Tokyo: Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-53940-7_4.

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Dorak, M. Tevfik. „Role of Natural Killer Cells and Killer Immunoglobulin-Like Receptor Polymorphisms“. In Bone Marrow and Stem Cell Transplantation, 123–44. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-223-6_10.

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Ghansah, Tomar, John M. Ninos und William G. Kerr. „A role for the SH2-containing inositol phosphatase in the biology of natural killer cells and stem cells“. In Activating and Inhibitory Immunoglobulin-like Receptors, 129–40. Tokyo: Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-53940-7_17.

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Rajalingam, Raja. „Overview of the Killer Cell Immunoglobulin-Like Receptor System“. In Methods in Molecular Biology™, 391–414. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-842-9_23.

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Schellekens, Jennifer, Katia Gagne und Steven G. E. Marsh. „Natural Killer Cells and Killer-Cell Immunoglobulin-Like Receptor Polymorphisms: Their Role in Hematopoietic Stem Cell Transplantation“. In Methods in Molecular Biology, 139–58. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9437-9_9.

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Konferenzberichte zum Thema "Killer cell immunoglobulin live receptors"

Chan, Daniel C., Zhiyong Zhang, Hong Wang, Xiaomei Sui, Tiffany T. Chan, Natalie Ahn, Lewis L. Lanier und Paul A. Bunn. „Abstract 1357: Role of Natural Killer-cell Immunoglobulin-like receptors KIR2DL1 and KIR3DL1 in immune resistance“. In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1357.

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Hagberg, N., D. Leonard, G. Nordmark und L. Rönnblom. „PS1:16 The presence of autoantibodies to multiple killer cell immunoglobulin-like receptors is associated with nephritis in sle patients“. In 11th European Lupus Meeting, Düsseldorf, Germany, 21–24 March 2018, Abstract presentations. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-abstract.64.

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Chan, Daniel C. F., Zhiyong Zhang, Hong Wang, Xiaomei Sui, Tiffany T. Y. Chan, Natalie Ahn, Joe Phillips, Kathryn Horwitz, Lewis Lanier und Paul Bunn. „Abstract 3656: Therapeutic effects of anti-KIR antibodies against metastatic cancer cells with aberrant expression of Natural Killer-Cell Immunoglobulin-like Receptors (KIRs)“. In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3656.

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Chan, Daniel C., Zhiyong Zhang, Di Zheng, Tiffany Chan, Mary Berg, Kathryn Horwitz, Natalie Ahn, Lewis Lanier und Paul Bunn. „Abstract 4836: Immune-tolerance due to aberrant expression of Natural Killer-Cell Immunoglobulin-like Receptors (KIRs) on cancer cells and enhanced cancer-platelet interactions“. In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4836.

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Mkorombindo, T., T. K. Tran-Nguyen, K. Yuan, J. Xue, J. M. Pilewski, M. L. N. McDonald, F. C. Sciurba und S. R. Duncan. „The Association of HLA-C and Killer Cell Immunoglobulin-Like Receptor Permutations on COPD Risk“. In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2545.

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Fontela, Miguel Gomez, Sebastian Snedal und Daniel Abate-Daga. „225 Killer cell immunoglobulin-like receptor 2DL2 (KIR2DL2) immune checkpoint as a modulator of T-cell effector function“. In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0225.

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Nasef, SI, AM Asker, HH Omar und HM Hassoba. „SAT0304 Stimulatory and inhibitory killer immunoglobulin-like receptors on natural killer T (NKT) cells in patients with systemic lupus erythematosus (SLE): relation to disease activity“. In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.3920.

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Berichte der Organisationen zum Thema "Killer cell immunoglobulin live receptors"

Varbanova, Viktoria, Snejina Mihailova, Elissaveta Naumova und Anastasiya Mihaylova. Distribution of Killer-cell Immunoglobulin-like Receptors (KIR) and their HLA Class I Ligands in the Bulgarian Population. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, Juli 2020. http://dx.doi.org/10.7546/crabs.2020.07.14.

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