Zeitschriftenartikel zum Thema „Isogenic cellular models“
Geben Sie eine Quelle nach APA, MLA, Chicago, Harvard und anderen Zitierweisen an
Machen Sie sich mit Top-50 Zeitschriftenartikel für die Forschung zum Thema "Isogenic cellular models" bekannt.
Neben jedem Werk im Literaturverzeichnis ist die Option "Zur Bibliographie hinzufügen" verfügbar. Nutzen Sie sie, wird Ihre bibliographische Angabe des gewählten Werkes nach der nötigen Zitierweise (APA, MLA, Harvard, Chicago, Vancouver usw.) automatisch gestaltet.
Sie können auch den vollen Text der wissenschaftlichen Publikation im PDF-Format herunterladen und eine Online-Annotation der Arbeit lesen, wenn die relevanten Parameter in den Metadaten verfügbar sind.
Sehen Sie die Zeitschriftenartikel für verschiedene Spezialgebieten durch und erstellen Sie Ihre Bibliographie auf korrekte Weise.
Paredes-Redondo, A., und Y. Y. Lin. „Developing novel human isogenic cellular models for Duchenne muscular dystrophy“. Neuromuscular Disorders 27 (März 2017): S6. http://dx.doi.org/10.1016/s0960-8966(17)30234-1.
Der volle Inhalt der QuelleZhang, Yuting, Emily Wilt und Xin Lu. „Human Isogenic Cell Line Models for Neutrophils and Myeloid-Derived Suppressor Cells“. International Journal of Molecular Sciences 21, Nr. 20 (18.10.2020): 7709. http://dx.doi.org/10.3390/ijms21207709.
Der volle Inhalt der QuelleBenarroch, Louise, Julia Madsen-Østerbye, Mohamed Abdelhalim, Kamel Mamchaoui, Jessica Ohana, Anne Bigot, Vincent Mouly, Gisèle Bonne, Anne T. Bertrand und Philippe Collas. „Cellular and Genomic Features of Muscle Differentiation from Isogenic Fibroblasts and Myoblasts“. Cells 12, Nr. 15 (03.08.2023): 1995. http://dx.doi.org/10.3390/cells12151995.
Der volle Inhalt der QuellePavan, Eleonora, Maximiliano Ormazabal, Paolo Peruzzo, Emilio Vaena, Paula Rozenfeld und Andrea Dardis. „CRISPR/Cas9 Editing for Gaucher Disease Modelling“. International Journal of Molecular Sciences 21, Nr. 9 (05.05.2020): 3268. http://dx.doi.org/10.3390/ijms21093268.
Der volle Inhalt der QuelleKarwacka, Marianna, und Marta Olejniczak. „Advances in Modeling Polyglutamine Diseases Using Genome Editing Tools“. Cells 11, Nr. 3 (02.02.2022): 517. http://dx.doi.org/10.3390/cells11030517.
Der volle Inhalt der QuelleKlementieva, Natalia, Daria Goliusova, Julia Krupinova, Vladislav Yanvarev, Alexandra Panova, Natalia Mokrysheva und Sergey L. Kiselev. „A Novel Isogenic Human Cell-Based System for MEN1 Syndrome Generated by CRISPR/Cas9 Genome Editing“. International Journal of Molecular Sciences 22, Nr. 21 (08.11.2021): 12054. http://dx.doi.org/10.3390/ijms222112054.
Der volle Inhalt der QuelleBoussaad, Ibrahim, Emily K. Dolezal, Fabiana Perna, Stephen D. Nimer und Eirini P. Papapetrou. „IPS Cells From Del(7q)-MDS Patients Display Impaired Proliferation and Hematopoietic Commitment“. Blood 120, Nr. 21 (16.11.2012): 174. http://dx.doi.org/10.1182/blood.v120.21.174.174.
Der volle Inhalt der QuelleMuto, Valentina, Federica Benigni, Valentina Magliocca, Rossella Borghi, Elisabetta Flex, Valentina Pallottini, Alessandro Rosa, Claudia Compagnucci und Marco Tartaglia. „CRISPR/Cas9 and piggyBac Transposon-Based Conversion of a Pathogenic Biallelic TBCD Variant in a Patient-Derived iPSC Line Allows Correction of PEBAT-Related Endophenotypes“. International Journal of Molecular Sciences 24, Nr. 9 (28.04.2023): 7988. http://dx.doi.org/10.3390/ijms24097988.
Der volle Inhalt der QuelleLi, Fenfang, Igor Cima, Jess Honganh Vo, Min-Han Tan und Claus Dieter Ohl. „Single Cell Hydrodynamic Stretching and Microsieve Filtration Reveal Genetic, Phenotypic and Treatment-Related Links to Cellular Deformability“. Micromachines 11, Nr. 5 (09.05.2020): 486. http://dx.doi.org/10.3390/mi11050486.
Der volle Inhalt der QuellePatel, Ronak, Shyanne Page und Abraham Jacob Al-Ahmad. „Isogenic blood-brain barrier models based on patient-derived stem cells display inter-individual differences in cell maturation and functionality“. Journal of Neurochemistry 142, Nr. 1 (14.05.2017): 74–88. http://dx.doi.org/10.1111/jnc.14040.
Der volle Inhalt der QuelleBoussaad, Ibrahim, Andriana Kotini, Emily K. Dolezal, Stephen Nimer und Eirini P. Papapetrou. „An iPSC-Based Model Of MDS For Phenotype-Driven Gene and Drug Discovery“. Blood 122, Nr. 21 (15.11.2013): 859. http://dx.doi.org/10.1182/blood.v122.21.859.859.
Der volle Inhalt der QuelleHaley, John A., Christian F. Ruiz, Emily D. Montal, Daifeng Wang, John D. Haley und Geoffrey D. Girnun. „Decoupling of Nrf2 Expression Promotes Mesenchymal State Maintenance in Non-Small Cell Lung Cancer“. Cancers 11, Nr. 10 (02.10.2019): 1488. http://dx.doi.org/10.3390/cancers11101488.
Der volle Inhalt der QuelleGhosal, Abhisek, Stefan Jellbauer, Rubina Kapadia, Manuela Raffatellu und Hamid M. Said. „Salmonellainfection inhibits intestinal biotin transport: cellular and molecular mechanisms“. American Journal of Physiology-Gastrointestinal and Liver Physiology 309, Nr. 2 (15.07.2015): G123—G131. http://dx.doi.org/10.1152/ajpgi.00112.2015.
Der volle Inhalt der QuelleCeballos-Garzon, Andres, Elvira Roman, Jesús Pla, Fabrice Pagniez, Daniela Amado, Carlos J. Alméciga-Díaz, Patrice Le Pape und Claudia M. Parra-Giraldo. „CRISPR-Cas9 approach confirms Calcineurin-responsive zinc finger 1 (Crz1) transcription factor as a promising therapeutic target in echinocandin-resistant Candida glabrata“. PLOS ONE 17, Nr. 3 (18.03.2022): e0265777. http://dx.doi.org/10.1371/journal.pone.0265777.
Der volle Inhalt der QuelleKálmán, Sára, Edit Hathy und János M. Réthelyi. „A Dishful of a Troubled Mind: Induced Pluripotent Stem Cells in Psychiatric Research“. Stem Cells International 2016 (2016): 1–21. http://dx.doi.org/10.1155/2016/7909176.
Der volle Inhalt der QuelleWang, Wei, Tiansu Wang, Andriana G. Kotini, Camelia Iancu-Rubin, Ronald Hoffman und Eirini P. Papapetrou. „Modeling Calreticulin-Mutant Myeloproliferative Neoplasms with Isogenic Induced Pluripotent Stem Cells“. Blood 132, Supplement 1 (29.11.2018): 4319. http://dx.doi.org/10.1182/blood-2018-99-111512.
Der volle Inhalt der QuelleХамидуллина, А. И., Э. Р. Гандалипов, Я. Е. Абраменко, К. В. Чернов, Т. А. Кирюхина, А. В. Брутер und В. В. Татарский. „Creation of A549 and MCF7 tumor sublines with knockout of TP53 using CRISPR/Cas9“. Nauchno-prakticheskii zhurnal «Medicinskaia genetika 22, Nr. 11 (19.12.2023): 27–34. http://dx.doi.org/10.25557/2073-7998.2023.11.27-34.
Der volle Inhalt der QuelleCharlebois, Daniel A., Kevin Hauser, Sylvia Marshall und Gábor Balázsi. „Multiscale effects of heating and cooling on genes and gene networks“. Proceedings of the National Academy of Sciences 115, Nr. 45 (19.10.2018): E10797—E10806. http://dx.doi.org/10.1073/pnas.1810858115.
Der volle Inhalt der QuelleLee, Hans C., Hua Wang, Bing-Zong Li, Zhiqiang Wang, Richard Julian Jones, Dongmin Gu, Sean O'Brien, Richard E. Davis und Robert Z. Orlowski. „CX-5461, a Novel RNA Polymerase I Inhibitor, Is Active Against Wild-Type and Mutant p53 Myeloma Models“. Blood 122, Nr. 21 (15.11.2013): 4438. http://dx.doi.org/10.1182/blood.v122.21.4438.4438.
Der volle Inhalt der Quellevan Waardenburg, Robert C. A. M., Laurina A. de Jong, Foke van Delft, Maria A. J. van Eijndhoven, Melanie Bohlander, Mary-Ann Bjornsti, Jaap Brouwer und Jan H. M. Schellens. „Homologous recombination is a highly conserved determinant of the synergistic cytotoxicity between cisplatin and DNA topoisomerase I poisons“. Molecular Cancer Therapeutics 3, Nr. 4 (01.04.2004): 393–402. http://dx.doi.org/10.1158/1535-7163.393.3.4.
Der volle Inhalt der QuelleRakotomalala, Andria, Paul Lewandowski, Quentin Bailleul, Clara Savary, Mélanie Arcicasa, Christine Bal, Maud Hamadou et al. „Abstract 1671: Engineering new cellular models to decipher H3.3K27M mutation role in DIPGs' resistance to therapies“. Cancer Research 82, Nr. 12_Supplement (15.06.2022): 1671. http://dx.doi.org/10.1158/1538-7445.am2022-1671.
Der volle Inhalt der QuelleDe Souza, Cristabelle, Jill A. Madden, Dennis Minn, Vigneshwari Easwar Kumar, Dennis J. Montoya, Roshni Nambiar, Zheng Zhu et al. „The P72R Polymorphism in R248Q/W p53 Mutants Modifies the Mutant Effect on Epithelial to Mesenchymal Transition Phenotype and Cell Invasion via CXCL1 Expression“. International Journal of Molecular Sciences 21, Nr. 21 (28.10.2020): 8025. http://dx.doi.org/10.3390/ijms21218025.
Der volle Inhalt der QuelleJungles, Kassidy M., Andrea M. Pesch, Nicole Hirsh, Anna R. Michmerhuizen, Kari Wilder-Romans, Benjamin C. Chandler, Meilan Liu et al. „Abstract 216: Expression of DNA damage response proteins modifies the efficacy of CDK4/6 inhibitor-mediated radiosensitization in breast cancer models“. Cancer Research 82, Nr. 12_Supplement (15.06.2022): 216. http://dx.doi.org/10.1158/1538-7445.am2022-216.
Der volle Inhalt der QuelleChakrabarty, Anindita, Sreeraj Surendran, Neil E. Bhola, Vishnu S. Mishra, Tasaduq Hussain Wani, Khemraj S. Baghel, Carlos L. Arteaga, Rohini Garg und Goutam Chowdhury. „The H1047R PIK3CA oncogene induces a senescence-like state, pleiotropy and acute HSP90 dependency in HER2+ mammary epithelial cells“. Carcinogenesis 40, Nr. 10 (18.06.2019): 1179–90. http://dx.doi.org/10.1093/carcin/bgz118.
Der volle Inhalt der QuelleCostamagna, Gianluca, Giacomo Pietro Comi und Stefania Corti. „Advancing Drug Discovery for Neurological Disorders Using iPSC-Derived Neural Organoids“. International Journal of Molecular Sciences 22, Nr. 5 (06.03.2021): 2659. http://dx.doi.org/10.3390/ijms22052659.
Der volle Inhalt der QuelleWenzl, Kerstin, Michelle Manske, Paola Martinez, Stephen Ansell, James R. Cerhan und Anne J. Novak. „Isogenic Loss of TNFAIP3 in Waldenstrom Macroglobulinemia Enhances MYD88L265P-Driven Signaling“. Blood 128, Nr. 22 (02.12.2016): 4100. http://dx.doi.org/10.1182/blood.v128.22.4100.4100.
Der volle Inhalt der QuelleFerri-Borgogno, Sammy, Sugata Barui, Amberly M. McGee, Tamara Griffiths, Pankaj K. Singh, Cortt G. Piett, Bidyut Ghosh et al. „Paradoxical Role of AT-rich Interactive Domain 1A in Restraining Pancreatic Carcinogenesis“. Cancers 12, Nr. 9 (21.09.2020): 2695. http://dx.doi.org/10.3390/cancers12092695.
Der volle Inhalt der QuelleHollywood, Jennifer A., Aneta Przepiorski, Randall F. D’Souza, Sreevalsan Sreebhavan, Ernst J. Wolvetang, Patrick T. Harrison, Alan J. Davidson und Teresa M. Holm. „Use of Human Induced Pluripotent Stem Cells and Kidney Organoids To Develop a Cysteamine/mTOR Inhibition Combination Therapy for Cystinosis“. Journal of the American Society of Nephrology 31, Nr. 5 (20.03.2020): 962–82. http://dx.doi.org/10.1681/asn.2019070712.
Der volle Inhalt der QuelleJung, Moonjung, Stefan Cordes, Jizhong Zou, Shiqin J. Yu, Xavi Guitart, So Gun Hong, Vinh Dang et al. „GATA2 deficiency and human hematopoietic development modeled using induced pluripotent stem cells“. Blood Advances 2, Nr. 23 (11.12.2018): 3553–65. http://dx.doi.org/10.1182/bloodadvances.2018017137.
Der volle Inhalt der QuelleSidhu, Ishnoor, Sonali P. Barwe, Raju K. Pillai und Anilkumar Gopalakrishnapillai. „Harnessing the Power of Induced Pluripotent Stem Cells and Gene Editing Technology: Therapeutic Implications in Hematological Malignancies“. Cells 10, Nr. 10 (09.10.2021): 2698. http://dx.doi.org/10.3390/cells10102698.
Der volle Inhalt der QuelleMalladi, Srinivas. „Abstract P1-06-08: Metabolic diversity determines metastatic fitness of breast cancer brain-tropic cells“. Cancer Research 82, Nr. 4_Supplement (15.02.2022): P1–06–08—P1–06–08. http://dx.doi.org/10.1158/1538-7445.sabcs21-p1-06-08.
Der volle Inhalt der QuelleGrantcharova, Nina, Verena Peters, Claudia Monteiro, Katherina Zakikhany und Ute Römling. „Bistable Expression of CsgD in Biofilm Development of Salmonella enterica Serovar Typhimurium“. Journal of Bacteriology 192, Nr. 2 (06.11.2009): 456–66. http://dx.doi.org/10.1128/jb.01826-08.
Der volle Inhalt der QuelleGerritsen, Jacqueline S., Joseph S. Faraguna, Rudy Bonavia, Frank B. Furnari und Forest M. White. „Predictive data-driven modeling of C-terminal tyrosine function in the EGFR signaling network“. Life Science Alliance 6, Nr. 8 (11.05.2023): e202201466. http://dx.doi.org/10.26508/lsa.202201466.
Der volle Inhalt der QuelleKotini, Andriana, Jeffrey J. Delrow, Timothy A. Graubert, Stephen Nimer und Eirini P. Papapetrou. „Functional Dissection of Chromosome 7q Loss and Haploinsufficient Gene Discovery Using iPSC Models of MDS“. Blood 124, Nr. 21 (06.12.2014): 524. http://dx.doi.org/10.1182/blood.v124.21.524.524.
Der volle Inhalt der QuelleLemaire, Sandrine, Klaudia Kosowska-Shick, Peter C. Appelbaum, Gunther Verween, Paul M. Tulkens und Françoise Van Bambeke. „Cellular Pharmacodynamics of the Novel Biaryloxazolidinone Radezolid: Studies with Infected Phagocytic and Nonphagocytic cells, Using Staphylococcus aureus, Staphylococcus epidermidis, Listeria monocytogenes, and Legionella pneumophila“. Antimicrobial Agents and Chemotherapy 54, Nr. 6 (12.04.2010): 2549–59. http://dx.doi.org/10.1128/aac.01724-09.
Der volle Inhalt der QuelleJavier, Rodrigo, und Craig Horbinski. „TAMI-48. THE KETOGENIC DIET IS INEFFECTIVE IN PRECLINICAL MODELS OF IDH1 WILD-TYPE AND IDH1 MUTANT GLIOMA“. Neuro-Oncology 23, Supplement_6 (02.11.2021): vi208. http://dx.doi.org/10.1093/neuonc/noab196.831.
Der volle Inhalt der QuelleFrank, Karen M., Tong Zhou, Liliana Moreno-Vinasco, Brian Hollett, Joe G. N. Garcia und Juliane Bubeck Wardenburg. „Host Response Signature to Staphylococcus aureus Alpha-Hemolysin Implicates Pulmonary Th17 Response“. Infection and Immunity 80, Nr. 9 (25.06.2012): 3161–69. http://dx.doi.org/10.1128/iai.00191-12.
Der volle Inhalt der QuelleLara-Chacón, Bárbara, Sandra L. Guerrero-Rodríguez, Karla J. Ramírez-Hernández, Angélica Yamilett Robledo-Rivera, Marco Antonio Velasco Velazquez, Roberto Sánchez-Olea und Mónica Raquel Calera. „Gpn3 Is Essential for Cell Proliferation of Breast Cancer Cells Independent of Their Malignancy Degree“. Technology in Cancer Research & Treatment 18 (01.01.2019): 153303381987082. http://dx.doi.org/10.1177/1533033819870823.
Der volle Inhalt der QuelleMy, Ilaria, und Elisa Di Pasquale. „Genetic Cardiomyopathies: The Lesson Learned from hiPSCs“. Journal of Clinical Medicine 10, Nr. 5 (09.03.2021): 1149. http://dx.doi.org/10.3390/jcm10051149.
Der volle Inhalt der QuelleManceau, Line, Julien Richard Albert, Pier-Luigi Lollini, Maxim V. C. Greenberg, Pascale Gilardi-Hebenstreit und Vanessa Ribes. „Divergent transcriptional and transforming properties of PAX3-FOXO1 and PAX7-FOXO1 paralogs“. PLOS Genetics 18, Nr. 5 (23.05.2022): e1009782. http://dx.doi.org/10.1371/journal.pgen.1009782.
Der volle Inhalt der QuelleKargaran, Parisa K., Jared M. Evans, Sara E. Bodbin, James G. W. Smith, Timothy J. Nelson, Chris Denning und Diogo Mosqueira. „Mitochondrial DNA: Hotspot for Potential Gene Modifiers Regulating Hypertrophic Cardiomyopathy“. Journal of Clinical Medicine 9, Nr. 8 (23.07.2020): 2349. http://dx.doi.org/10.3390/jcm9082349.
Der volle Inhalt der QuelleNtai, Ioanna, Luca Fornelli, Caroline J. DeHart, Josiah E. Hutton, Peter F. Doubleday, Richard D. LeDuc, Alexandra J. van Nispen et al. „Precise characterization of KRAS4b proteoforms in human colorectal cells and tumors reveals mutation/modification cross-talk“. Proceedings of the National Academy of Sciences 115, Nr. 16 (02.04.2018): 4140–45. http://dx.doi.org/10.1073/pnas.1716122115.
Der volle Inhalt der QuelleJacobs, Keith M., Sandeep Misri, Barbara Meyer, Suyash Raj, Cheri L. Zobel, Barry P. Sleckman, Dennis E. Hallahan und Girdhar G. Sharma. „Unique epigenetic influence of H2AX phosphorylation and H3K56 acetylation on normal stem cell radioresponses“. Molecular Biology of the Cell 27, Nr. 8 (15.04.2016): 1332–45. http://dx.doi.org/10.1091/mbc.e16-01-0017.
Der volle Inhalt der QuelleRosenthal, Dean S., Elijah Finn, Devin Teehan, Nusrat Islam, Veerupaxagouda Patil, Bonnie Carney, Scott S. Rosenthal, Lucia Dussan, Cynthia M. Simbulan-Rosenthal und Peter Sykora. „Abstract B035: Developing the UValidate platform to measure DNA damage and repair capacity in isogenic donor-derived skin keratinocytes, fibroblasts and melanocyte cell-lines with different Fitzpatrick phototypes“. Cancer Research 84, Nr. 1_Supplement (09.01.2024): B035. http://dx.doi.org/10.1158/1538-7445.dnarepair24-b035.
Der volle Inhalt der QuelleVötsch, Désirée, Maren Willenborg, Walter M. R. Oelemann, Graham Brogden und Peter Valentin-Weigand. „Membrane Binding, Cellular Cholesterol Content and Resealing Capacity Contribute to Epithelial Cell Damage Induced by Suilysin of Streptococcus suis“. Pathogens 9, Nr. 1 (30.12.2019): 33. http://dx.doi.org/10.3390/pathogens9010033.
Der volle Inhalt der QuelleMeng, Qingyuan, Xiao Ding, Xiaosong Liu, Hailong Wang, Ling Wang, Junwen Qiao, Hua Cao et al. „Abstract 503: ISM3412, a novel and selective MAT2A inhibitor for the treatment of cancer“. Cancer Research 83, Nr. 7_Supplement (04.04.2023): 503. http://dx.doi.org/10.1158/1538-7445.am2023-503.
Der volle Inhalt der QuelleJenkins, Samir V., Shruti Shah, Azemat Jamshidi-Parsian, Amir Mortazavi, Gunnar Boysen, Kieng B. Vang, Robert J. Griffin, Narasimhan Rajaram und Ruud P. Dings. „Abstract 1088: Acquired radiation resistance induces thiol-dependent cisplatin cross-resistance“. Cancer Research 83, Nr. 7_Supplement (04.04.2023): 1088. http://dx.doi.org/10.1158/1538-7445.am2023-1088.
Der volle Inhalt der QuelleTawil, Nadim, Rayhaan Bassawon, Brian Meehan, Laura Montermini, Dongsic Choi, Ali Nehme, Hamed Najafabadi et al. „OPTC-5. Molecular signatures of podoplanin expressing glioblastoma cell subsets with putative role in cancer associated thrombosis and microthrombosis“. Neuro-Oncology Advances 3, Supplement_2 (01.07.2021): ii7. http://dx.doi.org/10.1093/noajnl/vdab070.026.
Der volle Inhalt der QuelleMaurissen, Thomas L., Masahide Kawatou, Víctor López-Dávila, Kenji Minatoya, Jun K. Yamashita und Knut Woltjen. „Modeling mutation-specific arrhythmogenic phenotypes in isogenic human iPSC-derived cardiac tissues“. Scientific Reports 14, Nr. 1 (31.01.2024). http://dx.doi.org/10.1038/s41598-024-52871-1.
Der volle Inhalt der QuelleLebedeva, Irina V., Michelle V. Wagner, Sunil Sahdeo, Yi-Fan Lu, Anuli Anyanwu-Ofili, Matthew B. Harms, Jehangir S. Wadia, Gunaretnam Rajagopal, Michael J. Boland und David B. Goldstein. „Precision genetic cellular models identify therapies protective against ER stress“. Cell Death & Disease 12, Nr. 8 (August 2021). http://dx.doi.org/10.1038/s41419-021-04045-4.
Der volle Inhalt der Quelle