Thematische Bibliographien / Immune complex diseases Immunological aspects

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  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher

Auswahl der wissenschaftlichen Literatur zum Thema „Immune complex diseases Immunological aspects“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 1. Februar 2022

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Zeitschriftenartikel zum Thema "Immune complex diseases Immunological aspects"

1

Troshina, Ekaterina A., Maria A. Terekhova und Ravida R. Akhmatova. „Immunological aspects of papillary thyroid cancer. What's new?“ Clinical and experimental thyroidology 16, Nr. 4 (15.06.2021): 14–18. http://dx.doi.org/10.14341/ket12695.

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Studying of the common links of pathogenesis of endocrine, autoimmune and oncological diseases is the area of interest of researchers from all countries of the world. Comprehension of artificially created mutual influences of molecular-genetic, immune factors that underlie the development and progression of endocrine tumors, primarily thyroid cancer, is important for creation and application of innovative treatment methods in oncoendocrinology.Today, the question of considering autoimmune diseases as a potential cause of oncological processes or on the contrary to consider them as protective conditions in some types of malignant tumors, remains controversial.In particular, autoimmune thyropathies and papillary thyroid cancer is an interesting model for studying these complex relationships. . The purpose of this article is to discuss accumulated experience, review the literature devoted to the study of immunological aspects in the pathogenesis of papillary thyroid cancer, reconsider obtained material and form a conclusion.
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Podoprigora, Gennadiy Ignat'evich, Lyudmila Ivanovna Kafarskaya, Nikolay Alekseevich Baynov und Andrey Nikolaevich Shkoporov. „Bacterial Translocation from Intestine: Microbiological, Immunological and Pathophysiological Aspects“. Annals of the Russian academy of medical sciences 70, Nr. 6 (06.12.2015): 640–50. http://dx.doi.org/10.15690/vramn564.

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Bacterial translocation (BT) is both pathology and physiology phenomenon. In healthy newborns it accompanies the process of establishing the autochthonous intestinal microbiota and the host microbiome. In immunodeficiency it can be an aethio-pathogenetic link and a manifestation of infection or septic complications. The host colonization resistance to exogenous microbic colonizers is provided by gastrointestinal microbiota in concert with complex constitutional and adaptive defense mechanisms. BT may be result of barrier dysfunction and self-purification mechanisms involving the host myeloid cell phagocytic system and opsonins. Dynamic cell humoral response to microbial molecular patterns that occurs on the mucous membranes initiates receptor signaling pathways and cascade of reactions. Their vector and results are largely determined by cross-reactivity between microbiome and the host genome. Enterocyte barriers interacting with microbiota play leading role in providing adaptive, homeostatic and stress host reactivity. Microcirculatory ischemic tissue alterations and inflammatory reactions increase the intestinal barrier permeability and BT. These processes a well as mechanisms for apoptotic cells and bacteria clearance are justified to be of prospective research interest. The inflammatory and related diseases caused by alteration and dysfunction of the intestinal barrier are reasonably considered as diseases of single origin. Maternal microbiota affects the formation of the innate immune system and the microbiota of the newborn, including intestinal commensal translocation during lactation. Deeper understanding of intestinal barrier mechanisms needs complex microbiological, immunological, pathophysiological, etc. investigations using adequate biomodels, including gnotobiotic animals.
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Salmasi, J. M., A. N. Kazimirskii, I. V. Kukes, G. V. Poryadin und D. I. Pozdnyakov. „Interferoncontaining drugs: clinical, pharmacological, and immunological points of their use for respiratory diseases treatment“. Meditsinskiy sovet = Medical Council, Nr. 11 (12.08.2021): 210–20. http://dx.doi.org/10.21518/2079-701x-2021-11-210-220.

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Nowadays pharmacological group classified by the ATX L03AX code as immunostimulants is demand among doctors of various specialties. The main area of application of such drugs is infectious respiratory viral processes, which are associated not only with the pathogenetic action of viruses, but also with a high risk of bacterial complications. Thus, the practitioner is faced with the task of preventing such complications and choosing an immunomodulatory drug with the most pronounced pharmacodynamic properties in this regard. In Russia, there are many drugs belonging to the group of immunomodulators with different mechanisms of action and end pharmacological and immunological points of application. The emphasis of the mechanisms of action of such drugs is made on the effect on the systems of both innate and adaptive immunity. At the same time, the severity of the effect on both one and the other immune system in drugs that is strong enough may differ, which requires special attention from the doctor when choosing a drug in a particular situation. A special place in the group of immunomodulatory drugs used for infectious respiratory diseases is occupied by interferoncontaining drugs that contain interferon alfa-2b (IFN-a2b). In addition, there are combination of IFN-a2b with an immunoglobulin complex, which increases the effectiveness of this drug in the treatment of various infectious and inflammatory diseases. The article describes the theoretical and practical aspects of administration this combined drug in pediatric practice and presents own experimental studies.
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Kusters, Johannes G., Arnoud H. M. van Vliet und Ernst J. Kuipers. „Pathogenesis of Helicobacter pylori Infection“. Clinical Microbiology Reviews 19, Nr. 3 (Juli 2006): 449–90. http://dx.doi.org/10.1128/cmr.00054-05.

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SUMMARY Helicobacter pylori is the first formally recognized bacterial carcinogen and is one of the most successful human pathogens, as over half of the world's population is colonized with this gram-negative bacterium. Unless treated, colonization usually persists lifelong. H. pylori infection represents a key factor in the etiology of various gastrointestinal diseases, ranging from chronic active gastritis without clinical symptoms to peptic ulceration, gastric adenocarcinoma, and gastric mucosa-associated lymphoid tissue lymphoma. Disease outcome is the result of the complex interplay between the host and the bacterium. Host immune gene polymorphisms and gastric acid secretion largely determine the bacterium's ability to colonize a specific gastric niche. Bacterial virulence factors such as the cytotoxin-associated gene pathogenicity island-encoded protein CagA and the vacuolating cytotoxin VacA aid in this colonization of the gastric mucosa and subsequently seem to modulate the host's immune system. This review focuses on the microbiological, clinical, immunological, and biochemical aspects of the pathogenesis of H. pylori.
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Khammissa, R. A. G., R. Chandran, A. Masilana, J. Lemmer und L. Feller. „Adverse Immunologically Mediated Oral Mucosal Reactions to Systemic Medication: Lichenoid Tissue Reaction/Interface Dermatitis-Stomatitis, Autoimmune Vesiculobullous Disease, and IgE-Dependent and Immune Complex Reactions“. Journal of Immunology Research 2018 (10.06.2018): 1–10. http://dx.doi.org/10.1155/2018/7645465.

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Drug-induced hypersensitivity immune reactions are exaggerated immunoinflammatory responses to allergenic components of the medications that occur in genetically susceptible subjects. The type of hypersensitivity immune response generated, whether antibody mediated or T cell mediated, or an immune complex reaction is determined by multiple factors, including the molecular characteristics of the allergen, the route of administration of the medication, the manner of presentation of the allergen by antigen-presenting cells to naïve T cells, the repertoire of the T cell receptors, and the cytokine profile within the microenvironment. This review deals with the clinical and histopathological aspects of adverse immunologically mediated oral mucosal reactions to systemic medication. We elaborate on diseases showing features of lichenoid tissue reaction/interface dermatitis-stomatitis, autoimmune vesiculobullous oral lesions, and immunoglobulin E- (IgE-) and immune complex-mediated oral reactions to drugs.
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Signorile, Anna, Anna Ferretta, Maddalena Ruggieri, Damiano Paolicelli, Paolo Lattanzio, Maria Trojano und Domenico De Rasmo. „Mitochondria, Oxidative Stress, cAMP Signalling and Apoptosis: A Crossroads in Lymphocytes of Multiple Sclerosis, a Possible Role of Nutraceutics“. Antioxidants 10, Nr. 1 (28.12.2020): 21. http://dx.doi.org/10.3390/antiox10010021.

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Multiple sclerosis (MS) is a complex inflammatory and neurodegenerative chronic disease that involves the immune and central nervous systems (CNS). The pathogenesis involves the loss of blood–brain barrier integrity, resulting in the invasion of lymphocytes into the CNS with consequent tissue damage. The MS etiology is probably a combination of immunological, genetic, and environmental factors. It has been proposed that T lymphocytes have a main role in the onset and propagation of MS, leading to the inflammation of white matter and myelin sheath destruction. Cyclic AMP (cAMP), mitochondrial dysfunction, and oxidative stress exert a role in the alteration of T lymphocytes homeostasis and are involved in the apoptosis resistance of immune cells with the consequent development of autoimmune diseases. The defective apoptosis of autoreactive lymphocytes in patients with MS, allows these cells to perpetuate, within the CNS, a continuous cycle of inflammation. In this review, we discuss the involvement in MS of cAMP pathway, mitochondria, reactive oxygen species (ROS), apoptosis, and their interaction in the alteration of T lymphocytes homeostasis. In addition, we discuss a series of nutraceutical compounds that could influence these aspects.
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Campos-Valdez, Marina, Hugo C. Monroy-Ramírez, Juan Armendáriz-Borunda und Laura V. Sánchez-Orozco. „Molecular Mechanisms during Hepatitis B Infection and the Effects of the Virus Variability“. Viruses 13, Nr. 6 (18.06.2021): 1167. http://dx.doi.org/10.3390/v13061167.

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The immunopathogenesis and molecular mechanisms involved during a hepatitis B virus (HBV) infection have made the approaches for research complex, especially concerning the patients’ responses in the course of the early acute stage. The study of molecular bases involved in the viral clearance or persistence of the infection is complicated due to the difficulty to detect patients at the most adequate points of the disease, especially in the time lapse between the onset of the infection and the viral emergence. Despite this, there is valuable data obtained from animal and in vitro models, which have helped to clarify some aspects of the early immune response against HBV infection. The diversity of the HBV (genotypes and variants) has been proven to be associated not only with the development and outcome of the disease but also with the response to treatments. That is why factors involved in the virus evolution need to be considered while studying hepatitis B infection. This review brings together some of the published data to try to explain the immunological and molecular mechanisms involved in the different stages of the infection, clinical outcomes, viral persistence, and the impact of the variants of HBV in these processes.
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Alagawany, Mahmoud, Shaaban S. Elnesr, Mayada R. Farag, Mohamed E. Abd El-Hack, Asmaa F. Khafaga, Ayman E. Taha, Ruchi Tiwari et al. „Use of Licorice (Glycyrrhiza glabra) Herb as a Feed Additive in Poultry: Current Knowledge and Prospects“. Animals 9, Nr. 8 (07.08.2019): 536. http://dx.doi.org/10.3390/ani9080536.

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Supplementation of livestock and poultry diets with herbal plants containing bioactive components have shown promising reports as natural feed supplements. These additives are able to promote growth performance and improve feed efficiency, nutrient digestion, antioxidant status, immunological indices, and poultry health. Several studies have used complex herbal formulas with the partial inclusion of licorice. However, the individual use of licorice has been rarely reported. The major problem of the poultry industry is the epidemiological diseases, mainly confined to the respiratory, digestive, and immune systems. Licorice has certain bioactive components such as flavonoids and glycyrrhizin. The roots of this herb contain 1 to 9% glycyrrhizin, which has many pharmacological properties such as antioxidant, antiviral, anti-infective and anti-inflammatory properties. Licorice extracts (LE) have a positive effect on the treatment of high-prevalence diseases such as the immune system, liver, and lung diseases. Studies showed that adding LE to drinking water (0.1, 0.2, or 0.3 g/L) reduced serum total cholesterol (p < 0.05) of broiler chickens. Moreover, LE supplementation in poultry diets plays a significant role in their productive performance by enhancing organ development and stimulating digestion and appetite. Along with its growth-promoting effects, licorice has detoxifying, antioxidant, antimicrobial, anti-inflammatory, and other health benefits in poultry. This review describes the beneficial applications and recent aspects of the Glycyrrhiza glabra (licorice) herb, including its chemical composition and role in safeguarding poultry health.
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Figueroa, J. E., und P. Densen. „Infectious diseases associated with complement deficiencies.“ Clinical Microbiology Reviews 4, Nr. 3 (Juli 1991): 359–95. http://dx.doi.org/10.1128/cmr.4.3.359.

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The complement system consists of both plasma and membrane proteins. The former influence the inflammatory response, immune modulation, and host defense. The latter are complement receptors, which mediate the cellular effects of complement activation, and regulatory proteins, which protect host cells from complement-mediated injury. Complement activation occurs via either the classical or the alternative pathway, which converge at the level of C3 and share a sequence of terminal components. Four aspects of the complement cascade are critical to its function and regulation: (i) activation of the classical pathway, (ii) activation of the alternative pathway, (iii) C3 convertase formation and C3 deposition, and (iv) membrane attack complex assembly and insertion. In general, mechanisms evolved by pathogenic microbes to resist the effects of complement are targeted to these four steps. Because individual complement proteins subserve unique functional activities and are activated in a sequential manner, complement deficiency states are associated with predictable defects in complement-dependent functions. These deficiency states can be grouped by which of the above four mechanisms they disrupt. They are distinguished by unique epidemiologic, clinical, and microbiologic features and are most prevalent in patients with certain rheumatologic and infectious diseases. Ethnic background and the incidence of infection are important cofactors determining this prevalence. Although complement undoubtedly plays a role in host defense against many microbial pathogens, it appears most important in protection against encapsulated bacteria, especially Neisseria meningitidis but also Streptococcus pneumoniae, Haemophilus influenzae, and, to a lesser extent, Neisseria gonorrhoeae. The availability of effective polysaccharide vaccines and antibiotics provides an immunologic and chemotherapeutic rationale for preventing and treating infection in patients with these deficiencies.
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Amend, Anaïs, Natalie Wickli, Anna-Lena Schäfer, Dalina T. L. Sprenger, Rudolf A. Manz, Reinhard E. Voll und Nina Chevalier. „Dual Role of Interleukin-10 in Murine NZB/W F1 Lupus“. International Journal of Molecular Sciences 22, Nr. 3 (29.01.2021): 1347. http://dx.doi.org/10.3390/ijms22031347.

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As a key anti-inflammatory cytokine, IL-10 is crucial in preventing inflammatory and autoimmune diseases. However, in human and murine lupus, its role remains controversial. Our aim was to understand regulation and immunologic effects of IL-10 on different immune functions in the setting of lupus. This was explored in lupus-prone NZB/W F1 mice in vitro and vivo to understand IL-10 effects on individual immune cells as well as in the complex in vivo setting. We found pleiotropic IL-10 expression that largely increased with progressing lupus, while IL-10 receptor (IL-10R) levels remained relatively stable. In vitro experiments revealed pro- and anti-inflammatory IL-10 effects. Particularly, IL-10 decreased pro-inflammatory cytokines and slowed B cell proliferation, thereby triggering plasma cell differentiation. The frequent co-expression of ICOS, IL-21 and cMAF suggests that IL-10-producing CD4 T cells are important B cell helpers in this context. In vitro and in vivo effects of IL-10 were not fully concordant. In vivo IL-10R blockade slightly accelerated clinical lupus manifestations and immune dysregulation. Altogether, our side-by-side in vitro and in vivo comparison of the influence of IL-10 on different aspects of immunity shows that IL-10 has dual effects. Our results further reveal that the overall outcome may depend on the interplay of different factors such as target cell, inflammatory and stimulatory microenvironment, disease model and state. A comprehensive understanding of such influences is important to exploit IL-10 as a therapeutic target.
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Dissertationen zum Thema "Immune complex diseases Immunological aspects"

1

Wootton, Andrew. „The glomerular basement membrane and nephritis /“. Title page, contents and abstract only, 1985. http://web4.library.adelaide.edu.au/theses/09PH/09phw918.pdf.

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Cheung, Ka-wa Benny, und 張嘉華. „Immune regulation in response to mycobacterial infection“. Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39634206.

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Waight, Sharma Agnes Phyllis. „The intestinal immune response to Giardia in the rat“. Title page, abstract and contents only, 1988. http://web4.library.adelaide.edu.au/theses/09PH/09phw138.pdf.

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Yip, Ming-shum, und 葉名琛. „Immune responses of human respiratory epithelial cells to respiratory syncytial virus and human metapneumovirus“. Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B3955725X.

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Metcalfe, Hannah Jane. „Activation of TLR5 by Salmonella-derived flagellins“. Thesis, Royal Veterinary College (University of London), 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572477.

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Abdukalykova, Saule. „Cellular and humoral immune responses in birds fed different levels of Arginine and vitamin E“. Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=100753.

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The effects of vitamin E (VE) and Arginine (ARG) on humoral and cellular immunity in chickens were investigated in two experiments. The humoral immunity was measured by antibody responses to sheep red blood cells (SRBC) and maternal antibody titers to the infectious bursal disease virus (IBDV), while the cellular immunity was studied using the cutaneous basophil hypersensitivity test to phytogemagglutinin (PHA) and by counting subpopulations of T-lymphocytes. We used two levels of ARG: normal (NARG, 1.2% in feed) and high ARG (HARG, additional 0.3% in drinking water or 1% in feed in experiments 1 and 2, respectively); and three levels of VE were given: 40, 80, and 400 IU/kg feed in experiment 1, and 40, 80, and 200 in experiment 2.
HARG improved the antibody response to SRBC compared with NARG ( P<0.01 for experiment 1 and P<0.013 for experiment 2) 4 days after injection in both experiments. In experiment 1, the VE80 birds maintained higher antibody titers to SRBC (P<0.001) than the VE40 and VE400 birds 4, 8 and 16 d after inoculation. In experiment 2, the antibody titers to SRBC were higher in the VE80 birds compared with the VE200 birds at days 5, 8, and 12 after inoculation (P<0.001). Maternal antibody titers (log10) to the IBDV were higher in the HARG than in the NARG diet in 17-day-old birds (P<0.001) and higher in the VE80 than in the VE40 birds (P<0.001), yet similar to those of the VE200 birds. No interactions were found between ARG and VE.
Naive birds fed HARG exhibited a higher response than NARG birds (P<0.05) to PHA-P at d 17 and to PHA-M at d 41, but, after a second exposure, high ARG levels did not have an effect. Also, in naive birds, the effects of VE were not significant at d 17, but showed an influence after a second exposure in 41-d-old birds.
The percentage of T-helper (Th) and T-cytotoxic (Tc) cells in the blood of 29-d-old birds were not different between ARG levels (P=0.07 and P=0.06, respectively), but Th cells were higher in the VE80 and VE200 birds than in the VE40 birds, and Tc was higher in the VE80 than in the VE40 birds (P=0.02). The B-cell:T-cell ratio was higher in the HARG than the NARG birds (P=0.01) and in the VE40 compared with the VE80 and VE200 birds (P<0.001). Neither ARG nor VE had an effect on the ratio of Th:Tc cells, nor on the percentage of immature T-lymphocytes.
A combination of high levels of ARG and high levels of VE (80 IU/kg of BW) has an important immunomodulation effect on the cellular and humoral immune responses in broiler chickens, improving both maternal antibody titers against the IBDV and antibody titers against SRBC. A combination of ARG and VE increases the proportions of Th and Tc cells, the B-cell:T-cell ratio, and growth performance. The evidence suggests that ARG and VE play complementary and regulatory role on immune response and may enhance the resistance of broilers to infectious diseases.
Key words. Arginine, vitamin E, humoral immunity, cell-mediated immunity, lymphocyte, ELISA.
L'effet de la vitamine E (VE) et l'arginine (ARG) sur les systèmes hummoraireet cellulaire de l'immunité a était évalué chez la volaille dans deux recherches. Lesystème hummoraire de l'immunité a était évalué en utilisant les paramètres tels que laproduction d'anticorps après une injection des globules rouge provenant des moutons(SRBC) et le niveau d'anticorps maternelle après une infection avec les virus causantla maladie 'infectious bursal disease' (lBDV), tandis que les effets sur le systemcellulaire de l'immunité avaient aussi été évalués en utilisant les paramètres comme'cutaneous basophil hypersensitivity test to phytogemagglutinin (PHA)' et endéterminant la concentration des lymphocytes T. Deux concentrations de ARG avaientété utilisées: normale (NARG, 1.2 % de la diète) et une concentration élevée (HARG,additionel 0.3 % dans l'eau ou 1 % dans les diètes); et 3 concentrations de VE: 40, 80et 400 lU/kg dans les diètes dans la première recherche et 40, 80, et 200 lU/kg dans ladeuxième recherche.
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Alberts, Terri Lynn. „Chronic fatigue and immune dysfunction syndrome: its relationship to underlying emotional and psychological issues“. CSUSB ScholarWorks, 1997. https://scholarworks.lib.csusb.edu/etd-project/1181.

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This post-positivist research study explored the possible relationship between Chronic Fatigue and Immune Dysfunction Syndrome (CFIDS) and the presence of underlying psychological and emotional issues. An exploratory design with naturalistic methods of inquiry was utilized to investigate whether the presence, or absence, of these issues had any impact on the overall disease process.
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Kodituwakku, Aruna Poojitha. „Antigen specific B cells in the immune response to Haemophilus influenzae type b PRP conjugate vaccine /“. Title page, table of contents and summary only, 2004. http://web4.library.adelaide.edu.au/theses/09PH/09phk769.pdf.

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Wootton, Andrew. „The glomerular basement membrane and nephritis“. 1986. http://web4.library.adelaide.edu.au/theses/09PH/09phw918.pdf.

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„The association of various HLA-A, -B and -DR loci with membranous glomerulonephritis, IgA nephropathy, and focal segmental glomerulosclerosis in KwaZulu-Natal renal patients“. Thesis, 2007. http://hdl.handle.net/10413/1789.

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This KwaZulu-Natal (KZN) based study investigates hypertension, glomerulonephritides and the rarity of IgA Nephropathy (IgAN) in Africans in association with the Human Leukocyte Antigen (HLA). A retrospective hypertensive study found a positive association with HLA-B40 (P c<0.05) and HLA-B15 (Pc<0.02) in Indians and Africans respectively. No association was found in Whites. A prospective study showed glomerulonephritides to be positively associated with HLA-A33 in Indians (Pc 0.049). No associations were found with glomerulonephritides in Africans and Whites. Combined Race groups show no HLA associations. HLA-A30; HLA-A34; HLA-A29; HLA-B42; HLA-B58; HLA-B70 and HLA-DR11 were extremely significantly higher in Africans compared to Indians and Whites (all P<0.0001). In conclusion, HLA-B40 and I 1LA-B15 are possible disease susceptibility markers in Indian and African hypertensives; HLA-A33 is a possible disease susceptibility marker for glomerulonephritides in Indians and alleles in linkage might be responsible for the rarity of IgAN in Africans but further studies need to be employed.
Thesis (M.Med)-University of KwaZulu-Natal, 2007.
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Bücher zum Thema "Immune complex diseases Immunological aspects"

1

Mak, Tak W. Handbook of immune response genes. New York: Plenum Press, 1998.

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Marchuk, G. I. Mathematical modelling of immune response in infectious diseases. Dordrecht: Kluwer Academic Publishers, 1997.

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International Symposium of the Immune Response to Viral Infections (1988 Florence, Italy). The immune response to viral infections. New York: Plenum Press, 1989.

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Stress, immune function, and health: The connection. New York: Wiley-Liss, 1999.

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Galoyan, Armen. Brain immune system signal molecules in protection from aerobic and anaerobic infections. New York: Springer, 2012.

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Green Cross International Symposium (1st 1992 Kyoto). Molecular basis of immune responses: [proceedings of the First Green Cross International Symposium "immunology for tomorrow" 1992]. Tokyo: Academic Press, 1993.

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7

Janice, Dietert, Hrsg. Strategies for protecting your child's immune system: Tools for parents and parents-to-be. Hackensack, NJ: World Scientific, 2010.

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Killer cell dynamics: Mathematical and computational approaches to immunology. New York, NY: Springer, 2007.

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Healing HIV: How to rebuild your immune system. Mill Valley, Calif: HealthFirst Press, 1999.

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TH17 cells in health and disease. New York: Springer, 2011.

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