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Thematische Bibliographien / Hydrocortisone

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Zeitschriftenartikel
Dissertationen
Bücher
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Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Hydrocortisone“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 9. Juni 2025

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Zeitschriftenartikel zum Thema "Hydrocortisone"

1

&NA;. "Hydrocortisone see Methylprednisolone/hydrocortisone." Reactions Weekly &NA;, no. 374 (October 1991): 5. http://dx.doi.org/10.2165/00128415-199103740-00021.

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&NA;. "Hydrocortisone see Tetracaine/sulfacetamide/hydrocortisone." Reactions Weekly &NA;, no. 329 (December 1990): 6. http://dx.doi.org/10.2165/00128415-199003290-00022.

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&NA;. "Hydrocortisone." Reactions Weekly &NA;, no. 1380 (December 2011): 22. http://dx.doi.org/10.2165/00128415-201113800-00078.

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&NA;. "Hydrocortisone." Reactions Weekly &NA;, no. 1382 (December 2011): 23. http://dx.doi.org/10.2165/00128415-201113820-00081.

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&NA;. "Hydrocortisone." Reactions Weekly &NA;, no. 1384 (January 2012): 30. http://dx.doi.org/10.2165/00128415-201213840-00120.

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&NA;. "Hydrocortisone." Reactions Weekly &NA;, no. 712 (August 1998): 9. http://dx.doi.org/10.2165/00128415-199807120-00023.

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&NA;. "Hydrocortisone." Reactions Weekly &NA;, no. 1189 (February 2008): 20. http://dx.doi.org/10.2165/00128415-200811890-00060.

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&NA;. "Hydrocortisone." Reactions Weekly &NA;, no. 1140 (February 2007): 12. http://dx.doi.org/10.2165/00128415-200711400-00042.

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&NA;. "Hydrocortisone." Reactions Weekly &NA;, no. 1143 (March 2007): 14. http://dx.doi.org/10.2165/00128415-200711430-00045.

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&NA;. "Hydrocortisone." Inpharma Weekly &NA;, no. 1174 (February 1999): 16. http://dx.doi.org/10.2165/00128413-199911740-00029.

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Dissertationen zum Thema "Hydrocortisone"

1

WYTS, LAURENCE. "Aminoglutethimide et hydrocortisone dans les adenocarcinomes prostatiques." Lille 2, 1991. http://www.theses.fr/1991LIL2M333.

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Garcia, Monica. "Differential Effects of Hydrocortisone on PTSD Symptom Clusters." Kent State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=kent1523196739368854.

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Hamitouche, Noureddine. "Modélisation pharmacocinétique - pharmacodynamique de la fludrocotisone par approche de population." Thesis, Rennes 1, 2017. http://www.theses.fr/2017REN1B026/document.

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Introduction. Les corticoïdes sont supposés avoir des effets bénéfiques à faibles doses chez les patients en choc septique réfractaire. Ces effets ont été retrouvés dans plusieurs études utilisant l’hydrocortisone. Mais des données similaires n’existent cependant pas pour la fludrocortisone. Pourtant, l’hypothèse est que ces effets seraient liés en partie à l’action minéralocorticoïde. Or, la fludrocortisone est considérée comme un puissant minéralocorticoïde. Avant une éventuelle évaluation de la fludrocortisone chez les patients en choc septique, il est nécessaire dans un premier temps d’étudier sa pharmacocinétique (PK) et sa relation pharmacocinétique-pharmacodynamique (PK-PD) afin de mieux cerner ses effets et de sélectionner sa posologie efficace. Méthodes. Pour répondre aux objectifs, plusieurs travaux ont été réalisés. La modélisation en pharmacométrie par approche de population a été utilisée dans ces travaux pour caractériser la PK et la relation PK-PD de la fludrocortisone chez des volontaires sains après administration unique et répétée et la PK chez les patients en choc septique. Résultats. Chez les volontaires sains en administration unique seule ou en association avec l’hydrocortisone, la fludrocortisone a montré une demi-vie courte et proche de celle de l’hydrocortisone. Par ailleurs, la fludrocortisone présentait une puissance de l’effet minéralocorticoïde environ 200 fois plus importante que celle de l’hydrocortisone. Les simulations ont montré que la fludrocortisone nécessiterait d’être administrée à raison de quatre fois par jour. Chez les patients en choc septique, l’absorption de la fludrocortisone était très variable (7/21 des patients n’absorbaient pas la molécule) ce qui suggérait la nécessité de mettre au point une forme administrable par voie intraveineuse. A nouveau chez les volontaires sains mais en administration répétée pendant plusieurs jours, la fludrocortisone a montré une demi-vie et des paramètres pharmacocinétiques semblables à ceux retrouvés lors de la première étude et sur le plan pharmacodynamique, des effets hémodynamiques favorables (sur la réactivité vasculaire, la pression artérielle…) montrés pour la première fois avec cette molécule. Conclusion. La fludrocortisone a montré qu’elle pouvait induire les effets biologiques et hémodynamiques recherchés. Les effets hémodynamiques sur la réactivité vasculaire et la pression artérielle ont été observés après une imprégnation de 5 jours d’administration répétée de la fludrocortisone notamment avec la dose de 400 µg/jour. Une évaluation de l’efficacité de la fludrocortisone chez les patients en choc septique et maintenant envisageable et nécessaire pour confirmer les résultats obtenus<br>Introduction. Low doses of corticosteroids showed beneficial effects in septic shock patients. These favorable effects may be partly result from the stimulation of the mineralocorticoid receptors. This finding has led us to explore the pharmacokinetic and the effects on hemodynamic and biologic parameters of fludrocortisone which is a potent mineralocorticoid. Methods. In this work, a population approach modeling (nonlinear mixed effects modeling) was used to characterize the pharmacokinetic and the pharmacokinetic-pharmacodynamic relationship of fludrocortisone in healthy volunteers and the pharmacokinetic in septic shock patients. Results. In healthy volunteers after single oral administration alone or in combination with hydrocortisone, fludrocortisone 50 µg showed a short and similar plasma elimination half-life that intravenous hydrocortisone. Fludrocortisone plasma concentrations and effect on urinary sodium/potassium ratio had a higher inter-individual variability as compared to hydrocortisone. Simulations suggested that the administration regimen of fludrocortisone should be reconsidered. In septic shock patient, a single oral dose of fludrocortisone at 50 µg yielded detectable plasma drug concentrations in two-thirds of adults with septic shock. Fludrocortisone pharmacokinetics showed a short plasma elimination half-life and a large inter-individual variability. These results suggested that an intravenous formulation of fludrocortisone would be useful to reduce its pharmacokinetic variability in septic patients. In healthy volunteers again, after 5 days of repeated oral administration, fludrocortisone improved pressor response to phenylephrine. This effect was observed only at the dose of 400 µg/day, suggesting that fludrocortisone at higher doses than previously administered (50 µg/day) may be useful to be effective. Furthermore, we showed that fludrocortisone had a short plasma half-live (1.94 h) which is consistent with our previously published study. After 5 day of repeated administration, fludrocortisone significantly increased blood pressure. This effect was more marked at the dose of 400 µg/day. Conclusion. Our results argue in favor of potential beneficial effects that fludrocortisone could have in septic shock patients. An evaluation of the effectiveness of fludrocortisone in these patients is now possible and necessary to confirm our results

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Hall, Clifford Michael. "Relative efficacy of hydrocortisone and methylprednisolone in acute severe asthma." Thesis, University of Cape Town, 1993. http://hdl.handle.net/11427/25562.

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Bosela, Ahmed Abdalla. "The effect of gamma-radiation on hydrocortisone in solutions and topical preparations." Thesis, University of Bath, 1987. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.377371.

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Gochenour, Lori L. "Cortisol responsivity association with fear and pain related to root canal therapy /." Morgantown, W. Va. : [West Virginia University Libraries], 2003. http://etd.wvu.edu/templates/showETD.cfm?recnum=2946.

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Thesis (M.S.)--West Virginia University, 2003.<br>Title from document title page. Document formatted into pages; contains ix, 57 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references (p. 43-46).

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Woods, Diana Lynn. "The effect of therapeutic touch on glucocortcoids and agitated behaviour in individuals with dementia of the Alzheimer type /." Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/7298.

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Santema, Peter. "Conflict, cooperation and cortisol in meerkats." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608222.

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Loric, Sylvain. "Transcortine sérique : méthode de dosage par mesure de sa capacité de liaison pour le cortisol." Paris 5, 1989. http://www.theses.fr/1989PA05P151.

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Muller, Caroline. "Etude du mécanisme d'action anti-glucocorticoïde des dérivés 7-hydroxylés de la déhydroépiandrostérone." Paris, CNAM, 2006. http://www.theses.fr/2006CNAM0554.

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Des expériences de microscopie confocale réalisées dans la lignée cellulaire COS transfectée par le récepteur glucocorticoïde humain (hGR) ont montré que ni la déhydroépiandrostérone (DHEA) ni ses dérivés 7-hydroxylés ne modifient la localisation du récepteur et n'empêchent son transfert nucléaire. Ces stéroïdes sont également sans effet sur l'activité transactivatrice du hGR. La voie d’action génomique impliquant le hGR ne serait donc pas la cible de l’action de ces stéroïdes. Une analyse enzymologique des réactions catalysées par la 11β-hydroxystéroïde déshydrogénase 1 humaine (h11β-HSD1) exprimée dans la levure a mis en évidence une interconversion des métabolites de la DHEA, en parallèle de la réaction d’oxydo-réduction du cortisol et de la cortisone. Une compétition s’opère donc entre les glucocorticoïdes, immuno-suppresseurs, et les anti-glucocorticoïdes, immuno-stimulateurs, pour la liaison à la h11β-HSD1 au sein des tissus où coexistent ces deux types de substrats<br>Experiments with a confocal microscope with a COS cell line transfected with the human glucocorticoid receptor (hGR) showed that dehydroepiandrosterone (DHEA) and its 7-hydroxylated derivatives do not modify the location of the receptor and do not prevent the nuclear transfer of hGR. These steroids had no effect on the transactivation activity of hGR. Thus, these neurosteroids were inactive on this model and their genomic action through the GR could be excluded. The human 11β-hydroxysteroid dehydrogenase type 1 (h11β-HSD1) was expressed in the yeast and each of the enzyme-catalyzed reactions were analyzed. The inter-conversion of the 7-hydroxylated DHEA metabolites was demonstrated as well as the oxidoreduction of cortisol and cortisone. A competition for the binding with the h11β-HSD1 may take place between these steroids and the glucocorticoid activation process, the former triggering immunity and the latter suppressing it

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Bücher zum Thema "Hydrocortisone"

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Savage, Edward Bruce. Hydrocortisone induces aortic rupture in inbred blotchy mice: Implications for abdominal aortic aneurysmal disease in humans. [New Haven: s.n.], 1985.

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Clarke, Dara. The control of androgen production in human adrenals: Examination of the effects of pro-opiomelanocortin-derived peptides on androgen and cortisol production in isolated human adrenal cells. Dublin: University College Dublin, 1996.

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K, Lüdecke Dieter, Chrousos George P, Tolis George, and International Symposium on Challenges of Hypersecretion: ACTH, Cushing's Syndrome, and Other Hypercortisolemic States (2nd : 1989 : Crete, Greece), eds. ACTH, Cushing's syndrome, and other hypercortisolemic states. New York: Raven Press, 1990.

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Justin, Lam, and Lam Carrie, eds. Advanced symptoms of adrenal fatigue syndrome: A metabolic perspective. Loma Linda, CA: Adrenal Institute Press, 2016.

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Khalsa, Dharma Singh. Brain Longevity. New York: Grand Central Publishing, 2001.

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Blokdijk, G. J. Hydrocortisone Butyrate; Third Edition. CreateSpace Independent Publishing Platform, 2018.

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Blokdijk, G. J. Hydrocortisone Valerate; Second Edition. CreateSpace Independent Publishing Platform, 2018.

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Patel, J. J. The degradation of Hydrocortisone and Dexamethasone. 1986.

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R, Chandra. The degradation of hydrocortisone and dexamethasone. 1986.

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Cortisol: Physiology, Regulation and Health Implications. Nova Science Publishers, Incorporated, 2012.

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Buchteile zum Thema "Hydrocortisone"

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Woods, Douglas W., Matthew R. Capriotti, Madison Pilato, Carolyn A. Doyle, Christopher J. McDougle, Beth Springate, Deborah Fein, et al. "Hydrocortisone." In Encyclopedia of Autism Spectrum Disorders, 1523. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_100679.

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de Groot, Anton C. "Hydrocortisone." In Monographs In Contact Allergy, 500–502. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003158004-247.

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Lascelles, P. T., and D. Donaldson. "Hydrocortisone Suppression Test." In Diagnostic Function Tests in Chemical Pathology, 84–85. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-1846-7_44.

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de Groot, Anton C. "Hydrocortisone Sodium Phosphate." In Monographs In Contact Allergy, 503–4. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003158004-248.

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Longhini, Federico, Eugenio Garofalo, and Andrea Bruni. "Hydrocortisone in Sepsis." In Reducing Mortality in Critically Ill Patients, 93–98. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71917-3_10.

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Isaksson, Marléne. "Budesonide, Tixocortol Pivalate, and Hydrocortisone-17-Butyrate." In Management of Positive Patch Test Reactions, 63–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55706-4_13.

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Dehshibi, Mohammad Mahdi, Alessandro Chiolerio, Anna Nikolaidou, Richard Mayne, Antoni Gandia, Mona Ashtari-Majlan, and Andrew Adamatzky. "On Stimulating Fungi Pleurotus Ostreatus with Hydrocortisone." In Emergence, Complexity and Computation, 105–21. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-38336-6_9.

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Blankenstein, Oliver. "Hydrocortisone Replacement in Disorders of Sex Development." In Understanding Differences and Disorders of Sex Development (DSD), 160–71. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000363752.

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Bertossi, Mirella, Beatrice Nico, Luisa Roncali, Daniela Virgintino, Lucia Mancini, Domenico Ribatti, and Pasquale Coratelli. "The Effects of Hydrocortisone on the Mesonephros Proximal Tubule Cells." In Advances in Experimental Medicine and Biology, 357–64. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-8953-8_36.

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Briegel, J., E. Kilger, and G. Schelling. "Stress Doses of Hydrocortisone in Septic Shock: Beyond the Hemodynamic Effects." In Yearbook of Intensive Care and Emergency Medicine, 189–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-662-13453-5_18.

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Konferenzberichte zum Thema "Hydrocortisone"

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"Effect of Hydrocortisone On Some Physiological and Morphological Changes in Mice Fetus." In XII. International Scientific Congress of Pure, Applied and Technological Sciences. Rimar Academy, 2024. https://doi.org/10.47832/minarcongress12-25.

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The purpose of the current study was to investigate the effects of hydrocortisone during the prenatal stage and its subsequent effects on various phenotypic abnormalities and morphologies in pregnant mice and their offspring. Rats were given hydrocortisone acetate treatment from the first day of pregnancy to day eighteen, at varying doses of 0.10– 0.15–0.20 mg/day. An experiment using thirty-two pregnant mice was conducted. Three groups of eight pregnant mice each make up each experimental set-up. Eight untreated pregnant mice (control group) were included in the first group; eight pregnant mice treated with hydrocortisone dosage of 0.20 milligrams per kilogram, these animals treated with a body weight of 0.15 mg/kg, and these mice treated with 0.01 mg/Kg of hydrocortisone were included in the second group. The statistical analysis showed that the typical embryo's length, weight, and quantity in pregnant mice decreased significantly in contrast to the group under authority. Additionally, the organs of thepregnat mice— The placenta, brain, spleen, liver, heart, kidney, lung, and reproductive system all significantly decreased. The dosage of hydrocortisone mostly causes the following physiologic malformations: head cap atrophy, tissue invasions, many bleeding sites, particularly the bloody area at the mid-dorsum, and hooked tails. Based on the previously provided information, we deduced that the administration of hydrocortisone resulted in the malformation of several organs. With the exception of the (0.2 mg/Kg) group, all embryos exhibited decreased mother weight upon adsorption. Pregnant women tend to become sedentary with accumulated bulk in their stomach and broiler, particularly when they are on 0.20 mg/kg of medication, during the behavior terms with greater attention. suggesting that the central nervous system has been partially defeminized and masculinized

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Ambalavanan, Namasivayam, and Rachel Giles. "Hydrocortisone does not help preterm infants." In ATS 2022 International Conference, edited by Richard Dekhuijzen. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/07799c07.

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Lu, De-Zhao, Ping-Er Wang, Ji Zhu, Tao-Qi Lin, and Xing-De Wo. "Effect of Hydrocortisone on Liver Mitochondria Proteome of Rats." In 2012 International Conference on Biomedical Engineering and Biotechnology (iCBEB). IEEE, 2012. http://dx.doi.org/10.1109/icbeb.2012.168.

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Deshpande, Madhura, and Tara Pouyani. "Hydrocortisone enhances and inhibits hyaluronate synthesis in a human dermal equivalent." In 2009 IEEE 35th Annual Northeast Bioengineering Conference. IEEE, 2009. http://dx.doi.org/10.1109/nebc.2009.4967637.

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Whitman, C., and C. Rose. "Vitamin C, Thiamine, and Hydrocortisone in Sepsis: A Single-Centered Experience." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3513.

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Livint, Vlad-Petru, Andrei-Eugen Bulgariu, Anca Mihaela Mocanu, and Laura Bulgariu. "Retention of Hydrocortisone from Aqueous Solution by Adsorption on PET Fibers." In 2022 E-Health and Bioengineering Conference (EHB). IEEE, 2022. http://dx.doi.org/10.1109/ehb55594.2022.9991686.

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Radulova-Mauersberger, O., F. Oehme, N. Mibelli, F. von Bechtolsheim, J. Weitz, M. Distler, and S. Hempel. "Does hydrocortisone reduce acute pancreatitis (PPAP) and morbidity after pancreatic resections?" In Viszeralmedizin 2024. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1789753.

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Albasha, W., and T. Ardiles. "Hydrocortisone, Ascorbic Acid, and Thiamine Therapy for Post-Cardiopulmonary Bypass Vasoplegia Syndrome." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3427.

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JUNG, Boris, Noemie Clavieras, Gerald Chanques, Fouad Belafia, and Samir Jaber. "Etomidate Is Not Associated With Prognosis In Hydrocortisone-Treated Septic Shock Patients." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4685.

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Arabi, Y., A. Al Jumah, O. Dabbagh, H. Tamim, A. Rishu, A. Al Abdulkareem, B. Al Knawy, A. Hajeer, W. Al-Tamimi, and A. Cherfan. "Hydrocortisone for Cirrhotic Patients with Septic Shock: Double-Blind Placebo-Controlled Randomized Trial." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1561.

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Berichte der Organisationen zum Thema "Hydrocortisone"

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Yehuda, Rachel. Improving PTSD Outcomes in OIF/OEF Returnees: A Randomized Clinical Trial of Hydrocortisone Augmentation of Prolonged Exposure Therapy. Fort Belvoir, VA: Defense Technical Information Center, August 2012. http://dx.doi.org/10.21236/ada603134.

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Chen, Chi-Hsin, Edward Pei-Chuan Huang, James d’Etienne, Eric H. Chou, Yen-Hsing Liu, Yu-Sheng Huang, Jia-How Chang, Pei-Chun Lai, and Yen-Ta Huang. Triple Combination of Hydrocortisone, Ascorbic Acid, and Thiamine (HAT) for Sepsis/Septic Shock: Meta-analysis and Trial Sequential Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2020. http://dx.doi.org/10.37766/inplasy2020.9.0097.

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song, lin, CHUN LIU, and Kangrui Fu. Early use of low-dose hydrocortisone can reduce in-hospital mortality in patients with septic shock:A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2024. http://dx.doi.org/10.37766/inplasy2024.8.0070.

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Lai, Pei-Chun, Chao-Han Lai, Edward Chia-Cheng Lai, and Yen-Ta Huang. Do we need to administer fludrocortisone in addition to hydrocortisone for adult patients with septic shock? A Bayesian network meta-analysis of randomized controlled trials and observational study with target trial emulation. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2023. http://dx.doi.org/10.37766/inplasy2023.6.0053.

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