Thematische Bibliographien / Frusemide

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  2. Dissertationen
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Auswahl der wissenschaftlichen Literatur zum Thema „Frusemide“

Autor: Grafiati
Veröffentlicht am 18. Mai 2024

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Zeitschriftenartikel zum Thema "Frusemide"

1

HINCHCLIFF, K. W., und K. H. McKEEVER. „Frusemide“. Equine Veterinary Journal 27, S18 (10.06.2010): 256–58. http://dx.doi.org/10.1111/j.2042-3306.1995.tb04932.x.

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Johnson, Valerie E., und P. J. Hilton. „Frusemide-Sensitive Sodium and Potassium Transport by Human Leucocytes“. Clinical Science 68, Nr. 1 (01.01.1985): 89–91. http://dx.doi.org/10.1042/cs0680089.

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1. Frusemide-sensitive sodium and potassium transport by normal human leucocytes has been studied in vitro by both isotopic and net flux techniques. 2. In physiological media the leucocyte exhibits a frusemide-sensitive influx of sodium and potassium of equal magnitude compatible with a 1:1 co-transport system. 3. Cells exposed to zero external sodium and potassium (osmolality maintained with choline) demonstrated a frusemide-sensitive sodium and potassium efflux. 4. Frusemide-sensitive potassium influx was dependent on the presence of external sodium but frusemide-sensitive sodium influx persisted unchanged in the absence of external potassium. 5. Frusemide-sensitive potassium influx was dependent on external chloride but frusemide-sensitive sodium influx was chloride-independent. 6. These last two observations make it likely that the frusemide-sensitive pathway is capable of operating in modes other than sodium-potassium co-transport.
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3

Jeffrey, R. F., T. M. MacDonald, M. Rutter, S. Freestone, J. Brown, R. R. Samson und M. R. Lee. „The effect of intravenous frusemide on urine dopamine in normal volunteers: Studies with indomethacin and carbidopa“. Clinical Science 73, Nr. 2 (01.08.1987): 151–57. http://dx.doi.org/10.1042/cs0730151.

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1. The urine dopamine response to intravenous frusemide (30 mg) was investigated in 15 salt replete male volunteers. The effects of oral indomethacin (100 mg) and oral carbidopa (100 mg) given before intravenous frusemide were studied in the same group of subjects. 2. Frusemide produced a significant increase in urine dopamine output within 15 min. 3. Indomethacin attenuated the natriuretic and renin responses to frusemide, but did not alter urine dopamine output. 4. Carbidopa lowered urine dopamine to undetectable levels, but did not significantly affect the natriuretic and renin responses to frusemide. 5. We conclude that urine dopamine excretion after frusemide is not directly related to increased sodium excretion or renin response and it is not mediated by the prostaglandins. In addition, dopamine does not contribute to the renal actions of frusemide under normal conditions.
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4

Molimard, M., und C. Advenier. „Effect of frusemide on bradykinin- and capsaicin-induced contraction of the guinea-pig trachea“. European Respiratory Journal 6, Nr. 3 (01.03.1993): 434–39. http://dx.doi.org/10.1183/09031936.93.06030434.

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Frusemide, a loop diuretic, inhibits the bronchial response to various bronchoconstrictor stimuli in asthmatic subjects. The underlying mechanisms remain unclear. In order to determine whether frusemide inhibits pharmacologically induced C-fibre stimulation, we investigated the effect of frusemide on bradykinin-, capsaicin-, neurokinin A-, and substance P-induced contraction of the guinea-pig isolated trachea. Frusemide 10(-5) and 10(-4) M produced a significant inhibition of concentration-response curves to bradykinin, which was markedly reduced by indomethacin 10(-6) M. Frusemide significantly reduced capsaicin-induced contraction only in the presence of indomethacin 10(-6) M. Neurokinin A- and substance P-induced contractions were not affected by frusemide and/or indomethacin. Our data suggest that a cyclo-oxygenase pathway is involved in the inhibition by frusemide of the bradykinin-induced contraction, but not in the inhibition of the capsaicin-induced contraction.
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5

Melki, T. S., M. L. Foegh und P. W. Ramwell. „Implication of thromboxane in frusemide diuresis in rats“. Clinical Science 71, Nr. 6 (01.12.1986): 647–50. http://dx.doi.org/10.1042/cs0710647.

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1. Administration of the thromboxane A2 (TXA2) synthase inhibitor OKY 1581 to rats significantly increased the urine output elicited by the loop diuretic frusemide. 2. The administration of the TXA2 receptor antagonist SQ 29548 significantly increased the diuretic effect of frusemide. 3. Another TXA2 receptor antagonist L-640,035 increased significantly the diuretic effect of frusemide. 4. Both the sodium excretion rate and urine osmolar excretion rate were significantly increased in rats treated with the TXA2 synthase inhibitor OKY 1581 and frusemide. 5. The data suggest that TXA2 is released during frusemide-induced diuresis in rats, and the released TXA2 has an opposing antidiuretic effect.
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6

Boles Ponto, Laura L., und Ronald D. Schoenwald. „Furosemide (Frusemide)“. Clinical Pharmacokinetics 18, Nr. 5 (Mai 1990): 381–408. http://dx.doi.org/10.2165/00003088-199018050-00004.

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Boles Ponto, Laura L., und Ronald D. Schoenwald. „Furosemide (Frusemide)“. Clinical Pharmacokinetics 18, Nr. 6 (Juni 1990): 460–71. http://dx.doi.org/10.2165/00003088-199018060-00003.

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8

Cardan, E. „Intrathecal frusemide“. Anaesthesia 40, Nr. 10 (Oktober 1985): 1025. http://dx.doi.org/10.1111/j.1365-2044.1985.tb10581.x.

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Yeo, CT, BJ O'Connor, M. Chen-Worsdell, PJ Barnes und KF Chung. „Protective effect of loop diuretics, piretanide and frusemide, against sodium metabisulphite-induced bronchoconstriction in asthma“. European Respiratory Journal 5, Nr. 10 (01.11.1992): 1184–88. http://dx.doi.org/10.1183/09031936.93.05101184.

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We determined whether the loop diuretic, piretanide, had a similar inhibitory action against sodium metabisulphite (MBS)-induced bronchoconstriction in asthmatic subjects as frusemide and, if so, its duration of action. In the first study, we compared the effect of inhaled placebo, piretanide (24 mg), or frusemide (40 mg), on the provocative concentration of MBS needed to cause a 20% fall in baseline forced expiratory volume in one second (FEV1) (PC20MBS) in 12 mild asthmatic subjects before, immediately after, and at 1.5, 3, 6, and 24 h, after inhalation. Both piretanide and frusemide induced a significant diuresis lasting at least 24 h. Frusemide caused a mean 3.8 fold (95% confidence interval: 2.3-6.3 fold), piretanide a 2.5 fold (1.8-3.4 fold) and placebo a 1.7 fold (1.5-1.9 fold) increase in PC20MBS. The effects of frusemide and piretanide were significantly greater than that of placebo. At later time points, tachyphylaxis to the bronchoconstrictor effects of MBS was observed during the placebo limb. In the second study, we measured PC20MBS at 90 min after inhalation of either placebo, piretanide (24 mg), or frusemide (40 mg). No significant difference in PC20MBS was observed. We conclude that piretanide in addition to frusemide significantly inhibits MBS-induced bronchoconstriction and that this action is short-lived over less than 90 min. Frusemide was more potent in inhibiting MBS-induced bronchoconstriction despite causing a smaller diuretic effect than piretanide. The basic mechanism of action of the loop diuretics in the airways remains unclear.
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J. Jawad, Fatima. „A Study on the Stability of Different Frusemide Liquid Dosage Formulas: Oral Solution, Syrup, Elixir, Suspension and Emulsion“. Iraqi Journal of Pharmaceutical Sciences ( P-ISSN: 1683 - 3597 , E-ISSN : 2521 - 3512) 17, Nr. 2 (30.03.2017): 1–8. http://dx.doi.org/10.31351/vol17iss2pp1-8.

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The present study aim at preparing frusemide in liquid form suitable for oral use. This is achieved through preparing different liquid forms of frusemide. The frusemide liquid is prepared in the following forms: oral solution, syrup and elixir with intensity of 1, 0.4 and 0.8% weight /volume respectively and in combination with potassium carbonate, polysorbate 80, alcohol and phosphate buffer solution of pH8 to dissolve the frusemide in the above mentioned forms. The different forms of the prepared medicine have been stored in glass bottles that can provide protection against light and at 40, 50, 600C for four months. Besides the pH has been checked to decide the period of validity. The results show that the expiration date of frusemide have lasted for 1.8, 1.07 and 1.22 years respectively for the oral solution, the syrup and the elixir. The suspensions of frusemide are formulated in combination with the following: polyvinyl pyrolidine, xanthan gum, the combination of (xanthan gum and sodium carboxymethyl cellulose), the combination of (xanthan: methyl cellulose) and chitosan. The formulas which give suitable release of the drug are chosen for assessment according to the following considerations: The rat of sedimentation and apparent zero order degradation constant at 250C. In conclusion, it is found the best formula is that which includes poly vinylpyrolidine, tween20, glycerol, sorbitol, cocoa syrup and parabens at pH7. the fluidity of this chosen formula is psendoplastic type and its validity has lasted for about three years. The emulsion of frusemide is also prepared extemporaneously by using the commercial frusemide tablets in combination with acacia and olive oil. This should be consumed within 45 days of the date of production. Key word: frusemide, elixir, suspension, emulsion.
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Dissertationen zum Thema "Frusemide"

1

Overend, Tim. „The effect of frusemide on intrapulmonary sensory receptors“. Thesis, Liverpool John Moores University, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337788.

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Upsdell, Stephen Mark. „A critical appraisal of the use of frusemide in diuresis renography“. Thesis, Imperial College London, 1990. http://hdl.handle.net/10044/1/46589.

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Martin, Una. „Drug disposition in chronic renal failure : studies with paracetamol and frusemide“. Thesis, University of Edinburgh, 1992. http://hdl.handle.net/1842/19990.

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Patients with renal failure react inappropriately to many drugs and have an increased incidence of side effects which may be due to changes in drug absorption, distribution, metabolism and excretion. Active or inactive polar drug metabolites which are normally excreted in the urine will accumulate and patients with end stage disease may depend completely on haemodialysis or continuous ambulatory peritoneal dialysis (CAPD) for their elimination. Despite several single dose studies little information exists about the disposition of paracetamol and frusemide during chronic dosing in patients with renal failure including those maintained on dialysis and this has been investigated further in the present study. Six patients with end stage disease maintained on CAPD were given 1 g of oral paracetamol. Absorption was normal but plasma concentrations of the glucuronide and sulphate conjugates were greatly increased with little change during the observation period. The extraction capacity of the peritoneal membrane was low and the peritoneal clearance was < 7ml.min-1 for paracetamol and these conjugates. This disposition of paracetamol was then compared in 6 healthy volunteers and 6 conservatively managed patients with chronic renal failure taking 1 g 3 times a day for 10 days. Before dosing, the daily plasma concentrations of unchanged paracetamol were significantly higher in the patients (3.1 ± 0.6 versus 1.1 ± 0.3 mg.l-1) suggesting enterohepatic recycling with regeneration by hydrolysis of the conjugates and reabsorption of the parent compound. There was marked accumulation of the glucuronide conjugate (87.0 ± 69.0 versus 3.0 ± 0.5 mg.l-1 in the volunteers) which was dependent on the severity of the renal failure. The concentrations of the sulphate conjugate (25.0 ± 19.0 mg.l^-1) did not accumulate as predicted possibly due to depletion of inorganic sulphate. Patients with end stage renal failure maintained on haemodialysis were also treated with a similar regime of paracetamol. Neither the glucuronide nor sulphate conjugate reached the predicted mean plasma concentrations of 569 ± 150 and 434 ± 92 mg.l^-1 and extraction ratios of paracetamol and its conjugates were less than 50%.
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McCrindle, Jennifer. „Factors affecting the absorption and disposition of frusemide and bumetanide in man“. Thesis, University of Edinburgh, 1996. http://hdl.handle.net/1842/20670.

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Drug absorption, elimination and effect may be influenced by many factors including type of formulation, particle size, dissolution and by the presence of other drugs or food and fluid ingestion. The influence of some of these factors on the absorption and effect of frusemide and bumetanide have been investigated in the present studies. The effect of food on the absorption of oral frusemide (40 mg) was determined in 8 healthy volunteers. Food significantly reduced peak plasma concentrations (2.35±0.49 versus 0.51±0.19 mg/l) and delayed the time to peak. The bioavailability of frusemide was also significantly reduced from 76% fasting to 42% after food. The study was then repeated in 9 healthy volunteers using oral bumetanide (2 mg) given with and without food. The peak concentration was reduced after food (96.9±15.1 versus 36.1±11.5 μg/l) and the time to peak concentration delayed. However the mean bioavailability of bumetanide was not significantly reduced by food. A survey carried out in two medical wards within the Edinburgh Royal Infirmary then provided information on the general use of frusemide and bumetanide and showed that most of the patients were taking a single oral dose of diuretic with or in close proximity to breakfast, a situation which could potentially alter diuretic absorption and effect. Consequently, the effect of hospital breakfast on the absorption and efficacy of frusemide was studied in 10 medical inpatients. However when frusemide was administered 2 hours after breakfast, no significant improvement in area under the plasma concentration time curve, urinary recovery of frusemide or total natriuretic and diuretic response was found.
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Passmore, Anthony Peter. „An investigation of the acute diuretic and renal haemodynamic effects of frusemide, bemetanide and caffeine“. Thesis, Queen's University Belfast, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317051.

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D'Souza, Gwyn Ann Ophilda. „Frusemide in the horse and its effects on the metabolism, pharmacokinetics and detectability of co-administered drugs“. Thesis, Imperial College London, 1991. http://hdl.handle.net/10044/1/46751.

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Johnston, George Dennis. „An investigation of some of the factors modifying the acute peripheral non-diuretic vascular effects of frusemide“. Thesis, Queen's University Belfast, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.329394.

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Bücher zum Thema "Frusemide"

1

Doherty, Christopher. Microenvironmental role of pharmaceutical excipients in drug dissolution control: Studies of physicochemical andcrystallographic properties of frusemide, including a novel crystalline form.... Bradford, 1986.

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Nuhu, Shem Zagbayi. Comparative pharmacology of three diuretics: Effect of flurbiprofen : a comparison in rats and rabbits of some aspects of the pharmacology of piretanide, frusemide and bumetanide, with particular reference to the involvement of prostanoids. Bradford, 1985.

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Buchteile zum Thema "Frusemide"

1

Darlington, L. Gail. „Study to Compare the Relative Hyperuricaemic Effects of Frusemide and Bumetanide“. In Purine and Pyrimidine Metabolism in Man V, 333–39. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5104-7_57.

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Rossoni, G., F. Berti, G. Zuccari, A. Buschi, M. Robuschi, L. M. Villa und O. Caratozzolo. „Frusemide Inhalation Prevents Immunological Mediator Release and Respiratory Changes in Guinea-Pig“. In Asthma Treatment, 261–71. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3446-4_24.

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Passavanti, G., M. Giannattasio, G. Mazzia und P. Coratelli. „Muzolimine vs Frusemide in Chronic Renal Failure: Short Term Effect in Double-Blind Study“. In Diuretics: Basic, Pharmacological, and Clinical Aspects, 385–87. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2067-8_93.

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Hamdy, R. C., und B. Murnane. „The effects of frusemide and Dorbanex on the pharmacokinetic profile of benoxaprofen in elderly subjects“. In Side-Effects of Anti-Inflammatory Drugs, 277–79. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-010-9772-7_22.

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„Frusemide.“ In Drugs Handbook 2012–2013. Bloomsbury Academic, 2011. http://dx.doi.org/10.5040/9781350363595.art-778.

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„frusemide, n.“ In Oxford English Dictionary. 3. Aufl. Oxford University Press, 2023. http://dx.doi.org/10.1093/oed/4951549334.

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Ainsworth, Sean. „F“. In Neonatal Formulary, herausgegeben von Sean Ainsworth, 308–48. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198840787.003.0019.

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This chapter presents information on neonatal drugs that begin with F, including use, pharmacology, adverse effects, fetal and infant implications of maternal treatment, treatment, and supply of Fentanyl, Fibrin sealants and cyanoacrylate tissue adhesives, Flecainide, Flucloxacillin (also cloxacillin and dicloxacillin), Fluconazole, Flucytosine, Fludrocortisone, Folic acid (pteroylglutamic acid), Formula milks for babies with intolerance/allergy, Formula milks for preterm babies, Fosfomycin, Fresh frozen plasma and cryoprecipitate, and Furosemide = Frusemide (former BAN)
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frca, o. boyd mrcp. „The high-risk surgical case“. In Critical Care Cases, 63–66. Oxford University PressNew York, NY, 1997. http://dx.doi.org/10.1093/oso/9780192625847.003.0015.

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Abstract A 75-year-old female presented to the surgical team with a 12-hour history of abdominal pain. She took regular NSAIDS. for osteoarthritis. One year previously she was admitted to the same hospital with a myocardial infarction, since when she had taken regular frusemide and enalapril for shortness of breath. She lived in a bungalow with social services support. Pulse rate 120/min; BP 100/50 mmHg; temperature 38°C; respiratory rate 25/min with shallow breaths and decreased air entry at the bases. Auscul tation revealed a gallop rhythm and the abdomen was tense and silent.
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