Thematische Bibliographien / Escherichia coli

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Escherichia coli“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 30. Juli 2024

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Zeitschriftenartikel zum Thema "Escherichia coli"

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Haubrich, William S. „Escherich of Escherichia coli“. Gastroenterology 122, Nr. 1 (Januar 2002): 54. http://dx.doi.org/10.1016/s0016-5085(02)80065-8.

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Schnabel, Uta. „Inactivation of Escherichia coliK-12 and enterohemorrhagic Escherichia coli (EHEC) by atmospheric pressure plasma“. Journal of Agricultural Science and Applications 03, Nr. 03 (04.09.2014): 81–91. http://dx.doi.org/10.14511/jasa.2014.030305.

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Starčič Ejravec, Marjanca, Matija Rijavec und Darja Žagur-Bertok. „Kolicini zbirke uropatogenih bakterij Escherichia coli“. Acta Biologica Slovenica 49, Nr. 2 (01.12.2006): 13–21. http://dx.doi.org/10.14720/abs.49.2.15098.

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110 uropathogenic Escherichia coli (UPEC) strains were screened for colicin production and 42 (38%) of the tested UPEC strains were found to be colicinogenic. The ColA, ColB, ColD, ColE2, ColE3, ColE4, ColE5, ColE6, ColE7, ColIa, ColIb, ColK, ColN, MccB17, ColS4, MccC7 and ColE6-J colicin producer strains from Pugsley’s collection of colicinogenic strains were lysed by all colicinogenic UPEC strains, the ColM and ColE1 producer strains by 93% of the UPEC colicinogenic strains and the ColV producer strain by only 81% of the UPC colicinogenic strains. 67% of the colicinogenic UPEC strains were able to lyse all 20 used coli- cin producer strains and 33% of the colicinogenic UPEC strains were able to lyse 19 Pugsley’s strains. Hence, a majority (67%) of the studied UPEC strains encode and produce either more than one colicin, or a colicin not tested. Colicins of UPEC strains producing only one colicin were identified; 8 strains(19% of the colicinogenic strains) produced only ColV, 3 strains (7%) ColM and 3 strains (7%) ColE1. Plasmids were found in 88% of the colicinogenic strains. 11 DL strains were found to harbour conjugative plasmids encoding antibiotic resistance(s) and colicinogenicity. Further, 19% of the haemolytic UPEC strains and 44% of non-haemolytic strains were also colicinogenic, 28% of the cnf encoding strains and 41% of the strains not encoding cnf were colicinogenic, while 40% of ibeA encoding strains and 38% of strains not encoding ibeA were colicinogenic.
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Bahri, Saiful, Saiku Rokhim und Yosi Setia Prasiska. „Kontaminasi Bakteri Escherichia coli pada Sampel Daging“. Journal of Health Science and Prevention 3, Nr. 1 (22.04.2019): 62–67. http://dx.doi.org/10.29080/jhsp.v3i1.195.

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Pangan yang berasal dari ternak sangat kita butuhkan karena mengandung protein penting dan mudah dicerna, daging merupakan salah satu pangan yang berasal dari ternak dan sangat bermanfaat bagi tubuh karena mengandung gizi yang lengkap sehingga perlu untuk dilakukan pencegahan kontaminasi terutama oleh bakteri Eschericia coli. Tujuan dari penelitian ini adalah untuk mengetahui kontaminasi bakteri Escherichia coli pada beberapa sampel daging. Penelitian ini termasuk dalam penelitian eksperimental untuk mengidentifikasi bakteri Escherichia coli pada beberapa daging segar menggunakan metode pengenceran dan lama waktu inkubasi yang berbeda dari setiap sampel. Berdasarkan penelitian yang telah dilakukan diperoleh jumlah kontaminasi bakteri Escherichia coli tertinggi pada sampel daging ayam daripada daging sapi dan daging kambing. Hasil tertinggi kontaminasi bakteri Escherichia coli daging ayam yang melebihi BMCM adalah sebesar 1108 : 1 x 109 dengan kode DA 049-DA 058. Kesimpulan berdasarkan hasil yang diperoleh bahwa 18 sampel daging ayam sebanyak (50%) sampel terkontaminasi Escherichia coli, 13 sampel daging sapi sebanyak (38%) terkontaminasi Escherichia coli. Sedangkan 1 daging kambing (0%) tidak terkontaminasi bakteri Escherichia coli.
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Goldman, R., und H. M. Adam. „Escherichia coli“. Pediatrics in Review 27, Nr. 3 (01.03.2006): 114–15. http://dx.doi.org/10.1542/pir.27-3-114.

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Lorenz, E., M. D. Plamann und G. V. Stauffer. „Escherichia coli“. MGG Molecular & General Genetics 250, Nr. 1 (1996): 81. http://dx.doi.org/10.1007/s004380050053.

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Morosini, M. I. „Escherichia coli“. International Journal of Infectious Diseases 14 (März 2010): e23-e24. http://dx.doi.org/10.1016/j.ijid.2010.02.1539.

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Goodsell, David S. „Escherichia coli“. Biochemistry and Molecular Biology Education 37, Nr. 6 (November 2009): 325–32. http://dx.doi.org/10.1002/bmb.20345.

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LEENANON, B., und M. A. DRAKE. „Acid Stress, Starvation, and Cold Stress Affect Poststress Behavior of Escherichia coli O157:H7 and Nonpathogenic Escherichia coli†“. Journal of Food Protection 64, Nr. 7 (01.07.2001): 970–74. http://dx.doi.org/10.4315/0362-028x-64.7.970.

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The effects of acid shock, acid adaptation, starvation, and cold stress of Escherichia coli O157:H7 (ATCC 43895), an rpo S mutant (FRIK 816-3), and nonpathogenic E. coli (ATCC 25922) on poststress heat resistance and freeze–thaw resistance were investigated. Following stress, heat tolerance at 56°C and freeze–thaw resistance at −20 to 21°C were determined. Heat and freeze–thaw resistance of E. coli O157:H7 and nonpathogenic E. coli was enhanced after acid adaptation and starvation. Following cold stress, heat resistance of E. coli O157:H7 and nonpathogenic E. coli was decreased, while freeze–thaw resistance was increased. Heat and freeze–thaw resistance of the rpoS mutant was enhanced only after acid adaptation. Increased or decreased tolerance of acid-adapted, starved, or cold-stressed E. coli O157:H7 cells to heat or freeze–thaw processes should be considered when processing minimally processed or extended shelf-life foods.
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Firmansyah, Dedy. „EFEK LISTRIK TEGANGAN RENDAH TERHADAP PERTUMBUHAN BAKTERI Escherichia Coli“. Jurnal Kedokteran YARSI 30, Nr. 2 (04.08.2023): 82–90. http://dx.doi.org/10.33476/jky.v30i2.3103.

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Bakteri Escherichia coli merupakan salah satu jenis bakteri gram negatif dan bersifat fakultatif anaerob.Bakteri yang ditemukan oleh Theodor Escherich ini biasanya hidup di usus besar manusia berfungsi untuk menjaga kesehatan sistem pencernaan. Bakteri ini umumnya tidak berbahaya,walaupun begitu tingkat kewaspadaan terhadap Escherichia coli perlu tetap diterapkan.Prevalensi infeksi karena bakteri Escherichia coli sangat tinggi di negara berkembang dengan perkiraan angka kejadian lebih dari 100 kasus per 100.000 penduduk (WHO, 2006). Penelitian ini merupakan penelitian eksperimental dengan tujuan untuk mengetahui efek listrik tegangan rendah terhadap pertumbuhan bakteri Escherichia coli.Perlakuan menggunakan perlakuan sebanyak lima batch yang masing-masing batch terdiri dari lima tabung. Setiap 1 batch penelitian menggunakan tegangan listrik yang sama selama 1 jam dengan mengevaluasi pemantauan setiap 20 menit, 40 menit, dan 60 menit.Pada batch 1,digunakan tegangan listrik 0,4 volt sebanyak lima tabung,batch ke-2 dilanjutkan dengan tegangan listrik 0,5 volt sebanyak lima tabung,batch ke-3 dengan menggunakan tegangan listrik 0,7 volt,dan batch ke-4 dengan menggunakan tegangan listrik 0,8 volt dan pada batch ke-5 dengan menggunakan tegangan listrik 1 volt sebanyak lima tabung.Dari hasil penelitian ini didapatkan hasil bahwa listrik tegangan rendah dapat membunuh bakteri Eschericia coli dengan tegangan listrik optimal sebesar 0,5 volt,arus listrik 40 mA dan waktu pemantauan optimal yaitu 20 menit.
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Dissertationen zum Thema "Escherichia coli"

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Schnepel, Jörn. „Klonierung, Expression und Charakterisierung kryptischer Fibronektin-Proteinasen“. [S.l. : s.n.], 2000. http://deposit.ddb.de/cgi-bin/dokserv?idn=969330472.

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Rang, Camilla. „Molecular and physiological characteristics of Escherichia coli growth in vitro and in the gastrointestinal tract of mice“. Göteborg : [Dept. of General and Marine Microbiology, Göteborg University], 1997. http://catalog.hathitrust.org/api/volumes/oclc/38988157.html.

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Rosko, Jerko. „Osmotaxis in Escherichia coli“. Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/28947.

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Bacterial motility, and in particular repulsion or attraction towards specific chemicals, has been a subject of investigation for over 100 years, resulting in detailed understanding of bacterial chemotaxis and the corresponding sensory network in many bacterial species including Escherichia coli. E. Coli swims by rotating a bundle of flagellar filaments, each powered by an individual rotary motor located in the cell membrane. When all motors rotate counter-clockwise (CCW), a stable bundle forms and propels the cell forward. When one or more motors switch to clock-wise (CW) rotation, their respective filaments fall out of the bundle, leading to the cell changing orientation. Upon switching back to CCW, the bundle reforms and propels the cell in a new direction. Chemotaxis is performed by the bacterium through prolonging runs by suppressing CW rotation when moving towards nutrients and facilitating reorientation by increasing CW bias when close to a source of a harmful substance. Chemicals are sensed through interaction with membrane bound chemosensors. These proteins can interact with a very specific set of chemicals and the concentrations they are able to sense are in the range between 10-⁶ and 10-² M. However, experiments have shown that the osmotic pressure exerted by large (> 10-¹ M) concentrations of solutes, which have no specificity for binding to chemosensors (e.g. sucrose), is able to send a signal down the chemotactic network. Additionally, clearing of bacterial density away from sources of high osmolarity has been previously observed in experiments with agar plates. This behaviour has been termed osmotaxis. The aim of this doctoral thesis work is to understand how different environmental cues influence the tactic response and ultimately, combine at the network output to direct bacterial swimming. As tactic responses to chemical stimuli have been extensively studied, I focus purely on the response to non-specific osmotic stimuli, using sucrose to elevate osmolarity. I monitor the chemotactic network output, the rotation of a single bacterial flagellar motor, using Back Focal Plane Interferometry over a variety of osmotic conditions. Additionally, in collaboration with Vincent Martinez, I studied the effect of elevated osmolality on swimming speed of large (104) bacterial populations, using differential dynamic microscopy (DDM). I have found that sudden increases in media osmolarity lead to changes of both motor speed and motor clockwise bias, which is the fraction of time it spends rotating clockwise. Changes in CW Bias proceed in two phases. Initially, after elevating the osmolarity, CW Bias drops to zero, indicating that the motor is exclusively in the ‘cell run’ mode. This phase lasts from 2-5 minutes depending on the magnitude of the change in solute concentration. What follows then is a distinct second phase where the CW Bias is elevated with respect to the initial levels and this phase lasts longer than 15-20 minutes. In comparison, for defined chemical stimuli, the motor output resets after several seconds, a behaviour termed perfect adaptation. For changes of 100 mOsm/kg and 200 mOsm/kg in magnitude the motors speed up, often by as much as a factor of two, before experiencing a gradual slow down. Despite the slow down, motors still rotate faster 15-20 minutes after the change in osmolarity, than they did before. For changes of 400 mOsm/Kg in magnitude the motors decrease sharply in speed, coming to a near halt, recovering after 5 minutes and eventually, on average, speeding up. DDM studies of free swimming bacteria have shown that elevated osmolality leads to higher swimming speeds, in agreement with single motor data. Using theoretical models of bacterial swimming from the literature, it is discussed how this motor output, although different to what is expected for chemotaxis, is able to drive bacteria away from regions of space with high osmolalities. Additionally, I have started extending the work done with sucrose, to another solute often used to elevate osmolality, sodium chloride. While sucrose is outer membrane impermeable, NaCl can cross the outer membrane into the periplasmic space. Another layer of complexity is that NaCl has some specificty for the chemoreceptors. The preliminary results are shown and qualitatively agree with those obtain with sucrose.
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Rosser, Tracy. „Pathogenic potential of Escherichia coli O26 and sorbitol-fermenting Escherichia coli O157:NM“. Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/4427.

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Verocytotoxin-producing Escherichia coli (VTEC) are important human pathogens that may cause diarrhoea, haemorrhagic colitis and haemolytic uremic syndrome (HUS). Worldwide, non-sorbitol-fermenting (NSF) VTEC O157:H7 is the most common serogroup associated with HUS but several non-O157:H7 serogroups have emerged as causes of this disease. This research investigated the pathogenic potential of two non-O157:H7 serogroups: O26 and sorbitol-fermenting (SF) O157:NM. While VTEC O26 have emerged as a significant cause of HUS in continental Europe, human infections associated with this pathogen are uncommon in Scotland and generally only result in simple diarrhoea. The study characterised E. coli O26 isolates recovered from human infections in Europe and Scotland and isolates collected from Scottish cattle with the objectives to identify factors which may allow strains to cause more serious clinical disease and to investigate the potential of bovine VTEC O26 in Scotland to cause human infection. MLST analysis of housekeeping genes found little genetic variation in the genomic ‘backbone’ among the vast majority of E. coli O26 isolates. The gene for verocytotoxin 2 (vtx2) alone was carried by VTEC O26 isolates recovered from patients in continental Europe but was found in no Scottish human isolate, where the majority of isolates did not harbour a vtx gene. It was demonstrated that among the European VTEC O26 human isolates, 67% carried a specific allele within the promoter region for LEE1 and 87% harboured the tccP2 gene. In contrast, no Scottish VTEC O26 human isolate carried this allele or the tccP2 gene. The impact these genotypic characteristics have on the pathogenic potential of a strain remains uncertain. There were no clear differences in verocytotoxin titres, levels of LEEencoded protein secretion or levels of adherence to Caco-2 cells between VTEC O26 isolates recovered from human infections of varying severity. However, levels of LEE-encoded protein secretion from cattle isolates were generally higher than those from many of the human isolates. The differences in pathogenic potential between isolates are likely to be due to horizontally acquired DNA, including vtx2 carriage and the O-island-phage-associated effector protein repertoire. Further work is required to determine if the differences identified may also impact on shedding levels from cattle and therefore the likelihood of transmission to humans. Since 1988, SF VTEC O157:NM strains have emerged and have been associated with a higher incidence of progression to HUS than NSF VTEC O157:H7. This study investigated bacterial factors that may account for the increased pathogenic potential of SF VTEC O157:NM. While no evidence of toxin or toxin expression differences between the two VTEC O157 groups was found, the SF VTEC O157:NM strains adhered at significantly higher levels to a human colonic cell line. Under the conditions tested, curli were shown to be the main factor responsible for the increased adherence to Caco-2 cells. The capacity of SF VTEC O157:NM strains to express curli at 37C may have relevance to the epidemiology of human infections as curliated strains could promote higher levels of colonization and inflammation in the human intestine. In turn this could lead to increased toxin exposure and an increased likelihood of progression to HUS.
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Solecki, Olivia. „Explaining the urban and rural differences of Escherichia coli 0157 human infection in Grampian“. Thesis, Available from the University of Aberdeen Library and Historic Collections Digital Resources, 2008. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=25203.

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Wu, Gilbert Kar Po. „Signal transduction responses to enteropathogenic Escherichia coli and Shiga toxin-producing Escherichia coli infections“. Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0007/MQ46054.pdf.

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Hesterberg, Micaela. „Charakterisierung einer unbekannten Redoxgruppe sowie der Konformation der NADH:Ubichinon-Oxidoreduktase (Komplex I) aus Escherichia coli“. [S.l.] : [s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=964821672.

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Grabowski, Gabriele. „Untersuchungen zum Mechanismus der Katalyse und zu In-vivo-Selektionssystemen für die Anreicherung von Mutanten der Restriktionsendonuklease EcoRI mit veränderter DNA-Bindungs- oder Spaltspezifität“. [S.l. : s.n.], 1998. http://deposit.ddb.de/cgi-bin/dokserv?idn=954823028.

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Uhland, Kerstin. „Biochemische und genetische Charakterisierung der Trehalasen TreA und TreF von Escherichia coli“. [S.l. : s.n.], 1997. http://deposit.ddb.de/cgi-bin/dokserv?idn=958497885.

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Franke, Sylvia. „Das Kupfer-transportierende CusCFBA-Efflux-System aus Escherichia coli“. [S.l. : s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=967128110.

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Bücher zum Thema "Escherichia coli"

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Hilary, Babcock, Hrsg. Escherichia coli infections. 2. Aufl. New York: Chelsea House, 2010.

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Wiwanitkit, Viroj. Escherichia coli Infections. North Charleston: CreateSpace Independent Publishing Platform, 2011.

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Manning, Shannon D. Escherichia coli infections. Herausgegeben von Alcamo I. Edward und Heymann David L. Philadelphia: Chelsea House, 2005.

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Manning, Shannon D. Escherichia coli infections. Herausgegeben von Babcock Hilary. 2. Aufl. New York: Chelsea House, 2010.

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Kay, Miller Ellen. Escherichia coli O157. Beltsville, Md: National Agricultural Library, 1993.

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Kay, Miller Ellen. Escherichia coli O157. Beltsville, Md: National Agricultural Library, 1992.

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Kasʹi͡anenko, A. M. Gigienicheskie i ėpidemiologicheskie aspekty ėsherikhiozov. Kiev: Nauk. dumka, 1988.

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United States. Animal and Plant Health Inspection Service. Veterinary Services. Centers for Epidemiology and Animal Health., Hrsg. Escherichia coli O157:H7: Issues and ramifications. Fort Collins, Colo: USDA:APHIS:VS Centers for Epidemiology and Animal Health, 1994.

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United States. Animal and Plant Health Inspection Service. Veterinary Services. Centers for Epidemiology and Animal Health. Escherichia coli O157:H7: Issues and ramifications : executive summary. Fort Collins, Colo: USDA:APHIS:VS, Centers for Epidemiology and Animal Health, 1994.

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Torres, Alfredo G., Hrsg. Escherichia coli in the Americas. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-45092-6.

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Buchteile zum Thema "Escherichia coli"

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Gyles, C. L., und J. M. Fairbrother. „Escherichia Coli“. In Pathogenesis of Bacterial Infections in Animals, 267–308. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9780470958209.ch15.

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Weinstock, George M. „Escherichia coli“. In Bacterial Genomes, 651–53. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-6369-3_60.

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Altenbuchner, Josef, und Ralf Mattes. „Escherichia coli“. In Production of Recombinant Proteins, 7–43. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2005. http://dx.doi.org/10.1002/3527603670.ch2.

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Coia, John, und Heather Cubie. „Escherichia coli“. In The Immunoassay Kit Directory, 757–61. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0359-3_16.

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Rabinowitz, Ronald P., und Michael S. Donnenberg. „Escherichia coli“. In Infectious Agents and Pathogenesis, 101–31. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0313-6_6.

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Ullmann, Uwe. „Escherichia coli“. In Lexikon der Infektionskrankheiten des Menschen, 297–300. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_334.

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Feng, Peter. „Escherichia Coli“. In Guide to Foodborne Pathogens, 222–40. Oxford: John Wiley & Sons, 2013. http://dx.doi.org/10.1002/9781118684856.ch14.

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Schultz, Michael. „Escherichia coli“. In Therapeutic Microbiology, 83–96. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815462.ch7.

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Bhunia, Arun K. „Escherichia coli“. In Foodborne Microbial Pathogens, 249–69. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7349-1_14.

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Pitout, Johann D. D. „Escherichia Coli“. In Molecular Techniques for the Study of Hospital-Acquired Infection, 179–92. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118063842.ch11.

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Konferenzberichte zum Thema "Escherichia coli"

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Xiao Liang und Xiao Liang. „Method To Partition Between Freely Suspended Escherichia coli and Escherichia coli attached to clay particles“. In 2012 Dallas, Texas, July 29 - August 1, 2012. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2012. http://dx.doi.org/10.13031/2013.41852.

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Hirayama, Kayoko, Yun Jung Heo und Shoji Takeuchi. „Formation of cross-shaped Escherichia coli“. In 2014 IEEE 27th International Conference on Micro Electro Mechanical Systems (MEMS). IEEE, 2014. http://dx.doi.org/10.1109/memsys.2014.6765602.

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Freire, Higor, Larissa Gomes, Jose de Carvalho, Francilayne Barbosa und Diego Pereira. „Escherichia Coli Diarreiogeníca: uma Revisão Literária“. In XXI I Congresso Brasileiro de Nutrologia. Thieme Revinter Publicações Ltda, 2018. http://dx.doi.org/10.1055/s-0038-1674671.

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Sahlan, Muhamad, Ihsan Wiratama, Heri Hermansyah, Anondho Wijarnako, Mohamad Teguh Gumelar und Masafumi Yohda. „Apoptin gene optimization in Escherichia coli“. In SECOND INTERNATIONAL CONFERENCE OF MATHEMATICS (SICME2019). Author(s), 2019. http://dx.doi.org/10.1063/1.5096733.

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Balaev, Aleksei E., K. N. Dvoretski und Valeri A. Doubrovski. „Refractive index of escherichia coli cells“. In Saratov Fall Meeting 2001, herausgegeben von Valery V. Tuchin. SPIE, 2002. http://dx.doi.org/10.1117/12.475627.

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Zeljkovic, V., C. Drazgalski und P. Mayorga. „Algorithmic escherichia coli bacteria incidence evaluation“. In 2018 Global Medical Engineering Physics Exchanges/Pan American Health Care Exchanges (GMEPE/PAHCE). IEEE, 2018. http://dx.doi.org/10.1109/gmepe-pahce.2018.8400732.

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Tanba, C., S. Bandaru und C. J. Haas. „Double Trouble: Escherichia Coli Bilateral Empyema“. In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a2110.

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Hanif, Sarmad, Urmi Bajpai, Bhakti Chavan, Ritam Das und Sanket Shah. „Lysins as antibacterials against Uropathogenic Escherichia coli“. In International Symposium on Immunobiologicals. Instituto de Tecnologia em Imunobiológicos, 2022. http://dx.doi.org/10.35259/isi.2022_52167.

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9

Bousba, Houssem Eddine, Salah Sahli, Wail Seif, Eddine Namous, Lyes Benterrouche und Mouna Saoudi. „Inactivation of Escherichia coli in water using cold atmospheric plasma jet“. In 2022 2nd International Conference on Advanced Electrical Engineering (ICAEE). IEEE, 2022. http://dx.doi.org/10.1109/icaee53772.2022.9962110.

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Martin, T., und C. Paul. „Induced Polarisation (IP) Laboratory Measurements on Escherichia Coli (E. Coli)-Sand Mixtures“. In 25th European Meeting of Environmental and Engineering Geophysics. European Association of Geoscientists & Engineers, 2019. http://dx.doi.org/10.3997/2214-4609.201902481.

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Berichte der Organisationen zum Thema "Escherichia coli"

1

Acott, Jedidiah. Interstrand Crosslink Resistance in Escherichia Coli. Portland State University, Mai 2018. http://dx.doi.org/10.15760/mem.1.

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2

Clark, D. P. Regulation of alcohol fermentation by Escherichia coli. Office of Scientific and Technical Information (OSTI), Januar 1990. http://dx.doi.org/10.2172/7206403.

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3

Clark, D. P. Regulation of alcohol fermentation by Escherichia coli. Office of Scientific and Technical Information (OSTI), Januar 1989. http://dx.doi.org/10.2172/7279319.

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4

Flowers, Ann M. Secretion of Heterologous Proteins from Escherichia coli. Fort Belvoir, VA: Defense Technical Information Center, Dezember 2000. http://dx.doi.org/10.21236/ada391190.

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5

Dr. David Nunn. Improvements In Ethanologenic Escherichia Coli and Klebsiella Oxytoca. Office of Scientific and Technical Information (OSTI), September 2010. http://dx.doi.org/10.2172/992134.

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6

Mingorance, Jesús. Escherichia coli O104:H4, ¿es nueva la "nueva bacteria"? Sociedad Española de Bioquímica y Biología Molecular (SEBBM), Juli 2011. http://dx.doi.org/10.18567/sebbmdiv_rpc.2011.07.2.

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7

Baehr, A., G. Dunham, Hideo Matsuda, G. Michaels, R. Taylor, R. Overbeek, K. E. Rudd et al. An integrated database to support research on Escherichia coli. Office of Scientific and Technical Information (OSTI), Januar 1992. http://dx.doi.org/10.2172/5865388.

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8

Baehr, A., G. Dunham, Hideo Matsuda, G. Michaels, R. Taylor, R. Overbeek, K. E. Rudd et al. An integrated database to support research on Escherichia coli. Office of Scientific and Technical Information (OSTI), Januar 1992. http://dx.doi.org/10.2172/10122295.

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9

Wendel, Brian. Completion of DNA Replication in Escherichia coli. Portland State University Library, Januar 2000. http://dx.doi.org/10.15760/etd.6290.

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10

Monson, Melissa S., Michael G. Kaiser und Susan J. Lamont. Variation in Avian Pathogenic Escherichia coli Colonization Levels in Chickens. Ames (Iowa): Iowa State University, Januar 2016. http://dx.doi.org/10.31274/ans_air-180814-232.

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