Zeitschriftenartikel: „Enteral feeding“

1

Weissman, T. E., B. K. Wershil und H. M. Adam. „Enteral Feeding“. Pediatrics in Review 29, Nr. 3 (01.03.2008): 105–6. http://dx.doi.org/10.1542/pir.29-3-105.

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Weissman, Taryn E., und Barry K. Wershil. „Enteral Feeding“. Pediatrics In Review 29, Nr. 3 (01.03.2008): 105–6. http://dx.doi.org/10.1542/pir.29.3.105.

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3

Jeejeebhoy, Khursheed N. „Enteral feeding“. Current Opinion in Gastroenterology 18, Nr. 2 (März 2002): 209–12. http://dx.doi.org/10.1097/00001574-200203000-00010.

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4

Jeejeebhoy, Khursheed N. „Enteral feeding“. Current Opinion in Gastroenterology 19, Nr. 2 (März 2003): 140–43. http://dx.doi.org/10.1097/00001574-200303000-00007.

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5

Jeejeebhoy, Khursheed N. „Enteral feeding“. Current Opinion in Gastroenterology 20, Nr. 2 (März 2004): 110–13. http://dx.doi.org/10.1097/00001574-200403000-00010.

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6

Jeejeebhoy, Khursheed N. „Enteral feeding“. Current Opinion in Internal Medicine 2, Nr. 1 (Februar 2003): 77–80. http://dx.doi.org/10.1097/00132980-200302010-00014.

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7

DeLegge, Mark H. „Enteral feeding“. Current Opinion in Gastroenterology 24, Nr. 2 (März 2008): 184–89. http://dx.doi.org/10.1097/mog.0b013e3282f4dbab.

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8

&NA;. „Enteral feeding“. Current Opinion in Gastroenterology 28, Nr. 2 (März 2012): 185. http://dx.doi.org/10.1097/mog.0b013e3283516692.

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9

Jeejeebhoy, Khursheed N. „Enteral feeding“. Current Opinion in Clinical Nutrition and Metabolic Care 5, Nr. 6 (November 2002): 695–98. http://dx.doi.org/10.1097/00075197-200211000-00013.

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10

Brody, Jane K. „Enteral Feeding“. AJN, American Journal of Nursing 110, Nr. 1 (Januar 2010): 13. http://dx.doi.org/10.1097/01.naj.0000366033.05712.54.

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11

DeLong, Colin G., und Eric M. Pauli. „Enteral Feeding“. Advances in Surgery 54 (September 2020): 231–49. http://dx.doi.org/10.1016/j.yasu.2020.05.009.

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12

Jeejeebhoy, Khursheed N. „Enteral feeding“. Current Opinion in Gastroenterology 21, Nr. 2 (März 2005): 187–91. http://dx.doi.org/10.1097/01.mog.0000153345.57239.2c.

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13

Haddad, Rami Y., und David R. Thomas. „Enteral nutrition and enteral tube feeding“. Clinics in Geriatric Medicine 18, Nr. 4 (November 2002): 867–81. http://dx.doi.org/10.1016/s0749-0690(02)00035-6.

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14

Holden, Chris. „Home enteral feeding“. Paediatric Nursing 2, Nr. 6 (Juli 1990): 14–16. http://dx.doi.org/10.7748/paed.2.6.14.s15.

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15

Wireko, BM, und Tim Bowling. „Enteral tube feeding“. Clinical Medicine 10, Nr. 6 (Dezember 2010): 616–19. http://dx.doi.org/10.7861/clinmedicine.10-6-616.

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16

&NA;. „Sucralfate/enteral feeding“. Reactions Weekly &NA;, Nr. 459 (Juli 1993): 12. http://dx.doi.org/10.2165/00128415-199304590-00059.

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17

Maltman, Mark. „Enteral feeding tubes“. Veterinary Nursing Journal 21, Nr. 11 (November 2006): 20–21. http://dx.doi.org/10.1080/17415349.2006.11013524.

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18

Leães, Dória M., Elza Mello, Mariur Beghetto und Michelli Cristina de Silva Assis. „Enteral feeding tubes“. Nutrition and Health 21, Nr. 3 (Juli 2012): 193–200. http://dx.doi.org/10.1177/0260106012459937.

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Patients in whom oral energy intake is insufficient for daily needs may develop malnutrition and its complications, such as increased infection rates, increased length of hospitalization, and death. Enteral feeding is beneficial for these patients. However, this therapy is not without complications related to the insertion and placement of enteral feeding tubes. This review aims to identify from the literature different techniques for insertion and the methods used to evaluate the placement of enteral feeding tubes.

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19

Paccagnella, Agostino, Maria Lisa Marcon, Stefania Rebuffi, Antonella Garna, Alessandra Mauri, Daniela Maccari, Patrizia Paiusco und Nello Spinella. „Home Enteral Feeding“. Quality Management in Health Care 22, Nr. 3 (2013): 248–66. http://dx.doi.org/10.1097/qmh.0b013e31829a6d55.

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20

Cattan, Stéphane, und Jacques Cosnes. „Enteral feeding techniques“. Current Opinion in Clinical Nutrition and Metabolic Care 1, Nr. 3 (Mai 1998): 287–90. http://dx.doi.org/10.1097/00075197-199805000-00009.

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21

Matarese, Laura E. „Enteral Feeding Solutions“. Gastrointestinal Endoscopy Clinics of North America 8, Nr. 3 (Juli 1998): 593–609. http://dx.doi.org/10.1016/s1052-5157(18)30251-4.

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22

Edmonds, M. „Establish enteral feeding“. BMJ 310, Nr. 6975 (04.02.1995): 309. http://dx.doi.org/10.1136/bmj.310.6975.309.

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23

KASHIWABARA, Norio. „Enteral feeding formulas.“ Kagaku To Seibutsu 28, Nr. 4 (1990): 238–45. http://dx.doi.org/10.1271/kagakutoseibutsu1962.28.238.

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24

NOBEL, JOEL J. „Enteral feeding pumps“. Pediatric Emergency Care 12, Nr. 2 (April 1996): 128–36. http://dx.doi.org/10.1097/00006565-199604000-00017.

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25

Wolf, Steven E. „Enteral Feeding Intolerance“. Archives of Surgery 132, Nr. 12 (01.12.1997): 1310. http://dx.doi.org/10.1001/archsurg.1997.01430360056010.

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26

Muthiah, Alli, Sridevi Karthikeyan, Barath Raj Kumar und Aruna M. „EARLY ENTERAL FEEDING AND DELAYED ENTERAL FEEDING- A COMPARATIVE STUDY“. Journal of Evidence Based Medicine and Healthcare 4, Nr. 22 (14.03.2017): 1241–45. http://dx.doi.org/10.18410/jebmh/2017/243.

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27

O’Keefe, Stephen J. D., Ronzo B. Lee, Jing Li, Wen Zhou, Barbara Stoll und Qianyu Dang. „Trypsin and splanchnic protein turnover during feeding and fasting in human subjects“. American Journal of Physiology-Gastrointestinal and Liver Physiology 290, Nr. 2 (Februar 2006): G213—G221. http://dx.doi.org/10.1152/ajpgi.00170.2005.

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Knowledge of the stimulatory effects of enteral and parenteral (intravenous) feeding on the synthesis and turnover of trypsin would help in the management of acute pancreatitis, because the disease is caused by the premature activation of trypsin. To investigate this, we labeled intravenous infusions with [1-13C]leucine and enterals with [2H]leucine and measured isotope enrichment of plasma, secreted trypsin, and duodenal mucosal proteins over 6 h by duodenal perfusion/aspiration and endoscopic biopsy. Thirty healthy volunteers were studied during fasting ( n = 7), intravenous feeding ( n = 6), or postpyloric enteral feeding [duodenal polymeric ( n = 6), elemental duodenal ( n = 6), and jejunal elemental ( n = 5)]. All diets provided 1.5 g·kg−1·day−1 protein and 40 kcal·kg−1·day−1 energy. Results demonstrated that compared with fasting, enteral feeding increased the rate of appearance (71 ± 4 vs. 91 ± 5 min, P = 0.01) and secretion (546 ± 80 vs. 219 ± 37 U/h, P = 0.01) of newly labeled trypsin and expanded zymogen stores (1,660 ± 237 vs. 749 ± 133 units, P = 0.03). These differences persisted whether the feedings were polymeric or elemental, duodenal, or jejunal. In contrast, intravenous feeding had no effect on basal rates. Differential labeling of the plasma amino acid pool by enteral and intravenous isotope infusions suggested that 35% of absorbed amino acids were retained within the splanchnic bed during enteral feeding and that mucosal protein turnover increased from a fasting rate of 34 ± 6 to 108 ± 8%/day ( P < 0.05) compared with no change after intravenous feeding. In conclusion, all common forms of enteral feeding stimulate the synthesis and secretion of pancreatic trypsin, and only parenteral nutrition avoids it.

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Stamenkovic, Dusica, Marica Basic, I. Palibrk und Zorica Jankovic. „Enteral nutrition and surgical patient“. Acta chirurgica Iugoslavica 50, Nr. 4 (2003): 109–13. http://dx.doi.org/10.2298/aci0304109s.

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Enterai nutrition can be applicated alone or in combination with, parenteral nutrition. Enterai feeding should be applicated as early as possible in preoperative preparation or in postoperative period in respect of contraindications and everyday evaluation of patients. Immunomodulatory substances like arginin, 3-o-fat acids, ribonucleic acid and glutamine are incorporated in "ready to use" solution for enterai feeding. Enterai feedings oral or via tubes are safe if some precautions are taken: like sitting position and control of feeding tubes position. Use of jejunostomy and promotility agents improved enterai feeding after major abdominal surgery and acute pancreatitis. Enterai feeding and immunonutrition improved postoperative course in reduction of hospital stay, incidence of postoperative complications especially infections. The aim of this review article is to validate pro and con for enterai nutrition in preoperative and postoperative course.

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Chatterjee, Souvik, Sujoy Kumar Bala, Partha Chakraborty, Rajesh Dey, Santanu Sinha, Ramdip Ray und Abdur Rahed. „A comparative study between early enteral feeding (within 24 hours) versus conventional enteral feeding after enteric anastomosis“. Bangladesh Journal of Medical Science 11, Nr. 4 (13.11.2012): 273–83. http://dx.doi.org/10.3329/bjms.v11i4.12597.

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Background: Traditionally, enteric feeds are withheld for a period of 48-72 hrs, sometimes even more following enteric anastomosis depending upon return of full peristaltic sounds. This results in a period of nonstimulation of gut Gut Rest, which was supposed to result in better anastomotic healing. But this same also deprives the intestinal mucosa of surface nutrients as well as prolongs parenteral fluid therapy, thereby depriving the patients of adequate nutrition. Along with it, prolonged parenteral therapy also keeps the patients bound to bed with its resultant complications like, prolonged hospital stay and increased cost of therapy. Objectives: To compare the benefits of early enteral feeding over conventional enteric feeding following enteric anastomosis with special regards to patients recovery and complications. Methods and materials: The selection of patients into group A (60) and group B (60) was done after having fulfilled inclusion and exclusion criteria. Informed consent was obtained. The patients of group A were fed via enteral route within 24 hrs of enteric anastomosis. The patients of group B were fed via enteral route after 48-72 hrs or appearance of full peristaltic sounds following enteric anastomosis. These patients were followed in post operative period for their drain output, any nausea, vomiting or significant abdominal distension, prolonged ileus, post operative duration of shospital stay, post operative infective complications (e.g. wound infection, UTI, RTI), and different haematological and biochemical examinations. Results: This study shows that post operative nausea-vomiting, anastomotic leakage rate, re-exploration, wound infection and RTI rates are higher in group A than those of group B. In this study, the incidence of UTI in post operative period is higher in group B. But the differences in above mentioned variables are not statistically significant. Whereas appearance of intestinal peristaltic sound is earlier in group A (42.8 ± 10.68 hours) compare to that of group B (52.6 ± 13.46 hours). Here, the difference is statistically significant (p value = 0.000022) The duration of post operative hospital stay is shorter in group A (8.45 ± 5.143 days) than that of group B (10.533 ± 4.952 days). The difference of duration post operative hospital stay is statistically significant (p value = 0.0257). Removal of nasogastric tube, resumption of oral feeding, and passage of first flatus and/or defecation were earlier in the group A than that of the group B; the differences were statistically significant. The post operative day-5 albumin level is better in group A (3.147 ± 0.4409 gm/dl) than that of group B (2.935 ± 0.3124 gm/dl). This difference is also statistically significant (p value = 0.0029). There are three mortalities in group Awhereas one mortality in group B. This difference in mortality in two groups is not statistically significant. DOI: http://dx.doi.org/10.3329/bjms.v11i4.12597 Bangladesh Journal of Medical Science Vol. 11 No. 04 Oct12

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Triana, Miryam, Rosario Madero, Steven Abrams, Leopoldo Martinez, Maria Amesty, Miguel Pipaón und Carlos Zozaya. „Predicting Full Enteral Feeding in the Postoperative Period in Infants with Congenital Diaphragmatic Hernia“. European Journal of Pediatric Surgery 27, Nr. 05 (12.01.2017): 431–36. http://dx.doi.org/10.1055/s-0036-1597655.

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Introduction The objective of the study is to examine the factors associated with time to achieve full enteral feeding after repair of congenital diaphragmatic hernia. Materials and Methods Demographic, clinical, and therapeutic data were retrospectively assessed, and uni- and multivariate Cox regression were performed to examine factors predictive of achieving full enteral feeding that was defined as time to achieve120 mL/kg/d after surgical repair. Results Of 78 infants, 66 underwent intervention before hospital discharge. All infants who survived had reached full enteral feeding at the time of hospital discharge by a median of 22 days (range: 2–119 days) after surgery and 10 days (range: 1–91) after initiation of postoperative enteral feedings. Independent risk factors associated with a longer time to reach full enteral feeding achievement included gastroesophageal reflux and days of antibiotics in the postoperative period. Daily stool passage preoperatively predicted earlier enteral tolerance. Conclusion Infants who survive congenital diaphragmatic hernia generally are able to achieve full enteral feedings after surgical repair. A longer time to full feeding is needed in the most severe cases, but some specific characteristics can be used to help identify patients at higher risk. Although some of these characteristics are unavoidable, others including rational antibiotic usage and active gastroesophageal reflux prevention and treatment are feasible and may improve enteral tolerance.

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Silk, D. B. A. „Formulation of enteral diets for use in jejunal enteral feeding“. Proceedings of the Nutrition Society 67, Nr. 3 (23.05.2008): 270–72. http://dx.doi.org/10.1017/s0029665108007155.

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Nasogastric enteral feeding is not tolerated in patients with gastric atony and in many critically-ill patients in whom gastric emptying may be delayed and in whom gastro-oesophageal regurgitation may lead to pulmonary aspiration of enteral feed and the development of pneumonia. Initial attempts to overcome these problems led to the development of post pyloric enteral feeding techniques with the infusion port of the tubes positioned in the duodenum. In many centres this technique is still the most practised post-pyloric enteral feeding technique. Nasoduodenal feeding tubes often retroperistalse into the stomach. The technique of choice, therefore, in these difficult patients is to position the infusion port of the feeding tube well distal to the ligament of trietz (post ligament of trietz nasojejunal enteral tube feeding). While nasogastric and nasoduodenal enteral feeding techniques have been shown to elicit a stimulatory exocrine pancreatic response, distal jejunal enteral feeding does not. During this mode of feeding the ileal brake is activated and pancreatic exocrine pancreatic secretion inhibited by the action of the released peptide YY and glucagon-like peptide-1 hormones, in turn the inhibition of pancreatic secretion being the result of inhibition of trypsin secretion. In the light of the findings showing the absence of a stimulatory pancreatic exocrine response to nasojejunal enteral feeding these patients should receive a predigested rather than a polymeric enteral diet.

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Bjornvad, Charlotte R., Thomas Thymann, Nicolaas E. Deutz, Douglas G. Burrin, Søren K. Jensen, Bent B. Jensen, Lars Mølbak et al. „Enteral feeding induces diet-dependent mucosal dysfunction, bacterial proliferation, and necrotizing enterocolitis in preterm pigs on parenteral nutrition“. American Journal of Physiology-Gastrointestinal and Liver Physiology 295, Nr. 5 (November 2008): G1092—G1103. http://dx.doi.org/10.1152/ajpgi.00414.2007.

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Preterm neonates have an immature gut and metabolism and may benefit from total parenteral nutrition (TPN) before enteral food is introduced. Conversely, delayed enteral feeding may inhibit gut maturation and sensitize to necrotizing enterocolitis (NEC). Intestinal mass and NEC lesions were first recorded in preterm pigs fed enterally (porcine colostrum, bovine colostrum, or formula for 20–40 h), with or without a preceding 2- to 3-day TPN period ( n = 435). Mucosal mass increased during TPN and further after enteral feeding to reach an intestinal mass similar to that in enterally fed pigs without TPN (+60–80% relative to birth). NEC developed only after enteral feeding but more often after a preceding TPN period for both sow's colostrum (26 vs. 5%) and formula (62 vs. 39%, both P < 0.001, n = 43–170). Further studies in 3-day-old TPN pigs fed enterally showed that formula feeding decreased villus height and nutrient digestive capacity and increased luminal lactic acid and NEC lesions, compared with colostrum (bovine or porcine, P < 0.05). Mucosal microbial diversity increased with enteral feeding, and Clostridium perfringens density was related to NEC severity. Formula feeding decreased plasma arginine, citrulline, ornithine, and tissue antioxidants, whereas tissue nitric oxide synthetase and gut permeability increased, relative to colostrum (all P < 0.05). In conclusion, enteral feeding is associated with gut dysfunction, microbial imbalance, and NEC in preterm pigs, especially in pigs fed formula after TPN. Conversely, colostrum milk diets improve gut maturation and NEC resistance in preterm pigs subjected to a few days of TPN after birth.

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Martin, Leigh, und Susanne Cox. „Enteral feeding: practice guidance“. Paediatric Nursing 12, Nr. 1 (01.02.2000): 28–33. http://dx.doi.org/10.7748/paed.12.1.28.s21.

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&NA;. „Levodopa/enteral feeding interaction“. Reactions Weekly &NA;, Nr. 1323 (Oktober 2010): 26–27. http://dx.doi.org/10.2165/00128415-201013230-00087.

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35

Bingham, Hal G. „Enteral feeding in sepsis“. Plastic and Reconstructive Surgery 78, Nr. 1 (Juli 1986): 138. http://dx.doi.org/10.1097/00006534-198607000-00092.

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36

Rombeau, John L., Michael D. Caldwell und Russell J. Merritt. „Enteral and Tube Feeding“. Journal of Pediatric Gastroenterology and Nutrition 4, Nr. 3 (Juni 1985): 510. http://dx.doi.org/10.1097/00005176-198506000-00036.

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Tisherman, Samuel A., Paul E. Marik und Juan Ochoa. „Promoting enteral feeding 101 *“. Critical Care Medicine 30, Nr. 7 (Juli 2002): 1653–54. http://dx.doi.org/10.1097/00003246-200207000-00044.

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38

Shatz, David V., Ernest F. J. Block und Mark Kligman. „ALTERNATIVE IN ENTERAL FEEDING“. Southern Medical Journal 86, Supplement (September 1993): 120. http://dx.doi.org/10.1097/00007611-199309001-00343.

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Volicer, Ladislav. „Vignette on Enteral Feeding“. Journal of the American Geriatrics Society 41, Nr. 6 (Juni 1993): 687–88. http://dx.doi.org/10.1111/j.1532-5415.1993.tb06748.x.

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40

Mohler, Pamela A. „Phenytoin and Enteral Feeding“. Journal of Parenteral and Enteral Nutrition 16, Nr. 1 (Januar 1992): 95–96. http://dx.doi.org/10.1177/014860719201600195.

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Fang, John. „Appendix-Enteral Feeding Devices“. Journal of Parenteral and Enteral Nutrition 30, Nr. 1_suppl (Januar 2006): S96—S97. http://dx.doi.org/10.1177/01486071060300s1s96.

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Lau, Mary Theresa, und Joanne ,. Girard. „Ensuring safer enteral feeding“. Nursing Management (Springhouse) 42, Nr. 12 (Dezember 2011): 39–43. http://dx.doi.org/10.1097/01.numa.0000407579.93705.e9.

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43

Moxness, Karen. „Enteral and Tube Feeding“. Mayo Clinic Proceedings 60, Nr. 5 (Mai 1985): 360. http://dx.doi.org/10.1016/s0025-6196(12)60557-6.

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Kaminski, Mitchell V. „Enteral and Tube Feeding“. Journal of the American College of Nutrition 10, Nr. 4 (August 1991): 396. http://dx.doi.org/10.1080/07315724.1991.10738182.

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Kozlowski, Julie. „Enteral Feeding Tube Technique“. AJN, American Journal of Nursing 110, Nr. 2 (Februar 2010): 13. http://dx.doi.org/10.1097/01.naj.0000368030.64212.9b.

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46

Lender, Menahem. „Enteral and tube feeding“. Gastroenterology 88, Nr. 3 (März 1985): 851. http://dx.doi.org/10.1016/0016-5085(85)90170-2.

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Burdall, Oliver C., Lucy J. Howarth, Alison Sharrard und Alex C. H. Lee. „Paediatric enteral tube feeding“. Paediatrics and Child Health 27, Nr. 8 (August 2017): 371–77. http://dx.doi.org/10.1016/j.paed.2017.05.001.

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48

Evans, S., A. MacDonald und C. Holden. „Home enteral feeding audit“. Journal of Human Nutrition and Dietetics 17, Nr. 6 (Dezember 2004): 537–42. http://dx.doi.org/10.1111/j.1365-277x.2004.00569.x.

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Baxter, Janet P. „Home care enteral feeding“. Journal of Human Nutrition and Dietetics 18, Nr. 4 (August 2005): 319. http://dx.doi.org/10.1111/j.1365-277x.2005.00628.x.

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OʼRourke, Kevin. „Enteral feeding after Cruzan“. Topics in Clinical Nutrition 6, Nr. 3 (Juli 1991): 68–71. http://dx.doi.org/10.1097/00008486-199106000-00010.

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Zeitschriftenartikel zum Thema „Enteral feeding“

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