Thematische Bibliographien / Endocrine and paracrine regulations

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Auswahl der wissenschaftlichen Literatur zum Thema „Endocrine and paracrine regulations“

Autor: Grafiati

Veröffentlicht am 7. Dezember 2024

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Zeitschriftenartikel zum Thema "Endocrine and paracrine regulations"

Martin, Thomas John. „Endocrine and paracrine regulation of bone“. Future Rheumatology 3, Nr. 2 (April 2008): 117–19. http://dx.doi.org/10.2217/17460816.3.2.117.

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TAYLOR, ROBERT N., DAN I. LEBOVIC, DANIELA HORNUNG und MICHAEL D. MUELLER. „Endocrine and Paracrine Regulation of Endometrial Angiogenesis“. Annals of the New York Academy of Sciences 943, Nr. 1 (September 2001): 109–21. http://dx.doi.org/10.1111/j.1749-6632.2001.tb03795.x.

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SAEZ, JOSÉ M. „Leydig Cells: Endocrine, Paracrine, and Autocrine Regulation“. Endocrine Reviews 15, Nr. 5 (Oktober 1994): 574–626. http://dx.doi.org/10.1210/edrv-15-5-574.

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Salustri, Antonietta, Antonella Camaioni und Cristina D'Alessandris. „Endocrine and paracrine regulation of cumulus expansion“. Zygote 4, Nr. 04 (November 1996): 313–15. http://dx.doi.org/10.1017/s0967199400003312.

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In a Graafian follicle, granulosa cells are classified into two principal cell subpopulations: cumulus cells, which are closely associated with the oocyte to form the cumulus cell-oocyte complex (COC), and mural granulosa cells, which are organised as a stratified epithelium at the periphery of the follicle. Following the preovulatory gonadotropin surge, cumulus cells lose contact with mural granulosa cells and start to synthesise and secrete a large amount of hyaluronan (HA), a glycosaminoglycan with high molecular weight and large hydrodynamic domains (Salustriet al., 1992). Proteins derived from serum (Chenet al., 1992, 1994) and synthesised by cumulus cells (Camaioniet al., 1993, 1996) organise the strands of HA into an intercellular elastic network that traps the cumulus cells and the oocyte in a unit which can not be mechanically dissociated – a process also referred to as cumulus expansion. At ovulation, the expanded COC is released through the ruptured follicle wall and transferred to the oviduct. The matrix in the expanded COC facilitates its extrusion from the follicle and its capture by oviductal fimbria, and provides, together with the cumulus cells, a suitable microenvironment for sperm penetration and fertilisation (for references see Salustriet al., 1993).

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Sims, Natalie A., und Ling Yeong Chia. „Regulation of Sclerostin Expression by Paracrine and Endocrine Factors“. Clinical Reviews in Bone and Mineral Metabolism 10, Nr. 2 (16.11.2011): 98–107. http://dx.doi.org/10.1007/s12018-011-9121-7.

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Carbillon, Lionel, Michele Uzan, Jean-Claude Challier, Philippe Merviel und Serge Uzan. „Fetal-Placental and Decidual-Placental Units: Role of Endocrine and Paracrine Regulations in Parturition“. Fetal Diagnosis and Therapy 15, Nr. 5 (2000): 308–18. http://dx.doi.org/10.1159/000021027.

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Biswas, Subhasri, Urmi Mukherjee und Sudipta Maitra. „Endocrine disruption and female reproductive health: Implications on cross-talk between endocrine and autocrine/paracrine axes in the ovary“. Journal of Reproductive Health and Medicine 3 (10.11.2020): 2. http://dx.doi.org/10.25259/jrhm_11_2020.

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Female reproduction is a blend of neuroendocrine, endocrine, and autocrine/paracrine factors that maintain the appropriate ovarian micro-environment. The growing urbanization prompted exposure to a myriad of environmental toxins carrying the ability to interfere with reproductive processes governed by endogenous hormones, making reproductive health a major global concern. These environmental anthropogenic contaminants, popularly termed as endocrine-disrupting chemicals (EDCs), can disrupt the ovarian homeostasis leading to serious perturbations, namely, anovulation, infertility, estrogen deficiency, and premature ovarian failure. Although gonadotropin action, biosynthesis of gonadal steroids vis-à-vis growth factors comprise the essential modulators within the ovary, the redox balance along with inflammatory and cell death response can dramatically influence the framework of ovarian dynamics; however, details of which remain relatively less understood. The present overview provides an update on candidates (endocrines and autocrine/paracrine) of oogenesis, and the potential impact of EDCs on diverse intra-ovarian entities including but not limited to gonadotropin action, steroidogenic potential, expression of growth factors, and modulation of maturational competence. Moreover, the relative importance of free radical-induced stress, inflammation, and elevated cell death (follicular atresia), in the regulation of ovarian functions and how these intricate yet conjoined mechanisms may alter the reproductive performance of a female will be an issue of discussion.

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Polidoro, Juliano Z., Weverton M. Luchi, Antonio Carlos Seguro, Gerhard Malnic und Adriana C. C. Girardi. „Paracrine and endocrine regulation of renal K⁺ secretion“. American Journal of Physiology-Renal Physiology 322, Nr. 3 (01.03.2022): F360—F377. http://dx.doi.org/10.1152/ajprenal.00251.2021.

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The seminal studies conducted by Giebisch and coworkers in the 1960s paved the way for understanding the renal mechanisms involved in K+ homeostasis. It was demonstrated that differential handling of K+ in the distal segments of the nephron is crucial for proper K+ balance. Although aldosterone had been classically ascribed as the major ion transport regulator in the distal nephron, thereby contributing to K+ homeostasis, it became clear that aldosterone per se could not explain the ability of the kidney to modulate kaliuresis in both acute and chronic settings. The existence of alternative kaliuretic and antikaliuretic mechanisms was suggested by physiological studies in the 1980s but only gained form and shape with the advent of molecular biology. It is now established that the kidneys recruit several endocrine and paracrine mechanisms for adequate kaliuretic response. These mechanisms include the direct effects of peritubular K+, a gut-kidney regulatory axis sensing dietary K+ levels, the kidney secretion of kallikrein during postprandial periods, the upregulation of angiotensin II receptors in the distal nephron during chronic changes in K+ diet, and the local increase of prostaglandins by low-K+ diet. This review discusses recent advances in the understanding of endocrine and paracrine mechanisms underlying the modulation of K+ secretion and how these mechanisms impact kaliuresis and K+ balance. We also highlight important unknowns about the regulation of renal K+ excretion under physiological circumstances.

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Challis, J. R. G. „Endocrine and Paracrine Regulation of Birth at Term and Preterm“. Endocrine Reviews 21, Nr. 5 (01.10.2000): 514–50. http://dx.doi.org/10.1210/er.21.5.514.

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Challis, John R. G., Stephen G. Matthews, William Gibb und Stephen J. Lye. „Endocrine and Paracrine Regulation of Birth at Term and Preterm*“. Endocrine Reviews 21, Nr. 5 (01.10.2000): 514–50. http://dx.doi.org/10.1210/edrv.21.5.0407.

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Abstract We have examined factors concerned with the maintenance of uterine quiescence during pregnancy and the onset of uterine activity at term in an animal model, the sheep, and in primate species. We suggest that in both species the fetus exerts a critical role in the processes leading to birth, and that activation of the fetal hypothalamic-pituitary-adrenal axis is a central mechanism by which the fetal influence on gestation length is exerted. Increased cortisol output from the fetal adrenal gland is a common characteristic across animal species. In primates, there is, in addition, increased output of estrogen precursor from the adrenal in late gestation. The end result, however, in primates and in sheep is similar: an increase in estrogen production from the placenta and intrauterine tissues. We have revised the pathway by which endocrine events associated with parturition in the sheep come about and suggest that fetal cortisol directly affects placental PGHS expression. In human pregnancy we suggest that cortisol increases PGHS expression, activity, and PG output in human fetal membranes in a similar manner. Simultaneously, cortisol contributes to decreases in PG metabolism and to a feed-forward loop involving elevation of CRH production from intrauterine tissues. In human pregnancy, there is no systemic withdrawal of progesterone in late gestation. We have argued that high circulating progesterone concentrations are required to effect regionalization of uterine activity, with predominantly relaxation in the lower uterine segment, allowing contractions in the fundal region to precipitate delivery. This new information, arising from basic and clinical studies, should further the development of new methods of diagnosing the patient at risk of preterm labor, and the use of scientifically based strategies specifically for the management of this condition, which will improve the health of the newborn.

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Dissertationen zum Thema "Endocrine and paracrine regulations"

Jeanne, Fabian. „Evοlutiοns des systèmes GΝRΗ et des hοrmοnes glycοprοtéiques dans les cοntrôles endοcrine et paracrine de la spermatοgénèse chez la rοussette, Scyliοrhinus canicula“. Electronic Thesis or Diss., Normandie, 2024. http://www.theses.fr/2024NORMC225.

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La spermatogenèse est un processus hautement spécialisé de prolifération et de différenciation cellulaire conduisant à la production de spermatozoïdes haploïdes à partir de cellules souches spermatogoniales diploïdes. Chez les Gnathostomes, les fonctions testiculaires sont principalement contrôlées de façon endocrine par l’axe hypothalamo-hypophyso-gonadique (HHG) impliquant notamment les GnRHs hypothalamiques et les hormones gonadotropes, FSH et LH, dont l’émergence s’est produite à la racine des Vertébrés cartilagineux. De plus, des fonctions paracrines des GnRHs et de la thyrostimuline ont été explorées au niveau gonadique chez des Vertébrés osseux. L’objet de cette thèse était de caractériser les régulations endocrines et paracrines de la spermatogenèse médiées par les GnRHs et les hormones gonadotropes chez un modèle Élasmobranche, la roussette Scyliorhinus canicula. Ce travail a été étendu à la caractérisation de GPA2 et GPB5, constituant la thyrostimuline, qui correspondent aux orthologues des ancêtres moléculaires des sous-unités des hormones glycoprotéiques. Dans ce travail, l’évolution du protéome testiculaire au cours de la spermatogenèse de S. canicula a été décrite, les neuropeptides GnRHs, les hormones glycoprotéiques FSH, LH, TSH, GPA2 et GPB5, et leurs récepteurs associés ont fait l’objet d’analyses in silico et d’expressions dans différents tissus avec un focus au cours de la spermatogenèse. Il a été observé une expression à tous les stades de la spermatogenèse de fshr, lhr et des récepteurs aux GnRHs au niveau germinale et sertolien, de tshr et gpb5 au niveau sertolien et de gpa2 au niveau germinal, avec des abondances plus importantes associées aux stades spermatides. Ce travail a été complété par des tests fonctionnels in vitro qui ont montré que FSHR pouvait être activé par FSH et LH, LHR uniquement par LH, et que GPB5-GPA2 pouvait activer les trois récepteurs FSHR, LHR et TSHR, suggérant ainsi un rôle paracrine de la thyrostimuline au niveau testiculaire. L’ensemble de ce travail permet de proposer un modèle de régulation de la spermatogenèse chez les Élasmobranches qui associe des hormones endocrines, avec les GnRHs et gonadotropines circulantes, et des hormones paracrines, avec GPA2, GPB5 et les stéroïdes. Ce modèle apparaît cohérent et intermédiaire dans l’évolution des systèmes régulateurs de la spermatogenèse qui seraient passés d’une régulation paracrine prépondérante chez les Bilatériens non-Vertébrés à une régulation endocrine prépondérante chez les Vertébrés osseux avec la mise en place de l’axe hypothalamo-hypophyso-gonadique
Spermatogenesis is a highly specialized process of cell proliferation and differentiation leading to the production of haploid spermatozoa from diploid spermatogonial stem cells. In Gnathostomes, testicular functions are mainly controlled by the endocrine hypothalamic-pituitary-gonadal (HHG) axis, involving hypothalamic GnRHs and the gonadotropic hormones FSH and LH, which emerged at the root of cartilaginous vertebrates. In addition, paracrine functions of GnRHs and thyrostimulin have been explored at the gonadic level in bony vertebrates. The aim of this thesis was to characterize the endocrine and paracrine regulation of spermatogenesis exerted by GnRHs and gonadotropic hormones in an Elasmobranch model, the catshark Scyliorhinus canicula. This work has been extended to the characterization of GPA2 and GPB5, constituting thyrostimulin, which correspond to orthologs of the molecular ancestors of glycoprotein hormone subunits. In this work, the evolution of the testicular proteome during spermatogenesis in S. canicula was described, and the neuropeptides GnRHs, the glycoprotein hormones FSH, LH, TSH, GPA2 and GPB5, as well as their associated receptors were studied by in silico and expression analyses in different tissues, with a focus on spermatogenesis. Expressions were observed at all stages of spermatogenesis for fshr, lhr and GnRH receptors associated at germinal and sertolian levels, for tshr and gpb5 at sertolian level and for gpa2 at germinal level, with higher abundances associated with spermatid stages. This work was complemented by in vitro functional tests which showed that FSHR could be activated by FSH and LH, LHR only by LH, and that GPB5-GPA2 could activate all three receptors FSHR, LHR and TSHR, suggesting a paracrine role for thyrostimulin at the testicular level. Taken together, this work proposes a model for the regulation of spermatogenesis in Elasmobranchs that combines endocrine hormones, with circulating GnRHs and gonadotropins, and paracrine hormones, with GPA2, GPB5 and steroids. This model appears consistent and intermediate in the evolution of spermatogenesis regulating systems, which has shift from predominantly paracrine regulation in non-vertebrate bilaterians to predominantly endocrine regulation in bony vertebrates, with the establishment of the hypothalamic-pituitary-gonadal axis

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Cabrera, Over. „Autocrine/paracrine interactions modulating hormone release in the endocrine pancreas /“. Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-378-8/.

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Myers, Michelle. „In vitro modelling of paracrine and endocrine interactions in the human ovary“. Thesis, University of Edinburgh, 2008. http://hdl.handle.net/1842/29292.

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Women with regular cycles having hysterectomy for non-malignant conditions and women undergoing oocyte retrieval for assisted conception were used in the current study. Novel primary cultures and co-cultures of lutinised granulosa cells and fibroblast-like cells allowed the mechanistic in vivo interactions of the corpus luteum to be mimicked with an in vitro system. Herein, activin A is identified as a regulated molecule that may promote tissue remodelling during luteolysis. Activin A is secreted by luteal steroidogenic cells and at physiological concentrations it up regulates MMP-2 activity and expression in luteal fibroblast-like cells. HCG can inhibit activin A through several mechanisms including up regulating activin inhibitors, inhibin A and follistatin. Results suggest that activin is an excellent anti-luteal molecule whose paracrine/endocrine actions are to remove or potentially inhibit luteal tissue formation, and moreover to facilitate human luteolysis. However, the biological actions of activin A are inhibited during maternal recognition of pregnancy in the presence of conceptus-derived hCG which has marked and disparate changes on surrounding cell types that do not expression the hCG receptor. Consequently, luteolysis is presented, such that luteal fibroblast-like MMP-2 is inhibited as hCG-derived cortisol production promotes the maternal recognition of pregnancy.

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Cadranel, Jacques. „1,25 (OH) 2d3 médiateur endocrine et paracrine au cours des granulomatoses pulmonaires“. Paris 5, 1993. http://www.theses.fr/1993PA05CD03.

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Dans ce travail, nous montrons tout d'abord une production de 1,25 (OH)2D3 par les cellules pulmonaires fraîchement recueillies par lavage bronchoalvéolaire (LBA) chez des sujets tuberculeux hypercalcémiques (annexe I). Ensuite, après avoir constaté une corrélation entre les capacités de production de la 1,25 (OH)2D3 par ces cellules alvéolaires et les anomalies du bilan phosphocalciques (annexe II), nous avons identifié les cellules responsables de cette production chez des sujets tuberculeux non hypercalcémiques (annexe III). Ainsi, nous avons montré qu'une activité 25(OH)D3-1-a-hydroxylase est toujours retrouvée dans les macrophages T pulmonaires alors qu'elle est inconstante dans les macrophages et paraît absente ou considérablement réduite dans les cellules circulantes. Parallèlement, nous avons montré qu'une fraction des lymphocytes T alvéolaires expriment le récepteur de la 1,25 (OH)2D3 au site de la tuberculose, récepteur absent des lymphocytes circulants (annexe IV). La poursuite de notre recherche dans ce domaine vise à apprécier les interactions entre la 1,25 (OH)2D3 et les cellules (lymphocytes, monocytes) produisant des médiateurs impliqués dans la formation et la régulation des activités du granulome (TNFa, PGE2, IGF1, gIFN. . . ) (annexes V-VII).

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Amer, Shehla. „Endocrine and paracrine control of renal function in the in situ perfused trunk of the rainbow trout“. Thesis, University of Exeter, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261617.

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Cullon, Donna Lynn. „Evidence of endocrine disruption in neogastropods by organotins, 10 years after the 1989 regulations“. Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ62012.pdf.

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Schmitt, Matthias. „Endocrine and paracrine aspects of vascular control : the effects of natriuretic peptides on human capacitance in health and chronic heart failure“. Thesis, Cardiff University, 2004. http://orca.cf.ac.uk/55541/.

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The effects of natriuretic peptides on vascular control were investigated. In the major part of this thesis emphasis is placed on the effects of natriuretic peptides, in particular ANP, on regulation of regional vascular volume and venous tone in healthy volunteers (Chapter 3) and patients with chronic heart failure (Chapter 4). The second part of this thesis investigates the effects and mechanisms of action of ANP, BNP and CNP on large artery function in an ovine hind limb model (Chapter 5). Finally, the actions of the latest members of the natriuretic peptide family, namely DNP and NNP are investigated in vitro using rings of rabbit aorta in organ bath experiments (Chapter 6). The important new findings of this thesis are 1. ANP regulates regional vascular volume and venous tone over a wide range of physiological and pathophysiological plasma levels without affecting compliance. 2. Most importantly, basal ANP plasma levels contribute significantly to regulation of resting vascular tone. 3. The rank order of potency of NP in the forearm capacitance vasculature of patients with chronic heart failure is ANP BNP>CNP/Urodilatin. 4. Venous ANP responsiveness is preserved in patients with chronic heart failure despite marked impairment in the resistance vasculature. This preservation may be due to preserved venous endothelial function. 5. ANP acting locally modifies pulse wave velocity via the NPRa receptor. Neither CNP (acting via the NPRb receptor) nor cANF (acting via NPRc) elicit any immediate vasoactive effects. 6. Novel natriuretic peptide (NNP), a newly isolated NP from the venom of the green mamba snake (Dendroaspis angusepticus) has arterial vasorelaxant properties similar to those of ANP and DNP.

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Bainbridge, David Robert James. „Endocrine regulations of luteal regression and maternal recognition of pregnancy in the Red Deer (Cervus elaphus)“. Thesis, Royal Veterinary College (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522164.

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Lafont, Anne-Gaëlle. „Caractérisation et rôle fonctionnel d'une famille de peptides calciotropes (CT et CGRP) : nouvelles données chez un téléostéen et deux mollusques céphalopodes“. Paris 6, 2006. http://www.theses.fr/2006PA066190.

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Afin de mieux comprendre leur histoire évolutive, nous avons recherché la présence de la CT, du CGRP et de leurs organes cibles, chez un téléostéen, Anguilla anguilla, et deux céphalopodes, Nautilus macromphalus et Sepia officinalis. Contrairement à l’anguille, aucune molécule biologiquement apparentée à la CT n’a été détectée chez les céphalopodes. La co-localisation du CGRP et de son récepteur dans le système nerveux central de l’anguille et de la seiche, suggère que ce peptide pourrait jouer un rôle de neuromédiateur ou neurotransmetteur comme décrit chez les mammifères. L’implication du CGRP dans les mécanismes de régulation ionique, de façon endocrine dans les branchies et autocrine/paracrine dans le système rénal, représenterait une fonction ancienne, partagée par les téléostéens et les céphalopodes. Il semble que le CGRP ait une origine ancestrale intervenue avant l’émergence des deutérostomiens. La CT constituerait un peptide apparu plus tardivement au cours de l’évolution.

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Schote-Frese, Andrea [Verfasser]. „Nuclear receptors: variants and their role in neuro-endocrine immune regulations / vorgelegt von Andrea Schote-Frese“. 2009. http://d-nb.info/992981875/34.

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Bücher zum Thema "Endocrine and paracrine regulations"

Reproductive Endocrinology Study Section Workshop on Autocrine and Paracrine Mechanisms in Reproductive Endocrinology (1988 Shrewsbury, Mass.). Autocrine and paracrine mechanisms in reproductive endocrinology. New York: Plenum Press, 1989.

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Nikula, Hannu. Endocrine and paracrine modulation of gonadotropin action in the rat testis. Turku: Turun Yliopisto, 1990.

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Walter, Helen Jane. The spatio-temporal modulation of the insulin-like growth factor[s] axis in the lesioned central nervous system: Implications for endocrine, paracrine and autocrine activities. Birmingham: University of Birmingham, 1996.

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F, Piva, und International Symposium on "Cell to Cell Communication in Endocrinology" (1987 : Florence, Italy), Hrsg. Cell to cell communication in endocrinology. New York: Raven Press, 1988.

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Ezrin, Calvin. The endocrine control diet: How to beat the metabolic trap and lose weight permanently. New York: Harper & Row, 1990.

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1927-, Assenmacher Ivan, Boissin Jean und Centre d'études biologiques des animaux sauvages (France), Hrsg. Endocrine regulations as adaptive mechanisms to the environment =: Régulations endocriniennes et adaptations à l'environnement : Colloque international, Centre d'études biologiques des animaux sauvages, Forêt de Chizé, 1er-5 juillet 1985. Paris: Editions du Centre national de la recherche scientifique, 1986.

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Guraya, Sardul S. Comparative Cellular and Molecular Biology of Testis in Vertebrates: Trends in Endocrine, Paracrine and Autocrine Regulation of Structure and Functions. Science Publishers, 2001.

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Goldberg, Erwin, Barry Zirkin, Vassilios Papadopoulos und Polina V. Lishko, Hrsg. Endocrine and Paracrine Regulation of Spermatogenesis - A Collection of Up to Date Research Contributions on Testis Formation and Function. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-859-2.

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Krey, L. Autocrine and Paracrine Mechanisms in Reproductive Endocrinology. Springer London, Limited, 2012.

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Krey, L. Autocrine and Paracrine Mechanisms in Reproductive Endocrinology. Springer London, Limited, 2013.

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Buchteile zum Thema "Endocrine and paracrine regulations"

Welle, Stephen. „Endocrine, Paracrine, and Autocrine Regulation“. In Human Protein Metabolism, 124–60. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4612-1458-8_6.

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Setchell, B. P., T. Hertel und O. S�der. „Postnatal Testicular Development, Cellular Organization and Paracrine Regulation“. In Endocrine Development, 24–37. Basel: KARGER, 2003. http://dx.doi.org/10.1159/000069295.

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Dayton, William R., und Marcia R. Hathaway. „Autocrine, Paracrine, and Endocrine Regulation of Myogenesis“. In Animal Growth Regulation, 69–90. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-8872-2_4.

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Huhtaniemi, Ilpo, und Jorma Toppari. „Endocrine, Paracrine and Autocrine Regulation of Testicular Steroidogenesis“. In Advances in Experimental Medicine and Biology, 33–54. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-0952-7_3.

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Habibi, Hamid R. „Gonadotropin-Releasing Hormone as a Paracrine Regulator of Ovarian Function“. In Neural Regulation in the Vertebrate Endocrine System, 101–10. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4805-8_7.

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Sharpe, Richard M., Michael R. Millar, Simon Maddocks und Jacqui Clegg. „Transport Mechanisms for Endocrine and Paracrine Factors in the Testis“. In Cellular and Molecular Regulation of Testicular Cells, 249–59. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-2374-0_16.

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Hess, Rex A., Qing Zhou und Rong Nie. „The Role of Estrogens in the Endocrine and Paracrine Regulation of the Efferent Ductules, Epididymis and Vas Deferens“. In The Epididymis: From Molecules to Clinical Practice, 317–37. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0679-9_18.

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Tota, B., und M. C. Cerra. „The Endocrine–Paracrine Control of the Cardiovascular System“. In Cardio-Respiratory Control in Vertebrates, 317–79. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-93985-6_14.

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Stouffer, Richard L. „Endocrine, Paracrine, and Autocrine Regulators of the Macaque Corpus Luteum“. In Signaling Mechanisms and Gene Expression in the Ovary, 68–83. New York, NY: Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4612-3200-1_6.

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Gomez, F. „Effects of Estrogens on Various Endocrine Regulations“. In Estrogens and Antiestrogens I, 379–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-58616-3_20.

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Konferenzberichte zum Thema "Endocrine and paracrine regulations"

Nevdakh, A. S., und T. V. Dashkevich. „ENSURING RADIATION SAFETY DURING RADON THERAPY PROCEDURES“. In SAKHAROV READINGS 2022: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2022. http://dx.doi.org/10.46646/sakh-2022-2-258-261.

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Radon therapy is a type of medical treatment using natural radon gas in certain amounts to relieve chronic pain and fatigue. Therapeutic dosages of radon procedures should be in the range between the minimum effective and maximum permissible. This is necessary for effective treatment and at the same time to ensure the radiation safety of patients. Alpha radiation has a direct effect on the cells of the barrier, as well as other organs, and through the irritation of various receptors in these organs also indirectly, due to the activation of the central links of the nervous, endocrine and immune systems. Individual control of radiation doses is mandatory for the personnel supervising the procedure. Personnel must know and strictly follow the instructions, safety regulations, radiation safety standards, fire safety and industrial sanitation.

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Schiele, Nathan R., Douglas B. Chrisey und David T. Corr. „Novel Method of Laser Direct Writing for Precise Patterning of Human Dermal Fibroblasts“. In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19675.

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The ability to control a cell’s location, pattern geometry, and proximity to neighboring cells, in vitro, is highly desired to gain insight into cell-cell interactions, such as the modes of cellular signaling (direct cell contact, paracrine, or endocrine). A laser-based cell patterning technique, laser direct write, enables the precise spatial placement of living cells, with all the advantages of CAD/CAM control [1]. However, this technique is limited in usefulness due to the dependence on Matrigel® (BD Biosciences, Bedford, MA). The growth factor constituents of Matrigel® may interfere with many cellular processes under investigation and may preclude or greatly limit the utility of laser direct writing for precise cell cultures [2]. Therefore, to address this limitation, the objective of this study was to develop a Matrigel®-free laser direct writing method. Through the use of customized gelatin coatings on both the ribbon and receiving substrate, we effectively adapted the direct write technique to precisely pattern cells without the use of Matrigel®, as demonstrated with human dermal fibroblasts. The gelatin partially encapsulates the trypsinized cells on the ribbon, providing a volitization zone to protect the cells, and on the receiving substrate cushions the impact of transfer while maintaining moisture. Gelatin liquefies at 37°C, which allows it to be removed from the growth surface ensuring cellular proliferation, uninhibited by growth surface treatments. This represents a fundamental change from the original direct write technique in which cells must first form initial attachments to the ribbon via Matrigel® and then are written to a Matrigel® coated receiving substrate for their sustained growth. Additionally, we have developed a method to monitor the location of the patterned cells post-transfer to show that a gelatin coated-receiving substrate is effective as a patterning surface and ensures the registry of the pattern until cell attachment, even after the gelatin has been removed with the first growth medium application. This precise patterning technique can now be used in many biomedical applications, including those that involve cell types highly sensitive to growth factors, such as stem cells and cancer cells.

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Kinaci, Emre, Giuseppe Palmese, Joseph Stanzione und Sarah Salazar. „Evaluation of hybridized bio-based building blocks as coating materials“. In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/oyjf3899.

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Commercial epoxy resins are often the oligomeric reaction products of bisphenol A (BPA) with epichlorohydrin. BPA is a toxic petrochemical and a well-known endocrine disruptor which can mimic the bodyâ€™s own hormones and may lead to several negative health effects. The most common curing agents are also the derivatives of toxic amines such as aniline, ethylene di-amine, and ammonia etc. which are under strict regulations and restrictions by TSCA and (REACH) agencies. Therefore; there is a growing demand to develop epoxy and amine systems from non- or less- toxic and annually renewable precursors derived from bio-mass to replace depleting petrochemicals. Within this context, cardanol molecule is combined with vanillyl alcohol through electrophilic substitution reaction to synthesize a bis-phenol type resin (VAC). Di-glycidyl ether of the VAC (EVAC) is mixed with di-glycidyl ether of bisphenol A (DGEBA) at varying ratios and cured with di-furan di-amine (DFDA) which is previously a well-studied bio-based diamine derived from furfurylamine. Thermally cured blends demonstrated improved modulus and char yields relative to aliphatic and cyloaliphatic amines due the furanic nature of DFDA as demonstrated via thermo-physical and mechanical tests. Although, the replacement of EVAC with DGEBA resulted in a reduction in glass transition temperature (Tg) and the modulus of the network, desired flexibility and toughness for coatings applications were imparted due to the aliphatic nature of the cardanol molecule. In addition, rheological studies demonstrated improved gel time and lower viscosity with increasing EVAC content along with improved vapor barrier properties. These bio-derived epoxy-amine formulations demonstrated promising performance to completely or partially replace the BPA and other toxic amines in the thermoset networks for surface coating applications.

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