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Thematische Bibliographien / Element activation / Bücher

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Bücher zum Thema „Element activation“

Autor: Grafiati

Veröffentlicht am 23. Juni 2021

Zuletzt aktualisiert am 4. Februar 2022

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1

A, Bulman Robert, Cooper John R, Commission of the European Communities. Directorate-General for Science, Research, and Development. und Great Britain. National Radiological Protection Board., Hrsg. Speciation of fission and activation products in the environment. London: Elsevier Applied Science, 1986.

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2

Baryshev, Ruslan. Proactive library in the information and educational environment of the University. ru: INFRA-M Academic Publishing LLC., 2020. http://dx.doi.org/10.12737/1123649.

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In the monograph, the University library is presented as a complex system that includes elements of various properties and varying complexity. As in any system, structural change inevitably affects the performance of all its components. In this regard, the library is an element of the information and educational environment of the University, which is designed to support and improve the effectiveness of educational and scientific activities. The article reveals the concept of active University library" as a system for providing information services to the reader in any form and on any medium based on classical and network forms of service based on query advance services. The article analyzes the opportunities provided by the active University library for its users. The mechanism of activation of an electronic library through selective provision of information is considered, and the principle of the influence of an active electronic library on its proactivity is approved. For all those interested in librarianship and Informatization of education."

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3

Jikkenjo), "Kyōdairo (KUR) ni okeru Sōgōteki Biryō Genso Keisoku Shisutemu no Kōchiku to Ōyō" Senmon Kenkyūkai (2011 Kyōto Daigaku Genshiro. "Kyōdairo (KUR) ni okeru Sōgōteki Biryō Genso Keisoku Shisutemu no Kōchiku to Ōyō oyobi Hōshaka Bunseki o Mochiita Biryō Genso Bunseki no Genjō" Senmon Kenkyūkai hōkokusho. Ōsaka-fu Sennan-gun Kumatori-chō: Kyōto Daigaku Genshiro Jikkenjo, 2012.

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4

"Kyōdairo (KUR) ni okeru Sōgōteki Biryō Genso Keisoku Shisutemu no Kōchiku to Ōyō" Senmon Kenkyūkai (2010 Kyōto Daigaku Genshiro Jikkenjo). ""Kyōdairo (KUR) ni okeru Sōgōteki Biryō Genso Keisoku Shisutemu no Kōchiku to Ōyō" Senmon Kenkyūkai hōkokusho. Ōsaka-fu Sennan-gun Kumatori-chō: Kyōto Daigaku Genshiro Jikkenjo, 2011.

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5

Zaafarany, Ishaq A. Determination of trace elements in natural zeolites by neutron activation analysis (NAA). Salford: University of Salford, 1987.

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6

Poole, M. A. Effect of activator elements on the anodic behaviour ofaluminium. Manchester: UMIST, 1994.

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7

Kulm, LaVerne D. Elemental content of heavy-mineral concentrations on the Continental Shelf off Oregon and northernmost California. Portland, Or: Oregon, Dept. of Geology and Mineral Industries, 1988.

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8

Azmi, Peter B. Bacterially-derived DNA elements from the gene GPT can block enhancer-dependent transcriptional activation of an adjacent gene in a position-dependent manner. Ottawa: National Library of Canada, 2002.

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9

Heydorn. Neutron Activation Analysis for Clinical Trace Element Research. Taylor & Francis Group, 2018.

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10

Scholle, Carol Curio. Rapid Response Team Organization and Activation (DRAFT). Herausgegeben von Raghavan Murugan und Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0002.

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The Rapid Response System (RRS) is organized into four basic components. These components include an activation limb, a response limb, a quality assurance infrastructure, and an administrative component. These components remain consistent despite campus size, physical layout, patient population, available technical resources, and personnel. Oversight of the RRS is provided by the patient safety, risk management experts, as well as clinical experts to maintain high quality of care delivered to acutely ill patients. Administrative support in the development of policy, allocation of resources, and communicating a strong and clear message regarding the mission and vision of the RRS is invaluable. In this chapter, we review each element of the RRS.

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11

Heemers, Hannelore. Androgen-Induced Activation of Sterol Regulatory Element-Binding Proteins (Srebps) & Enhanced Lipogenesis in Tumor Cells. Leuven Univ Pr, 2003.

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12

Agency, International Atomic Energy, Hrsg. Handbook on nuclear activation data. Vienna: International Atomic Energy Agency, 1987.

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13

B, Alfassi Zeev, und Peisach Max, Hrsg. Elemental analysis by particle accelerators. Boca Raton: CRC Press, 1992.

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14

Badimon, Lina, Felix C. Tanner, Giovanni G. Camici und Gemma Vilahur. Pathophysiology of thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0018.

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Ischaemic heart disease and stroke are major causes of death and morbidity worldwide. Coronary and cerebrovascular events are mainly a consequence of a sudden thrombotic occlusion of the vessel lumen. Arterial thrombosis usually develops on top of a disrupted atherosclerotic plaque because of the exposure of thrombogenic material, such as collagen fibrils and tissue factor (TF), to the flowing blood. TF, either expressed by subendothelial cells, macrophage- and/or vascular smooth muscle-derived foam-cells in atherosclerotic plaques, is a key element in the initiation of thrombosis due to its ability to induce thrombin formation (a potent platelet agonist) and subsequent fibrin deposition at sites of vascular injury. Adhered platelets at the site of injury also play a crucial role in the pathophysiology of atherothrombosis. Platelet surface receptors (mainly glycoproteins) interact with vascular structures and/or Von Willebrand factor triggering platelet activation signalling events, including an increase in intracellular free Ca2+, exposure of a pro-coagulant surface, and secretion of platelet granule content. On top of this, interaction between soluble agonists and platelet G-coupled protein receptors further amplifies the platelet activation response favouring integrin alpha(IIb)beta(3) activation, an essential step for platelet aggregation. Blood-borne TF and microparticles have also been shown to contribute to thrombus formation and propagation. As thrombus evolves different circulating cells (red-blood cells and leukocytes, along with occasional undifferentiated cells) get recruited in a timely dependent manner to the growing thrombus and further entrapped by the formation of a fibrin mesh.

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15

Grom, Alexei A., und Athimalaipet V. Ramanan. Macrophage activation syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0168.

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Macrophage activation syndrome (MAS) is a life-threatening condition caused by excessive activation and proliferation of T lymphocytes and haemophagocytic macrophages. Although MAS has been reported in association with almost any rheumatic disease, it is by far most common in systemic juvenile idiopathic arthritis. Flares of the underlying disease or infection are most common triggers of MAS. The pathognomonic feature of MAS is typically found in bone marrow: numerous, well-differentiated macrophagic histiocytes phagocytosing normal haematopoietic elements. The expansion of these histiocytes leads to a massive systemic inflammatory reaction associated with three cardinal clinical features: severe cytopenias, liver dysfunction, and coagulopathy consistent with disseminated intravascular coagulation. Clinically, MAS is strikingly similar to the autosomal recessive disorders collectively known as familial haemophagocytic lymphohistiocytosis (FHLH). FHLH has been associated with various genetic defects affecting the cytolytic pathway. Cytolytic function is profoundly depressed in MAS patients as well, and this abnormality is caused by both genetic and acquired factors. Studies in animals suggest that uncontrolled expansion of activated CD8+ T lymphocytes secreting cytokines that activate macrophages is central to the pathophysiology of haemophagocytic syndromes. Consistent with this view, the combination of steroids and ciclosporin, an immunosuppressant that preferentially inhibits T lymphocytes, is an effective treatment for the majority of MAS patients. Patients in whom MAS remains active despite this treatment present a serious challenge and require more aggressive immunosuppression. However, in MAS triggered by infection, the optimal level of immunosuppression is difficult to determine. As a result, reported mortality rates reach 20%.

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16

Ebifegha, Michael Ebipa. Nuclear activation and magnetic resonance studies of body elements. 1986.

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17

Alamin, Mohamed Bachir. Bulk material elemental analysis using neutron activation techniques. 1995.

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18

Forrest, R. A., und D. A. J. Endacott. Activation Data for Some Elements Relevant to Fusion Reactors (Reports). AEA Technology Plc, 1989.

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19

Radiochemical Neutron Activation Analysis for 36 Elements in Geological Material. Washington, D.C.: National Academies Press, 1988. http://dx.doi.org/10.17226/19165.

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20

Grażyna, Domańska, Hrsg. Oznaczanie pierwiastków śladowych w geologicznych materiałach odniesienia przy zastosowaniu instrumentalnej neutronowej analizy aktywacyjnej (NAA) =: Opredelenie sledowykh ėlementov v geologicheskikh materialakh otnoshenii͡a︡ s ispolʹzovaniem metoda instrumentalʹnogo neĭtronnogo aktivat͡s︡ionnogo analiza = Determination of trace elements in geological SRM with the use of instrumental neutron activation analysis. Kraków: Instytut Fizyki i Techniki Jądrowej AGH, 1988.

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21

Truax, Jerome. Linking Laboratory and in Situ Activation Analysis of Rock-Forming Elements Using a 14 Mev Neutron Source. Delft Univ Pr, 1995.

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22

Henriksen, Niels Engholm, und Flemming Yssing Hansen. Unimolecular Reactions. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198805014.003.0007.

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This chapter considers unimolecular reactions; photo-induced reactions, that is, true unimolecular reactions; and reactions initiated by collisional activation, that is, apparent unimolecular reactions where it is assumed that the time scales for activation and subsequent reaction are well separated. Elements of classical and quantum dynamical descriptions are discussed, including Slater theory and the quantum mechanical description of photo-induced reactions. Statistical theories aiming at the calculation of micro-canonical as well as canonical rate constants are discussed, including a detailed discussion of RRKM theory. It concludes with a discussion of femtochemistry, that is, the observation and control of chemical dynamics using femtosecond pulses of electromagnetic radiation, focusing on the control of unimolecular reactions via the interaction with coherent light; that is, laser control.

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23

Bevan, Julie. The production of Sn-117m by the thermal neutron activation of Sn116, Sn117 and the natural elemental tin. 1995.

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24

Butterworth, G. J. Concentrations of Radiologically - Important Tramp Elements in Standard-grade Stainless Steels with Particular Relevance to Activation of the First Wall of NET (Reports). AEA Technology Plc, 1989.

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25

Johnston, Michael V. Coffin-Lowry Syndrome. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0057.

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Coffin-Lowry syndrome (CLS) is a relatively rare (1:50,000-100,000 incidence) sex-linked neurodevelopmental disorder that includes severe intellectual disability, dysmorphic features including facial and digital abnormalities, growth retardation, and skeletal changes. Most cases are sporadic with only 20% to 30% of cases having an additional family member. CLS is caused by variable loss of function mutations in the RPS6KA3 gene that maps to Xp22.2 and codes for the hRSK2 S6 kinase that phosphorylates the transcription factor CREB (cAMP response element binding protein) as well as other nuclear transcription factors. Phosphorylated CREB (pCREB) plays a major role in memory formation in fruit flies and mammals by activating specific genes through epigenetic histone acetylation.

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26

Biewener, Andrew A., und Shelia N. Patek, Hrsg. Muscles and Skeletons. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198743156.003.0002.

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Animal locomotion depends on the organization, physiology and biomechanical properties of muscles and skeletons. Musculoskeletal systems encompass the mechanical interactions of muscles and skeletal elements that ultimately transmit force for movement and support. Muscles not only perform work by contracting and shortening to generate force, they can also operate as brakes to slow the whole body or a single appendage. Muscles can also function as struts (rod-like) to maintain the position of a joint and facilitate elastic energy storage and recovery. Skeletal muscles share a basic organization and all rely on the same protein machinery for generating force and movement. Variation in muscle function, therefore, depends on the underlying mechanical and energetic components, enzymatic properties, and activation by the nervous system. Muscles require either an internal, external or hydrostatic skeletal system to transmit force for movement and support.

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27

Schott, Christopher K., und Jessica A. Fozard. Hypotension and Shock (DRAFT). Herausgegeben von Raghavan Murugan und Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0008.

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Hypotension is a common cause of rapid response team (RRT) activation. It is critical to be able to rapidly identify the etiology of hypotension. In the setting of a rapid response team call, there is often limited time and information available when first encountering a hypotensive patient. With attention to key elements in the patient’s history of present illness, physical exam, and findings of predominant changes in systolic, diastolic, and pulse pressures, RRTs can rapidly narrow their differential diagnosis. We will discuss the initial evaluation and treatment recommendations based on the etiology of hypotension and shock. Resuscitation should continue until circulatory homeostasis occurs, as guided by a patient’s exam, vital signs, and trends in laboratory values. This chapter provides a framework on how to quickly differentiate between the causes of hypotension or shock when evaluating patients during a rapid response scenario to most accurately guide therapy.

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