Thematische Bibliographien / Dyshidrosis / Zeitschriftenartikel
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Zeitschriftenartikel zum Thema „Dyshidrosis“

Autor: Grafiati
Veröffentlicht am 28. Juni 2021
Zuletzt aktualisiert am 2. Februar 2022

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1

Wollina, Uwe. „Pompholyx or dyshidrosis“. Expert Review of Dermatology 4, Nr. 4 (August 2009): 403–11. http://dx.doi.org/10.1586/edm.09.34.

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2

Wilson, M., und T. Maglietta. „Pompholyx (dyshidrosis). A case study“. Journal of the American Podiatric Medical Association 75, Nr. 3 (01.03.1985): 155–58. http://dx.doi.org/10.7547/87507315-75-3-155.

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3

Sudo, K., N. Fujiki, S. Tsuji, M. Ajiki, T. Higashi, M. Niino, S. Kikuchi, F. Moriwaka und K. Tashiro. „Focal (segmental) dyshidrosis in syringomyelia“. Journal of Neurology, Neurosurgery & Psychiatry 67, Nr. 1 (01.07.1999): 106–8. http://dx.doi.org/10.1136/jnnp.67.1.106.

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4

Lofgren, Sabra M., und Erin M. Warshaw. „Dyshidrosis: Epidemiology, Clinical Characteristics, and Therapy“. Dermatitis 17, Nr. 4 (Dezember 2006): 165–81. http://dx.doi.org/10.2310/6620.2006.05021.

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5

YERAGANI, VIKRAM K., HITEN PATEL und M. S. KESHAVAN. „Effectiveness of Desipramine in the Treatment of Dyshidrosis“. Journal of Clinical Psychopharmacology 8, Nr. 1 (Februar 1988): 76. http://dx.doi.org/10.1097/00004714-198802000-00038.

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6

Kutzner, Heinz, Reinhard M. Wurzel und Helmut H. Wolff. „Are Acrosyringia Involved in the Pathogenesis of "Dyshidrosis"?“ American Journal of Dermatopathology 8, Nr. 2 (April 1986): 109–16. http://dx.doi.org/10.1097/00000372-198604000-00004.

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7

Hsu, C. Y., Y. C. Wang und C. H. Kao. „Dyshidrosis is a risk factor for herpes zoster“. Journal of the European Academy of Dermatology and Venereology 29, Nr. 11 (27.04.2015): 2177–83. http://dx.doi.org/10.1111/jdv.13175.

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8

Chang, Y. Y., J. van der Velden, G. van der Wier, D. Kramer, G. F. H. Diercks, M. van Geel, P. J. Coenraads, P. L. J. M. Zeeuwen und M. F. Jonkman. „Keratolysis exfoliativa (dyshidrosis lamellosa sicca): a distinct peeling entity“. British Journal of Dermatology 167, Nr. 5 (05.10.2012): 1076–84. http://dx.doi.org/10.1111/j.1365-2133.2012.11175.x.

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9

Radloff, Petra, Dieter Vieluf, Hartwig Mensing und Johannes Ring. „Erstmanifestation eines bullösen Pemphigoids unter dem Bild einer Dyshidrosis palmoplantaris“. Der Hautarzt 45, Nr. 2 (Februar 1994): 97–99. http://dx.doi.org/10.1007/pl00013265.

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10

Brady, Margaret. „Patient management exchange Hands and feet that blister and peel: Dyshidrosis“. Journal of Pediatric Health Care 7, Nr. 1 (Januar 1993): 37–38. http://dx.doi.org/10.1016/0891-5245(93)90025-d.

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11

KIHARA, MIKIHIRO. „Medicine disease and autonomous nerve syndrome. Medicine disease and autonomic neuropathy. Dyshidrosis.“ Nihon Naika Gakkai Zasshi 84, Nr. 9 (1995): 1435–37. http://dx.doi.org/10.2169/naika.84.1435.

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12

ALTUNIŞIK, Nihal. „A Dyshidrosis Case Report That Characterized by Diffuse Blisters on the Bilateral Hands“. Turkiye Klinikleri Journal of Medical Sciences 37, Nr. 4 (2017): 215–16. http://dx.doi.org/10.5336/medsci.2017-57651.

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13

Perretta, Fernando, Norberto Antongiovanni und Sebastián Jaurretche. „Early Renal Involvement in a Girl with Classic Fabry Disease“. Case Reports in Nephrology 2017 (2017): 1–4. http://dx.doi.org/10.1155/2017/9543079.

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Fabry disease is an X-linked lysosomal storage disorder resulting from the deficiency or absence of the enzyme alpha galactosidase A; this defect leads to the systemic accumulation of globotriaosylceramide and its metabolites. Organic involvement in men is well known, but in women it is controversial, mainly due to the random X-chromosome inactivation in each of their cells (Lyon hypothesis). This would explain why women (heterozygotes) present a wide variability in the severity of their phenotype. The manifestations are multisystemic and begin in early childhood, reaching a severe compromise in adulthood. Typical acroparesthesia in hands and feet, gastrointestinal symptoms, angiokeratomas, dyshidrosis, hearing loss, arrhythmias, hypertrophic cardiomyopathy, cerebrovascular accidents, and renal failure can be observed. Nephropathy is one of the major complications of Fabry disease. Glomerular and vascular changes are present before progression to overt proteinuria and decreased glomerular filtration rate, even in pediatric patients. A case of incipient renal involvement in a girl with classic Fabry disease is reported.
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14

Steele, Haley R., Yanyan Xing, Yuyan Zhu, Henry B. Hilley, Katy Lawson, Yeseul Nho, Taylor Niehoff und Liang Han. „MrgprC11+ sensory neurons mediate glabrous skin itch“. Proceedings of the National Academy of Sciences 118, Nr. 15 (05.04.2021): e2022874118. http://dx.doi.org/10.1073/pnas.2022874118.

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Itch arising from glabrous skin (palms and soles) has attracted limited attention within the field due to the lack of methodology. This is despite glabrous itch arising from many medical conditions such as plantar and palmar psoriasis, dyshidrosis, and cholestasis. Therefore, we developed a mouse glabrous skin behavioral assay to investigate the contribution of three previously identified pruriceptive neurons in glabrous skin itch. Our results show that MrgprA3+ and MrgprD+ neurons, although key mediators for hairy skin itch, do not play important roles in glabrous skin itch, demonstrating a mechanistic difference in itch sensation between hairy and glabrous skin. We found that MrgprC11+ neurons are the major mediators for glabrous skin itch. Activation of MrgprC11+ neurons induced glabrous skin itch, while ablation of MrgprC11+ neurons reduced both acute and chronic glabrous skin itch. Our study provides insights into the mechanisms of itch and opens up new avenues for future glabrous skin itch research.
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15

Brzezinski, Piotr, und Lorenzo Martini. „A phytotherapeutic approach to hand and feet dyshidrosis using vegetables (espcially mashed celeriac applied topically)“. Our Dermatology Online 10, e (01.12.2019): e41.1-e41.2. http://dx.doi.org/10.7241/ourd.2019e.41.

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16

Siedler, Gabriela, Ann-Kathrin Káhn, Frank Weidemann, Christoph Wanner, Claudia Sommer und Nurcan Üçeyler. „Dyshidrosis is associated with reduced amplitudes in electrically evoked pain-related potentials in women with Fabry disease“. Clinical Neurophysiology 130, Nr. 4 (April 2019): 528–36. http://dx.doi.org/10.1016/j.clinph.2019.01.008.

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17

Ruhin, B., V. Martinot, P. Lafforgue, B. Catteau, S. Manouvrier-Hanu und J. Ferri. „Pure Ectodermal Dysplasia: Retrospective Study of 16 Cases and Literature Review“. Cleft Palate-Craniofacial Journal 38, Nr. 5 (September 2001): 504–18. http://dx.doi.org/10.1597/1545-1569_2001_038_0504_pedrso_2.0.co_2.

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Objective: To review the possible craniomaxillofacial deformative consequences associated with ectodermal dysplasias and embryonic malformations, which include dental ageneses. Setting: Oral and Maxillofacial Surgery Department, University Hospital, Lille, France. Patients: Sixteen patients (seven boys and nine girls, aged 4 to 34 years) with pure ectodermal dysplasia (no ectodermal dysplasia syndromes). Interventions: All patients had a clinical examination. Seven (two boys and five girls, aged 4 to 25 years) had undergone plaster casts and radiographic and Delaire's cephalometric studies before being treated. Main Outcome Measures: All patients had tooth ageneses (from hypodontia to anodontia), associated with cutaneous dyshidrosis and hair and nail dystrophy. Most of them had a short face, with an unusual facial concavity, a maxillary retrusion, and a relative mandibular protrusion. Management Results and Discussion: Depending on their ages and their orthopedic abnormalities, patients underwent either dental or prosthodontic, orthodontic, orthopedic, orthognathic, or implant treatment. So as not to interfere with the growth pattern, we preferred to reserve implant and orthognathic surgery for full-grown cases. Conclusions: Oral and maxillofacial surgeons must undertake a comprehensive approach to these patients to improve their dental, masticatory, growing, and orthognathic conditions.
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18

Hotta, Moeko, Teruki Yanagi, Tatsuro Sugai, Shinichi Nakazato, Kentaro Izumi, Hiroo Hata, Kohei Okada, Hideki Goto, Yoshihiro Matsuno und Hiroshi Shimizu. „Case of peripheral T-cell lymphoma, not otherwise specified, presenting dyshidrosis-like eruptions as the first clinical manifestation“. Journal of Dermatology 44, Nr. 12 (04.09.2017): e359-e360. http://dx.doi.org/10.1111/1346-8138.14029.

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19

Bittencourt, Achiléa L., Karla Mota, Rodrigo F. Oliveira und Lourdes Farré. „A Dyshidrosis-Like Variant of Adult T-Cell Leukemia/Lymphoma With Clinicopathological Aspects of Mycosis Fungoides. A Case Report“. American Journal of Dermatopathology 31, Nr. 8 (Dezember 2009): 834–37. http://dx.doi.org/10.1097/dad.0b013e3181ac04fe.

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20

Kawada, Morihiro, Yasuhiko Tamada, Hirokazu Simizu, Takeshi Yanagishita, Noriko Yamashita, Natsuko Ishida, Daisuke Watanabe et al. „Reduction in QSART and vasoactive intestinal polypeptide expression in the skin of Parkinson’s disease patients and its relation to dyshidrosis“. Journal of Cutaneous Pathology 36, Nr. 5 (Mai 2009): 517–21. http://dx.doi.org/10.1111/j.1600-0560.2008.01086.x.

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21

Bose, Reetesh, und Jennifer Beecker. „Dyshidrotic eczema in two patients on secukinumab for plaque psoriasis: A case report“. SAGE Open Medical Case Reports 8 (Januar 2020): 2050313X2090456. http://dx.doi.org/10.1177/2050313x20904561.

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Secukinumab was the first fully human anti-interleukin-17a monoclonal antibody and successfully treated moderate-severe psoriasis. These new, targeted, medications are becoming more ubiquitous, but long-term side effects are not fully known. Post-market surveillance is crucial to identify delayed adverse events, analogous to the paradoxical development of pustular psoriasis in a subset of patients treated with the anti-tumor necrosis factor-alpha class drugs. Dyshidrotic eczema and pompholyx are rare variants of dermatitis characterized by vesicles or bullae on the palms, soles and sides of the fingers. The etiology of dyshidrotic eczema is not always known, but medications have been implicated in a minority of patients. Herein, we present two cases of dyshidrotic eczema developing in patients on secukinumab for psoriasis. Extended follow-up and larger numbers of patients are needed to fully understand the potential association between secukinumab and dyshidrotic eczema.
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22

Martini, Lorenzo, und Texia Research. „The revival of an ancestral galenic preparation called “Ceratum” apt to re-establish the normal viscoelasticity of dermis in perilous skin diseases like iatrogenic dyshidrosis and to treat scars after milia removal.“ IOSR Journal of Pharmacy and Biological Sciences 11, Nr. 05 (Mai 2016): 38–41. http://dx.doi.org/10.9790/3008-1105023841.

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23

Guillet, M. H., Y. Le Ru, G. Guillet und Morvan Chu. „Resultats d'exploration de 111 dyshidroses“. Revue Française d'Allergologie et d'Immunologie Clinique 41, Nr. 1 (Januar 2001): 125. http://dx.doi.org/10.1016/s0335-7457(01)80053-8.

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24

Tchernev, Georgi, Matteo Zanardelli, Cristiana Voicu, Ilko Bakardzhiev, Torello Lotti, Jacopo Lotti, Katlein França, Atanas Batashki und Uwe Wollina. „Impetiginized Dyshidrotic Eczema“. Open Access Macedonian Journal of Medical Sciences 5, Nr. 4 (19.07.2017): 539–40. http://dx.doi.org/10.3889/oamjms.2017.081.

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A 16 years old female patient, affected by atopic dermatitis and rhinoconjunctivitis allergica since childhood, requested a dermatologic consultation for lesions which had appeared after 3 months of local treatment with clobethasole propionate. The histological analysis confirmed the diagnosis of dyshidrotic eczema and the microbiological smears demonstrated a significant infection with Staphylococcus aureus. The risk of developing corticosteroids’ side-effects depends on the potency of the product, extended period of use and the volume of product applied. Clobetasol propionate is a group I- highly potent corticosteroid, which should be used for a maximum period of 2 weeks. Several authors have found that this agent has cumulative depot effect, persisting in the epidermis for 4 days after only one application. Taking together these observations, sustained by the clinical case presented above, we can conclude that the infectious risks associated with topical corticosteroid treatment must not be neglected, particularly since treated patients are fragile, and frequently have multiple well-known risk factors.
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25

Diehl, Joseph, G. Peter Sarantopoulos und Melvin W. Chiu. „Dyshidrotic mycosis fungoides“. Journal of Cutaneous Pathology 38, Nr. 7 (24.02.2011): 590–92. http://dx.doi.org/10.1111/j.1600-0560.2011.01682.x.

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26

Semhoun-Ducloux, Sylvie, Didier Ducloux und Jean-Philippe Miguet. „Mycophenolate Mofetil-Induced Dyshidrotic Eczema“. Annals of Internal Medicine 132, Nr. 5 (07.03.2000): 417. http://dx.doi.org/10.7326/0003-4819-132-5-200003070-00015.

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27

Jakob, Thilo, Markus Tiemann, Christoph Kuwert, Dietrich Abeck, Hartwig Mensing und Johannes Ring. „Dyshidrotic cutaneous T-cell lymphoma“. Journal of the American Academy of Dermatology 34, Nr. 2 (Februar 1996): 295–97. http://dx.doi.org/10.1016/s0190-9622(96)80139-3.

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28

Lee, K. C., und B. Ladizinski. „Dyshidrotic eczema following intravenous immunoglobulin treatment“. Canadian Medical Association Journal 185, Nr. 11 (03.12.2012): E530. http://dx.doi.org/10.1503/cmaj.121036.

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29

Anonide, Antonio, David Usiglio, Andrea Pestarina und Luigi Massone. „Droxicam photosensitivity with dyshidrotic hand dermatitis“. International Journal of Dermatology 36, Nr. 4 (28.06.2008): 318–20. http://dx.doi.org/10.1111/j.1365-4362.1997.tb03059.x.

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30

Pickenacker, A. „Dyshidrotic Eczema Treated With Mycophenolate Mofetil“. Archives of Dermatology 134, Nr. 3 (01.03.1998): 378–79. http://dx.doi.org/10.1001/archderm.134.3.378.

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31

Vocks, E., S. G. Plötz und J. Ring. „The Dyshidrotic Eczema Area and Severity Index – A Score Developed for the Assessment of Dyshidrotic Eczema“. Dermatology 198, Nr. 3 (1999): 265–69. http://dx.doi.org/10.1159/000018127.

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32

Dosseur-Hauteville, Béatrice. „Traitement de la dyshidrose par aromathérapie“. Revue du Podologue 15, Nr. 86 (März 2019): 29–31. http://dx.doi.org/10.1016/j.revpod.2019.01.012.

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33

Chang, Claire H., und Philip R. Cohen. „Ipsilateral post-cast hypertrichosis and dyshidrotic dermatitis“. Archives of Physical Medicine and Rehabilitation 76, Nr. 1 (Januar 1995): 97–100. http://dx.doi.org/10.1016/s0003-9993(95)80050-6.

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34

Lehucher-Michel, M. P., M. C. Koeppel, A. Lanteaume und J. Sayag. „Dyshidrotic eczema and occupation: a descriptive study“. Contact Dermatitis 43, Nr. 4 (Oktober 2000): 200–205. http://dx.doi.org/10.1034/j.1600-0536.2000.043004200.x.

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35

Letić, Milorad. „Use of Sunlight to Treat Dyshidrotic Eczema“. JAMA Dermatology 149, Nr. 5 (01.05.2013): 634. http://dx.doi.org/10.1001/jamadermatol.2013.2772.

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36

Stuckert, Joseph, und Susan Nedorost. „Low-cobalt diet for dyshidrotic eczema patients“. Contact Dermatitis 59, Nr. 6 (Dezember 2008): 361–65. http://dx.doi.org/10.1111/j.1600-0536.2008.01469.x.

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37

KHLEBNIKOVA, A. N. „ROLE OF PROTECTIVE TOPICAL AGENTS IN THERAPY OF CHRONIC DERMATOSES“. Russian Journal of Allergy 11, Nr. 3 (15.12.2014): 47–52. http://dx.doi.org/10.36691/rja512.

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Epidermal barrier insufficiency results in skin dryness and fissures in different chronic dermatoses. Barrier dysfunction is due either to genetic problems or lipid deficiency or damage of tight junction proteins. Here we discuss various abnormalities of epidermal barrier in eczema and psoriasis which necessitate to prescribe pro- tective and moisturizing agents to restore skin barrier. We give our own practice data of using Bariederm cream and balm in combined therapy of dyshidrotic eczema, plantar eczema and palmoplantar psoriasis.
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38

Kotan, D., T. Erdem, B. A. Acar und A. Boluk. „Dyshidrotic eczema associated with the use of IVIg“. Case Reports 2013, feb15 1 (15.02.2013): bcr2012008001. http://dx.doi.org/10.1136/bcr-2012-008001.

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39

Fowler, Joseph F., und Frances J. Storrs. „Nickel Allergy and Dyshidrotic Eczema: Are They Related?“ Dermatitis 12, Nr. 2 (Juni 2001): 119–21. http://dx.doi.org/10.1097/01206501-200106000-00011.

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40

Fowler, Joseph F., und Frances J. Storrs. „Nickel Allergy and Dyshidrotic Eczema: Are They Related?“ American Journal of Contact Dermatitis 12, Nr. 2 (Juni 2001): 119–21. http://dx.doi.org/10.1097/01634989-200106000-00011.

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41

Reis, Joel, Inês Lobo, André Coelho und Manuela Selores. „Successful treatment of recalcitrant dyshidrotic eczema with oxybutynin“. International Journal of Dermatology 59, Nr. 8 (09.05.2020): 1010–12. http://dx.doi.org/10.1111/ijd.14928.

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42

Fowler, J. „Nickel allergy and dyshidrotic eczema: Are they related?“ American Journal of Contact Dermatitis 12, Nr. 2 (Juni 2001): 119–21. http://dx.doi.org/10.1053/ajcd.2001.23123.

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43

Bienvenu, Anne-Lise, und Stéphane Picot. „La dyshidrose : de la sueur et des larmes“. Revue du Podologue 13, Nr. 74 (März 2017): 29–30. http://dx.doi.org/10.1016/j.revpod.2016.12.011.

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44

Bizunova, M. A., N. G. Ilina, D. V. Demina, Yu M. Krinitsyna und I. G. Sergeeva. „Food allergy and sensitization in patients with dyshidrotic eczema“. Klinicheskaya dermatologiya i venerologiya 16, Nr. 5 (2017): 31. http://dx.doi.org/10.17116/klinderma201716531-34.

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45

Shin, Yoon-Jin, und Jong-Woo Lee. „Clinical Analysis on the 219 cases of Dyshidrotic Eczema“. Journal of Korean Medicine Ophthalmology and Otolaryngology and Dermatology 27, Nr. 4 (25.11.2014): 58–66. http://dx.doi.org/10.6114/jkood.2014.27.4.058.

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46

Letić, Milorad. „Exposure to sunlight as adjuvant therapy for dyshidrotic eczema“. Medical Hypotheses 73, Nr. 2 (August 2009): 203–4. http://dx.doi.org/10.1016/j.mehy.2008.08.035.

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47

Klein, Arnold William. „Treatment of dyshidrotic hand dermatitis with intradermal botulinum toxin“. Journal of the American Academy of Dermatology 50, Nr. 1 (Januar 2004): 153–54. http://dx.doi.org/10.1016/s0190-9622(03)01674-8.

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48

Basseri, Sana, Thai Yen Ly und Peter R. Hull. „Dyshidrotic Bullous Pemphigoid: Case Report and Review of Literature“. Journal of Cutaneous Medicine and Surgery 22, Nr. 6 (05.03.2018): 614–17. http://dx.doi.org/10.1177/1203475418763544.

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Background: Dyshidrotic pemphigoid (DP) is a rare variant of bullous pemphigoid (BP) that affects the hands and feet and may resemble an acute vesicular eczema. While it can remain confined to hands and feet, spread that involves the entire body is described. BP and DP are associated with autoantibodies directed against hemidesmosomal proteins BP180 (collagen XVII) and BP230 (dystonin), which are transmembrane and intracellular proteins in the basement membrane zone, respectively. Case Summary: We present a case of DP in a 78-year-old woman who was diagnosed based on histopathologic and immunofluorescence findings and subsequently successfully treated. Conclusion: DP is an unusual form of localized BP. While the pathogenesis is still unclear, it may involve differential expression of BP antigens in the cutaneous basement membrane of the hands and feet. The clinical presentation is a diagnostic challenge, and skin biopsies with immunofluorescence studies are required for diagnosis.
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49

Sherertz, Elizabeth F., und Katherine B. Medford. „Patch Testing May be Valuable in Chronic Dyshidrotic Eczema“. Dermatitis 5, Nr. 3 (September 1994): 180–81. http://dx.doi.org/10.1097/01206501-199409000-00014.

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50

Sherertz, Elizabeth F., und Katherine B. Medford. „Patch Testing May be Valuable in Chronic Dyshidrotic Eczema“. American Journal of Contact Dermatitis 5, Nr. 3 (September 1994): 180–81. http://dx.doi.org/10.1097/01634989-199409000-00014.

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