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Zeitschriftenartikel zum Thema „DoS Resistance“

Autor: Grafiati

Veröffentlicht am 28. Juni 2021

Zuletzt aktualisiert am 13. Februar 2022

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1

Pink, D. A. C., and P. Hand. "Plant resistance and strategies for breeding resistant varieties." Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (2002): S9—S14. http://dx.doi.org/10.17221/10310-pps.

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An explanation of the ‘boom-bust’ cycle of resistance breeding was provided by the gene-for-gene relationship between a pathogen and its host. Despite this understanding, most R genes continued to be deployed singly and resistance has been ephemeral. The reasons for breeding ‘single R gene’ varieties are discussed. Alternative strategies for the deployment of R genes and the use of quantitative race non-specific resistance have been advocated in order to obtain durable resistance. The feasibility of both of these approaches is discussed taking into account the impact of technologies such as plant transformation and marker-assisted selection. A change in focus from durability of the plant phenotype to that of the crop phenotype is advocated.

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2

Çelik, Cem. "Increasing antimicrobial resistance in nosocomial pathogens; multidrug-resistant extensively drug-resistant and pandrug-resistant Acinetobacter baumannii." Journal of Microbiology and Infectious Diseases 4, no. 1 (2014): 7–12. http://dx.doi.org/10.5799/ahinjs.02.2014.01.0116.

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3

Андреевская, С. Н., Т. Г. Смирнова, Е. Е. Ларионова, И. Ю. Андриевская, Л. Н. Черноусова та А. Эргешов. "Изониазид-резистентные Mycobacterium tuberculosis: частота выявления, спектры резистентности и генетические детерминанты устойчивости". Phylogenetic Analysis, № 1;2020 (2020): 22–28. http://dx.doi.org/10.24075/vrgmu.2020.001.

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Отсутствие ускоренной диагностики туберкулеза с устойчивостью возбудителя к изониазиду с сохраненной чувствительностью к рифампицину (ИР-ТБ) может быть причиной низкой эффективности терапии и приводить к амплификации лекарственной резистентности, в том числе к формированию множественной лекарственной устойчивости. Целью работы было определить частоту встречаемости ИР-ТБ в современной популяции, охарактеризовать фенотипическую чувствительность и генетические детерминанты устойчивости к изониазиду представителей этой группы M. tuberculosis на репрезентативном материале. Анализировали результаты определения лекарственной чувствительности, полученные при исследовании молекулярно- генетическими и/или культуральными методами изолятов M. tuberculosis / ДНК M. tuberculosis, выделенных от больных туберкулезом легких из клинических отделений Центрального научно-исследовательского институа туберкулеза за период 2011–2018 гг. Частота ИР-ТБ составила 12% от всех выявленных случаев туберкулеза. M. tuberculosis с ИР были как монорезистентными к изониазиду (45%), так и полирезистентными (устойчивыми к 2–6 противотуберкулезным препаратам), а устойчивость к изониазиду была обусловлена мутациями в гене katG, приводящими к высокому уровню резистентности. На основании анализа литературных данных и собственных наблюдений подчеркивается важность разработки и внедрения новых простых молекулярных тестов для определения устойчивости одновременно к рифампицину и изониазиду.

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4

Gokalp, Deniz, Alpaslan Tuzcu, Ster Irmak, Mithat Bahceci, and Ozlem Demirpence. "FREQUENCY OF ASPIRIN RESISTANCE IN PATIENTS WITH TYPE 1 AND 2 DIABETES MELLITUS AND ITS ASSOCIATION WITH METABOLIC PARAMETERS." International Journal of Surgery and Medicine 2, no. 3 (2016): 122. http://dx.doi.org/10.5455/ijsm.aspirin-resistance-diabetes.

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5

Singh, Amresh Kumar. "Resistance patterns and trends of extensively drug-resistant tuberculosis: 5-year experience." Journal of Microbiology and Infectious Diseases 03, no. 04 (2013): 169–75. http://dx.doi.org/10.5799/ahinjs.02.2013.04.0103.

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6

Kumar, Sinha Ritesh, and Chandra Satish. "Insulin resistance." Asian Pacific Journal of Health Sciences, Supplimentary 2014 (2014): 71–78. http://dx.doi.org/10.21276/apjhs.2014.1.1s.15.

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7

Shahina, Sk jasmine, Sheeba Ali Siddiqui, and Dr Padma Krishnan. "Nasal Carriage of Methicillin Resistant Staphylococci with Inducible Clindamycin Resistance and Pvl Gene." International Journal of Scientific Research 3, no. 6 (2012): 392–94. http://dx.doi.org/10.15373/22778179/june2014/132.

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8

Wang, S. C., and P. S. Wei. "Modeling Dynamic Electrical Resistance During Resistance Spot Welding." Journal of Heat Transfer 123, no. 3 (2000): 576–85. http://dx.doi.org/10.1115/1.1370502.

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Dynamic electrical resistance during resistance spot welding has been quantitatively modeled and analyzed in this work. A determination of dynamic resistance is necessary for predicting the transport processes and monitoring the weld quality during resistance spot welding. In this study, dynamic resistance is obtained by taking the sum of temperature-dependent bulk resistance of the workpieces and contact resistances at the faying surface and electrode-workpiece interface within an effective area corresponding to the electrode tip where welding current primarily flows. A contact resistance is composed of constriction and film resistances, which are functions of hardness, temperature, electrode force, and surface conditions. The temperature is determined from the previous study in predicting unsteady, axisymmetric mass, momentum, heat, species transport, and magnetic field intensity with a mushy-zone phase change in workpieces, and temperature and magnetic fields in the electrodes of different geometries. The predicted nugget thickness and dynamic resistance versus time show quite good agreement with available experimental data. Excluding expulsion, the dynamic resistance curve can be divided into four stages. A rapid decrease of dynamic resistance in stage 1 is attributed to decreases in contact resistances at the faying surface and electrode-workpiece interface. In stage 2, the increase in dynamic resistance results from the primary increase of bulk resistance in the workpieces and an increase of the sum of contact resistances at the faying surface and electrode-workpiece interface. Dynamic resistance in stage 3 decreases, because increasing rate of bulk resistance in the workpieces and contact resistances decrease. In stage 4 the decrease of dynamic resistance is mainly due to the formation of the molten nugget at the faying surface. The molten nugget is found to occur in stage 4 rather than stage 2 or 3 as qualitatively proposed in the literature. The effects of different parameters on the dynamic resistance curve are also presented.

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9

Cohen, Y. "Systemic induced resistance." Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (2002): S122—S125. http://dx.doi.org/10.17221/10334-pps.

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Biotic and abiotic agents may induce resistance in plants against pathogens. Abiotic agents may be synthetic or natural. The natural, non-protein amino acid BABA (DL-β-aminobutyric acid) induces systemic resistance in crop plants against pathogens. Dry, killed mycelia of Penicillium chrysogenum (DM) induces local resistance in plants against soil-borne pathogens. The activity of BABA and DM are described here in detail. Both products were shown to effectively control plant disease in nature.

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10

Manninger, K. "Effective Resistance Genes as Sources of Resistance against Hungarian Eheat Rusts." Czech Journal of Genetics and Plant Breeding 38, No. 3-4 (2012): 153–54. http://dx.doi.org/10.17221/6257-cjgpb.

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11

Šliková, S., E. Gregová, P. Bartoš, and A. Hanzalová. "Utilization of alien leaf rust resistance genes for development of resistant wheat genotypes by backross." Czech Journal of Genetics and Plant Breeding 41, Special Issue (2012): 321. http://dx.doi.org/10.17221/6209-cjgpb.

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12

Wrzecioniarz, Piotr, Wojciech Ambroszko, and Aleksandra Pindel. "Limitations of vehicle movement resistances: aerodynamic resistance." AUTOBUSY – Technika, Eksploatacja, Systemy Transportowe 19, no. 12 (2018): 252–55. http://dx.doi.org/10.24136/atest.2018.393.

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In the paper limitations and exemplary methods of aerodynamic drag force minimization in different kinds of vehicles are described. Changes of value of aerodynamic drag coefficient during years are present-ed. Conclusions about future possible new solutions in different kinds of vehicles are formulated.

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13

Wrzecioniarz, Piotr, Wojciech Ambroszko, and Aleksandra Pindel. "Limitations of vehicle movement resistances: rolling resistance." AUTOBUSY – Technika, Eksploatacja, Systemy Transportowe 19, no. 12 (2018): 256–59. http://dx.doi.org/10.24136/atest.2018.394.

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In the paper limitations and exemplary methods of rolling resistance minimization are described. Changes of value of rolling resistance coefficient during years and values for exemplary rolling pairs are presented. Conclusions about future progress are formulated.

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Yusuf, Yenni. "ANTI-MALARIAL DRUG RESISTANCE." Majalah Kedokteran Andalas 37, no. 1 (2015): 64. http://dx.doi.org/10.22338/mka.v37.i1.p64-69.2014.

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AbstrakTujuan studi ini adalah untuk menjelaskan mekanisme resistensi parasit malaria danusaha-usaha yang dapat dilakukan untuk menghadapi munculnya strain parasit yangresisten terhadap artemisinin. Metode yang digunakan adalah studi kepustakaan. ResistensiP.falciparum terhadap obat-obat anti malaria disebabkan oleh perubahan spontan yangterjadi pada beberapa gen seperti P.falciparum multi drug resistance1 (Pfmdr1), P.falciparumchloroquine transporter (Pfcrt), P.falciparum dihydropteroate synthase (Pfdhps), P.falciparumdihydrofolate reductase (Pfdhfr), and P.falciparum multidrug resistance-associated proteins(Pfmrp). Penyebaran resistensi tersebut dipengaruhi oleh tingkat transmisi di sebuah wilayah.WHO telah menjalankan usaha untuk menanggulangi penyebaran resistensi tersebut misalnyadengan merekomendasikan penghentian monoterapi artemisinin, dan pemberian anti malariasetelah konfirmasi laboratorium. Selain itu, perlu adanya penggunaan obat kombinasi, produksirejimen dosis tetap, dan pengembangan obat anti malaria baru. Kesimpulan dari hasil studiini ialah munculnya malaria resisten terhadap artemisinin akan menghambat usaha eradikasimalaria karena itu diperlukan usaha-usaha untuk menanggulanginya.AbstractThe objective of this study was to describe the development of anti-malarial drug resistanceof the parasites and the efforts taken to contain the emergence of artemisinin resistant malaria.This was a literature study. The development of resistance to anti-malarial drugs are due tospontaneous changes in certain genes such as of P.falciparum multi drug resistance1 (Pfmdr1),P.falciparum chloroquine resistance transporter (Pfcrt), P.falciparum dihydropteroate synthase(Pfdhps), P.falciparum dihydrofolate reductase (Pfdhfr), and P.falciparum multidrug resistanceassociatedproteins (Pfmrp). The spread of the resistance depends on the transmission ratewithin each area. WHO has established a global plan to contain the spread of this resistance,such as recommendation to withdraw artemisinin-based monotherapies and administrationof treatment after laboratory confirmation. In addition, administration of anti-malarial drugcombination, production of fixed dose regimen and development of new drugs are necessary.The Conclusion is emergence of artemisinin resistant malaria will threaten malaria eradicationthus some efforts are necessarily needed to contain it.Afiliasi penulis: Bagian Parasitologi Fakultas Kedokteran Universitas Hasanudin

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15

Talebiyan, Reza, Mehdi Kheradmand, Faham Khamesipour, and Mohammad Rabiee-Faradonbeh. "Multiple Antimicrobial Resistance ofEscherichia coliIsolated from Chickens in Iran." Veterinary Medicine International 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/491418.

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Antimicrobial agents are used extremely in order to reduce the great losses caused byEscherichia coliinfections in poultry industry. In this study, 318 pathogenicEscherichia coli(APEC) strains isolated from commercial broiler flocks with coli-septicemia were examined for antimicrobials of both veterinary and human significance by disc diffusion method. Multiple resistances to antimicrobial agents were observed in all the isolates. Resistance to the antibiotics was as follows: Tylosin (88.68%), Erythromycin (71.70%), Oxytetracycline (43.40%), Sulfadimethoxine-Trimethoprim (39.62%), Enrofloxacin (37.74%), Florfenicol (35.85%), Chlortetracycline (33.96%), Doxycycline (16.98%), Difloxacin (32.08%), Danofloxacin (28.30%), Chloramphenicol (20.75%), Ciprofloxacin (7.55%), and Gentamicin (5.66%). This study showed resistance against the antimicrobial agents that are commonly applied in poultry, although resistance against the antibiotics that are only applied in humans or less frequently used in poultry was significantly low. This study emphasizes on the occurrence of multiple drug resistantE. coliamong diseased broiler chickens in Iran. The data revealed the relative risks of using antimicrobials in poultry industry. It also concluded that use of antibiotics must be limited in poultry farms in order to reduce the antibiotic resistances.

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16

Chapman, John S. "Disinfectant resistance mechanisms, cross-resistance, and co-resistance." International Biodeterioration & Biodegradation 51, no. 4 (2003): 271–76. http://dx.doi.org/10.1016/s0964-8305(03)00044-1.

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17

Mora, Christophe, and Karyn Le Hur. "Universal resistances of the quantum resistance–capacitance circuit." Nature Physics 6, no. 9 (2010): 697–701. http://dx.doi.org/10.1038/nphys1690.

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18

D.P, Singh, Sharma A.K, Babu K.S, Sharma Indu, and Nagarajan S. "Multiple Diseases, Insect Pests Resistant Genotypes and Their Utilization in Breeding for Resistance in Wheat and Triticale." Greener Journal of Agricultural Sciences 4, no. 4 (2014): 150–65. http://dx.doi.org/10.15580/gjas.2014.4.042914214.

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19

Pamplona, Marisa, Mathias Kocher, Rolf Snethlage, and Luís Aires Barros. "Drilling resistance: overview and outlook." Zeitschrift der Deutschen Gesellschaft für Geowissenschaften 158, no. 3 (2007): 665–79. http://dx.doi.org/10.1127/1860-1804/2007/0158-0665.

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20

Rubiales, D. "Legume breeding for broomrape resistance." Czech Journal of Genetics and Plant Breeding 50, No. 2 (2014): 144–50. http://dx.doi.org/10.17221/106/2013-cjgpb.

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Legume cultivation is hampered in Mediterranean regions by the occurrence of the root parasitic weeds Orobanche crenata (crenate broomrape) and Orobanche foetida (foetida broomrape). Strategies of control have been developed but only marginal successes have been achieved. Most control methods are unfeasible, uneconomical, and hard to achieve or result in incomplete protection. Breeding for resistance is possible, but is hampered by the lack of sufficient levels of resistance, the complexity of its inheritance and the unreliability of available screening methods. Recent achievements in the identification of resistance levels and their deployment in breeding programmes will be presented and critically discussed.

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21

Valkonen, J. P. T. "Mechanisms of resistance to viruses." Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (2002): S132—S135. http://dx.doi.org/10.17221/10337-pps.

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Resistance associated with a hypersensitive response (HR) and subsequent development of necrotic lesions (cell death) at the sites of virus infection can restrict virus movement in plants. Genes for HR are dominant and act on a gene-for-gene basis. Many viral proteins triggering HR have been identified. Also, several genes for HR-based virus resistance, or virus-induced cell death without resistance, have been isolated and characterized in plants, which provides novel insights to the mechanisms of virus resistance. Another international, major research frontier has formed more recently around RNA silencing, a universal RNA surveillance system and inducible virus defence mechanism in multicellular organisms. Many viral proteins interfere with different phases of RNA silencing. The data provide novel insights to break-down of resistance in mixed virus infections (viral synergism), resistance to virus movement, and recovery of plants from virus infection.

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22

Tooming, Uku. "Imaginative resistance as imagistic resistance." Canadian Journal of Philosophy 48, no. 5 (2018): 684–706. http://dx.doi.org/10.1080/00455091.2017.1378534.

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AbstractWhen we are invited to imagine an unacceptable moral proposition to be true in fiction, we feel resistance when we try to imagine it. Despite this, it is nonetheless possible to suppose that the proposition is true. In this paper, I argue that existing accounts of imaginative resistance are unable to explain why only attempts to imagine (rather than to suppose) the truth of moral propositions cause resistance. My suggestion is that imagination, unlike supposition, involves mental imagery and imaginative resistance arises when imagery that one has formed does not match unacceptable propositions.

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23

Zhang, Li, Tao Liu, Pei-Yong Zheng, and Guang Ji. "Leptin resistance and insulin resistance." World Chinese Journal of Digestology 17, no. 15 (2009): 1534. http://dx.doi.org/10.11569/wcjd.v17.i15.1534.

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24

Jelesijević, Nenad. "Resistance against aestheticisation of resistance." Maska 29, no. 169 (2015): 32–47. http://dx.doi.org/10.1386/maska.29.169-171.32_1.

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25

Jelesijević, Nenad. "Resistance against aestheticisation of resistance." Maska 30, no. 169 (2015): 32–47. http://dx.doi.org/10.1386/maska.30.169-171.32_1.

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26

Szanto, Thomas. "Imaginative Resistance and Empathic Resistance." Topoi 39, no. 4 (2019): 791–802. http://dx.doi.org/10.1007/s11245-019-09642-9.

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27

Schmidt, B. J., and F. M. Bartlett. "Review of resistance factor for steel: resistance distributions and resistance factor calibration." Canadian Journal of Civil Engineering 29, no. 1 (2002): 109–18. http://dx.doi.org/10.1139/l01-082.

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Changes in the Canadian steel industry warrant a review of the steel resistance factor in CSA Standard S16 (formerly S16.1) "Limit states design of steel structures", originally calibrated in the landmark study by Kennedy and Gad Aly in 1980. This paper presents statistical parameters for the bending, compression, and tension resistances of W, WWF, and HSS components produced since 1999 that have been derived from geometric and material properties presented in a companion paper. The resistance factor for steel was recalibrated for the live and dead load combination in the 1995 National Building Code of Canada. A resistance factor of 0.95 is suitable for laterally supported beams, stocky columns, and tension members failing by yield of the gross section, whereas the current value of 0.90 is appropriate for intermediate columns and tension members failing by fracture of the net section.Key words: code calibration, member resistance, reliability, resistance factor, safety, structural steel.

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Lilja, Mona, Mikael Baaz, Michael Schulz, and Stellan Vinthagen. "How resistance encourages resistance: theorizing the nexus between power, ‘Organised Resistance’ and ‘Everyday Resistance’." Journal of Political Power 10, no. 1 (2017): 40–54. http://dx.doi.org/10.1080/2158379x.2017.1286084.

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29

Drake-Stowe, Katherine, Nicolas Bakaher, Simon Goepfert, et al. "Multiple Disease Resistance Loci Affect Soilborne Disease Resistance in Tobacco (Nicotiana tabacum)." Phytopathology® 107, no. 9 (2017): 1055–61. http://dx.doi.org/10.1094/phyto-03-17-0118-r.

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Phytophthora nicotianae and Ralstonia solanacearum are two of the most important pathogens affecting tobacco worldwide. Greater insight regarding genetic systems controlling resistance to these two soilborne pathogens, as well as identification of DNA markers associated with genomic regions controlling this resistance, could aid in variety development. An evaluation of 50 historical tobacco lines revealed a high positive correlation between resistances to the two pathogens, preliminarily suggesting that some genomic regions may confer resistance to both pathogens. A quantitative trait loci (QTL) mapping experiment designed to investigate the genetic control of soilborne disease resistance of highly resistant ‘K346’ tobacco identified four QTL significantly associated with resistance to P. nicotianae (explaining 60.0% of the observed phenotypic variation) and three QTL to be associated with R. solanacearum resistance (explaining 50.3% of the observed variation). The two QTL with the largest effect on Phytophthora resistance were also found to be the QTL with the greatest effects on resistance to Ralstonia. This finding partially explains previously observed associations between resistances to these two pathogens among U.S. current cultivars and within breeding populations. Further study is needed to determine whether these relationships are due to the same genes (i.e., pleiotropy) or favorable coupling-phase linkages that have been established over time.

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Vignaroli, C., G. M. Luna, C. Rinaldi, A. Di Cesare, R. Danovaro, and F. Biavasco. "New Sequence Types and Multidrug Resistance among Pathogenic Escherichia coli Isolates from Coastal Marine Sediments." Applied and Environmental Microbiology 78, no. 11 (2012): 3916–22. http://dx.doi.org/10.1128/aem.07820-11.

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ABSTRACTThe spread of antibiotic-resistant microorganisms is widely recognized, but data about their sources, presence, and significance in marine environments are still limited. We examined 109Escherichia colistrains from coastal marine sediments carrying virulence genes for antibiotic susceptibility, specific resistance genes, prevalence of class 1 and 2 integrons, and sequence type. Antibiotic resistance was found in 35% of strains, and multiple resistances were found in 14%; the resistances detected most frequently were against tetracycline (28%), ampicillin (16.5%), trimethoprim-sulfamethoxazole (13%), and streptomycin (7%). The highest prevalence of resistant strains was in phylogenetic group A, whereas phylogroup B2 exhibited a significantly lower frequency than all the other groups. Sixty percent of multiresistant strains harbored class 1 or 2 integrase genes, and about 50% carried resistance genes (particularlydfrAandaadA) linked to a class 1 integron. Multilocus sequence typing of 14 selected strains identified eight different types characteristic of extraintestinal pathogens and three new allelic combinations. Our data suggest that coastal marine sediment may be a suitable environment for the survival of pathogenic and antimicrobial-resistantE. colistrains capable of contributing to resistance spread via integrons among benthic bacteria, and they highlight a role for these strains in the emergence of new virulent genotypes.

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Choi, Jaehyeon, Jaebong Sim, Hwanyeong Oh, and Kyoungdoug Min. "Resistance Separation of Polymer Electrolyte Membrane Fuel Cell by Polarization Curve and Electrochemical Impedance Spectroscopy." Energies 14, no. 5 (2021): 1491. http://dx.doi.org/10.3390/en14051491.

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The separation of resistances during their measurement is important because it helps to identify contributors in polymer electrolyte membrane (PEM) fuel cell performance. The major methodologies for separating the resistances are electrochemical impedance spectroscopy (EIS) and polarization curves. In addition, an equivalent circuit was selected for EIS analysis. Although the equivalent circuit of PEM fuel cells has been extensively studied, less attention has been paid to the separation of resistances, including protonic resistance in the cathode catalyst layer (CCL). In this study, polarization curve and EIS analyses were conducted to separate resistances considering the charge transfer resistance, mass transport resistance, high frequency resistance, and protonic resistance in the CCL. A general solution was mathematically derived using the recursion formula. Consequently, resistances were separated and analyzed with respect to variations in relative humidity in the entire current density region. In the case of ohmic resistance, high frequency resistance was almost constant in the main operating load range (0.038–0.050 Ω cm2), while protonic resistance in the CCL exhibited sensitivity (0.025–0.082 Ω cm2) owing to oxygen diffusion and water content.

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Potter, Carol. "Resistance." Iowa Review 16, no. 2 (1986): 112–13. http://dx.doi.org/10.17077/0021-065x.3392.

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33

MIYAMOTO, Toru, Tadashi MIYATA, Kazuyuki SAKATA, and Yasuhiko KONNO. "RESISTANCE." Journal of Pesticide Science 19, Special (1994): S340—S345. http://dx.doi.org/10.1584/jpestics.19.special_s340.

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34

Sartre, Jean-Paul. "Resistance." Sartre Studies International 8, no. 2 (2002): 1–19. http://dx.doi.org/10.3167/135715502781824993.

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35

Heck, Mauro. "Resistance." Iowa Journal of Literary Studies 10, no. 1 (1989): 50. http://dx.doi.org/10.17077/0743-2747.1297.

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36

Seymour, Susan. "Resistance." Anthropological Theory 6, no. 3 (2006): 303–21. http://dx.doi.org/10.1177/1463499606066890.

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37

Sheaffer-Jones, Caroline. "Resistance." Romanic Review 108, no. 1-4 (2017): 89–91. http://dx.doi.org/10.1215/26885220-108.1-4.89.

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Thomas Haley. "Resistance." Fourth Genre: Explorations in Nonfiction 4, no. 1 (2002): 34–48. http://dx.doi.org/10.1353/fge.2013.0150.

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Subbiah, Shanmuga, and R. Parthasarathy. "Resistance." World Literature Today 68, no. 2 (1994): 252. http://dx.doi.org/10.2307/40150134.

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Deetz, Stanley. "Resistance." Management Communication Quarterly 21, no. 3 (2008): 387–92. http://dx.doi.org/10.1177/0893318907310943.

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Murray, Jeff B., and Zafeirenia Brokalaki. "Resistance." Journal of Customer Behaviour 18, no. 4 (2019): 338–47. http://dx.doi.org/10.1362/147539219x15774563471928.

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Beutler, Larry E., Francisco Rocco, Carla M. Moleiro, and Hani Talebi. "Resistance." Psychotherapy: Theory, Research, Practice, Training 38, no. 4 (2001): 431–36. http://dx.doi.org/10.1037/0033-3204.38.4.431.

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Bulajic, Snezana, Zora Mijacevic, and Radoslava Savic-Radovanovic. "Antibiotic resistance of lactic acid bacteria." Veterinarski glasnik 62, no. 5-6 (2008): 329–40. http://dx.doi.org/10.2298/vetgl0806329b.

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Annotation:

Knowledge on the antibiotic resistance of lactic acid bacteria is still limited, possibly because of the large numbers of genera and species encountered in this group, as well as variances in their resistance spectra. The EFSA considers antibiotic resistances, especially transferable resistances, an important decision criterion for determining a strain's QPS status. There are no approved standards for the phenotypic or genotypic evaluation of antibiotic resistances in food isolates. Also, the choice of media is problematic, as well as the specification of MIC breakpoint values as a result of the large species variation and the possible resulting variation in MIC values between species and genera. The current investigations in this field showed that we might end up with a range of different species- or genus-specific breakpoint values that may further increase the current complexity. Another problem associated with safety determinations of starter strains is that once a resistance phenotype and an associated resistance determinant have been identified, it becomes difficult to show that this determinant is not transferable, especially if the resistance gene is not located on a plasmid and no standard protocols for showing genetic transfer are available. Encountering those problems, the QPS system should allow leeway for the interpretations of results, especially when these relate to the methodology for resistance phenotype determinations, determinations of MIC breakpoints for certain genera, species, or strains, the nondeterminability of a genetic basis of a resistance phenotype and the transferability of resistance genes.

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Paramana, Katerina. "On Resistance through Ruptures and the Rupture of Resistances." Performance Research 19, no. 6 (2014): 81–89. http://dx.doi.org/10.1080/13528165.2014.985112.

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Noda, Shunichi. "Combined Resistance of Resistance Lattice Circuit." IEEJ Transactions on Fundamentals and Materials 119, no. 1 (1999): 62–68. http://dx.doi.org/10.1541/ieejfms1990.119.1_62.

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Gopalan, Suresh, and Sheng Yang He. "Disease resistance: beyond the resistance genes." Trends in Plant Science 3, no. 6 (1998): 207–8. http://dx.doi.org/10.1016/s1360-1385(98)01243-6.

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Hooper, David C., and George A. Jacoby. "Mechanisms of drug resistance: quinolone resistance." Annals of the New York Academy of Sciences 1354, no. 1 (2015): 12–31. http://dx.doi.org/10.1111/nyas.12830.

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Tran, Truc T., Jose M. Munita, and Cesar A. Arias. "Mechanisms of drug resistance: daptomycin resistance." Annals of the New York Academy of Sciences 1354, no. 1 (2015): 32–53. http://dx.doi.org/10.1111/nyas.12948.

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Moyle, G. "Resistance and cross-resistance to abacavir." HIV Medicine 2, no. 3 (2001): 154–62. http://dx.doi.org/10.1046/j.1468-1293.2001.00069.x.

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Coyle, Maureen. "Understanding Resistance to Climate Change Resistance." International Journal of Aging and Human Development 80, no. 1 (2014): 76–86. http://dx.doi.org/10.1177/0091415015591111.

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