Auswahl der wissenschaftlichen Literatur zum Thema „Division et mort cellulaire“
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Zeitschriftenartikel zum Thema "Division et mort cellulaire"
Golstein, P. „Mort programmée et terrain cellulaire“. médecine/sciences 7, Nr. 7 (1991): 681. http://dx.doi.org/10.4267/10608/4434.
Der volle Inhalt der QuelleHerbomel, Philippe. „Mort cellulaire programmée et embryogenèse“. Revue Française des Laboratoires 1999, Nr. 311 (März 1999): 31–34. http://dx.doi.org/10.1016/s0338-9898(99)80034-1.
Der volle Inhalt der QuelleLafaurie-Janvore, Julie, und Matthieu Piel. „Mécanique et division cellulaire“. médecine/sciences 29, Nr. 12 (Dezember 2013): 1089–91. http://dx.doi.org/10.1051/medsci/20132912009.
Der volle Inhalt der QuelleAmeisen, Jean Claude. „« Nous vivons dans l’oubli de nos métamorphoses… »: La mort et la sculpture du vivant“. Annales. Histoire, Sciences Sociales 62, Nr. 6 (Dezember 2007): 1251–83. http://dx.doi.org/10.1017/s0395264900036209.
Der volle Inhalt der QuelleNougarède, Arlette, Pierre Rondet, Pierre Landré und Jacques Rembur. „Effet d'un traitement par l'acide abscisique sur la division cellulaire, les teneurs en ADN et l'élongation du bourgeon cotylédonaire de plants de pois décapités“. Canadian Journal of Botany 65, Nr. 5 (01.05.1987): 907–15. http://dx.doi.org/10.1139/b87-125.
Der volle Inhalt der QuelleBauchot, Roland. „Encéphalisation et mort cellulaire chez les Vertébrés“. Geobios 22 (Januar 1989): 59–66. http://dx.doi.org/10.1016/s0016-6995(89)80007-5.
Der volle Inhalt der QuelleHasler, Julien, Claude Penel, Thomas Gaspar und Michèle Crèvecœur. „Mort cellulaire programmée, apoptose, …et cellules végétales“. L’Année Biologique 40 (Dezember 2001): 75–95. http://dx.doi.org/10.1016/s0003-5017(01)72086-7.
Der volle Inhalt der QuelleFavaudon, V. „Régulation du cycle cellulaire et de la mort cellulaire radio-induite“. Cancer/Radiothérapie 4, Nr. 5 (September 2000): 355–68. http://dx.doi.org/10.1016/s1278-3218(00)00009-3.
Der volle Inhalt der QuelleMagusto, Julie, Amine Majdi und Jérémie Gautheron. „Les mécanismes de mort cellulaire dans la stéatohépatite non alcoolique“. Biologie Aujourd’hui 214, Nr. 1-2 (2020): 1–13. http://dx.doi.org/10.1051/jbio/2020002.
Der volle Inhalt der QuelleBirsen, Rudy, Eric Grignano, Nicolas Chapuis und Didier Bouscary. „Ferroptose et cancer“. médecine/sciences 37, Nr. 8-9 (August 2021): 726–34. http://dx.doi.org/10.1051/medsci/2021108.
Der volle Inhalt der QuelleDissertationen zum Thema "Division et mort cellulaire"
Op, De Beeck Anne. „Etude du mode d'action cytotoxique de la protéine non structurale NS1 du parvovirus oncolytique MVMp: interférence avec la division cellulaire ou Chronique d'une mort annoncée“. Doctoral thesis, Universite Libre de Bruxelles, 1996. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/212389.
Der volle Inhalt der QuelleFarinas, Benoît. „L'algue Ostreococcus tauri, modèle émergent pour la caractérisation des principaux acteurs du cycle de division et de la mort cellulaire programmée dans la lignée verte“. Perpignan, 2006. http://www.theses.fr/2006PERP0699.
Der volle Inhalt der QuelleThe unicellular marine alga Ostreococcus tauri (Prasinophyceae) diverged early in evolution of green plants. O. Tauri has a very simple cellular organisation, a simple binary division and a minimal yet complete set of cell cycle control genes. Heterotrophic growth in darkness was investigated but no proliferation of O. Tauri was observed with the carbon sources tested. However, glycerol increased survival in the dark, albeit without cell division. In all conditions, cell death was observed and our results suggest occurrence of programmed cell death. Cell culture techniques were developed to optimise further work, for example for gene expression studies. Axenic cultures, with natural synchronisation by light/dark cycles was necessary for studying cell cycle regulation. O. Tauri division appeared to be fairly typical of eukaryotes. A PSTAIRE-type CDK (CDKA in plants) in the O. Tauri genome was found to be implicated in cell division. The relevant mRNA and proteins were present constitutively but with a peak of histone H1 activity early in cell cycle, suggesting a role in the G1/S transition and perhaps in the G2/M transition. As in other members of the plant kingdom, O. Tauri possesses a B-type CDK, highly regulated throughout the cell cycle, with a kinase activity occurring after OtCDKA activation, suggesting a mitotic role of OtCDKB. Although the PSTAIRE-type CDK is phosphorylated to inhibit the G2/M transition in animals, surprisingly, a tyrosine phosphorylation was detected only on OtCDKB
Chalabi, Asma. „Processus d'analyse dynamique pour l'imagerie de cellules vivantes permettant la détection des réponses cellulaires aux anticancéreux, par traitement de l'image et du signal et apprentissage automatique profond“. Electronic Thesis or Diss., Université Côte d'Azur, 2024. http://www.theses.fr/2024COAZ6004.
Der volle Inhalt der QuelleCell division and cell death are the main indicators to evaluate cancer drug action, and only their accurate measures can reveal the actual potency and efficacy of a compound. The detection of cell division and cell death events in live-cell assays has the potential to produce robust metrics of drug pharmacodynamics and return a more comprehensive understanding of tumor cells responses to cancer therapeutic combinations. Knowing precisely when a cell death or a cell division occurs in a live-cell experiment allows to study the relative contribution of different drug effects -such as cytotoxic or cytostatic effects, on a cell population. Yet, classical methods require dyes to measure cell viability as an end-point assay with whole population counts, where the proliferation rates can only be estimated when both viable and dead cells are labeled simultaneously.Live-cell imaging is a promising cell-based assay to determine drug efficacies, with the main limitation being the accuracy and depth of the analyses to detect and predict automatically cellular response phenotypes (cell death and division, which share some morphological features).This thesis introduces a method integrating deep learning using neural networks, and image and signal processing to perform dynamic image analyses of single-cell events in time-lapse microscopy experiments of drug pharmacological profiling. This method works by automatically tracking the cells, extracting radiometric and morphologic cell features, and analyzing the temporal evolution of these features for each cell so as to detect cellular events such as division and cell death, as well as acquiring signaling pathway dynamics.A case of study comprising the analyses of caspase-8 single-cell dynamics and other cell responses to cancer drugs is presented. The aim is to achieve automatically, at a large scale the necessary analyses to augment the phenotype prediction method available in the lab (Fateseq) and to apply it to various cancer cell lines of a human cancer cell line panel to improve our live-cell OMICS profiling approaches, and, in a longer term, to scale up pharmacological screening of new cancer drugs
FERNANDEZ, PIERRE-ALAIN. „Analyse cellulaire et moleculaire de la mort cellulaire programmee des vertebres“. Paris 6, 1995. http://www.theses.fr/1995PA066321.
Der volle Inhalt der QuelleDEPRAETERE, VALERIE. „Recherche de molecules signalisatrices de la mort cellulaire developpementale et de la mort cellulaire induite par irradiation“. Aix-Marseille 2, 1999. http://www.theses.fr/1999AIX22016.
Der volle Inhalt der QuelleRaoul, Cédric. „Mort développementale et pathologique du motoneurone spinal : Implication du récepteur de mort Fas (CD95)“. Aix-Marseille 2, 2002. http://www.theses.fr/2002AIX22018.
Der volle Inhalt der QuelleMorel, Jean-Benoît. „Analyse genetique et moleculaire de la mort cellulaire et de suppresseurs de la mort cellulaire en relation avec la reponse hypersensible chez arabidopsis thaliana“. Paris 11, 1998. http://www.theses.fr/1998PA112236.
Der volle Inhalt der QuelleMoreira, Wilfried. „Stress oxydatif, différentiation et mort cellulaire chez le parasite Leishmania“. Thesis, Université Laval, 2011. http://www.theses.ulaval.ca/2011/28186/28186.pdf.
Der volle Inhalt der QuelleOizel, Kristell. „Métabolisme et sensibilité à la mort cellulaire dans le glioblastome“. Nantes, 2015. https://archive.bu.univ-nantes.fr/pollux/show/show?id=a1401556-8497-464e-bdda-d0068fe3297e.
Der volle Inhalt der QuelleTumor cells undergo metabolic adaptations allowing them to sustain a high proliferative rate and to resist to cell death signals, especially increasing aerobic glycolysis and glutaminolysis. Glioblastoma multiforme (GBM), the most common brain tumor in adults, is characterized by a strong resistance to therapeutic treatments and the presence of cancer stem cells. This PhD project investigated the link between metabolism and cell death sensitivity in GBM. First, we studied the impact of isocitrate dehydrogenase (IDH) mutation recently identified in GBM patients. We show that mutated IDH induces a reduced sensitivity to etoposide-induced cell death mediated through a mitochondrial NADH pool reduction. Second, we aimed to determine if glutaminolysis inhibition could modulate cell death response in GBM tumor model. We show that epigallocatechin gallate (EGCG), an inhibitor of glutamate dehydrogenase (GDH), can sensitize GBM cell lines to cell death. Furthermore, in primary cultures models, EGCG sensitize the GBM mesenchymal subtype to cell death. This cellular model derived directly from patients tumors allows to keep the initial tumor heterogeneity, in particular the presence of cancer stem cells. These results show a direct link between metabolism and cell death resistance and open new therapeutic strategies. Thus EGCG could be a good candidate as an adjuvant in current GBM therapy in the context of personalized treatment
Larmonier, Nicolas. „Mort des cellules cancereuses et réponse immunitaire antitumorale“. Dijon, 2004. http://www.theses.fr/2004DIJOMU01.
Der volle Inhalt der QuelleBücher zum Thema "Division et mort cellulaire"
Desaint, Nilsy. Mort du père et place de la femme au Japon: Crise du modèle patriarcal et égalité des sexes dans le Japon contemporain. Paris: Harmattan, 2007.
Den vollen Inhalt der Quelle findenBourguet, Julien, A. Berkaloff und J. C. Lacroix. Biologie et physiologie cellulaires, tome 3. Chloroplastes, peroxysomes, division cellulaire. Hermann, 1997.
Den vollen Inhalt der Quelle findenHopkins, William D., Cheryl D. Stimpson und Chet C. Sherwood. Social cognition and brain organization in chimpanzees (Pan troglodytes) and bonobos (Pan paniscus). Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198728511.003.0014.
Der volle Inhalt der QuelleBuchteile zum Thema "Division et mort cellulaire"
Silveira, Léa. „V. Suicide cellulaire et pulsion de mort. À partir de La sculpture du vivant, de J.C. Ameisen“. In Pulsion de mort, 83–92. Hermann, 2023. http://dx.doi.org/10.3917/herm.santo.2023.01.0083.
Der volle Inhalt der QuelleFrançois, Arnaud. „La division de la vie : création, conservation et pulsion de mort chez Bergson et Freud“. In Bergson et Freud, 121. Presses Universitaires de France, 2014. http://dx.doi.org/10.3917/puf.sitbo.2014.01.0121.
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