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Thematische Bibliographien / Diseases / Zeitschriftenartikel

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Zeitschriftenartikel zum Thema „Diseases“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 30. Juli 2024

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1

Limpert, E., und P. Bartoš. „Wind-Dispersed Nomadic Diseases: Conclusions for Disease Resistance“. Czech Journal of Genetics and Plant Breeding 38, No. 3-4 (01.08.2012): 150–52. http://dx.doi.org/10.17221/6256-cjgpb.

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2

Dehuri, Priyadarshini, Debasis Gochhait, Debdatta Basu und Neelaiah Siddaraju. „Rosai-Dorfman Disease: An Imposter of Plasma Cell Rich Diseases“. Annals of Pathology and Laboratory Medicine 2, Nr. 12 (17.12.2018): C178–181. http://dx.doi.org/10.21276/apalm.2102.

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3

Balaji, SM. „Noncommunicable diseases and dental diseases“. Indian Journal of Dental Research 29, Nr. 6 (2018): 699. http://dx.doi.org/10.4103/ijdr.ijdr_895_18.

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4

Heinz, Don J., und Steve A. Ferreira. „Diseases of sugarcane. Major diseases“. Field Crops Research 19, Nr. 1 (August 1988): 77–78. http://dx.doi.org/10.1016/0378-4290(88)90037-8.

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5

MATSUMOTO, KEIZO. „Emerging infectious diseases and insensible bacillus infectious diseases. Emerging infectious diseases. Influenza virus infection diseases.“ Nihon Naika Gakkai Zasshi 86, Nr. 11 (1997): 2033–38. http://dx.doi.org/10.2169/naika.86.2033.

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6

Kadirovna, Muratova Saodat, Shukurova Nodira Tillayevna, Baratov Bobur und Teshayev Shoxjahon. „PREDICTIVE MODELING OF THE PROBABILITY OF DEVELOPING PERIODONTAL DISEASES IN PATIENTS WITH CARDIOVASCULAR DISEASE“. European International Journal of Multidisciplinary Research and Management Studies 4, Nr. 4 (01.04.2024): 65–70. http://dx.doi.org/10.55640/eijmrms-04-04-10.

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Currently, the forecast of the development of pathology is an important part of all branches of healthcare. [3,4,5]. However, despite the importance and scientific and practical significance of forecasting in dentistry, at present we have not found information about predictive models of individual risk of developing periodontitis in patients with hypertension.

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7

&NA;. „Diseases“. Inpharma Weekly &NA;, Nr. 872 (Januar 1993): 5. http://dx.doi.org/10.2165/00128413-199308720-00007.

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8

Kumagai, Shunichi. „7. Collagen Diseases and Allergic Diseases“. Nihon Naika Gakkai Zasshi 97, Nr. 12 (2008): 2991–97. http://dx.doi.org/10.2169/naika.97.2991.

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9

Tatsumi, Koichiro. „Respiratory diseases as lifestyle-related diseases“. Health Evaluation and Promotion 39, Nr. 6 (2012): 821–28. http://dx.doi.org/10.7143/jhep.39.821.

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10

Arma, Utmi, und Nadhifah Salsabila. „Peri-Implant Diseases and Gastrointestinal Diseases“. Archives of Orofacial Sciences 16, Supp. 1 (22.09.2021): 1–4. http://dx.doi.org/10.21315/aos2021.16.s1.1.

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Peri-implant diseases are serious problems that plagues today’s dentistry, both in terms of therapy and epidemiology. With the expansion of implantology practice and the increasing number of implants placed annually, the frequency of peri-implant diseases has greatly expanded. The clinical manifestations, in the absence of a globally established classification, are peri-implant mucositis and peri-implantitis, the counterparts of gingivitis and periodontitis, respectively. However, many doubts remain about their features. Official diagnostic criteria, globally recognised by the dental community, have not yet been introduced. The review presented possible association between gastrointestinal diseases and peri-implant diseases. Previous studies had revealed the association with significantly higher levels of bacteria in patient’s gastrointestinal disease at either gingivitis or in periodontitis site. Additionally, pathogenesis of the periodontitis is similar to peri-implant diseases.

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11

Yokoyama, Takashi, und Shirou Mohri. „Prion Diseases and Emerging Prion Diseases“. Current Medicinal Chemistry 15, Nr. 9 (01.04.2008): 912–16. http://dx.doi.org/10.2174/092986708783955437.

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12

Gorbunova, Victoria N. „Congenital metabolic diseases. Lysosomal storage diseases“. Pediatrician (St. Petersburg) 12, Nr. 2 (11.08.2021): 73–83. http://dx.doi.org/10.17816/ped12273-83.

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The classification and epidemiology of hereditary metabolic disorders are presented. That is a large group consisting from more them 800 monogenic diseases, each of which caused by inherited deficiency of certain metabolic fate. Many of these disorders are extremely rare, but their total incidence in the population is close to 1:10005000. Lysosomal storage diseases (LSD) resulting from inherited deficiency in lysosomal functions occupy a special place among hereditary metabolic disorders. The defects of catabolism cause the accumulation of undigested or partially digested macromolecules in lysosomes (that is, storage), which can result in cellular damage. About 60 diseases take part in this group with total incidence of about 1:70008000. LSDs typically present in infancy and childhood, although adult-onset forms also occur. Most of them have a progressive neurodegenerative clinical course, although symptoms in other organ systems are frequent. The etiology and pathogenetic aspects of their main clinical entities: mucopolysaccharidosis, glycolipidosis, mucolipidosis, glycoproteinosis, etc, are presented. Mucopolysaccharidoses caused by malfunctioning of lysosomal enzymes needed to break down glycosaminoglycans are more frequent among LSD. Sphingolipidoses caused by defects of lipid catabolism are second for frequency group of LSD. The state-of-art in field of newborn screening. clinical, biochemical and molecular diagnostics of these grave diseases are discussed. The main directions of modern lysosomal storage diseases therapy are characterized: transplantation of hematopoietic stem cells; enzyme replacement therapy; therapy with limitation of substrate synthesis (substrate-reducing therapy); pharmacological chaperone therapy. Perspective directions for LSD therapy are gene therapy and genome editing which are at advanced preclinical stages.

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13

Paramjyothi, G. „Respiratory Diseases and Coronary Artery Diseases“. Indian Journal of Cardiovascular Disease in Women WINCARS 02, Nr. 01 (März 2017): 056–67. http://dx.doi.org/10.1055/s-0038-1656461.

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14

Wurtz, Rebecca. „Psychiatric Diseases Presenting as Infectious Diseases“. Clinical Infectious Diseases 26, Nr. 4 (April 1998): 924–32. http://dx.doi.org/10.1086/513936.

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15

Ravilochan, K., und Kenneth Tyler. „Human Transmissible Neurodegenerative Diseases (Prion Diseases)“. Seminars in Neurology 12, Nr. 03 (September 1992): 178–92. http://dx.doi.org/10.1055/s-2008-1041174.

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16

Lee, Jeongmin, Su Yeon Kim, Kyu Jam Hwang, Young Ran Ju und Hee-Jong Woo. „Prion Diseases as Transmissible Zoonotic Diseases“. Osong Public Health and Research Perspectives 4, Nr. 1 (Februar 2013): 57–66. http://dx.doi.org/10.1016/j.phrp.2012.12.008.

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17

Mizuno, T. „1299 – Are mental diseases synaptic diseases?“ European Psychiatry 28 (Januar 2013): 1. http://dx.doi.org/10.1016/s0924-9338(13)76357-0.

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18

IWAMOTO, AIKICHI. „Infectious diseases. 1. Emerging infectious diseases and re-merging infectious diseases.“ Nihon Naika Gakkai Zasshi 88, Nr. 3 (1999): 517–21. http://dx.doi.org/10.2169/naika.88.517.

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19

Deiana, Giovanna, Antonella Arghittu, Marco Dettori und Paolo Castiglia. „One World, One Health: Zoonotic Diseases, Parasitic Diseases, and Infectious Diseases“. Healthcare 12, Nr. 9 (29.04.2024): 922. http://dx.doi.org/10.3390/healthcare12090922.

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When we take into account how the boundaries between human, animal, and environmental health are inextricably linked and increasingly intertwined, it comes as no surprise that the One Health approach has assumed an unprecedented level of importance over the past decade [...]

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20

Prasad, Dr S. R. „Neglected Tropical Diseases“. JOURNAL OF CLINICAL AND BIOMEDICAL SCIENCES 03, Nr. 1 (15.03.2013): 1–2. http://dx.doi.org/10.58739/jcbs/v03i1.7.

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21

Smith, John A. „Noninfectious Diseases, Metabolic Diseases, Toxicities, and Neoplastic Diseases of South American Camelids“. Veterinary Clinics of North America: Food Animal Practice 5, Nr. 1 (März 1989): 101–43. http://dx.doi.org/10.1016/s0749-0720(15)31006-9.

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22

Ramamurthy, Mahesh babu, Daniel Y. T. Goh und Michael Teik Chung Lim. „Rare Lung Diseases: Interstitial Lung Diseases and Lung Manifestations of Rheumatological Diseases“. Indian Journal of Pediatrics 82, Nr. 10 (20.08.2015): 956–61. http://dx.doi.org/10.1007/s12098-015-1867-3.

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23

Sari, Refik Ali. „Interstitial Lung Diseases In Collagen Vascular Diseases“. Güncel Göğüs Hastalıkları Serisi 2, Nr. 3 (18.01.2016): 358–73. http://dx.doi.org/10.5152/gghs.2014.026.

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24

Wang, Chrong-Reen, und Hung-Wen Tsai. „Autoimmune liver diseases in systemic rheumatic diseases“. World Journal of Gastroenterology 28, Nr. 23 (21.06.2022): 2527–45. http://dx.doi.org/10.3748/wjg.v28.i23.2527.

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25

Moutsopoulos, Haralampos M. „Autoimmune rheumatic diseases: One or many diseases?“ Journal of Translational Autoimmunity 4 (2021): 100129. http://dx.doi.org/10.1016/j.jtauto.2021.100129.

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26

Kouanda, Bakey, Zeeshan Sattar und Patrick Geraghty. „Periodontal Diseases: Major Exacerbators of Pulmonary Diseases?“ Pulmonary Medicine 2021 (02.11.2021): 1–10. http://dx.doi.org/10.1155/2021/4712406.

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Periodontal diseases are a range of polymicrobial infectious disorders, such as gingivitis and periodontitis, which affect tooth-supporting tissues and are linked to playing a role in the exacerbation of several pulmonary diseases. Pulmonary diseases, such as pneumonia, chronic obstructive pulmonary disease (COPD), asthma, tuberculosis, COVID-19, and bronchiectasis, significantly contribute to poor quality of life and mortality. The association between periodontal disease and pulmonary outcomes is an important topic and requires further attention. Numerous resident microorganisms coexist in the oral cavity and lungs. However, changes in the normal microflora due to oral disease, old age, lifestyle habits, or dental intervention may contribute to altered aspiration of oral periodontopathic bacteria into the lungs and changing inflammatory responses. Equally, periodontal diseases are associated with the longitudinal decline in spirometry lung volume. Several studies suggest a possible beneficial effect of periodontal therapy in improving lung function with a decreased frequency of exacerbations and reduced risk of adverse respiratory events and morbidity. Here, we review the current literature outlining the link between the oral cavity and pulmonary outcomes and focus on the microflora of the oral cavity, environmental and genetic factors, and preexisting conditions that can impact oral and pulmonary outcomes.

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27

Jeong, Sung Hwan. „Interstitial Lung Diseases in Collagen Vascular Diseases“. Journal of the Korean Medical Association 52, Nr. 1 (2009): 30. http://dx.doi.org/10.5124/jkma.2009.52.1.30.

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28

Linssen, A., und T. E. Feltkamp. „B27 positive diseases versus B27 negative diseases.“ Annals of the Rheumatic Diseases 47, Nr. 5 (01.05.1988): 431–39. http://dx.doi.org/10.1136/ard.47.5.431.

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29

Levine, Jerome F. „Infectious Diseases: Hot Topics:Infectious Diseases: Hot Topics“. Clinical Infectious Diseases 38, Nr. 8 (15.04.2004): 1197–98. http://dx.doi.org/10.1086/383064.

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30

Mandal, B. K., und P. F. Schofield. „ABC of colorectal diseases. Tropical colonic diseases.“ BMJ 305, Nr. 6854 (12.09.1992): 638–41. http://dx.doi.org/10.1136/bmj.305.6854.638.

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31

Sand, Freja Lærke, und Simon Francis Thomsen. „Skin diseases of the vulva: Infectious diseases“. Journal of Obstetrics and Gynaecology 37, Nr. 7 (11.04.2017): 840–48. http://dx.doi.org/10.1080/01443615.2017.1306696.

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32

Aronson, Jeff. „Rare diseases, orphan drugs, and orphan diseases“. BMJ 333, Nr. 7559 (13.07.2006): 127. http://dx.doi.org/10.1136/bmj.333.7559.127.

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33

Dorri, M. „Periodontal diseases: New classification for periodontal diseases“. British Dental Journal 225, Nr. 8 (Oktober 2018): 686. http://dx.doi.org/10.1038/sj.bdj.2018.941.

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34

Saudubray, J. M. „Status of lysosomal diseases amongst metabolic diseases“. La Revue de Médecine Interne 27 (März 2006): S11—S13. http://dx.doi.org/10.1016/s0248-8663(06)80004-4.

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35

FUKUDA, Yoshihiro. „Cytokines and diseases. Cytokines and liver diseases.“ Nihon Naika Gakkai Zasshi 80, Nr. 9 (1991): 1409–13. http://dx.doi.org/10.2169/naika.80.1409.

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36

Kanegane, Hirokazu. „Inflammatory bowel diseases and primary immunodeficiency diseases“. Immunological Medicine 41, Nr. 4 (02.10.2018): 154–61. http://dx.doi.org/10.1080/25785826.2018.1556025.

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37

Weidlich, Patrícia, Renata Cimões, Claudio Mendes Pannuti und Rui Vicente Oppermann. „Association between periodontal diseases and systemic diseases“. Brazilian Oral Research 22, suppl 1 (August 2008): 32–43. http://dx.doi.org/10.1590/s1806-83242008000500006.

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38

Shaw, Elizabeth A., und Anne M. Stevens. „Are pediatric autoimmune diseases primarily genetic diseases?“ Current Opinion in Rheumatology 20, Nr. 5 (September 2008): 589–94. http://dx.doi.org/10.1097/bor.0b013e328307f283.

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39

Withrock, Isabelle C., Stephen J. Anderson, Matthew A. Jefferson, Garrett R. McCormack, Gregory S. A. Mlynarczyk, Aron Nakama, Jennifer K. Lange et al. „Genetic diseases conferring resistance to infectious diseases“. Genes & Diseases 2, Nr. 3 (September 2015): 247–54. http://dx.doi.org/10.1016/j.gendis.2015.02.008.

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40

Casella, Giovanni, Vincenzo Villanacci, Camillo Di Bella, Elisabetta Antonelli, Vittorio Baldini und Gabrio Bassotti. „Pulmonary diseases associated with inflammatory bowel diseases“. Journal of Crohn's and Colitis 4, Nr. 4 (Oktober 2010): 384–89. http://dx.doi.org/10.1016/j.crohns.2010.02.005.

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41

PR, Nandeesh Kumar. „Anti-neurofascin demyelinating diseases (Anti-NF diseases)“. International Journal of Advance Research in Medical Surgical Nursing 6, Nr. 1 (01.01.2024): 26–29. http://dx.doi.org/10.33545/surgicalnursing.2024.v6.i1a.164.

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42

Ha, Tai-You. „MicroRNAs in Human Diseases: From Autoimmune Diseases to Skin, Psychiatric and Neurodegenerative Diseases“. Immune Network 11, Nr. 5 (2011): 227. http://dx.doi.org/10.4110/in.2011.11.5.227.

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43

KATSUMATA, TATSUYA. „Emerging infectious diseases and insensible bacillus infectious diseases. Emerging infectious diseases. Cryptosporidium infection.“ Nihon Naika Gakkai Zasshi 86, Nr. 11 (1997): 2058–63. http://dx.doi.org/10.2169/naika.86.2058.

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44

Oxley, Dwight K. „Endocrine Diseases“. Archives of Pathology & Laboratory Medicine 127, Nr. 3 (01.03.2003): 381–82. http://dx.doi.org/10.5858/2003-127-0381-ed.

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45

Zelmer, Derek A., D. D. Despommier, R. W. Gwadz, P. J. Hotez und C. A. Knirsch. „Parasitic Diseases“. Journal of Parasitology 87, Nr. 6 (Dezember 2001): 1414. http://dx.doi.org/10.2307/3285310.

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46

Araujo, Abelardo Q.-C. „Prionic diseases“. Arquivos de Neuro-Psiquiatria 71, Nr. 9B (September 2013): 731–37. http://dx.doi.org/10.1590/0004-282x201301461.

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Prion diseases are neurodegenerative illnesses due to the accumulation of small infectious pathogens containing protein but apparently lacking nucleic acid, which have long incubation periods and progress inexorably once clinical symptoms appear. Prions are uniquely resistant to a number of normal decontaminating procedures. The prionopathies [Kuru, Creutzfeldt-Jakob disease (CJD) and its variants, Gerstmann-Sträussler-Scheinker (GSS) syndrome and fatal familial insomnia (FFI)] result from accumulation of abnormal isoforms of the prion protein in the brains of normal animals on both neuronal and non-neuronal cells. The accumulation of this protein or fragments of it in neurons leads to apoptosis and cell death. There is a strong link between mutations in the gene encoding the normal prion protein in humans (PRNP) - located on the short arm of chromosome 20 – and forms of prion disease with a familial predisposition (familial CJD, GSS, FFI). Clinically a prionopathy should be suspected in any case of a fast progressing dementia with ataxia, myoclonus, or in individuals with pathological insomnia associated with dysautonomia. Magnetic resonance imaging, identification of the 14-3-3 protein in the cerebrospinal fluid, tonsil biopsy and genetic studies have been used for in vivo diagnosis circumventing the need of brain biopsy. Histopathology, however, remains the only conclusive method to reach a confident diagnosis. Unfortunately, despite numerous treatment efforts, prionopathies remain short-lasting and fatal diseases.

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47

Gamage, R. „Prion diseases“. Ceylon Medical Journal 45, Nr. 1 (20.01.2015): 3. http://dx.doi.org/10.4038/cmj.v45i1.7941.

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48

Rameev, V. V., L. V. Lysenko (Kozlovskaya), M. V. Bogdanova und S. V. Moiseev. „Autoinflammatory diseases“. Clinical pharmacology and therapy 29, Nr. 4 (15.11.2020): 49–60. http://dx.doi.org/10.32756/0869-5490-2020-4-49-60.

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Autoinflammatory diseases are a group of disorders caused by a dysregulation of the innate immune system. Unlike autoimmune diseases, they are not associated with changes in humoral or cellular immunity. The authors review the current classification, clinical manifestations and treatment of various systemic autoinflammatory diseases, including cryopirinassociated periodic syndrome, familial Mediterranean fever, HIDS, and TRAPS.

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49

Palm, Mary E., und R. J. Hillocks. „Cotton Diseases“. Mycologia 86, Nr. 2 (März 1994): 305. http://dx.doi.org/10.2307/3760659.

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50

Perry, Martha, und Bianca A. Allison. „Gonorrheal Diseases“. Pediatrics in Review 39, Nr. 8 (August 2018): 427–29. http://dx.doi.org/10.1542/pir.2017-0120.

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