Dissertationen zum Thema „Diabetes resistance“
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Pickavance, Lucy Cecilia. „Thiazolidinedione treatment in models of insulin resistance“. Thesis, University of Liverpool, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367553.
Der volle Inhalt der QuelleAng, Christine W. M. „Diabetes and the maternal resistance vasculature“. Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.395080.
Der volle Inhalt der QuelleHale, P. J. „Insulin resistance in diabetes mellitus and obesity“. Thesis, University of Oxford, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.371516.
Der volle Inhalt der QuelleEason, Robert C. „Treating type 2 diabetes through insulin resistance“. Thesis, Aston University, 2002. http://publications.aston.ac.uk/10953/.
Der volle Inhalt der QuelleReali, Federico. „Dynamical models for diabetes: insights into insulin resistance and type 1 diabetes“. Doctoral thesis, Università degli studi di Trento, 2017. https://hdl.handle.net/11572/369184.
Der volle Inhalt der QuelleReali, Federico. „Dynamical models for diabetes: insights into insulin resistance and type 1 diabetes“. Doctoral thesis, University of Trento, 2017. http://eprints-phd.biblio.unitn.it/1962/1/Reali_PhDThesis.pdf.
Der volle Inhalt der QuelleLindmark, Stina. „Neurohormonal mechanisms in insulin resistance and type 2 diabetes“. Doctoral thesis, Umeå : Univ, 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-225.
Der volle Inhalt der QuelleHunter, Steven J. „Insulin resistance : underlying mechanisms and influence of treatment“. Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337044.
Der volle Inhalt der QuelleRelton, Caroline L. „The identification and characterisation of genes associated with insulin resistance“. Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.264425.
Der volle Inhalt der QuelleVella, Sandro. „Pharmacological modulation of insulin resistance : benefits and harms“. Thesis, University of Dundee, 2013. https://discovery.dundee.ac.uk/en/studentTheses/4428eca5-5018-4816-95d0-30c0a3043409.
Der volle Inhalt der QuelleLee, Katie Ting Yan. „Novel wild-derived mouse models of obesity and diabetes resistance“. Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/41985.
Der volle Inhalt der QuelleMarakis, Georgios. „Natural therapy for insulin-resistance syndrome and type II diabetes“. Thesis, University of Reading, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340013.
Der volle Inhalt der QuelleJärvi, Anette. „Carbohydrate-rich foods in the treatment of the insulin resistance syndrome : studies of the importance of the glycaemic index and dietary fibre /“. Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5166-7/.
Der volle Inhalt der QuelleClarke, Stephen. „Liver steatosis and insulin-resistance : reversal by Sutherlandia frutescens“. Thesis, Nelson Mandela Metropolitan University, 2014. http://hdl.handle.net/10948/d1020788.
Der volle Inhalt der QuellePhillips, Amanda S. „The Influence of Perceived Stress on Insulin Resistance in Adults with Type 2 Diabetes“. Thesis, University of North Texas, 2014. https://digital.library.unt.edu/ark:/67531/metadc699935/.
Der volle Inhalt der QuelleKershner, David. „Oral Glucose Insulin Secretion Test for Identifying Patients with Insulin Resistance“. ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/5634.
Der volle Inhalt der QuelleBagstaff, Stephanie M. „Hyperinsulinemia and insulin resistance in cultured human muscle cells“. Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270177.
Der volle Inhalt der QuelleGelcich, Sarah. „Exploring risk, resistance and closing talk in chronic diabetes routine consultations“. Thesis, University of Sheffield, 2017. http://etheses.whiterose.ac.uk/19167/.
Der volle Inhalt der QuelleFryer, Lee George Daniel. „Studies into the role of glucose transporter function in insulin resistance“. Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265007.
Der volle Inhalt der QuelleCrawford, Lynne Mary. „The regulation of triglyceride metabolism in the liver and adipose tissue“. Thesis, University of Glasgow, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263457.
Der volle Inhalt der QuelleRanasinghe, Dilip Chathuranga. „The effects of supervised aerobic and resistance exercise training on Sri Lankan adults with type 2 diabetes mellitus: Sri Lanka diabetes aerobic and resistance training (SL-DART) study“. Thesis, Queensland University of Technology, 2018. https://eprints.qut.edu.au/115457/1/115457_9304177_ranasinghe_dilip_chathuranga_thesis.pdf.
Der volle Inhalt der QuelleAndrews, Tara Jane. „The role of endothelial function and oxidant stress in a model of insulin resistance“. Thesis, Queen Mary, University of London, 2003. http://qmro.qmul.ac.uk/xmlui/handle/123456789/28587.
Der volle Inhalt der QuelleMcClean, Paula. „Inhibition of GIP signalling alleviates obesity, insulin resistance and type 2 diabetes“. Thesis, University of Ulster, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.494369.
Der volle Inhalt der QuelleWiggam, Malcolm Ivan. „Aspects of insulin resistance in essential hypertension and insulin-dependent diabetes mellitus“. Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387971.
Der volle Inhalt der QuelleCairncross, Joy Claudia. „The effect of 14 weeks of strength training on insulin resistance“. Thesis, Nelson Mandela Metropolitan University, 2013. http://hdl.handle.net/10948/d1018276.
Der volle Inhalt der QuelleJun, Lucy Soo Yon. „Thyroid hormone-regulated skeletal muscle Glut4 glucose transporter trafficking during fasting in diet-induced obesity and insulin resistance“. CSUSB ScholarWorks, 2005. https://scholarworks.lib.csusb.edu/etd-project/2869.
Der volle Inhalt der QuelleKuhl, Jeanette. „Pathogenesis of type 2 diabetes with emphasis on the mechanism of insulin resistance /“. Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-839-8/.
Der volle Inhalt der QuelleStone, Monique Lee Women's & Children's Health Faculty of Medicine UNSW. „Poor glycaemic control in adolescents with type 1 diabetes“. Publisher:University of New South Wales. Women's & Children's Health, 2008. http://handle.unsw.edu.au/1959.4/41281.
Der volle Inhalt der QuelleErickson, Andrea Rose. „The Association Between Dairy Consumption and Insulin Resistance“. BYU ScholarsArchive, 2013. https://scholarsarchive.byu.edu/etd/4262.
Der volle Inhalt der QuelleWinnick, Jason Joseph. „Effect of aerobic exercise on peripheral glucose uptake and endogenous glucose production in type 2 diabetes mellitus“. Columbus, Ohio : Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1157551296.
Der volle Inhalt der QuelleNg, Foong Loo Yvonne Biotechnology & Biomolecular Sciences Faculty of Science UNSW. „Insulin action: unravelling AKT signalling in Adipocytes“. Awarded by:University of New South Wales. Biotechnology & Biomolecular Sciences, 2009. http://handle.unsw.edu.au/1959.4/44628.
Der volle Inhalt der QuelleMoncrieft, Ashley E. „Inactivity, Inflammation, and Insulin Resistance in Type 2 Diabetes and the Metabolic Syndrome“. Scholarly Repository, 2011. http://scholarlyrepository.miami.edu/oa_theses/300.
Der volle Inhalt der QuelleSong, Xiao Mei. „Insulin signal transduction in skeletal muscle : special consideration for insulin resistance and diabetes /“. Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4502-0/.
Der volle Inhalt der QuelleDanielsson, Anna. „Insulin signalling in human adipocytes : mechanisms of insulin resistance in type 2 diabetes“. Doctoral thesis, Linköping : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-10327.
Der volle Inhalt der QuellePuthanveetil, Prasanth Nair. „Role of cardiac FoxO1 in conditions of insulin resistance, nutrient excess, and diabetes“. Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/41051.
Der volle Inhalt der QuellePlatt, Thomas. „LEPTIN RESISTANCE INDUCED OBESITY AND DIABETES PROMOTE NEUROPATHOLOGICAL CHANGES IN THE AGING BRAIN“. UKnowledge, 2014. http://uknowledge.uky.edu/biochem_etds/18.
Der volle Inhalt der QuelleTurner, Daniel. „Impact of acute resistance exercise on glycaemia in individuals with type 1 diabetes“. Thesis, Swansea University, 2015. https://cronfa.swan.ac.uk/Record/cronfa43149.
Der volle Inhalt der QuelleKudiarasu, Christine. „Effects of eccentric versus concentric resistance training in adults with Type 2 Diabetes“. Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2020. https://ro.ecu.edu.au/theses/2297.
Der volle Inhalt der QuelleFrangioudakis, Georgia St Vincent's Clinical School UNSW. „Insulin signal transduction in vivo in states of lipid-induced insulin resistance“. Awarded by:University of New South Wales. St Vincent's Clinical School, 2004. http://handle.unsw.edu.au/1959.4/27419.
Der volle Inhalt der QuelleSharma, Divya. „Drug Delivery Systems for Treatment of Diabetes Mellitus“. Diss., North Dakota State University, 2019. https://hdl.handle.net/10365/31745.
Der volle Inhalt der QuelleNational Institutes of Health (NIH) grant R15GM114701
ND EPSCoR seed award FAR0030636
McConnell, Elizabeth Mae. „Increased vascular disease in adult hypopituitarism : studies of mortality, insulin resistance and endothelial function“. Thesis, Queen's University Belfast, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301743.
Der volle Inhalt der QuelleTalbot, Nicola A. „Obesity, inflammation and insulin resistance in skeletal muscle“. Thesis, Royal Veterinary College (University of London), 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618327.
Der volle Inhalt der QuelleAndersen, Ditte K. „The role of microRNAs in skeletal muscle insulin resistance“. Thesis, Royal Veterinary College (University of London), 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701676.
Der volle Inhalt der QuelleNowak, Christoph. „Insulin Resistance : Causes, biomarkers and consequences“. Doctoral thesis, Uppsala universitet, Molekylär epidemiologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-316891.
Der volle Inhalt der QuelleAlkhalidy, Hana Awwad. „Flavonol kaempferol in the regulation of glucose homeostasis in diabetes“. Diss., Virginia Tech, 2016. http://hdl.handle.net/10919/82485.
Der volle Inhalt der QuellePh. D.
Negrato, Carlos Antonio [UNESP]. „Hiperglicemia gestacional leve como fator de risco para síndrome metabólica na gravidez e morbilidade perinatal“. Universidade Estadual Paulista (UNESP), 2006. http://hdl.handle.net/11449/104174.
Der volle Inhalt der QuelleFundação para o Desenvolvimento Médico e Hospitalar (Famesp)
Em 1988, Reaven descreveu a síndrome X, uma junção de diversos fatores de risco para as doenças cardiovasculares, sendo a resistência à insulina o ponto de partida para o estabelecimento de tais fatores. Várias denominações foram dadas a este grupo de comorbidades e hoje ela é conhecida como Síndrome Metabólica. Seu estudo tem sido dificultado pela ausência de consenso na sua definição. Sua prevalência varia de acordo com o critério diagnóstico utilizado e com as características da população estudada, sendo mais encontrada em portadores de obesidade central. A prevalência em mulheres varia de 10,7% a 40,5%, e é um preditor para o desenvolvimento de complicações cardiovasculares, estando associada a duas importantes entidades nosológicas bastante estudadas em ginecologia e obstetrícia: a síndrome dos ovários policísticos e o diabetes gestacional. Embora não façam parte dos critérios diagnósticos, várias condições clínicas e fisiopatológicas estão freqüentemente a ela associadas, tais como: acantose nigricans, doença hepática gordurosa não-alcoólica, microalbuminúria, estados pró trombóticos (elevação dos níveis de PAI-1 e fibrinogênio), estados pró- inflamatórios (elevação dos níveis de IL-6, TNF-a, resistina e PC-R), disfunção endotelial e hiperuncemia. Os autores fazem uma revisão sobre o diagnóstico laboratorial e os fatores ligados a SM evidenciando que a Critósos diagnósticos atuais da síndrome metabólica gravidez é um modelo da SM compensada e sugerem que o diabetes gestacional e a hipergilcemia materna diária devam ser acrescentados aos critérios diagnósticos.
In 1988, Reaven described the Syndrome X, a junction of several risk factors for cardiovascular diseases, being the insulin resistance the set point for the establishment of such factors. Many denominations were given to this group of co-morbidities and today it is known as metabolic syndrome. lts study has been difficult by the absence of a consensus for its definition. lts prevalence vanes according to the diagnostic criterion used and with the characteristics of the studíed population, being found mainly in people with central obesity. The prevalence in women varies from 10.7% to 40.5%, and it is a predictor of cardiovascular complications associated with two important nosologic entities concerned with gynecology and obstetrics: the polycystic ovaries syndrome and gestational diabetes. A variety of clinical and physiopathoiogical conditions are frequently associated to it. These are: acanthosis nigricans, non alcoholic fatty tiver disease, microalbuminuria, prothrombotic states (elevation of PAI-1 and fibrinogen leveIs), proinflammatory states (elevation of IL-6, TNF-a, resistin and C-RP leveis), endothelial dysfunction and hyperuricemia. The authors make a revision on the laboratorial diagnosis and the factors Iinked to the metabolic syndrome and attest that pregnancy is a counterbalanced model of the syndrome Critérios diagnósticos atuaiS da 5ifldrOme metabólica and suggest that gestational diabetes and diurnal maternal hyperglycemia should be added to the diagnostic criteria.
Dumais, Valerie. „Insulin Resistance, Metabolic Syndrome and Diabetes in Women at High Risk for Breast Cancer“. Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31508.
Der volle Inhalt der QuelleMårdberg, Emelie. „Effect of Resistance Training in Patients with Type 2 Diabetes Mellitus : − A Systematic Review“. Thesis, Örebro universitet, Institutionen för läkarutbildning, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-37001.
Der volle Inhalt der QuelleAgbaje, Olorunsola Fatai. „Modelling methodologies to assess insulin resistance and insulin secretion in type 2 diabetes subjects“. Thesis, City University London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397933.
Der volle Inhalt der QuelleDirek, Kenan. „The role of mitochondria in the development of insulin resistance and type 2 diabetes“. Thesis, King's College London (University of London), 2013. https://kclpure.kcl.ac.uk/portal/en/theses/the-role-of-mitochondria-in-the-development-of-insulin-resistance-and-type-2-diabetes(30f426ed-9221-473f-b177-acefda85acaa).html.
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