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Zeitschriftenartikel zum Thema „Diabetes mellitus I“

Autor: Grafiati

Veröffentlicht am 28. Juni 2021

Zuletzt aktualisiert am 8. Februar 2022

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1

Doamekpor, Caleb. "Diabetes Mellitus." Acta Scientific Pharmaceutical Sciences 4, no. 2 (2020): 01–09. http://dx.doi.org/10.31080/asps.2020.04.diabetes-mellitus.

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2

GÜRBÜZ, Murat, and Menşure Nur ÇELİK. "Minerals and Diabetes Mellitus." Turkiye Klinikleri Journal of Internal Medicine 4, no. 2 (2019): 71–83. http://dx.doi.org/10.5336/intermed.2018-64388.

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3

Chaudhary, Nitin, and Nidhi Tyagi. "Diabetes mellitus: An Overview." International Journal of Research and Development in Pharmacy & Life Sciences 7, no. 4 (2018): 3030–33. http://dx.doi.org/10.21276/ijrdpl.2278-0238.2018.7(4).3030-3033.

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4

Ambulkar, Sunil, Parimal Tayde, Makarand Randive, and Mukund Ganeriwal. "Diabetes mellitus in pregnancy." New Indian Journal of OBGYN 4, no. 1 (2017): 4–9. http://dx.doi.org/10.21276/obgyn.2017.4.1.2.

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5

Begum, SA, R. Afroz, Q. Khanam, A. Khanom, and TS Choudhury. "Diabetes Mellitus and Gestational Diabetes Mellitus." Journal of Paediatric Surgeons of Bangladesh 5, no. 1 (2015): 30–35. http://dx.doi.org/10.3329/jpsb.v5i1.23887.

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Diabetes mellitus (DM), also known as simply diabetes, is a group of metabolic diseases in which there are high blood sugar levels over a prolonged period. Worldwide in 2012 and 2013 diabetes resulted in 1.5 to 5.1 million deaths per year, making it the 8th leading cause of death. Diabetes overall at least doubles the risk of death. This high blood sugar produces the symptoms of frequent urination, increased thirst, and increased hunger. Untreated, diabetes can cause many complications. Acute complications include diabetic ketoacidosis and nonketotic hyperosmolar coma. Serious long-term complications include heart disease, stroke, kidney failure, foot ulcers and damage to the eyes. The number of people with diabetes is expected to rise to 592 million by 2035. The economic costs of diabetes globally were estimated in 2013 at $548 billion and in the United States in 2012 $245 billion. [3]Globally, as of 2013, an estimated 382 million people have diabetes worldwide, with type 2 diabetes making up about 90% of the cases. This is equal to 8.3% of the adults population, with equal rates in both women and men. There are three main types of diabetes mellitus: In case of type 1 Diabetes mellitus, results from the bodys failure to produce enough insulin. This form was previously referred to as insulin-dependent diabetes mellitus (IDDM) or juvenile diabetes. The cause is unknown. Another type is type 2 diabetes mellitus begins with insulin resistance, a condition in which cells fail to respond to insulin properly. As the disease progresses a lack of insulin may also develop. This form was previously referred to as non insulin-dependent diabetes mellitus (NIDDM) or adult-onset diabetes. The primary cause is excessive body weight and not enough exercise. Gestational diabetes is the third main form and occurs when pregnant women without a previous history of diabetes develop a high blood glucose level. Gestational diabetes usually resolves after the birth of the baby. It occurs in about 210% of all pregnancies and may improve or disappear after delivery. However, after pregnancy approximately 510% of women with gestational diabetes are found to have diabetes mellitus, most commonly type 2. Gestational diabetes is fully treatable, but requires careful medical supervision throughout the pregnancy.J. Paediatr. Surg. Bangladesh 5(1): 30-35, 2014 (January)

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6

Daidano, Jeando Khan, Nazia Azam Yusfani, and Bilqees Daidano. "DIABETES MELLITUS." Professional Medical Journal 25, no. 06 (2018): 881–86. http://dx.doi.org/10.29309/tpmj/18.4535.

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7

Pelikánová, Terezie. "Diabetes mellitus and cardiovascular diseases." Cor et Vasa 53, no. 4-5 (2011): 242–48. http://dx.doi.org/10.33678/cor.2011.054.

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8

Goldmannová Ondřej Krystyník David Karásek, Dominika, and Josef Zadražil. "Diabetes mellitus after organ transplantation." Interní medicína pro praxi 21, no. 1 (2019): 20–23. http://dx.doi.org/10.36290/int.2019.003.

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9

Poskerová, H., P. Linhartová, J. Vokurka, A. Fassmann, and L. Hollá. "Diabetes Mellitus and Oral Health." Česká stomatologie/Praktické zubní lékařství 114, no. 5 (2014): 75–86. http://dx.doi.org/10.51479/cspzl.2014.018.

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10

Padmasri Devi, P., M. Mahalakshmi, V. Sarojini Devi, M. Kiran Deedi, Ch Ganapathi Swamy, and V. Thoyoja Durga. "Prevalence of Gestational Diabetes Mellitus." Indian Journal of Obstetrics and Gynecology 7, no. 2 (2019): 309–11. http://dx.doi.org/10.21088/ijog.2321.1636.7219.31.

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11

Poskerová, H., P. Linhartová, J. Vokurka, A. Fassmann, and L. Hollá. "Diabetes Mellitus and Oral Health." Česká stomatologie/Praktické zubní lékařství 114, no. 5 (2014): 75–86. http://dx.doi.org/10.51479/cspzl.2014.018.

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12

DEVI, Yuli Puspita, Rumaisah ABDILLAH, and Muthmainnah MUTHMAINNAH. "DETERMINANTS OF GESTATIONAL DIABETES MELLITUS." PERIÓDICO TCHÊ QUÍMICA 18, no. 37 (2021): 48–56. http://dx.doi.org/10.52571/ptq-v18-n73-pgi.48-2021.

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Background: Gestational Diabetes Mellitus (GDM) is a glucose tolerance disorder that first appears during pregnancy. GDM can cause a variety of obstetric and perinatal complications for pregnant women and their fetuses. The prevalence of GDM in the city of Surabaya in 2015 amounted to 2.29%, increasing to 3.88% in 2018. Aim: The purpose of this study was to analyze the determinants (family history of diabetes mellitus, BMI, history of birth weight, parity, and the age of the pregnant woman) that influence the GDM. Methods: This research is an analytical study with an observational approach. The research design was carried out using a case-control study. In this study, the number of samples was 36 people, 6 cases, and 30 people as controls, and were taken randomly. The data source obtained from secondary data (medical records of pregnant women) at Mulyorejo Health Center Surabaya. The analysis used in this study was a simple logistic regression test. Results and Discussion: The result showed an effect of a family history of diabetes mellitus (p = 0.035) on the incidence of GDM. Parity was a potential variable (p = 0.077) on the incidence of GDM. Meanwhile, BMI, history of birth weight, and mother's age did not affect the incidence of GDM. Conclusion: Pregnant women with a family history of diabetes who are not balanced with maintaining a good lifestyle can experience complications of developing GDM.

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13

Malinovská, Jana, Jana Urbanová, Michala Lustigová, Kristýna Kučera, and Jan Brož. "Diabetes mellitus and illicit drugs." Vnitřní lékařství 66, no. 5 (2020): e16-e19. http://dx.doi.org/10.36290/vnl.2020.092.

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14

Vohnout, Branislav, Jana Lisičanová, and Andrea Havranová. "PCSK9 inhibitors and diabetes mellitus." Vnitřní lékařství 64, no. 12 (2018): 1186–89. http://dx.doi.org/10.36290/vnl.2018.170.

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15

Sheth, Neeti R., Nabilah A. Sareshwala, Sushil G. Chaudhary, and Hiren D. Matai. "Awareness about diabetes mellitus and diabetic retinopathy in patients with diabetes mellitus." International Journal of Research in Medical Sciences 5, no. 8 (2017): 3570. http://dx.doi.org/10.18203/2320-6012.ijrms20173564.

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Background: Awareness about diabetes mellitus and diabetic retinopathy in patients with diabetes mellitus.Methods: 50 patients of diabetes mellitus attending Pandit Deendayal Upadhyay Medical College, Rajkot, Gujarat, India, were randomly selected during January 2017 to March 2017. 11-point questionnaire obtained on detailed search of literature on published reports were administered by interviewers well versed in English and Gujarati.Results: Total of 50 patients were randomly selected.42%- females and 58% - males. Mean age - 58.5 years. Age range 40 to 71 years. Patients with good sugar control -26% and poor control - 74%. 94% of the patients were taking treatment for DM and 6% were not on any treatment. 48 % of the patients were illiterate, 10% were graduate and 42% had education below 12th standard. 50% were aware about DM affecting the eye .38% had taken eye treatment and 62% had not. 26% were aware of DM affecting eye inspite of good control and 26 % aware of the need of check-up in poor control. 40% aware of the complications related to DM.Conclusions: Better literacy rates is contributory to public awareness, however trend for poor practice needs to be radically changed with aggressive public motivation emphasizing the necessity of screening and follow ups.

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16

SAARI, K. M. "DIABETES MELLITUS AND DIABETIC RETINOPATHY." Acta Ophthalmologica 62, S165 (2009): 98–104. http://dx.doi.org/10.1111/j.1755-3768.1984.tb03078.x.

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17

Spanheimer, Robert G. "Diabetes mellitus." Postgraduate Medicine 109, no. 4 (2001): 21–25. http://dx.doi.org/10.3810/pgm.2001.04.903.

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18

Ahdi, M. "Diabetes mellitus." Nederlands Tijdschrift voor Tandheelkunde 119, no. 2 (2012): 65–71. http://dx.doi.org/10.5177/ntvt.2012.02.11253.

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19

Kaufman, Francine Ratner. "Diabetes Mellitus." Pediatrics in Review 18, no. 11 (1997): 383–93. http://dx.doi.org/10.1542/pir.18-11-383.

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20

Uhl, Richard L., Andrew J. Rosenbaum, John A. DiPreta, James Desemone, and Michael Mulligan. "Diabetes Mellitus." Journal of the American Academy of Orthopaedic Surgeons 22, no. 3 (2014): 183–92. http://dx.doi.org/10.5435/jaaos-22-03-183.

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21

&NA;. "Diabetes mellitus." Drugs & Therapy Perspectives 6, no. 3 (1995): 14–16. http://dx.doi.org/10.2165/00042310-199506030-00007.

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22

Muneer, Khadija, Naheed Hashmat, Taimia Ayub, and Usman Abdul Ahad. "DIABETES MELLITUS." Professional Medical Journal 25, no. 12 (2018): 1869–75. http://dx.doi.org/10.29309/tpmj/18.4670.

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Background: Type I diabetes mellitus is a chronic illness in which autoimmune destruction of pancreatic beta cells results in the body’s inability to produce insulin. Vitamin D has several important roles regarding bone health however, recent studies suggest that Vitamin D3 (the active form of Vitamin D) has potent antiproliferative and immunomodulatory properties which has linked it to many autoimmune disease including Type I Diabetes Melltius. Vitamin D deficiency (serum levels less than 50 nmol/l) has a negative influence on insulin secretion in patients with Type I Diabetes Mellitus, thereby suggesting a role for vitamin D3 in the pathogenesis of Type I Diabetes Mellitus. Vitamin D deficiency is an increasingly recognized comorbidity in patients with Type I diabetes mellitus. We aim to determine the frequency of vitamin D deficiency in Type I Diabetes Mellitus in Pakistani population. Objectives: The objective of the study was to determine frequency of Vitamin D deficiency in patients havingType I Diabetes Mellitus in Pakistani Population. Study Design: Cross-sectional study. Setting: The study was conducted in Diabetes Management Center & Endocrinology Unit (DMC & EU) at Services Hospital Lahore. Period: From 20th May to 19th November (6 months). Materials and Methods: 200 patients having Type I Diabetes Mellitus presenting to Diabetic Management Center were recruited by non-probability purposive sampling. Informed consent was taken. Pro forma was filled by a skilled interviewer and blood sample for vitamin D levels was drawn. Data was entered in the pro forma given at the end and was analyzed in SPSS. Results: Of the 200 subjects 125 were males and 75 were females. Of the males 85.6% and females 88% were vitamin D deficient. The frequency of vitamin D deficiency in newly diagnosed Type I Diabetics is 86.5%. In this study it was seen that higher HbA1c levels are significantly associated with Vitamin D deficiency. Conclusions: These results conclude that vitamin D deficiency is significantly frequent at the onset of Type I Diabetes Mellitus . So vitamin D levels should be measured in all Type I diabetics on their first presentation to the hospital especially those with higher HbA1c levels and vitamin D should be replaced in deficient patients. Further prospectivestudies should be done to evaluate Vitamin D3 as a factor in managing glycemic control.

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23

Hafeez, Arsalan, Kashif Rehman, Aqib Rehman, and Abdul Hafeez Ch. "DIABETES MELLITUS." Professional Medical Journal 25, no. 09 (2018): 1406–12. http://dx.doi.org/10.29309/tpmj/18.4813.

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24

WEILAND, D. "DIABETES MELLITUS." Clinics in Family Practice 4, no. 3 (2002): 703–52. http://dx.doi.org/10.1016/s1522-5720(02)00023-5.

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25

Jolly, Gary Peter, and Thomas Zgonis. "Diabetes mellitus." Clinics in Podiatric Medicine and Surgery 20, no. 4 (2003): xiii—xiv. http://dx.doi.org/10.1016/s0891-8422(03)00075-2.

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26

Schuster, Dara P., and Vani Duvuuri. "Diabetes mellitus." Clinics in Podiatric Medicine and Surgery 19, no. 1 (2002): 79–107. http://dx.doi.org/10.1016/s0891-8422(03)00082-x.

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27

Mandracchia, Vincent J. "Diabetes mellitus." Clinics in Podiatric Medicine and Surgery 20, no. 4 (2003): xi—xii. http://dx.doi.org/10.1016/s0891-8422(03)00093-4.

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28

HAMERA, EDNA. "Diabetes Mellitus." Annual Review of Nursing Research 10, no. 1 (1992): 55–75. http://dx.doi.org/10.1891/0739-6686.10.1.55.

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29

&NA;. "Diabetes Mellitus." Disease Management & Health Outcomes 2, no. 4 (1997): 213. http://dx.doi.org/10.2165/00115677-199702040-00015.

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30

Gleeson, Jeremy M., Michael Foris, Marjorie Cypress, Donna J. Rodriguez, Neal Friedman, and Martha Kent. "Diabetes Mellitus." Disease Management and Health Outcomes 5, no. 2 (1999): 61–72. http://dx.doi.org/10.2165/00115677-199905020-00001.

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31

J. Kramer, Holly, and Sankar D. Navaneethan. "Diabetes Mellitus." Nephrology Self-Assessment Program 18, no. 4 (2019): 214–19. http://dx.doi.org/10.1681/nsap.2019.18.4.6.

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32

Jiménez, P. Gallardo, J. Martín-Carmona, and E. Lorenzo Hernández. "Diabetes mellitus." Medicine - Programa de Formación Médica Continuada Acreditado 13, no. 16 (2020): 883–90. http://dx.doi.org/10.1016/j.med.2020.09.010.

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33

Aznar Rodríguez, S., A. Lomas Meneses, R. P. Quílez Toboso, and I. Huguet Moreno. "Diabetes mellitus." Medicine - Programa de Formación Médica Continuada Acreditado 11, no. 17 (2012): 995–1002. http://dx.doi.org/10.1016/s0304-5412(12)70418-3.

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34

Teramoto, Tamio, Jun Sasaki, Shun Ishibashi, et al. "Diabetes Mellitus." Journal of Atherosclerosis and Thrombosis 21, no. 2 (2014): 93–98. http://dx.doi.org/10.5551/jat.19349.

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35

Han, Steven-Huy B., and Paul Martin. "Diabetes Mellitus." Journal of Clinical Gastroenterology 30, no. 3 (2000): 227–28. http://dx.doi.org/10.1097/00004836-200004000-00003.

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36

Carr, Marcus E. "Diabetes mellitus." Journal of Diabetes and its Complications 15, no. 1 (2001): 44–54. http://dx.doi.org/10.1016/s1056-8727(00)00132-x.

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37

Todd, John A. "Diabetes mellitus." Current Opinion in Genetics & Development 2, no. 3 (1992): 474–78. http://dx.doi.org/10.1016/s0959-437x(05)80160-1.

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38

GOLDEN, WILLIAM E., and ROBERT H. HOPKINS. "Diabetes Mellitus." Internal Medicine News 39, no. 9 (2006): 31. http://dx.doi.org/10.1016/s1097-8690(06)73427-8.

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39

Günther, K. P., J. Lützner, F. Beyer, S. Kirschner, and C. Hamann. "Diabetes mellitus." Osteologie 23, no. 02 (2014): 86–90. http://dx.doi.org/10.1055/s-0037-1620045.

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ZusammenfassungEine Totalendoprothese (TEP) der Hüfte stellt einen sicheren und sehr häufigen operativen Eingriff dar. Das perioperative Risiko und die Komplikationen während des stationären Aufenthalts werden wesentlich durch Begleiterkrankungen bestimmt. In dieser Studie wurde die Bedeutung eines Diabetes mellitus als Risikofaktor für perioperative Komplikationen sowie Komplikationen nach drei und zwölf Monaten analysiert. In der untersuchten Kohorte von 458 Patienten war das Risiko für perioperative Komplikationen und Komplikationen während des stationären Aufenthalts nach Hüft-TEP beim Vorliegen eines Diabetes mellitus um das Zwei- bis Dreifache gesteigert. Patienten mit Diabetes mellitus wiesen sowohl häufigere chirurgische als auch internistische Komplikationen auf, was im Mittel zu einer um einen Tag verlängerten stationären Verweildauer führte. Nach zwölf Monaten existierten hingegen keine signifikanten Unterschiede zwischen Patienten mit oder ohne Diabetes mellitus. Zusammenfassend stellt ein Diabetes mellitus bei Hüft-TEP einen Risikofaktor für stationäre Komplikationen dar und verlängert die stationäre Verweildauer. Nach zwölf Monaten weisen Patienten mit Diabetes mellitus nach Hüft-TEP jedoch nicht mehr Komplikationen auf als Patienten ohne Diabetes.

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40

Marg, Wolfgang. "Diabetes mellitus." Pädiatrie up2date 3, no. 03 (2008): 263–87. http://dx.doi.org/10.1055/s-2008-1077368.

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41

Long, R. "Diabetes mellitus." Veterinary Record 124, no. 25 (1989): 667. http://dx.doi.org/10.1136/vr.124.25.667-d.

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42

Milne, E. "Diabetes mellitus." Veterinary Record 125, no. 2 (1989): 49. http://dx.doi.org/10.1136/vr.125.2.49-b.

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43

Davies, M. "Diabetes mellitus." Veterinary Record 125, no. 4 (1989): 97. http://dx.doi.org/10.1136/vr.125.4.97-b.

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44

Khoury, Jane C., Dawn Kleindorfer, Kathleen Alwell, et al. "Diabetes Mellitus." Stroke 44, no. 6 (2013): 1500–1504. http://dx.doi.org/10.1161/strokeaha.113.001318.

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45

Kudva, Yogish C., and K. Sreekumaran Nair. "Diabetes Mellitus." Mayo Clinic Proceedings 95, no. 1 (2020): 15–21. http://dx.doi.org/10.1016/j.mayocp.2019.11.016.

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46

Kidambi, Srividya, and Shailendra B. Patel. "Diabetes Mellitus." Journal of the American Dental Association 139 (October 2008): 8S—18S. http://dx.doi.org/10.14219/jada.archive.2008.0364.

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47

Hamdy, Ronald C. "DIABETES MELLITUS." Southern Medical Journal 95, no. 1 (2002): 1–2. http://dx.doi.org/10.1097/00007611-200201000-00001.

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48

&NA;. "DIABETES MELLITUS." Southern Medical Journal 95, no. 1 (2002): 1–2. http://dx.doi.org/10.1097/00007611-200295010-00001.

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49

Luise, Mario. "Diabetes mellitus." Medical Journal of Australia 158, no. 9 (1993): 537–40. http://dx.doi.org/10.5694/j.1326-5377.1993.tb121869.x.

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50

Prior, Steven J. "DIABETES MELLITUS." Innovation in Aging 3, Supplement_1 (2019): S574. http://dx.doi.org/10.1093/geroni/igz038.2129.

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Abstract Nearly three-fourths of adults over 65 year of age are affected by impaired glucose tolerance or type 2 diabetes. Both aging and inactivity contribute to the numerous skeletal muscle changes that occur with insulin resistance and type 2 diabetes. These changes include reduced capillarization that can impaired glucose uptake and substrate delivery, resulting in metabolic abnormalities and metabolic inflexibility. These changes may ultimately contribute to reduced delivery of oxygen, nutrients, and hormones to the muscles leading to impairments in metabolism, muscle mass, and function. We will discuss current research on the role of vascular impairments and reduced skeletal muscle capillarization in the development of impaired muscle metabolism, fitness and function. Finally, we will discuss how exercise training may reverse these declines.

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