Auswahl der wissenschaftlichen Literatur zum Thema „Dette chronique de sommeil“
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Zeitschriftenartikel zum Thema "Dette chronique de sommeil"
Roca-Paixao, L., C. Bougard, P. Van Beers, P. Arnal, C. Drogou, G. Dispersyn, F. Sauvet, D. Leger, M. Chennaoui und A. Rabat. „Vulnérabilité cognitive à une dette chronique de sommeil : effet différentiel avec le matin/soir et les capacités de maintien de l’éveil“. Médecine du Sommeil 11, Nr. 1 (Januar 2014): 15. http://dx.doi.org/10.1016/j.msom.2014.01.033.
Der volle Inhalt der QuellePoudevigne, Laura, Louise Gaillard, Christine Le Coz, Mariette Gaudin, Mounir Chennaoui, Damien Léger, Aurélie Servonnet, Arnaud Rabat und Brice Faraut. „Étude de la fatigabilité attentionnelle associée à une dette chronique de sommeil : marqueurs biologiques et stratégie de récupération par la sieste“. Médecine du Sommeil 21, Nr. 1 (März 2024): 34. http://dx.doi.org/10.1016/j.msom.2023.12.087.
Der volle Inhalt der QuelleGuieu, J. D. „Dette de sommeil et obésité !“ Médecine du Sommeil 1, Nr. 2 (Dezember 2004): 47. http://dx.doi.org/10.1016/s1769-4493(04)70181-8.
Der volle Inhalt der QuelleDuforez, François. „Dette de sommeil et performance sportive“. Les Cahiers de l'INSEP 41, Nr. 1 (2008): 128–40. http://dx.doi.org/10.3406/insep.2008.965.
Der volle Inhalt der QuelleHaba-Rubio, José, Sophie de Seigneux und Raphael Heinzer. „Troubles du sommeil et maladie rénale chronique“. Néphrologie & Thérapeutique 8, Nr. 2 (April 2012): 74–80. http://dx.doi.org/10.1016/j.nephro.2011.07.408.
Der volle Inhalt der QuelleMarty, M., S. Rozenberg, B. Duplan, P. Thomas, B. Duquesnoy und F. Allaert. „TO41 - Qualité du sommeil et lombalgie chronique“. Douleurs : Evaluation - Diagnostic - Traitement 5 (November 2004): 29. http://dx.doi.org/10.1016/s1624-5687(04)94585-4.
Der volle Inhalt der QuelleHochner, I., JM Brogard, JF Blickle, JP Kantelip und J. Massol. „Le sommeil des diabétiques en déséquilibre glycémique chronique“. La Revue de Médecine Interne 17 (Januar 1995): S104. http://dx.doi.org/10.1016/0248-8663(96)86604-5.
Der volle Inhalt der QuelleFettal, Nadia. „L’impact de la bronchopneumopathie chronique obstructive sur le sommeil“. Médecine du Sommeil 13, Nr. 1 (Januar 2016): 28–29. http://dx.doi.org/10.1016/j.msom.2016.01.084.
Der volle Inhalt der QuelleCabane, J., I. Arnulf und JP Derenne. „Hoquet chronique et sommeil. Étude prospective sur huit cas“. La Revue de Médecine Interne 17 (Januar 1995): S27. http://dx.doi.org/10.1016/0248-8663(96)86498-8.
Der volle Inhalt der QuelleManus, Jean-Marie. „Augmentation de l’insulinorésistance et déficit chronique de sommeil en postménopause“. Revue Francophone des Laboratoires 2024, Nr. 560 (März 2024): 20–21. http://dx.doi.org/10.1016/s1773-035x(24)00104-7.
Der volle Inhalt der QuelleDissertationen zum Thema "Dette chronique de sommeil"
Melone, Marie-Anne. „Diagnοstic and therapeutic strategies οf circadian and sleep/wake rhythm disοrders in at-risk pοpulatiοns“. Electronic Thesis or Diss., Normandie, 2024. http://www.theses.fr/2024NORMR023.
Der volle Inhalt der QuelleSleep health guidelines advocate for 7 to 9 hours of nightly sleep for the general population, yet sleep debt persists, presenting significant health risks, including metabolic, cardiac, mental, and neurocognitive diseases. This widespread sleep debt is often attributed to the conflict between modern lifestyles—characterized by artificial lighting, shift work, and social obligations—and our innate circadian rhythms, leading to a condition known as circadian dysrhythmia. Circadian rhythms are the natural oscillations in physiological processes that are essential for aligning genetic, physiological, and behavioral patterns with solar time to anticipate changes in our environment. The misalignment of these rhythms is increasingly linked to various health disorders. Diagnosing circadian rhythms and sleep/wake disorders poses challenges, as part of its definition relies on subjective assessments and clinical evaluations of sleep quality. Moreover, sleep/wake timing or chronotype questionnaires, although validated, may not accurately reflect individual circadian clocks. While melatonin measurement is considered the gold standard, its practical implementation is difficult, making actigraphy and sleep logs more common tools for identifying circadian rhythms and sleep/wake disorders. This highlights the need for improved diagnostic methods. Potential therapeutic interventions could help improve circadian dysrhythmias related health outcomes. In this context, this manuscript delves into the prevalence, risk factors, and consequences of circadian rhythms and sleep/wake disorders, particularly focusing on at-risk populations like student-athletes and critically ill patients, where misaligned zeitgebers exacerbate health risks. This work includes three studies’ findings and one narrative review on circadian rhythm and sleep/wake disorders, their risk factors, consequences, and potential treatments in populations prioritizing performance (student-athletes) and recovery (critically ill patients)
PAYEN, PACCARD CHRISTINE. „La ronchopathie chronique“. Lyon 1, 1990. http://www.theses.fr/1990LYO1M290.
Der volle Inhalt der QuelleLaroche, Liny. „Relation entre l'insomnie chronique et le fonctionnement immunitaire“. Master's thesis, Université Laval, 1999. http://hdl.handle.net/20.500.11794/53135.
Der volle Inhalt der QuelleFrançois, Thierry. „Sommeil paradoxal, dépression et antidépresseurs : effets d'une administration chronique d'antidépresseurs tricycliques sur le sommeil paradoxal de patients déprimés“. Besançon, 1991. http://www.theses.fr/1991BESAM067.
Der volle Inhalt der QuelleBeaulieu, Philippe Ortho Marie-Pia d'. „Traitement cognitivo-comportemental de l'insomnie chronique“. Créteil : Université de Paris-Val-de-Marne, 2006. http://doxa.scd.univ-paris12.fr:80/theses/th0243099.pdf.
Der volle Inhalt der QuellePittaras, Elsa. „Marqueurs comportementaux et neurochimiques individuels de la prise de décision chez la souris et effets d'une dette de sommeil“. Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLS122/document.
Der volle Inhalt der QuelleAffective abilities that rely on the integrity of several neural circuits. In healthy subjects, inter-individual variability during decision-making exists due to genetic, epigenetic and environmental factors. Moreover, many psychiatric and neurobiological disorders are characterized by poor decision-making processes. Therefore, determining behavioral traits and neurobiological substrates involved in these processes is of major interest to unravel markers that could predict the emergence of neuropathologies.Based on the Iowa Gambling Task in humans, we developed a decision-making task in mice that assesses their ability to choose between several conflicting options under uncertainty. Thanks to a differential approach of mice’s behavior, we show that decision-making skills differed between mice: some mice exhibit a rigid behavior and avoid penalty (safe mice); others maintained exploratory behavior even if they took risks (risky mice); a majority of mice exhibit an intermediate behavior (average mice). We found that a combination of behavioral characteristics related to different psychopathologies in humans were specifically associated with extreme behavior in mice: safe mice exhibited a more anxious behavior, a lower prefrontal activation after the MGT than others subgroups of performance together with a lower basal rate of serotonin in the prefrontal cortex. Risky mice displayed a riskier behavior in various behavioral tasks, were less sensitive to reward, and had a lower basal rate of serotonin in the orbitofrontal cortex as well as a higher basal rate of serotonin, dopamine and noradrenaline in the hippocampus.To investigate the consequences of environmental changes on decision-making individual profiles, we performed the MGT on groups of mice either under Acute Sleep Dept (ASD) or under chronic sleep debt (CSD). We show that CSD didn't play any apparent effect but that ASD emphasized decision-making profiles: safe mice became drastically more rigid and avoided penalty; and risky mice chose systematically riskier options and developed rigid and unefficient decisions. These behavioral data could be explained by a decreased serotonin metabolism in the orbitofrontal cortex, an increase in the hippocampus and a high level of dopamine in the caudate putamen, the key brain area of habits.Therefore, in healthy inbred mice the MGT reveals individual inadapted decision-making strategies which are characterized by behavioral and neurobiological substrates exacerbated by an environmental stress. This paradigm also allows the determination of mice vulnerability to develop psychopathologies (e.g. depression, addiction) for which sleep debt could a trigger or a magnifier
Chenuel, Bruno Haouzi Philippe. „Etude des mécanismes physiopathologiques impliqués dans les apnées centrales du sommeil associées à l'insuffisance cardiaque chronique : données expérimentales animales et applications à l'Homme“. [S.l.] : [s.n.], 2006. http://www.scd.uhp-nancy.fr/docnum/SCD_T_2006_0237_CHENUEL.pdf.
Der volle Inhalt der QuelleLANDART, FREDERIQUE. „Decouverte d'episodes de desaturation en oxygene lors du sommeil chez un bronchopathe chronique a l'occasion de crises convulsives“. Amiens, 1991. http://www.theses.fr/1991AMIEM078.
Der volle Inhalt der QuelleJounieaux, Vincent. „Les evenements respiratoires du sommeil des bronchopneumopathies chroniques obstructives : etude comparative des patients (pa 02 comprise entre 60 et 70 mm hg) avec un groupe de sujets temoins“. Amiens, 1988. http://www.theses.fr/1988AMIEM055.
Der volle Inhalt der QuelleChenuel, Bruno. „Etude des mécanismes physiopathologiques impliqués dans les apnées centrales du sommeil associées à l'insuffisance cardiaque chronique : données expérimentales animales et applications à l'Homme“. Nancy 1, 2006. http://docnum.univ-lorraine.fr/public/SCD_T_2006_0237_CHENUEL.pdf.
Der volle Inhalt der QuelleDuring sleep, control of breathing is greatly influenced by metabolic information. Whether this chemical control of ventilation is involved in the genesis of central sleep apneas with CheyneStokes respiration in severe congestive heart patients is unknown and the subject of our work. It includes three series of experiments in animal models to clarify the mechanisms involved in the hypocapnia-induced apneas, the anatomical structures mediating such a ventilatory response and the specific effects of heart failure on the control of breathing, leading to an increased susceptibility to central apneas. More specifically a sleeping dog model was used, allowing the study of periodic breathing induced by a transient hyperventilation, with and without any acute pulmonary vascular congestion. We were interested in conditions capable of changing the apneic threshold defined as the arterial or end-expiratory partial carbon dioxide pressure below which ventilation ceases. Two mechanisms possibly involved in human pathology were investigated : 1- changes in the activity of the peripheral chemoreceptors 2- pulmonary vascular congestion. Whereas the arterial chemoreceptors are involved in the genesis of hypocapnia-induced apneas and the cardio-respiratory structures activated by a pulmonary edema facilitate the occurrence of central apneas, these mechanisms were not able to fully explain the description of the Cheyne-Stokes respiration in Humans. The relevance of these results must be evaluated in Human and the role played by the non-chemical information of the control of breathing in the genesis of central apneas must be more precisely studied
Bücher zum Thema "Dette chronique de sommeil"
Auclair, Huguette. La fibromyalgie: Comment se refaire une santé. Montréal: Édimag, 2000.
Den vollen Inhalt der Quelle findenGuité, Marcel. La fibromyalgie: Bien connaître pour mieux surmonter la douleur, la fatigue chronique et les troubles du sommeil, par Marcel Guité et Agathe Drouin Bégin = Fibromyalgia : an understanding to better deal with pain, chronic fatique and sleep disorders. Sudbury, Ont: Laurentian University, 2004.
Den vollen Inhalt der Quelle findenGRATTON, NICOLE. Dormez-Vous Assez?: Insomnie, Dette de Sommeil, Cure de Repos. FLAMMARION QUEBEC, 2006.
Den vollen Inhalt der Quelle findenFaraut, Brice. Sauvés par la sieste: Petits sommes et grandes victoires sur la dette de sommeil. ACTES SUD, 2021.
Den vollen Inhalt der Quelle findenFibromyalgie, Cahier. Mon Cahier de Suivi Fibromyalgie: Journal de Suivi Médical Pour l'évaluation de la Douleursuivi de l'humeur et des Médicaments Pour la Gestion des Maladies Chroniques Syndrome de Fatigue Chronique, Troubles du Sommeil, état dépressif, Carnet Suivi Santè. Independently Published, 2021.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Dette chronique de sommeil"
Hausser-Hauw, Chantal. „Épilepsie post-traumatique et hématome sous-dural chronique“. In Manuel d'EEG de l'adulte. Veille et sommeil, 201–2. Elsevier, 2007. http://dx.doi.org/10.1016/b978-2-294-07145-4.50044-1.
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