Auswahl der wissenschaftlichen Literatur zum Thema „Cardiomyopathie arythmogène du ventricule droit“
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Zeitschriftenartikel zum Thema "Cardiomyopathie arythmogène du ventricule droit"
Riad, Zakaria, und Jean-Luc Fellahi. „La cardiomyopathie arythmogène du ventricule droit“. Le Praticien en Anesthésie Réanimation 24, Nr. 5 (Oktober 2020): 269–74. http://dx.doi.org/10.1016/j.pratan.2020.08.005.
Der volle Inhalt der QuelleLanglois, S., B. Constans und A. S. Ducloy-Bouthors. „Cardiomyopathie arythmogène du ventricule droit et grossesse : étude rétrospective“. Annales Françaises d'Anesthésie et de Réanimation 33 (September 2014): A3. http://dx.doi.org/10.1016/j.annfar.2014.07.013.
Der volle Inhalt der QuelleChaimaa, Rhemimet, Ameur Asmaa, Souilk Houda, Rim Raissouni, Elharrak Meryem, Tabti Fatine, I. Fellet und Cherti a. „DYSPLASIE ARYTHMOGENE DE VENTRICULE DROIT REVELEE PAR UNE TACHYCARDIE VENTRICULAIRE CHEZ UN SUJET JEUNE“. International Journal of Advanced Research 11, Nr. 05 (31.05.2023): 1461–66. http://dx.doi.org/10.21474/ijar01/17014.
Der volle Inhalt der QuelleTabti, Fatine, Mariam El Harrak, Khaoula Aboubakr, Amal Baicha, Belghit El Hajjaj, Asmaa Ameur, Chaimaa Rhemmimet und Zakia Touati. „ARRHYTHMOGENIC RIGHT VENTRICULAR CARDIOMYOPATHYREVEALED BY AVENTRICULAR TACHYCARDIA“. International Journal of Advanced Research 11, Nr. 03 (31.03.2023): 1456–68. http://dx.doi.org/10.21474/ijar01/16591.
Der volle Inhalt der QuelleTissier, S., C. Barbary, A. Codreanu, E. Aliot und D. Regent. „Apport du scanner volumique avec synchronisation ECG dans le diagnostic de cardiomyopathie arythmogène du ventricule droit“. Journal de Radiologie 86, Nr. 12 (Dezember 2005): 1799–804. http://dx.doi.org/10.1016/s0221-0363(05)81525-2.
Der volle Inhalt der QuelleDonal, E. „Difficulté de la démarche diagnostique face à une suspicion de la cardiomyopathie arythmogène du ventricule droit. L’échocardiographie garde une valeur“. Annales de Cardiologie et d'Angéiologie 62, Nr. 2 (April 2013): 65–66. http://dx.doi.org/10.1016/j.ancard.2012.04.007.
Der volle Inhalt der QuelleChalabreysse, L., C. Camminada, F. Thivolet und P. Bouvagnet. „Cardiomyopathie arythmogène du ventricule droit : recherche de mutations sur le gène de la desmoplakine dans une série autopsique de 29 patients décédés de mort subite“. Annales de Pathologie 26 (November 2006): 137. http://dx.doi.org/10.1016/s0242-6498(06)78454-1.
Der volle Inhalt der QuelleGandjbakhch, E. „Dysplasie arythmogène du ventricule droit“. Archives des Maladies du Coeur et des Vaisseaux - Pratique 2016, Nr. 248 (Mai 2016): 3–10. http://dx.doi.org/10.1016/j.amcp.2016.03.002.
Der volle Inhalt der QuelleDumousset, E., A. Alfidja, D. Lamaison, J. Ponsonnaille, A. Ravel, J. M. Garcier und L. Boyer. „IRM et dysplasie arythmogène du ventricule droit (DAVD)“. Journal de Radiologie 85, Nr. 3 (März 2004): 313–20. http://dx.doi.org/10.1016/s0221-0363(04)97584-1.
Der volle Inhalt der QuelleJacquier, A., E. Bressollette, J. P. Laissy, J. Y. Gaubert, D. Crochet, G. Moulin und J. M. Bartoli. „IRM et dysplasie arythmogène du ventricule droit (DAVD)“. Journal de Radiologie 85, Nr. 6 (Juni 2004): 721–24. http://dx.doi.org/10.1016/s0221-0363(04)97673-1.
Der volle Inhalt der QuelleDissertationen zum Thema "Cardiomyopathie arythmogène du ventricule droit"
VAUTHIER, MARTINE. „Modalites evolutives des myocardiopathies arythmogenes du ventricule droit“. Rennes 1, 1993. http://www.theses.fr/1993REN1M077.
Der volle Inhalt der QuelleLaredo, Mikaël. „Nouveaux développements techniques et applications cliniques de l'imagerie par résonance magnétique cardiaque et tomodensitométrie pour le diagnostic et la caractérisation de la cardiomyopathie arythmogène“. Electronic Thesis or Diss., Sorbonne université, 2023. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2023SORUS568.pdf.
Der volle Inhalt der QuelleArrhythmogenic cardiomyopathies (ACM) constitute a spectrum of genetically-determined cardiac diseases of which the main consequence are ventricular arrhythmias potentially leading to sudden cardiac death. Arrhythmogenic cardiomyopathies have a diverse range of clinical-imaging phenotypes, and a continuously evolving landscape including concealed structural changes requiring multiparametric assessment. Cardiac magnetic resonance (CMR) is central in their evaluation, given the growing identification of biventricular and left-ventricular(LV)-predominant ACM variants. Multidetector computed tomography (MDCT), while not as established, offers potential utility as either an adjunct or alternative tool. In this thesis, we took advantage on our large ACM cohort to pursue three aims able to extend the boundaries of cardiac imaging for ACM diagnosis and functional characterization. First, we developed and evaluated a novel feature-tracking CMR parameter that integrates both longitudinal and radial right ventricular (RV) dynamics, which was effective in distinguishing ACM patients, particularly those lacking major structural criteria, from healthy subjects, implying that a two-dimensional representation of RV dynamics is crucial to capture the complex physiology of ACM. Second, we described the CMR features of Desmoplakin (DSP)-related ACM, a specific entity associated with particularly worse outcomes amidst the wide diversity of ACMs, and for the first time compared them to those found in ACM with LV involvement. We found that the presence of diffuse late gadolinium enhancement extending beyond the inferolateral LV combined with an end-systolic LV-to-RV volume ratio ≥0.8 may be considered as indicative of a high pre-genetical test results likelihood for DSP-ACM. Third, through a transversal multimodality study, we showed there was overall a low spatial concordance between the arrhythmogenic substrate and morpho-functional abnormalities or RV fat, at a segmental level. This finding paves the way for an integration of EP data into ACM diagnostic criteria and suggest that routine practice cardiac imaging cannot reliably exclude the presence of an EP substrate in a given RV location. Determinant improvements are currently being undertaken to increase the impact and generalizability of the three sets of results
BUISINE, LAURENCE. „Myocardiopathie du ventricule droit antenatale secondaire a la fermeture du canal arteriel par l'indometacine“. Amiens, 1992. http://www.theses.fr/1992AMIEM004.
Der volle Inhalt der QuellePerruc, François. „Analyse comparée des extra systoles ventriculaires sur cœur sain et sur dysplasie ventriculaire droite arythmogène“. Bordeaux 2, 1989. http://www.theses.fr/1989BOR25020.
Der volle Inhalt der QuelleAstanière, Françoise. „Évolution hémodynamique sévère d'un cas de dysplasie ventriculaire droite arythmogène avec hypoxie réfractaire : à propos d'une observation“. Bordeaux 2, 1997. http://www.theses.fr/1997BOR2M098.
Der volle Inhalt der QuelleBarahona-Dussault, Catherine. „Rôle du test génétique dans la cardiomyopathie arythmogène du ventricule droit. Étude sur une cohorte prospective unicentrique“. Thèse, 2008. http://hdl.handle.net/1866/2789.
Der volle Inhalt der QuelleArrhythmogenic right ventricular cardiomyopathy/dysplasia (ARVC/D) is a genetic disorder characterized by the presence of fibro-fatty replacement of the myocardium in the right ventricle. The disease is thought responsible for an important percentage of sudden cardiac death in the young. Hence the disease is usually difficult to diagnose with present clinical tools. ARVC/D is it caused in greater part by mutations in desmosomal proteins. The diagnosis of the genetic carriers bears important implications in family members, who unknowingly may be at risk for sudden death. In order to improve the diagnosis, a new tool, genetic testing, is increasingly being used. Hypothesis: In order to assess the value of genetic testing in complementing clinical testing in ARVC/D, we undertook the project to collect and perform clinical and genetic investigation in 23 probands with the disease. Methods: The probands were usually identified either after the death of a family member or after their clinical investigation for arrhythmias. The diagnosis of ARVC was made with clinical tools according to accepted criteria. Genetic analysis of desmosomal proteins previously associated with the disease was performed by sequencing the exons and intron-exon boundaries. Results: The clinical diagnosis was clear in 18/23 and suspicious in 5/23 individuals. We identified 15 different mutations in 10 probands. 64% of the mutations were not previously described. Interestingly we also observed the presence of double or triple mutants in 40% of the positive individuals. Individuals with mutations were younger and had more symptoms than individuals with no mutation. Conclusion: Genetic testing is useful in 43% of patients with ARVC. The use of mutation-based genetic technology has a very limited value due to the high percentage of previously unknown mutations in this disease. The presence of double and even triple mutants is not associated with a more severe phenotype but it indicates the need to have genetic testing performed for all genes for familial screening. Genetic testing is a useful tool to add to the clinical testing for the diagnosis of patients who do not completely fulfill the clinical criteria for the disease. Key words: genetic, ARVC/D, sudden death, desmosome
Buchteile zum Thema "Cardiomyopathie arythmogène du ventricule droit"
Prouteau, N., und V. Fuzier. „Dysplasie arythmogène du ventricule droit“. In Prise en charge des maladies rares en anesthésie et analgésie obstétricales, 218–20. Elsevier, 2015. http://dx.doi.org/10.1016/b978-2-294-74764-9.00066-2.
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