Thematische Bibliographien / BRAT1

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „BRAT1“

Autor: Grafiati
Veröffentlicht am 14. Dezember 2024

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Zeitschriftenartikel zum Thema "BRAT1"

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Ertl, Johanna, Ömer Güllülü, Stephanie Hehlgans, Franz Rödel, Donat Kögel und Benedikt Linder. „BRAT1 Impairs DNA Damage Repair in Glioblastoma Cell Lines“. Medical Sciences Forum 3, Nr. 1 (29.01.2021): 3. http://dx.doi.org/10.3390/iecc2021-09190.

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Glioblastomas (GBMs) are one of the most malignant brain tumors in adults. This is partly due to the potential presence of so-called glioma stem-like cells (GSCs), which are characterized by the expression of stemness markers and a resistance to radio- and chemotherapy. Previous work from us showed that after combination treatment of GSCs with arsenic trioxide and gossypol, the protein BRCA1-associated ATM-activator 1 (BRAT1) was one of the most downregulated proteins. This protein is largely undescribed, but it has been shown to regulate DNA damage signaling through interaction with ATM, BRCA1, and DNA-PKcs in initial stages of DNA damage response. An unpublished analysis of The Cancer Genome Atlas and The Human Protein Atlas databases showed an increased expression of BRAT1 in GBMs compared to healthy tissues and that an increased expression is negatively correlated with patient survival. Due to these findings, our goal is to analyze the radio-sensitizing effect of BRAT1 on FCS-grown (i.e., differentiated) highly radio-resistant GBM cells and GSCs. Here, using stable knockdowns of BRAT1, we show that it is needed for effective DNA repair after irradiation using a γH2AX-foci assay, whereas it is dispensable for cellular proliferation. A cell death analysis using Annexin V/propidium iodide staining revealed a first hint that BRAT1 downregulation sensitizes GBM cells to irradiation. Moreover, through immunofluorescent staining, we showed that BRAT1 is needed for BRCA1 recruitment to DNA damage sites. Future experiments will aim at systematically analyzing the downstream effects of BRAT1 depletion and to determine further interactors. Thus, we hope to gain a deeper understanding of the mechanism of radio-resistance in GSCs, also in order to individually determine the effectiveness of radiotherapy.
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Mahjoub, Areej, Zuzana Cihlarova, Martine Tétreault, Lauren MacNeil, Neal Sondheimer, Keith W. Caldecott, Hana Hanzlikova und Grace Yoon. „Homozygous pathogenic variant in BRAT1 associated with nonprogressive cerebellar ataxia“. Neurology Genetics 5, Nr. 5 (04.09.2019): e359. http://dx.doi.org/10.1212/nxg.0000000000000359.

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ObjectiveTo investigate the pathogenicity of a novel homozygous BRAT1 variant in 2 siblings with nonprogressive cerebellar ataxia (NPCA) through functional studies on primary and immortalized patient cell lines.MethodsBRAT1 protein levels and ataxia-telangiectasia mutated (ATM) kinase activity in patient-derived and control cell lines were assessed by Western blotting. The impact of the novel BRAT1 variants on mitochondrial function was also assessed, by comparing patient and control cell lines for rates of oxygen consumption and for phosphorylation (S293) of the E1⍺ subunit of pyruvate dehydrogenase (PDH).ResultsTwo male siblings with NPCA, mild intellectual disability, and isolated cerebellar atrophy were found to be homozygous for a c.185T>A (p.Val62Glu) variant in BRAT1 by whole exome sequencing. Western blotting revealed markedly decreased BRAT1 protein levels in lymphocytes and/or fibroblast cells from both affected siblings compared to control cell lines. There were no differences between the patient and control cells in ATM kinase activation, following ionizing radiation. Mitochondrial studies were initially suggestive of a defect in regulation of PDH activity, but there was no evidence of increased phosphorylation of the E1⍺ subunit of the PDH complex. Measurement of oxygen consumption rates similarly failed to identify differences between patient and control cells.ConclusionsBiallelic pathogenic variants in BRAT1 can be associated with NPCA, a phenotype considerably milder than previously reported. Surprisingly, despite the molecular role currently proposed for BRAT1 in ATM regulation, this disorder is unlikely to result from defective ATM kinase or mitochondrial dysfunction.
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Lin, En-Bing, und Eric Linton. „Comparisons of BRAT1 gene variants“. Communications in Information and Systems 19, Nr. 4 (2019): 375–90. http://dx.doi.org/10.4310/cis.2019.v19.n4.a2.

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Yeom, Gyeong Eun, Young Hwa Jung, Soo Yeon Kim, Sun Ah Choi, Hunmin Kim und Chang Won Choi. „First Successful Application of Preimplantation Genetic Diagnosis for Lethal Neonatal Rigidity and Multifocal Seizure Syndrome in Korea: A Case Report“. Neonatal Medicine 29, Nr. 4 (30.11.2022): 141–48. http://dx.doi.org/10.5385/nm.2022.29.4.141.

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Lethal neonatal rigidity and multifocal seizure syndrome (RMFSL) is a severe autosomal recessive epileptic encephalopathy characterized by rigidity, intractable multifocal seizures, microcephaly, apnea, and bradycardia immediately after birth. RMFSL is related to a mutation in breast cancer 1-associated ataxia telangiectasia mutated activation-1 protein (<i>BRAT1</i>). We report a case of a female infant born to non-consanguineous Korean parents who developed hypertonia, dysmorphic features, progressive encephalopathy with refractory seizures at birth, and worsening intermittent apnea, leading to intubation and death at 137 days of age. The initial repeated electroencephalographic findings were normal; however, a pattern of focal seizures emerged at 35 days of life. Rapid trio whole-exome sequencing revealed heterozygous mutations c.1313_1314delAG p.(Gln438Argfs*51) and c.1276C>T p. (Gln426*) in <i>BRAT1</i>. After genetic counseling for pregnancy planning, a preimplantation genetic diagnosis for targeted <i>BRAT1</i> mutations was successfully performed, and a healthy baby was born. To our knowledge, this is the first reported case of a Korean patient with compound heterozygous mutations in <i>BRAT1</i>. An early and accurate genetic diagnosis can help provide timely treatment to patients and indicate the need for reproductive counseling for parents for family planning.
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Srivastava, Siddharth, und Sakkubai Naidu. „Epileptic Encephalopathy Due to BRAT1 Pathogenic Variants“. Pediatric Neurology Briefs 30, Nr. 12 (01.12.2016): 45. http://dx.doi.org/10.15844/pedneurbriefs-30-12-1.

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Szymańska, Krystyna, Milena Laure-Kamionowska, Krzysztof Szczałuba, Agnieszka Koppolu, Mariusz Furmanek, Katarzyna Kuśmierska, Snir Boniel, Rafał Płoski und Małgorzata Rydzanicz. „Clinico-pathological correlation in case of BRAT1 mutation“. Folia Neuropathologica 56, Nr. 4 (2018): 362–71. http://dx.doi.org/10.5114/fn.2018.80870.

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Smith, Nicholas J., Jill Lipsett, Leanne M. Dibbens und Sarah E. Heron. „BRAT1-associated neurodegeneration: Intra-familial phenotypic differences in siblings“. American Journal of Medical Genetics Part A 170, Nr. 11 (02.08.2016): 3033–38. http://dx.doi.org/10.1002/ajmg.a.37853.

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Falcone, Grazia Maria Igea, Alessandra Tessa, Filippo Maria Santorelli, Graziana Tavilla, Antonio Toscano und Olimpia Musumeci. „Adult onset cerebellar ataxia due to novel mutations in BRAT1“. Journal of the Neurological Sciences 429 (Oktober 2021): 118261. http://dx.doi.org/10.1016/j.jns.2021.118261.

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Mundy, Sheraden A., Bryan L. Krock, Rong Mao und Joseph J. Shen. „BRAT1-related disease-identification of a patient without early lethality“. American Journal of Medical Genetics Part A 170, Nr. 3 (22.10.2015): 699–702. http://dx.doi.org/10.1002/ajmg.a.37434.

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Putoux, Audrey, Nicolas Chatron, Anne-Sophie Eyraud-Rousselle, Audrey Labalme, Nathalie Streichenberger, David Meyronet, Julitta De-Regnauld-De-Bellescize, Damien Sanlaville, Patrick Edery und Gaëtan Lesca. „Epileptic encephalopathy caused by BRAT1 mutations: Description of a novel patient“. European Journal of Paediatric Neurology 21 (Juni 2017): e53-e54. http://dx.doi.org/10.1016/j.ejpn.2017.04.901.

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Dissertationen zum Thema "BRAT1"

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Gordon, Derek, und David Rogoff. „Big Brat“. Digital Commons at Loyola Marymount University and Loyola Law School, 2011. https://digitalcommons.lmu.edu/etd/64.

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Borneskog, Ida. „Deconstructing the Brat phenomenon“. Thesis, Stockholms universitet, Centrum för modevetenskap, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-62776.

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Parker, Richard S. „BRAT - (Blender Rapid Animation Tool)“. Thesis, California State University, Long Beach, 2013. http://pqdtopen.proquest.com/#viewpdf?dispub=1523073.

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These are exciting times in the world of computer animation. Individual artists can now produce animations which in the past required teams of artists and programmers to create, but the obstacles to an individual artist can still be daunting in terms of required manual input or technical skills. The goal of this thesis is to demonstrate a simple software tool (BRAT - Blender Rapid Animation Tool) which allows an artist to create cartoon-like 3D animations using the artist's 2D drawings, at the touch of a button. It could be useful for any artist producing 3D animations. The tool is a Python script addon to the Blender 3D modeling system. It is a high level program which links to Blender's Python API library of low level calls to automate the manual work normally required by 3D artists to create an animation. The output of BRAT is a 3D animation file showing a character traversing a rectangular matrix of buildings.

This thesis compares the manual way of creating such an animation with the BRAT automated process. The comparison explains the benefits of using BRAT. This thesis also discusses the design of BRAT and compares it to some other existing 3D software tools whcih also attempt to simplify the 3D animation production process. The conclusion discusses some future enhancements to make BRAT more useful.

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Engel, Camille. „Description phénotypique de formes rares de trouble du développement intellectuel et caractérisation des mécanismes moléculaires impliqués“. Electronic Thesis or Diss., Bourgogne Franche-Comté, 2024. http://www.theses.fr/2024UBFCE006.

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L’avènement des nouvelles techniques de séquençage a permis d’augmenter de façon considérable le taux diagnostique des troubles du développement intellectuel (TDI) et plus de 2000 gènes impliqués sont aujourd’hui connus. Malgré ces progrès considérables, l’interprétation des variants identifiés par les techniques de séquençage reste parfois difficile et l’histoire naturelle des TDI nouvellement décrits est souvent méconnue. Notre travail a consisté à étudier quatre formes de TDI rares de modes de transmissionvariés sur les plans clinique et génétique afin de mieux comprendre ces affections et les mécanismes qui les sous-tendent. Nous avons d’une part précisé les tableaux cliniques associés aux variations de BRAT1, CNOT3 et MTOR et avons recherché l’existence d’éventuelles corrélations phénotype-génotype pour les variations de ces gènes. D’autre part, nous avons participé à la mise en place d’un test fonctionnel permettant de reclasser les variants de signification incertaine de PQBP1
The advent of new sequencing techniques has dramatically increased the diagnostic rate of intellectual disability (ID), and more than 2,000 genes are currently known to be involved. Despite these considerable progresses, interpreting the variants identified by sequencing methods remains challenging, and the natural history of newly described ID is often poorly understood. To better understand these disorders and their underlying mechanisms, we have studied four rare forms of ID with various inheritance patterns from both clinical and genetic perspectives. On one hand, we defined the clinical pictures associated with variations in BRAT1, CNOT3 and MTOR, and we investigated the existence of any phenotype-genotype correlations. On the other hand, we contributed to the design of a functional test to reclassify PQBP1 variants of uncertain significance
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Hunt, Andrew W. „Basic Expeditionary Airfield Resource (BEAR) Requirements Analysis Tool (BRAT)“. Quantico, VA : Marine Corps Command and Staff College, 2008. http://handle.dtic.mil/100.2/ADA491134.

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Brato, Thorsten [Verfasser]. „A sociophonetic study of Aberdeen English : Innovation and conservatism / Thorsten Brato“. Gießen : Universitätsbibliothek, 2012. http://d-nb.info/1064023126/34.

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Levesque, Lauren Patricia. „Media Culture, Artifact and Gender Identity: An Analysis of Bratz Dolls“. Thesis, University of Ottawa (Canada), 2010. http://hdl.handle.net/10393/28628.

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It could be argued that girl's play is witnessing a drastic transformation. This alteration is fostering much debate surrounding young girls and their notion of self identity. Neil Postman (1982) argues that childhood no longer exists as it has disappeared through the mass media. Likewise, Sharon Lamb (2001, 2006) argues that young girls are continually being sold the ideal attitude and a hyper-sexualized self identity through the media messages and products they consume. Such a problematic transformation raises several concerns with regards to girlhood studies. My research asks how MGA Entertainment's Bratz dolls place identity formation into question. By exploring the aforementioned notions, my research explores girl's play and identity and looks at how it contributes to the shaping of how a girl's choice in play impacts girlhood. I argue that such a claim would be best explored and answered through interviewing young girls and their mothers.
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Bergsager, Håkon, und Erlend Hillestad Bårgard. „Forankring av rørgater i bratt terreng : Forsøk med materialer og metoder“. Thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for konstruksjonsteknikk, 2012. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-18767.

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Denne oppgaven tar for seg forankring av nedgravde rørgater. Det er undersøkt interaksjonen mellom rør og jord, først i horisontalt terreng og deretter i bratt terreng med helning over 20 grader. I rapporten er det studert beregningsmodeller som benyttes for beregning av glidestabilitet, motstand mot oppløft og global knekking. Det er sett på både modeller som benyttes i vannkraftbransjen og modeller fra andre fagområder. For å undersøke hvorvidt de ulike beregningsmodellene gjengir virkeligheten ble det i atbeidet med oppgaven utført to typer forsøk. Modellforsøk med rør av polyvinylklorid (PVC) og sand og fullskala feltforsøk med rør av glassfiberarmert plast (GRP) med pukk som omfyllingsmasse. I modellforsøkene ble følgende undersøkt: - Aksiell friksjonskraft i horisontalt og bratt terreng - Motstand mot oppløft - Stivhet i jord og knekking Forsøksplanen for feltforsøkene ble utarbeidet på grunnlag av modellforsøkene, og i disse ble det sett nærmere på: - Aksiell friksjonskraft i horisontalt og bratt terreng - Motstand mot oppløft I feltforsøkene ble det også undersøkt i hvilken grad disse kreftene er avhengig av overdekningshøyde og komprimering. Beregning av knekking ble utført med elementmetodeprogrammene Focus Konstruksjon 2D og Abaqus. Dette for å se om programmene kan benyttes til å simulere knekking av rørgater i bratt terreng. Beregningene ble sammenlignet med modellforsøkene og viste at Abaqus er det mest egnede programmet. Verdiene fra 2D-modellen i Abaqus kan benyttes til gode overslagsberegninger, men for en fullverdig simulering av knekking av nedgravde rørgater bør det gjennomføres mer avanserte beregninger. Resultatene fra modellforsøkene virker til å følge samme trend som beregningsmodellene. Likevel er det noen avvik som kan komme av unøyaktigheter i målingene fra modellforsøkene. I feltforsøkene faller resulatene fra oppløftsforsøkene innforbi de beregnede verdiene. De aksielle friksjonskreftene avviker derrimot mye fra de beregnede verdiene. Sannsynligvis skyldes dette usikkerhet omkring friksjonsfaktoren mellom rør og omfyllingsmasse, og jordtrykket rundt røret. Det er anbefalt konkrete forslag for å studere temaet nærmere.
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Bossel, Hans Eirik Anders, und Mats Breien Haugen. „Forankring av rørgater i bratt terreng : Forsøk med materialer og metoder“. Thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for bygg, anlegg og transport, 2013. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-22363.

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Denne masteroppgaven omhandler forankring av nedgravde rørgater i bratt terreng. Oppgaven er en fortsettelse av et samarbeidsprosjekt mellom NVE og Institutt for vann- og miljøteknikk ved NTNU. Det siste året er det skrevet en masteroppgave og tre prosjektoppgaver om samme tema.Teorien er basert på arbeidet i de nevnte oppgavene. Det er presentert fem ulike beregningsmodeller for å beregne stabiliteten for en rørgate forankret på friksjon. Modellene blir evaluert med to ulike typer av forsøk, modellforsøk utført i laboratorium og fullskala forsøk i felt. Forsøkene er utført med rørsegmenter på 3 ? 6 m, og med diameter, d = 100, 300 og 500 mm. I tillegg er det utført laboratorieforsøk (friksjonsforsøk) for å bestemme friksjonskoeffisienten (&#956;) mellom rør og omfyllingsmasser (pukk). Modellforsøk i laboratorium viser at friksjonskraften mellom rør og omfyllingsmasse reduseres som en funksjon av helningen på røret. Alle beregningsmodeller ser ut til å underestimere friksjonskraften mellom rør og omfyllingsmasse for forsøk utført med horisontalt rør. En av modellene gir imidlertid gode resultater for forsøk utført med rør i 35o helning. Dette viser at en justering av beregningsmodellene med &#8730;(&#12310;tan&#12311;^2 (&#966;)-&#12310;tan&#12311;^2 (&#945;)), hvor &#966; er friksjonsvinkelen til omfyllingsmassen og &#945; er helningen av røret, er en god justering for å kalkulere friksjonen internt i omfyllingsmassen.Fullskala forsøk viser at beregningsmodellene overestimerer friksjonskraften mellom rør og omfyllingsmasser.Resultater fra modellforsøkene utført med horisontalt rør viser godt samsvar med resultater fra feltforsøkene når de skaleres opp med en faktor, k=2*D_fullskala/D_modell , hvor D er diameteren til rørene benyttet i de ulike forsøkene.Resultater fra friksjonsforsøk viser at friksjonskoeffisienten (&#956;) mellom rør og pukk er, &#956; > 0.60, hvilket er en høyere verdi enn hva som er vanlig å benytte i bransjen. Det er vist at fullskala forsøk, med oppsettet som er benyttet under arbeidet med denne oppgaven, kan gi usikre resultat. Det er derfor foreslått at fullskala forsøk som foretas på senere tidspunkt, utføres med samme oppsett som er benyttet i modellforsøkene.Det viktigste videre arbeidet som kan utføres ved en videreføring av oppgaven er arbeid for å undersøke skala- og laboratorieeffekter, både for modellforsøk utført i laboratorium og fullskala forsøk. En god metode for å skalere modellforsøk vil være av stor verdi for å fortsette arbeidet med å kalibrere beregningsmodellene.&#8195;
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Brato, Thorsten [Verfasser]. „Variation and Change in Aberdeen English : A Sociophonetic Study / Thorsten Brato“. Frankfurt a.M. : Peter Lang GmbH, Internationaler Verlag der Wissenschaften, 2016. http://d-nb.info/1123420548/34.

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Bücher zum Thema "BRAT1"

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Mushketyk, I͡Uriĭ. Na brata brat. Kyïv: Vyd-vo "Rada", 1996.

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Saaki︠a︡n, Aramais. Bratu--brat: Stikhi. Moskva: Sov. pisatelʹ, 1986.

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Nemchenko, Gariĭ. Brat, naĭdi brata: Roman i povesti. Moskva: Profizdat, 1985.

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Nemchenko, Gariĭ Leontʹevich. Brat naĭdi brata: Roman i povesti. Moskva: Profizdat, 1985.

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Nemchenko, Gariĭ. Brat, naĭdi brata: Roman i povesti. Moskva: Profizdat, 1985.

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universitet, Ulʹi︠a︡novskiĭ gosudarstvennyĭ tekhnicheskiĭ, Hrsg. Brat na brata: Grazhdanskai︠a︡ voĭna Ivana Usti︠u︡zhaninova. Ulʹi︠a︡novsk: UlGTU, 2014.

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Szczepański, Mieczysław Roman. Kiedy brata zabija brat: Losy żołnierza AK, Mieczysława Romana Szczepańskiego. Londyn: Oficyna Poetów i Malary, 1987.

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Kennedy, X. J. Brats. New York: Atheneum, 1986.

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Kennedy, X. J. Brats. New York: Trumpet Club, 1991.

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Mikhaĭlovich, Prokhorov Gelian, und Institut russkoĭ literatury (Pushkinskiĭ dom), Hrsg. Pisʹma: K materi brata, O.N. Vysotskoĭ, drugu, V.N. Abrosovu i bratu, O.N. Vysotskomu (1945-1991). Sankt-Peterburg: Izdatelʹstvo Pushkinskogo Doma, 2008.

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Buchteile zum Thema "BRAT1"

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Jackson-Bernitsas, Deborah, Kaiyi Li und Shiaw-Yih Lin. „BRIT1 Gene“. In Encyclopedia of Cancer, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_733-2.

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Jackson-Bernitsas, Deborah, Kaiyi Li und Shiaw-Yih Lin. „BRIT1 Gene“. In Encyclopedia of Cancer, 699–702. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_733.

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Jackson-Bernitsas, Deborah, Shiaw-Yih Lin und Kaiyi Li. „BRIT1 Gene“. In Encyclopedia of Cancer, 567–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_733.

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Anderson, Mary M. „Tij Brata“. In The Festivals of Nepal, 116–20. London: Routledge, 2024. http://dx.doi.org/10.4324/9781003540137-17.

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Frederick, Carl. „The Schrödinger Brat Paradox“. In Science Fiction by Scientists, 139–60. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-41102-6_11.

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Marinca, Vasile, Nicolae Herisanu und Bogdan Marinca. „Cylindrical Liouville-Bratu-Gelfand Problem“. In Optimal Auxiliary Functions Method for Nonlinear Dynamical Systems, 343–54. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75653-6_27.

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Großbröhmer, Christoph, Hanna Siebert, Lasse Hansen und Mattias P. Heinrich. „Employing ConvexAdam for BraTS-Reg“. In Brainlesion: Glioma, Multiple Sclerosis, Stroke and Traumatic Brain Injuries, 252–61. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-33842-7_22.

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Abreu-Torres, Dania, Rosana Blanco-Cano und Rita E. Urquijo-Ruiz. „La Mission (Peter Bratt, 2009, USA)“. In Latinidad and Film, 65–72. Cham: Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-56118-4_6.

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„BRAF1“. In Encyclopedia of Cancer, 608. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_100348.

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„BRAF1“. In Encyclopedia of Cancer, 476. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_703.

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Konferenzberichte zum Thema "BRAT1"

1

Levi, Riccardo, Aldo Marzullo, Giovanni Savini, Victor Savevski und Letterio S. Politi. „An Unet Boosting Training Strategy for the BraTS-ISBI 2024 Goat Challenge“. In 2024 IEEE International Symposium on Biomedical Imaging (ISBI), 1–4. IEEE, 2024. http://dx.doi.org/10.1109/isbi56570.2024.10635202.

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Zhang, Qimin, Weiwei Qi, Huili Zheng und Xinyu Shen. „CU-Net: A U-Net Architecture for Efficient Brain-Tumor Segmentation on BraTS 2019 Dataset“. In 2024 4th International Conference on Machine Learning and Intelligent Systems Engineering (MLISE), 255–58. IEEE, 2024. http://dx.doi.org/10.1109/mlise62164.2024.10674119.

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Datta, Gourab, Mohammad Jawad-Ul Kabir Chowdhury, S. A. Naimul Hoque und Md Aminul Islam. „Performance Analysis of U-Net and Unetr For 2D BraTS Within Consumer-Grade Hardware Limits“. In 2024 8th International Artificial Intelligence and Data Processing Symposium (IDAP), 1–7. IEEE, 2024. http://dx.doi.org/10.1109/idap64064.2024.10710974.

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Amor, Feriel, Hiba Mzoughi, Ines Njeh und Mohamed Ben Slima. „Review of MRI brain tumor segmentation and MGMT promoter classification methods on BraTs dataset based on Deep learning“. In 2024 IEEE 7th International Conference on Advanced Technologies, Signal and Image Processing (ATSIP), 249–54. IEEE, 2024. http://dx.doi.org/10.1109/atsip62566.2024.10638990.

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Neyaz, Zarqua, und Himanshu Mittal. „Integrating 3D U-Net and Attention U-Net for Brain Tumor Segmentation: Performance Evaluation on BRATS 2021 Dataset“. In 2024 15th International Conference on Computing Communication and Networking Technologies (ICCCNT), 1–6. IEEE, 2024. http://dx.doi.org/10.1109/icccnt61001.2024.10724970.

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Vega, L., D. Lederer, C. Hocq, B. Van Grambezen, V. Benoit, S. Flas, O. Danhaive und M. R. Cilio. „The Neonatal Presentation of BRAT1-Related Neonatal Rigidity and Multifocal Seizure Syndrome“. In Abstracts of the 48th Annual Meeting of the SENP (Société Européenne De Neurologie Pédiatrique). Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1746209.

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So, Eui Young, und Toru Ouchi. „Abstract 5407: The novel roles of BRCA1/ATM-associated BRAT1 in glucose/mitochondrial metabolism and tumor cell growth.“ In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5407.

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Akbar, Agus Subhan, Chastine Fatichah und Nanik Suciati. „Simple MyUnet3D for BraTS Segmentation“. In 2020 4th International Conference on Informatics and Computational Sciences (ICICoS). IEEE, 2020. http://dx.doi.org/10.1109/icicos51170.2020.9299072.

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Setiawan, Budiana. „Women's Leadership in Asta Brata“. In Proceedings of the 3rd International Symposium on Religious Life, ISRL 2020, 2-5 November 2020, Bogor, Indonesia. EAI, 2021. http://dx.doi.org/10.4108/eai.2-11-2020.2305079.

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Bozhkov, Yu, und S. Dimas. „The Liouville-Bratu-Gelfand problem“. In THE 5TH INTERNATIONAL CONFERENCE ON COMPUTATIONAL INTELLIGENCE IN INFORMATION SYSTEMS (CIIS 2022): Intelligent and Resilient Digital Innovations for Sustainable Living. AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0177433.

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Berichte der Organisationen zum Thema "BRAT1"

1

Pernice, M., und B. Gunney. Solution of the Modified Bratu Problem in SAMRAI. Office of Scientific and Technical Information (OSTI), Februar 2004. http://dx.doi.org/10.2172/15013877.

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2

Sung, Patrick M. Interactions Among Brac1, Brac2, and Components of the Recombination Machinery. Fort Belvoir, VA: Defense Technical Information Center, Juni 2001. http://dx.doi.org/10.21236/ada398099.

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Sung, Patrick M. Interactions Among Brac1, Brac2, and Components of the Recombination Machinery. Fort Belvoir, VA: Defense Technical Information Center, Juni 1999. http://dx.doi.org/10.21236/ada381339.

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