Auswahl der wissenschaftlichen Literatur zum Thema „Bones Pathophysiology“
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Zeitschriftenartikel zum Thema "Bones Pathophysiology":
Wiegerinck, R. F., R. Schreurs und F. W. Prinzen. „Pathophysiology of dyssynchrony: of squirrels and broken bones“. Netherlands Heart Journal 24, Nr. 1 (10.12.2015): 4–10. http://dx.doi.org/10.1007/s12471-015-0765-7.
Shapkin, Yu G., P. A. Seliverstov und E. A. Skripal. „THE PHENOMENON OF «SECOND HIT» AFTER OPERATIONS OF OSTEOSYNTHESIS IN CASE OF POLY-TRAUMA“. Medical Journal of the Russian Federation 23, Nr. 6 (15.12.2017): 331–36. http://dx.doi.org/10.18821/0869-2106-2017-23-6-331-336.
Thévenin-Lemoine, C., J. Vial, J. L. Labbé, B. Lepage, B. Ilharreborde und F. Accadbled. „MRI of acute osteomyelitis in long bones of children: Pathophysiology study“. Orthopaedics & Traumatology: Surgery & Research 102, Nr. 7 (November 2016): 831–37. http://dx.doi.org/10.1016/j.otsr.2016.06.014.
Tasis, Nikolaos, Ioannis Tsouknidas, Argyrios Ioannidis, Konstantinos Nassiopoulos und Dimitrios Filippou. „Left Functional Pneumonectomy Caused by a Very Rare Giant Intrathoracic Cystic Lesion in a Patient with Gorham–Stout Syndrome: Case Report and Review of the Literature“. Case Reports in Pulmonology 2018 (2018): 1–9. http://dx.doi.org/10.1155/2018/2406496.
Kuhweide, R., V. Van de Steene, S. Vlaminck und J. W. Casselman. „Ramsay Hunt syndrome: pathophysiology of cochleovestibular symptoms“. Journal of Laryngology & Otology 116, Nr. 10 (Oktober 2002): 844–48. http://dx.doi.org/10.1258/00222150260293691.
Puntillo, Filomena, Mariateresa Giglio, Antonella Paladini, Gaetano Perchiazzi, Omar Viswanath, Ivan Urits, Carlo Sabbà, Giustino Varrassi und Nicola Brienza. „Pathophysiology of musculoskeletal pain: a narrative review“. Therapeutic Advances in Musculoskeletal Disease 13 (Januar 2021): 1759720X2199506. http://dx.doi.org/10.1177/1759720x21995067.
Shams, Ramsha, Kelsey P. Drasites, Vandana Zaman, Denise Matzelle, Donald C. Shields, Dena P. Garner, Christopher J. Sole, Azizul Haque und Narendra L. Banik. „The Pathophysiology of Osteoporosis after Spinal Cord Injury“. International Journal of Molecular Sciences 22, Nr. 6 (17.03.2021): 3057. http://dx.doi.org/10.3390/ijms22063057.
Gore, Ashwini P., Soon Ho Kwon und Antine E. Stenbit. „A Roadmap to the Brittle Bones of Cystic Fibrosis“. Journal of Osteoporosis 2011 (2011): 1–10. http://dx.doi.org/10.4061/2011/926045.
Assi, Tarek, Sarah Watson, Bachar Samra, Elie Rassy, Axel Le Cesne, Antoine Italiano und Olivier Mir. „Targeting the VEGF Pathway in Osteosarcoma“. Cells 10, Nr. 5 (18.05.2021): 1240. http://dx.doi.org/10.3390/cells10051240.
Golovach, I. Yu. „Clinical significance of spondyloarthritis-attended enthesites: from pathophysiology to treatment (review)“. PAIN, JOINTS, SPINE 11, Nr. 1 (01.04.2021): 17–27. http://dx.doi.org/10.22141/2224-1507.11.1.2021.226905.
Dissertationen zum Thema "Bones Pathophysiology":
Anderson, Paul Hamill. „The regulation of Vitamin D metabolism in the kidney and bone“. Title page, contents and abstract only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09pha5486.pdf.
Huang, C. C. „Pathophysiology of post-transplantation bone disease : mechanisms of bone loss after orthotopic liver transplantation“. Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604707.
Chapurlat, Roland, Deborah Gensburger, Juan Jimenez-Andrade, Joseph Ghilardi, Marilyn Kelly und Patrick Mantyh. „Pathophysiology and medical treatment of pain in fibrous dysplasia of bone“. BioMed Central, 2012. http://hdl.handle.net/10150/610228.
Borazjani, Ali. „Pathophysiology of Pelvic Organ Prolapse“. Cleveland State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=csu1432745397.
Ismail, Medhat Mohamed. „Pathophysiology and therapy of bone marrow failure : studies of apoptosis and stem cell transplantation“. Thesis, St George's, University of London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269742.
Elkin, Sarah Louise. „The pathogenesis and pathophysiology of low bone mineral density in adults with cystic fibrosis“. Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.422707.
Edwards, Sarah. „Investigating the role of a novel ER molecular chaperone : Creld2 in the physiology and pathophysiology of endochondral bone growth“. Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/investigating-the-role-of-a-novel-er-molecular-chaperone-creld2-in-the-physiology-and-pathophysiology-of-endochondral-bone-growth(6fd49909-beec-42d1-a546-8b2411616e59).html.
Davies, Matthew Rhys. „The role of bone morphogenetic protein 7 in the pathophysiology and treatment of vascular calcification associated with chronic renal failure“. Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1444711/.
Ho, Ken Choong Khoon School of Medicine UNSW. „Characterization of critical size sheep cranial defect model for study of bone graft substitute“. 2007. http://handle.unsw.edu.au/1959.4/40499.
Dodge, Todd Randall. „Experimental and Computational Analysis of Dynamic Loading for Bone Formation“. Thesis, 2013. http://hdl.handle.net/1805/3670.
Bone is a dynamic tissue that is constantly remodeling to repair damage and strengthen regions exposed to loads during everyday activities. However, certain conditions, including long-term unloading of the skeleton, hormonal imbalances, and aging can disrupt the normal bone remodeling cycle and lead to low bone mass and osteoporosis, increasing risk of fracture. While numerous treatments for low bone mass have been devised, dynamic mechanical loading modalities, such as axial loading of long bones and lateral loading of joints, have recently been examined as potential methods of stimulating bone formation. The effectiveness of mechanical loading in strengthening bone is dependent both on the structural and geometric characteristics of the bone and the properties of the applied load. For instance, curvature in the structure of a bone causes bending and increased strain in response to an axial load, which may contribute to increased bone formation. In addition, frequency of the applied load has been determined to impact the degree of new bone formation; however, the mechanism behind this relationship remains unknown. In this thesis, the application of mechanical loading to treat osteoporotic conditions is examined and two questions are addressed: What role does the structural geometry of bone play in the mechanical damping of forces applied during loading? Does mechanical resonance enhance geometric effects, leading to localized areas of elevated bone formation dependent on loading frequency? Curvature in the structure of bone was hypothesized to enhance its damping ability and lead to increased bone formation through bending. In addition, loading at frequencies near the resonant frequencies of bone was predicted to cause increased bone formation, specifically in areas that experienced high principal strains due to localized displacements during resonant vibration. To test the hypothesis, mechanical loading experiments and simulations using finite element (FE) analysis were conducted to characterize the dynamic properties of bone. Results demonstrate that while surrounding joints contribute to the greatest portion of the damping capacity of the lower limb, bone absorbs a significant amount of energy through curvature-driven bending. In addition, results show that enhanced mechanical responses at loading frequencies near the resonant frequencies of bone may lead to increased bone formation in areas that experience the greatest principal strain during vibration. These findings demonstrate the potential therapeutic effects of mechanical loading in preventing costly osteoporotic fractures, and explore characteristics of bone that may lead to optimization of mechanical loading techniques. Further investigation of biomechanical properties of bone may lead to the prescribing of personalized mechanical loading treatments to treat osteoporotic diseases.
Bücher zum Thema "Bones Pathophysiology":
Medicine, Conference on Skeletal Biology and. Skeletal biology and medicine. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2007.
NATO Advanced Study Institute on Advances in Bone Regulatory Factors: Morphology, Biochemistry, Physiology, and Pharmacology (1989 Erice, Italy). Bone regulatory factors: Morphology, biochemistry, physiology, and pharmacology. New York: Plenum Press, 1990.
International Symposium on Bone Circulation (4th 1987 Toulouse, France). Bone circulation and bone necrosis: Proceedings of the IVth International Symposium on Bone Circulation, Toulouse (France), 17th-19th September 1987. Berlin: Springer-Verlag, 1990.
Conference on Skeletal Biology and Medicine (2nd 2007 New York, N.Y.). Skeletal biology and medicine. Boston, Mass: Blackwell Pub., on behalf of the New York Academy of Sciences, 2007.
Conference on Skeletal Biology and Medicine (4th 2011 New York, N.Y.). Skeletal biology and medicine II: Bone and cartilage homeostasis and bone disease. Herausgegeben von Zaidi Mone. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2011.
Behari, Jitendra. Biophysical bone behavior. Singapore: John Wiley, 2009.
N.Y.) Conference on Skeletal Biology and Medicine (3rd 2010 New York. Skeletal biology and medicine. Herausgegeben von Zaidi Mone und New York Academy of Sciences. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.
NATO Advanced Study Institute on Advances in Bone Regulatory Factors: Morphology, Biochemistry, Physiology, and Pharmacology (1989 Erice, Italy). Bone regulatory factors: Morphology, biochemistry, physiology, and pharmacology. New York: Plenum Press, 1990.
Lee, Diane. The pelvic girdle: An integration of clinical expertise and research. 4. Aufl. Edinburgh: Elsevier/Churchill Livingstone, 2011.
Lundon, Katie. Orthopedic rehabilitation science: Principles for clinical management of nonmineralized connective tissue. Amsterdam: Butterworth-Heinemann, 2003.
Buchteile zum Thema "Bones Pathophysiology":
Mori, Satoshi. „Bone Microdamage and Its Repair: Pathophysiology of Bone Fatigue“. In Mechanical Loading of Bones and Joints, 139–45. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-65892-4_14.
Roodman, G. David. „Pathophysiology of Bone Metastases“. In Bone Metastases, 31–50. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-1-4020-9819-2_2.
Chirgwin, John M., und G. David Roodman. „Pathophysiology of Bone Metastases“. In Bone Metastases, 3–17. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7569-5_1.
Van der Wall, Hans, Barry Elison, Clayton Frater, Warwick Bruce und Stephen Clarke. „Pathophysiology of Bone Metastases“. In Radionuclide and Hybrid Bone Imaging, 59–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02400-9_3.
Kerschan-Schindl, Katharina, Ursula Föger-Samwald und Peter Pietschmann. „Pathophysiology of Bone Fragility“. In Principles of Bone and Joint Research, 83–97. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58955-8_6.
Bartl, Reiner, und Christoph Bartl. „Classifying Bone Disorders According to Pathophysiology“. In Bone Disorders, 55–59. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29182-6_9.
Caniggia, Angelo. „Pathophysiology of Bone Formation and Resorption“. In Bone Regulatory Factors, 235–52. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-1508-8_14.
Jones, John Paul. „Pathophysiology of Osteonecrosis“. In Bone Circulation and Vascularization in Normal and Pathological Conditions, 249–61. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2838-8_28.
Pietschmann, P., R. Gruber und M. Peterlik. „Pathophysiology and Aging of Bone“. In Radiology of Osteoporosis, 3–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05235-8_1.
Thürlimann, Beat. „Physiology and Pathophysiology of Bone“. In Recent Results in Cancer Research, 1–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59845-6_1.
Konferenzberichte zum Thema "Bones Pathophysiology":
Hedrich, Christian. „SP0187 PATHOPHYSIOLOGY AND THERAPEUTIC CONSEQUENCES AUTO-INFLAMMATORY BONE DISORDERS“. In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.8494.
Slichter, Sherrill J. „PATHOPHYSIOLOGY OF THROMBOCYTOPENIA AND RESULTANT CLINICAL INDICATIONS FOR PLATELET TRANSFUSION“. In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643996.
de la Puente, Pilar, Rebecca Gilson, Barbara Muz, Feda Azab, Justin King, Samuel Achilefu, Ravi Vij und Abdel Kareem Azab. „Abstract 5356: 3D tissue-engineered bone marrow niche as novel method to study pathophysiology and drug resistance in multiple myeloma“. In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-5356.
Berichte der Organisationen zum Thema "Bones Pathophysiology":
Zhang, Jiwang. The Role of Necroptosis in the Pathophysiology of Bone Marrow Failure. Fort Belvoir, VA: Defense Technical Information Center, März 2014. http://dx.doi.org/10.21236/ada604192.