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1

Ahmed, Mohamed H. „Biochemical Markers“. American Journal of Clinical Pathology 127, Nr. 1 (Januar 2007): 20–22. http://dx.doi.org/10.1309/jxwum661t8vt1etx.

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2

Rothstein, Jeffrey. „Biochemical markers: Pro“. Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders 3, sup1 (September 2002): S81. http://dx.doi.org/10.1080/146608202320374381.

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3

Mitsumoto, Hiroshi. „Biochemical markers: Con“. Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders 3, sup1 (September 2002): S83. http://dx.doi.org/10.1080/146608202320374390.

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4

Strong, Michael J. „Biochemical markers: Summary“. Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders 3, sup1 (September 2002): S85—S90. http://dx.doi.org/10.1080/146608202320374408.

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5

Sandberg, Dr K. „3. Biochemical markers“. Animal Genetics 20, Nr. 1 (24.04.2009): 56–83. http://dx.doi.org/10.1111/j.1365-2052.1989.tb01910.x.

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6

Seishima, Mitsuru, Makiko Suzuki und Satoshi Maeda. „Atherosclerosis and biochemical markers.“ SEIBUTSU BUTSURI KAGAKU 48, Nr. 4 (2004): 143–46. http://dx.doi.org/10.2198/sbk.48.143.

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7

Selroos, Olof B. N. „Biochemical Markers in Sarcoidosis“. CRC Critical Reviews in Clinical Laboratory Sciences 24, Nr. 3 (Januar 1986): 185–216. http://dx.doi.org/10.3109/10408368609110273.

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8

Đerić, Mirjana, Sunčica Kojić-Damjanov, Velibor Čabarkapa und Nevena Eremić. „Biochemical Markers of Atherosclerosis“. Journal of Medical Biochemistry 27, Nr. 2 (01.01.2008): 148–53. http://dx.doi.org/10.2478/v10011-008-0008-1.

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Biochemical Markers of AtherosclerosisThis paper is a brief review of some lipid parameters and serum markers of inflammation in a view of their predictive relevance for the atherosclerotic disease. A discourse on the importance of measuring different lipids and lipoproteins, concentration of LDL particles and apolipoprotein levels is still underway. Also, the recommendations for apolipoprotein (a), phenotypization and other lipid markers have not yet been established. In recent years the recommendations imply simultaneous measuring of multiple markers and calculating the lipid index values such as lipid tetrad index (LTI), lipid pentad index (LPI) and atherogenic index of plasma (AIP). Several circulating markers of inflammation such as C-reactive protein, serum fibrinogen and elevated leukocyte number, are consistently associated with atherosclerosis. In spite of a lack of evidence on measuring the C-reactive protein in a wide population, the guidelines for its application in diagnostics and therapy of coronary heart disease were developed. Some proinflammatory cytokines, adhesion molecules and markers of leukocyte activation are promising markers, requiring, however, more detailed prospective evaluation. The question to be elucidated is if these inflammatory markers are directly involved in the pathogenic process.
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9

Kanis, John. „Biochemical markers in osteoporosis“. Scandinavian Journal of Clinical and Laboratory Investigation 57 (1997): 6–11. http://dx.doi.org/10.3109/00365519709168303.

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10

Ratcliffe, Anthony, und Markus J. Seibel. „Biochemical markers of osteoarthritis“. Current Opinion in Rheumatology 2, Nr. 5 (Oktober 1990): 770–76. http://dx.doi.org/10.1097/00002281-199002050-00014.

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11

Kanis, John A. „Biochemical markers in osteoporosis“. Scandinavian Journal of Clinical and Laboratory Investigation 57, sup227 (Januar 1997): 6–11. http://dx.doi.org/10.1080/00365519709168303.

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12

Jung, Rex E., Ronald A. Yeo, Stephen J. Chiulli, Wilmer L. Sibbitt, David C. Weers, Blaine L. Hart und William M. Brooks. „Biochemical markers of cognition“. NeuroReport 10, Nr. 16 (November 1999): 3327–31. http://dx.doi.org/10.1097/00001756-199911080-00014.

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13

Ikizler, T. Alp. „Biochemical markers: Clinical aspects“. Journal of Renal Nutrition 7, Nr. 2 (April 1997): 61–64. http://dx.doi.org/10.1016/s1051-2276(97)90039-x.

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14

Paselk, R. „Biochemical Markers for Cancer“. Biochemical Education 13, Nr. 1 (Januar 1985): 43. http://dx.doi.org/10.1016/0307-4412(85)90154-2.

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15

Stadtman, E. R. „Biochemical markers of aging“. Experimental Gerontology 23, Nr. 4-5 (Januar 1988): 327–47. http://dx.doi.org/10.1016/0531-5565(88)90036-8.

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16

Samoilova, I. G., M. V. Matveeva und D. E. Galyukova. „Biochemical markers of autism“. Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 124, Nr. 1 (2024): 55. http://dx.doi.org/10.17116/jnevro202412401155.

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17

Duffy, M. J. „Biochemical markers as prognostic indices in breast cancer“. Clinical Chemistry 36, Nr. 2 (01.02.1990): 188–91. http://dx.doi.org/10.1093/clinchem/36.2.188.

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Abstract Traditional prognostic markers in breast cancer include histological variables such as tumor size, grade, and axillary node status. In recent years some new potential prognostic markers of a biochemical nature have been described: estradiol receptors, progesterone receptors, epidermal growth factor receptors, erbB-2 proto-oncogene, and certain proteolytic enzymes. None of these new markers excels axillary node status as a prognostic marker. Biochemical markers can, however, be evaluated with use of minimal surgery and may help distinguish the minority of aggressive axillary-node-negative breast cancers.
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18

Watts, Nelson B. „Clinical Utility of Biochemical Markers of Bone Remodeling“. Clinical Chemistry 45, Nr. 8 (01.08.1999): 1359–68. http://dx.doi.org/10.1093/clinchem/45.8.1359.

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Abstract Remodeling is essential for bone health. It begins with resorption of old bone by osteoclasts, followed by the formation of new bone by osteoblasts. Remodeling is coupled (formation is linked to resorption). After middle age or perhaps beginning earlier, bone loss occurs because resorption exceeds formation. This imbalance is accentuated by estrogen deficiency as well as by many diseases and conditions. Biochemical markers that reflect remodeling and can be measured in blood or urine include resorption markers (e.g., collagen cross-links) and formation markers (e.g., alkaline phosphatase). Bone markers exhibit substantial short-term and long-term fluctuations related to time of day, phase of the menstrual cycle, and season of the year, as well as diet, exercise, and anything else that alters bone remodeling. These biological factors, in addition to assay imprecision, produce significant intra- and interindividual variability in markers. Bone marker measurements are noninvasive, inexpensive, and can be repeated often. Unfortunately, most of the studies that provided insight on clinical situations did not focus on markers as a primary endpoint. Bone markers have been useful in clinical practice and have been helpful in understanding the pathogenesis of osteoporosis and the mechanism of action of therapies. In clinical trials, markers aid in selecting optimal dose and in understanding the time course of onset and resolution of treatment effect. Clinical questions that might be answered by bone markers include diagnosing osteoporosis, identifying “fast bone losers” and patients at high risk of fracture, selecting the best treatment for osteoporosis, and providing an early indication of the response to treatment. Additional information is needed to define specific situations and cut points to allow marker results to be used with confidence in making decisions about individual patients.
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19

Cheung, K. L., S. E. Pinder, C. Paish, A. H. Sadozye, S. Y. Chan, A. J. Evans, R. W. Blamey und J. F. R. Robertson. „The Role of Blood Tumor Marker Measurement (Using a Biochemical Index Score and C-Erbb2) in Directing Chemotherapy in Metastatic Breast Cancer“. International Journal of Biological Markers 15, Nr. 3 (Juli 2000): 203–9. http://dx.doi.org/10.1177/172460080001500310.

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The role of blood tumor markers in monitoring response in advanced breast cancer is established in endocrine therapy and standard chemotherapy. This study examines marker levels in patients receiving new chemotherapy regimens. Thirty patients were recruited into two multicenter trials in which docetaxel-based regimens were used in 15 patients. The other 15 received doxorubicin-based regimens. Biochemical response calculated from a score using CA15.3, CEA and ESR was compared with UICC response. Marker changes at 2, 4 and 5 months correlated with UICC response at 3, 41/2 and 6 months, respectively (p < 0.03). Eleven patients achieved both clinical/radiological and biochemical response at the end of treatment; markers had not yet returned to below cutoffs in seven, suggesting a possible advantage to continue chemotherapy. No patient showed a biochemical response whilst judged clinically/radiologically progressive. Nineteen patients had progressed either clinically/radiologically or biochemically at six months; of these, eight showed progression assessed earlier by markers so that a median of four cycles of chemotherapy could have been saved. Measurements of serum c-erbB2 showed a correlation with tissue c-erbB2 staining in the primary tumor (p < 0.003). Among the patients with positive tissue staining, sequential changes in serum c-erbB2 completely paralleled initial response.
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20

Zaitseva, O. V. „Biochemical markers of bone collagen type I metabolism“. Ukrainian Biochemical Journal 87, Nr. 1 (27.02.2015): 21–32. http://dx.doi.org/10.15407/ubj87.01.021.

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21

Franco, Leonardo, und Mario Alejandro Ortíz Salazar. „Biochemical markers of bone metabolism“. Revista Estomatología 18, Nr. 1 (28.09.2017): 30–34. http://dx.doi.org/10.25100/re.v18i1.5707.

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The quantity and quality of bone tissue renewal are dependent on the generation of new bone (deposition) mediated by osteoblasts and the loss (resorption) mediated by osteoclasts. For each of these processes there are important markers that can be measured in serum or urine. Resorption markers are products of metabolic degradation of bone matrix in particu-lar of the type I collagen (hydroxyproline, pyridinoline and deoxypyridinoline). In addition, the resorptive activity can also be evaluated through the tartrate resistant acid phosphatase (TRAP) and calcium-creatinine ratio in urine. Bone formation markers are collagen proteins (ALP, OCN), non collagen (ONC, OPN, BSP) or fragments of collagen synthesis (procollagen peptides).
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22

Rosen, Clifford J., und Alan Tenenhouse. „Biochemical markers of bone turnover“. Postgraduate Medicine 104, Nr. 4 (Oktober 1998): 101–14. http://dx.doi.org/10.3810/pgm.1998.10.447.

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23

Accomando, Salvatore. „Biochemical markers in Celiac disease“. Frontiers in Bioscience S2, Nr. 1 (2010): 313–17. http://dx.doi.org/10.2741/s66.

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24

Bapayeva, G. B. „Biochemical markers of premature birth“. Journal of obstetrics and women's diseases 54, Nr. 3 (01.11.2005): 38–41. http://dx.doi.org/10.17816/jowd83449.

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Summary: In this article the results of the research of diagnostic importance of myoglobin determination in peripheral blood and amniotic fluid, fetal fibronectin examination in cervicovaginal composition contents of a pregnant woman are represented in prediction of premature birth.
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25

Koshul’ko, P. A., M. S. Kovalenko, Yu V. Abalenihina, A. I. Mirov, O. E. Golofast und E. D. Rokunov. „Biochemical markers of missed pregnancy“. Problemy reproduktsii 27, Nr. 6 (2021): 138. http://dx.doi.org/10.17116/repro202127061138.

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26

Lyubimova, N. V., und N. E. Kushlinskiy. „Biochemical markers of bone metastasis“. Advances in molecular oncology 2, Nr. 1 (02.06.2015): 061. http://dx.doi.org/10.17650/2313-805x.2015.2.1.061-075.

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27

Lyubimova, N. V., und N. E. Kushlinskiy. „Biochemical markers of bone metastasis“. Advances in molecular oncology 2, Nr. 1 (02.06.2015): 061. http://dx.doi.org/10.17650/2313-805x.2015.2.1.61-75.

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28

Kovářů, H., F. Kovářů, A. Fišerová, P. Zelníčková und E. Matalová. „Biochemical Markers of Lymphocyte Maturation“. Acta Veterinaria Brno 71, Nr. 4 (2002): 503–8. http://dx.doi.org/10.2754/avb200271040503.

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29

Delmas, Pierre D. „Biochemical markers of bone turnover“. Acta Orthopaedica Scandinavica 66, sup266 (Januar 1995): 176–82. http://dx.doi.org/10.3109/17453679509157687.

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30

Griesmacher, A., P. Peichl, P. Pointinger, R. Mateau und H. Broll. „Biochemical markers in menopausal women“. Scandinavian Journal of Clinical and Laboratory Investigation 57 (1997): 64–72. http://dx.doi.org/10.3109/00365519709168309.

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31

Apple, Fred. „Biochemical markers of thrombolytic success“. Scandinavian Journal of Clinical and Laboratory Investigation 59 (1999): 60–66. http://dx.doi.org/10.3109/00365519909168328.

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32

Kemp, M., J. Donovan, H. Higham und J. Hooper. „Biochemical markers of myocardial injury“. British Journal of Anaesthesia 93, Nr. 1 (Juli 2004): 63–73. http://dx.doi.org/10.1093/bja/aeh148.

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33

Kerrigan, Richard W., und Ian K. Ross. „Extracellular Laccases: Biochemical Markers forAgaricusSystematics“. Mycologia 80, Nr. 5 (September 1988): 689–95. http://dx.doi.org/10.1080/00275514.1988.12025602.

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34

Lyubimova, N. V., T. K. Churikova, M. G. Toms und N. E. Kushlinskiy. „BIOCHEMICAL MARKERS OF NEUROENDOCRINAL TUMORS“. Tambov University Reports. Series: Natural and Technical Sciences 21, Nr. 2 (2016): 494–510. http://dx.doi.org/10.20310/1810-0198-2016-21-2-494-510.

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35

Krstic, Danijela, Nenad Tomic, Branimir Radosavljevic, Natasa Avramovic, Vesna Dragutinovic, Sanja Radojevic Skodric und Mirjana Colovic. „Biochemical Markers of Renal Function“. Current Medicinal Chemistry 23, Nr. 19 (19.07.2016): 2018–40. http://dx.doi.org/10.2174/0929867323666160115130241.

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36

Demers, Laurence M., Luis Costa und Allan Lipton. „Biochemical Markers and Skeletal Metastases“. Clinical Orthopaedics and Related Research 415 (Oktober 2003): S138—S147. http://dx.doi.org/10.1097/01.blo0000092979.12414.54.

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37

Thompson, R. P. H. „Biochemical markers of liver disease“. Current Opinion in Gastroenterology 1, Nr. 3 (Mai 1985): 377–89. http://dx.doi.org/10.1097/00001574-198505000-00002.

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38

Matull, W. R. „Biochemical markers of acute pancreatitis“. Journal of Clinical Pathology 59, Nr. 4 (01.04.2006): 340–44. http://dx.doi.org/10.1136/jcp.2002.002923.

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39

Engler, H., B. Thürlimann und F. Riesen. „Biochemical Markers of Bone Remodelling“. Oncology Research and Treatment 19, Nr. 2 (1996): 126–31. http://dx.doi.org/10.1159/000218778.

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40

Griesmacher, A., P. Peichl, P. Pointinger, R. Mateau und H. Bröll. „Biochemical markers in menopausal women“. Scandinavian Journal of Clinical and Laboratory Investigation 57, sup227 (Januar 1997): 64–72. http://dx.doi.org/10.1080/00365519709168309.

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41

Apple, Fred S. „Biochemical markers of thrombolytic success“. Scandinavian Journal of Clinical and Laboratory Investigation 59, sup230 (Januar 1999): 60–66. http://dx.doi.org/10.1080/00365519909168328.

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42

Gao, F., und D. N. F. Harris. „Biochemical markers of cerebral damage“. European Journal of Anaesthesiology 14, Nr. 2 (März 1997): 113–17. http://dx.doi.org/10.1097/00003643-199703000-00001.

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43

De Jongh, R., C. De Deyne, G. Kenis, G. Vundelinckx, L. Merckx und R. Heylen. „Biochemical markers of brain ischaemia“. European Journal of Anaesthesiology 15, Supplement 17 (Januar 1998): 14–15. http://dx.doi.org/10.1097/00003643-199801001-00011.

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44

Crofton, P. M., H. F. Stirling, E. Schönau, S. F. Ahmed, W. H. B. Wallace, J. C. Wade, R. Magowan et al. „Biochemical Markers of Bone Turnover“. Hormone Research 45, Nr. 1 (1996): 55–58. http://dx.doi.org/10.1159/000184832.

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45

Rambotti, Pietro. „Biochemical markers in lymphoproliferative diseases“. Critical Reviews in Oncology/Hematology 2, Nr. 4 (Januar 1985): 297–321. http://dx.doi.org/10.1016/s1040-8428(85)80006-8.

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46

Shepherd, J. „Biochemical risk markers of CHD“. Atherosclerosis 144 (Mai 1999): 144. http://dx.doi.org/10.1016/s0021-9150(99)80558-9.

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47

Seibel, Markus J. „Biochemical markers of bone remodeling“. Endocrinology and Metabolism Clinics of North America 32, Nr. 1 (März 2003): 83–113. http://dx.doi.org/10.1016/s0889-8529(02)00077-4.

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48

Garnero, Patrick, und Pierre D. Delmas. „BIOCHEMICAL MARKERS OF BONE TURNOVER“. Endocrinology and Metabolism Clinics of North America 27, Nr. 2 (Juni 1998): 303–23. http://dx.doi.org/10.1016/s0889-8529(05)70007-4.

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49

Gavaghan, Mary. „Biochemical Markers in Myocardial Injury“. AORN Journal 70, Nr. 5 (November 1999): 839–54. http://dx.doi.org/10.1016/s0001-2092(06)61303-3.

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50

Yuasa, Isao, und Kazuo Umetsu. „Molecular aspects of biochemical markers“. Legal Medicine 7, Nr. 4 (Juli 2005): 251–54. http://dx.doi.org/10.1016/j.legalmed.2004.12.001.

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