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Zeitschriftenartikel zum Thema "Biochemical markers":
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Ahmed, Mohamed H. „Biochemical Markers“. American Journal of Clinical Pathology 127, Nr. 1 (Januar 2007): 20–22. http://dx.doi.org/10.1309/jxwum661t8vt1etx.
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Strong, Michael J. „Biochemical markers: Summary“. Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders 3, sup1 (September 2002): S85—S90. http://dx.doi.org/10.1080/146608202320374408.
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Selroos, Olof B. N. „Biochemical Markers in Sarcoidosis“. CRC Critical Reviews in Clinical Laboratory Sciences 24, Nr. 3 (Januar 1986): 185–216. http://dx.doi.org/10.3109/10408368609110273.
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Đerić, Mirjana, Sunčica Kojić-Damjanov, Velibor Čabarkapa und Nevena Eremić. „Biochemical Markers of Atherosclerosis“. Journal of Medical Biochemistry 27, Nr. 2 (01.01.2008): 148–53. http://dx.doi.org/10.2478/v10011-008-0008-1.
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Biochemical Markers of AtherosclerosisThis paper is a brief review of some lipid parameters and serum markers of inflammation in a view of their predictive relevance for the atherosclerotic disease. A discourse on the importance of measuring different lipids and lipoproteins, concentration of LDL particles and apolipoprotein levels is still underway. Also, the recommendations for apolipoprotein (a), phenotypization and other lipid markers have not yet been established. In recent years the recommendations imply simultaneous measuring of multiple markers and calculating the lipid index values such as lipid tetrad index (LTI), lipid pentad index (LPI) and atherogenic index of plasma (AIP). Several circulating markers of inflammation such as C-reactive protein, serum fibrinogen and elevated leukocyte number, are consistently associated with atherosclerosis. In spite of a lack of evidence on measuring the C-reactive protein in a wide population, the guidelines for its application in diagnostics and therapy of coronary heart disease were developed. Some proinflammatory cytokines, adhesion molecules and markers of leukocyte activation are promising markers, requiring, however, more detailed prospective evaluation. The question to be elucidated is if these inflammatory markers are directly involved in the pathogenic process.
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Kanis, John. „Biochemical markers in osteoporosis“. Scandinavian Journal of Clinical and Laboratory Investigation 57 (1997): 6–11. http://dx.doi.org/10.3109/00365519709168303.
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Ratcliffe, Anthony, und Markus J. Seibel. „Biochemical markers of osteoarthritis“. Current Opinion in Rheumatology 2, Nr. 5 (Oktober 1990): 770–76. http://dx.doi.org/10.1097/00002281-199002050-00014.
Allen, Marcus Christopher. „Biochemical markers of pulmonary oxygen toxicity“. Thesis, University of Aberdeen, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.277226.
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This study has investigated potential biochemical markers of pulmonary oxygen toxicity (POT). Toxicity was investigated in male Sprague Dawley rats exposed to oxygen partial pressures ranging from 0.21-2.5 bars. It is known that prolonged exposure to 1 bar of oxygen damages pulmonary endothelial cells and so the biochemical functions of these cells have been studied. Control isolated perfused rat lungs were able to clear and/or metabolise a wide range of substances including 5-HT, PGE2, bradykinin, and angiotensin 1, all endothelial cell functions. As expected 5-HT clearance was compromised in isolated perfused rat lung obtained from rats exposed to 1 bar of oxygen, confirming endothelial cell damage. However the clearance of 5-HT by lungs obtained from rats exposed to 2.5 bars was normal, implying that the site of toxicity is different at these partial pressures. In addition enhancement of toxicity by vitamin E deficiency was not associated with endothelial cell damage at 2.5 bar. At the molecular level oxygen free radicals are thought to be the causative agents of POT. The radicals are reputed to damage lipids, but a process of peroxidation. One of the lipid fragment products of ω6 polyunsaturated fatty acids is n-pentane, a compound which is excreted on the breath. Monitoring of this compound during exposure to 0.21, 1.0, 2.5 bars of oxygen even in vitamin E deficient rats did not show a rise in pentane expiration in response to oxygen exposure. This implies that peroxidation of ω6 polyunsaturated fatty acids did not take place, although other lipids may have been peroxidised. In conclusion the site of POT may depend on the partial pressure of oxygen. Endothelial cell damage is probably absent during exposure to 2.5 bars of oxygen. In addition n-pentane monitoring, a reputed marker of POT, failed to reveal lipid perodixation during exposure to hyperoxia.
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Israngkun, na Ayudthaya Porn Paul. „Potential biochemical markers for infantile autism /“. The Ohio State University, 1986. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487322984315317.
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The project objective was to develop a reliable selection procedure to match contact lens materials with individual wearers by the identification of a biochemical marker for assessment of in-eye performance of contact lenses. There is a need for such a procedure as one of the main reasons for contact lens wearers ceasing wearing contact lenses is poor end of day comfort i.e. the lenses become intolerable to the wearer as the day progresses. The selection of an optimal material for individual wearers has the potential benefit to reduce drop Qut, hence increasing the overall contact lens population, and to improve contact lens comfort for established wearers. Using novel analytical methods and statistical techniques, we were able to investigate the interactions between the composition of the tear film and of the biofilm deposited on the contact lenses and contact lens performance. The investigations were limited to studying the lipid components of the tear film; the lipid layer, which plays a key role in preventing evaporation and stabilising the tear film, has been reported to be significantly thinner and of different mixing characteristics during contact lens wear. Different lipid families were found to influence symptomatology, in vivo tear film structure and stability as well as ocular integrity. Whereas the symptomatology was affected by both the tear film lipid composition and the nature of the lipid deposition, the structure of the tear film and its stability were mainly influenced by the tear film lipid composition. The ocular integrity also appeared to be influenced by the nature of the lipid deposition. Potential markers within the lipid species have been identified and could be applied as follows: When required in order to identify a problematic wearer or to match the contact lens material to the contact lens wearer, tear samples collected by the clinician could be dispatched to an analytical laboratory where lipid analysis could be carried out by HPLC. A colorimetric kit based on the lipid markers could also be developed and used by clinician directly in the practice; such a kit would involve tear sampling and classification according to the colour into "Problem", "Border line" and "Good" contact lens wearers groups. A test kit would also have wider scope for marketing in other areas such as general dry-eye pathology.
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Branca, Francesco. „Biochemical markers of skeletal growth in children“. Thesis, University of Aberdeen, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.282683.
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This work focused on the application to growth studies of the assay for pyridinium crosslinks in urine. The study showed the existence of a nyctohermeral rhythm of crosslink excretion in children, with a peak during the night or early in the morning, and pointed out between-day fluctuations of excretion (17% for Pyd and 19% for Dpd). The study also showed that urinary crosslinks were significantly related to anthropometric estimates of skeletal mass and that such estimates should be used to account for size differences between individuals. Cross-sectional observations on 272 children (3-18 years) showed a parallelism between the height velocity curve and the age profile of crosslink excretion. In 3-5 year old children, the study pointed out a positive relationship with monthly height velocity (R = 0.20); 5-month integrated values of excretion were also correlated to 5-month height velocity (R = 0.78 for Pyd; R = 0.73 for Dpd). Urinary crosslinks were normal in 53 children (124±34 months) affected by miscellaneous conditions leading to growth defects, in 14 children (117±47 months) affected by coeliac disease, nor in 20 children affected by GH deficiency or short stature (aged 117±20 months); they were in the high range in 7 girls with Turner's syndrome, in 58 malnourished boys (14±14 months), urinary crosslinks were proportional to the degree of wasting and were positively correlated to the rate of height gain during hospitalisation. In children treated with Growth Hormone, except those affected by Turner's syndrome, crosslinks could predict 6-month growth after the first month of therapy.
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Åkesson, Kristina. „Fracture and biochemical markers of bone metabolism“. Lund : University of Lund, Dept. of Orthopaedics, Malmö General Hospital, Sweden, 1995. http://books.google.com/books?id=Ib9qAAAAMAAJ.
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McCoy, Paula K. „Psychological Hardiness and Biochemical Markers of Acute Stress“. Thesis, University of North Texas, 2001. https://digital.library.unt.edu/ark:/67531/metadc2884/.
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The establishment of physiological norms for psychologically hardy vs. non-hardy individuals was attempted by examination of levels of salivary cortisol and urinary norepinephrine before and after a mid-term examination stressor. Normative data was collected on the reported frequency of stressors and their severity one week prior to the examination, and self-reported ratings of stress immediately prior to the examination. Performance on the examination as a function of hardiness was explored. Associations between demographic variables and psychological hardiness were also studied. Results from this study were inconclusive in establishing physiological norms for psychologically hardy individuals. Associations were found between: 1) hardiness and frequency of stressors; 2) hardiness and age; and 3) self-reported ratings of stress and anxiety as measured by the State-Trait Anxiety Inventory (STAI).
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Morgan, Tanya G. „Biochemical and mobility markers in osteoarthritis of the knee“. Thesis, University of Sunderland, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269194.
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Glassbrook, Norman J. „Biochemical markers for the detection and classification of Aspergillus“. Thesis, Cardiff University, 2008. http://orca.cf.ac.uk/54802/.
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The genus Aspergillus includes a diverse group of filamentous fungi that are widely distributed in nature, commonly found in soil. The Aspergilli include species that can be beneficial or detrimental to humans, so detection and accurate identification of these organisms can be very important. Morphology and genetic sequence analysis are well established methods for classifying and identifying fungi, but morphology remains a widely used technique that generally works well for Aspergilli. However, some organisms may be misidentified due to atypical morphology and some hidden (cryptic) species may not be recognized as different from named species based on readily observable traits. In this study, reference strains of different Aspergillus species, Penicillium chrysogenum, Candida albicans, and Cryptococcus neoformans were characterized using LC/MS and GC/MS biochemical profiling techniques in order to find specific small molecules, peptides or biochemical profiles that can be used in addition to established methods to detect and classify Aspergilli to the species level. Subsequently, analytical methods developed for characterizing the reference strains were applied, along with morphology and PCR, to characterize and identify several laboratory and field isolates. Some unique compounds and biochemical patterns did emerge from small molecule profiling that could be used for classifying Aspergilli, but protein profiling by LC/MS/MS was a much more effective approach. Tandem mass spectra from LC/MS/MS of tryptic peptides from fungal proteins were searched against protein databases and matched to theoretical spectra derived from those databases. Many of the amino acid sequences detected were taxonomically diagnostic for classifying Aspergillus species. Protein profiling also provided a great deal of additional biochemical information on the test organisms by identifying the predominant enzymes and structural proteins present under different experimental conditions and may find broader application for identifying and studying other organisms.
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Stangland, Jenna Emily. „Biochemical Markers of Iron Status in Recreational Female Runners“. The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1374168831.
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Plata-Muñoz, Juan José. „Clinical, biochemical and molecular markers of injury before transplantation“. Thesis, University of Oxford, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572681.
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The use of organs from donors after circulatory death (DCD) has been recommended as one strategy to enlarge the donor pool and raise the transplant rate. However, DCD allografts had higher incidence of early post-transplant dysfunction. The general aim of this research project was to develop clinical and experimental strategies to reduce the incidence of early post-transplant dysfunction of kidney and liver allografts from DCD. First the ability of a clinical scoring system based on donor data for identifying DCD kidneys with high-risk of post-transplant dysfunction was evaluated using the Oxford and the UK National DCD kidney transplant cohorts. This works suggest that stratification of DCD kidneys before transplantation might allow early identification of kidneys in which lower graft function and survival could be expected if any additional therapeutic intervention is implemented. Second, as it has been suggested that hypothermic machine perfusion (HMP) may protect DCD kidneys from additional preservation injury and improve their outcome after transplantation, this work explored the benefit of HMP as preservation technique fo DCD kidneys in Oxford and discusses the potential of this technique for reducing the incidence of post-transplant dysfunction in DCD kidneys. The Oxford. Liver Group has provided evidence of the benefit of preservation with normothermic machine perfusion (NMP) on post-transplant function and survival of DCD liver allografts. In this work, the molecular mechanisms associated with this benefit were characterized using micro array technology. This analysis suggests that the beneficial effect ofNMP may be associated with the induction of the ischaemic preconditioning phenomenon and highlights a group of genes with potential for gene therapy. Finally, this works provides the "proof-of-concept" that the use of a non-mammalian viral vector for gene transfer of kidneys and livers during conventional cold preservation is feasible and is not associated with additional tissue injury.
R, Eastell, und Bone Markers: Biochemical and Clinical Perspectives Workshop (2000 : Geneva, Switzerland), Hrsg. Bone markers: Biochemical and clinical perspectives. London: Martin Dunitz, 2001.
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Practices, LLC Best. Incorporating biomarker research for R&D success: Access and intelligence for achieving world-class excellence. Chapel Hill, NC: Best Practices, LLC, 2004.
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Kim, Sang-gyŏm. Saenghwahakchŏk pʻyojija mit taeri kyŏlgwa pyŏnsu ŭi PK/PD modelling chŏgyong e kwanhan yŏnʼgu =: The study for evaluation of PK/PD modeling using biomarker and surrogate endpoint. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.
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1948-, Eganhouse Robert P., American Chemical Society. Division of Environmental Chemistry., American Chemical Society. Division of Geochemistry. und American Chemical Society Meeting, Hrsg. Molecular markers in environmental geochemistry. Washington, DC: American Chemical Society, 1997.
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Baak, Marleen A., Bernard Gutin, Kim A. Krawczewski Carhuatanta, Stephen C. Woods, Heinz W. Harbach, Megan M. Wenner, Nina S. Stachenfeld et al. „Overtraining-Biochemical Markers“. In Encyclopedia of Exercise Medicine in Health and Disease, 676–79. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_156.
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Jensen, J. E. B., H. A. Sørensen und O. H. Sørensen. „Biochemical Markers and Bone“. In Management of Fractures in Severely Osteoporotic Bone, 72–84. London: Springer London, 2000. http://dx.doi.org/10.1007/978-1-4471-3825-9_6.
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Wu, Alan H. B., und Robert G. McCord. „New Biochemical Markers for Heart Diseases“. In Cardiac Markers, 281–94. Totowa, NJ: Humana Press, 1998. http://dx.doi.org/10.1007/978-1-4612-1806-7_17.
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Hampson, Geeta. „Biochemical Markers in Bone Diseases“. In Radionuclide and Hybrid Bone Imaging, 109–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02400-9_5.
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Chu, T. M. „Biochemical Markers for Human Cancer“. In Current Topics in Pathology, 19–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71356-9_2.
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Blackwell, Penny, Ian Godber und Nigel Lawson. „Biochemical Markers of Bone Turnover“. In Clinical Trials in Osteoporosis, 221–40. London: Springer London, 2002. http://dx.doi.org/10.1007/978-1-4471-3710-8_13.
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Ramlawi, Basel, und Frank W. Sellke. „Biochemical Markers of Brain Injury“. In Brain Protection in Cardiac Surgery, 45–55. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84996-293-3_6.
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Greenblatt, Matthew B., Joy N. Tsai und Marc N. Wein. „Biochemical Markers of Bone Turnover“. In Osteoporosis, 169–84. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-69287-6_9.
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Rosen, Clifford J. „Biochemical Markers of Bone Turnover“. In Osteoporosis, 129–41. Totowa, NJ: Humana Press, 1996. http://dx.doi.org/10.1007/978-1-4612-0221-9_11.
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Konferenzberichte zum Thema "Biochemical markers":
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Greifová, H., E. Tvrdá, P. Červeňanská, E. Tušimová, A. Kováčik, K. Zbyňovská und N. Lukáč. „BIOCHEMICAL MARKERS OF INFLAMMATION IN DAIRY CATTLE“. In XVIII INTERNATIONAL SCIENTIFIC CONFERENCE RISK FACTORS OF FOOD CHAIN 2017. Uniwersytet Pedagogiczny w Krakowie, 2017. http://dx.doi.org/10.24917/9788380840973.6.
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Maciel Campos Da Silva, Ana Sofia, Rita Enriquez, Joana Duarte, Andreia Daniel, Inês Claro, Margarida Barata, Maria Alvarenga Santos, Teresa Martín, Nídia Caires und Filipa Todo Bom. „Biochemical markers as predictors of outcomes in COPD exacerbations“. In ERS International Congress 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/13993003.congress-2023.pa3638.
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Zolotavina, M. L., und E. A. Gaidabura. „CORRELATION ANALYSIS OF THE MAIN BIOCHEMICAL MARKERS OF IN-FLAMMATION IN COVID-19“. In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2022. http://dx.doi.org/10.47501/978-5-6044060-2-1.137-142.
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The article is devoted to determining the correlation between the biochemical parameters of the blood serum of patients with COVID-19. Concentrations of the main biochemical markers of inflammation at different stages of lung tissue damage by a new coronavirus infection with further correlation analysis of blood parameters were determined. The experiment of the study showed that there is a high correlation between the biochemical parameters of blood. Its sig-nificance helps to determine the sequence of the manifestation of biochemical markers of in-flammation and proves the mechanism of changes in the biochemical picture of blood in COVID-19, which we assume.
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Gomez Punter, Rosa Mar, Rosa Maria Giron, Emma Vazquez, Gilda Fernandes, Olga Rajas, Carolina Cisneros, Claudia Valenzuela und Julio Ancochea. „Biochemical Markers Of Bone Turnover In Adults With Cystic Fibrosis“. In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2810.
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Du, Shuyan, Xiangling Mao, Dikoma Shungu und Paul Sajda. „Blind recovery of biochemical markers of brain cancer in MRSI“. In Medical Imaging 2004, herausgegeben von J. Michael Fitzpatrick und Milan Sonka. SPIE, 2004. http://dx.doi.org/10.1117/12.534652.
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„Catalytically active bispecific antibodies – new biochemical markers of HIV/AIDS“. In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-258.
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Singh, S. P., und C. Murali Krishna. „Raman spectroscopy of oral tissues: correlation of spectral and biochemical markers“. In SPIE BiOS, herausgegeben von Bernard Choi, Nikiforos Kollias, Haishan Zeng, Hyun Wook Kang, Brian J. F. Wong, Justus F. Ilgner, Guillermo J. Tearney et al. SPIE, 2014. http://dx.doi.org/10.1117/12.2052797.
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Zemlyanukhina, O. A., E. N. Vasilchenko, N. N. Cherkasova und N. A. Karpechenko. „Physiological and biochemical markers in the study of sugar beet plants“. In CURRENT STATE, PROBLEMS AND PROSPECTS OF THE DEVELOPMENT OF AGRARIAN SCIENCE. Federal State Budget Scientific Institution “Research Institute of Agriculture of Crimea”, 2019. http://dx.doi.org/10.33952/09.09.2019.108.
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Varma, Vishal K., Samuel Ohlander, Peter Nguyen, Christopher Vendryes, Sujeeth Parthiban, Blake Hamilton, M. Chad Wallis et al. „Fourier transform infrared spectroscopic imaging identifies early biochemical markers of tissue damage“. In SPIE BiOS, herausgegeben von Anita Mahadevan-Jansen und Wolfgang Petrich. SPIE, 2014. http://dx.doi.org/10.1117/12.2040282.
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Abdulilah ABDULMAWJOOD, Sana, Eman Salem MAHMOUD und Mohammed I. MAJEED. „INFLAMMATORY MARKERS AND SOME BIOCHEMICAL PARAMETERS IN FEMALE RATS TREATED WITH QUERCETIN“. In VI.International Scientific Congress of Pure,Applied and Technological Sciences. Rimar Academy, 2022. http://dx.doi.org/10.47832/minarcongress6-29.
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The objective of this study was to determine whether quercetin, a polyphenol, could protect rats from nitrite's harmful effects. Methodologies: During the course of this investigation, twenty-one albino female rats have been used. The animals were placed in one of the three groups, which each had a total of seven rats. The groups were selected at random. Group, I received water throughout the duration of the experiment and was regarded as the healthy control group. The animals in Group II were given a solution containing 50 milligrams of sodium nitrite per kilogram of body weight via a gavage needle For the whole period of the study while those in Group III were given a solution containing both 50 milligrams of sodium nitrite per kilogram of body weight and 100 milligrams of quercetin per kilogram of rat weight. Blood samples were analyzed inflammatory markers including interleukin-1, interleukin-6, and tumor necrosis factor-alpha, as well as biochemical markers including liver enzymes , urea, and serum creatinine.
Berichte der Organisationen zum Thema "Biochemical markers":
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Nicholson, Ralph, Reuven Reuveni und Moshe Shimoni. Biochemical Markers for Disease Resistance in Corn. United States Department of Agriculture, Mai 1996. http://dx.doi.org/10.32747/1996.7613037.bard.
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The objective was to screen maize lines for their ability to express resistance based on biochemical traits. Cultivars were screened for retention of the hydroxamic acid DIMBOA and the synthesis of phenols (based on anthocyanin production) as markers for resistance. Lines were selected and inoculated with fungal pathogens (Exserohilum turcicum, Puccinia sorghi, Cochliobolus heterostraphus, Colletotricum graminicola.), and the Maize Dwarf Mosaic and Johnson Grass Mosaic viruses. Lines were screened in the field and greenhouse. Results showed that lines selected for augmented phenol synthesis do exhibit heightened levels of resistance to fungal pathogens. Isolation of mRNA followed by northern analyses for expression of A1 (dihydroflavanol reductase) and peroxidase confirmed that genes for these enzymes were turned on in response to inoculation of lines predicted to exhibit resistance. Peroxidase and b-1,3-glucanase were assayed in breeding lines having or lacking the se gene. A specific ionically-bound peroxidase isozyme and a b-1,3-glucanase isozyme were revealed in lines having the se gene. Data suggest that peroxidase and b-1,3-glucanase isozymes, may be considered as markers to identify resistance to E. turcicum in maize genotypes with the se gene.
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Hayes, Ronald L. Biochemical Markers of Brain Injury: An Integrated Proteomics-Based Approach. Fort Belvoir, VA: Defense Technical Information Center, Februar 2005. http://dx.doi.org/10.21236/ada437666.
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Lockridge, Oksana. Biochemical Markers for Exposure to Low Doses of Organophosphorus Insecticides. Fort Belvoir, VA: Defense Technical Information Center, August 2002. http://dx.doi.org/10.21236/ada408118.
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Lockridge, Oksana. Biochemical Markers for Exposure to Low Doses of Organophosphorus Insecticides. Fort Belvoir, VA: Defense Technical Information Center, August 2003. http://dx.doi.org/10.21236/ada419631.
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Hayes, Ronald L. Biochemical Markers of Brain Injury: An Integrated Proteomics Based Approach. Fort Belvoir, VA: Defense Technical Information Center, Februar 2004. http://dx.doi.org/10.21236/ada425658.
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Akhverdyan, Y., L. Seewordova, Y. Polyakova, B. Zavodovsky, E. Papichev und V. Pavlovskaya. BIOCHEMICAL MARKERS OF BONE REMODELING IN PATIENTS WITH RHEUMATOID ARTHRITIS. "PLANET", 2019. http://dx.doi.org/10.18411/978-5-907192-54-6-2019-xxxvi-19-23.
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Hayes, Ronald L. Biochemical Markers of Brain Injury: An Integrated Proteomics-Based Approach. Fort Belvoir, VA: Defense Technical Information Center, Dezember 2011. http://dx.doi.org/10.21236/ada561092.
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Hayes, Ronald L. Biochemical Markers of Brain Injury: An Integrated Proteomics-Based Approach. Fort Belvoir, VA: Defense Technical Information Center, Februar 2007. http://dx.doi.org/10.21236/ada474912.
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Wiles, Connor. Frozen, Old, or New? Comparing Biochemical Markers and Tissue Oxygenation in Transfused Blood. Portland State University Library, Januar 2014. http://dx.doi.org/10.15760/honors.34.
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Wegiel, Jetzy. Characterization of the Pathological and Biochemical Markers that Correlate to the Clinical Features of Autism. Fort Belvoir, VA: Defense Technical Information Center, Oktober 2009. http://dx.doi.org/10.21236/ada512506.
