Auswahl der wissenschaftlichen Literatur zum Thema „Auxiliary channels“
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Zeitschriftenartikel zum Thema "Auxiliary channels"
Dvorak, Nolan M., Paul A. Wadsworth, Pingyuan Wang, Jia Zhou und Fernanda Laezza. „Development of Allosteric Modulators of Voltage-Gated Na+ Channels: A Novel Approach for an Old Target“. Current Topics in Medicinal Chemistry 21, Nr. 10 (17.06.2021): 841–48. http://dx.doi.org/10.2174/1568026621666210525105359.
Der volle Inhalt der QuelleFlockerzi, Veit, und Bernd Fakler. „TR(i)P Goes On: Auxiliary TRP Channel Subunits?“ Circulation Research 134, Nr. 4 (16.02.2024): 346–50. http://dx.doi.org/10.1161/circresaha.123.323178.
Der volle Inhalt der QuelleHoshi, Toshinori, Rong Xu, Shangwei Hou, Stefan H. Heinemann und Yutao Tian. „A point mutation in the human Slo1 channel that impairs its sensitivity to omega-3 docosahexaenoic acid“. Journal of General Physiology 142, Nr. 5 (14.10.2013): 507–22. http://dx.doi.org/10.1085/jgp.201311061.
Der volle Inhalt der QuelleZhou, Zijing, Xiaonuo Ma, Yiechang Lin, Delfine Cheng, Navid Bavi, Genevieve A. Secker, Jinyuan Vero Li et al. „MyoD-family inhibitor proteins act as auxiliary subunits of Piezo channels“. Science 381, Nr. 6659 (18.08.2023): 799–804. http://dx.doi.org/10.1126/science.adh8190.
Der volle Inhalt der QuelleJones, Lisa P., Shao-kui Wei und David T. Yue. „Mechanism of Auxiliary Subunit Modulation of Neuronal α1E Calcium Channels“. Journal of General Physiology 112, Nr. 2 (01.08.1998): 125–43. http://dx.doi.org/10.1085/jgp.112.2.125.
Der volle Inhalt der QuelleJiao, Yunjing, Qijing Lin, Kun Yao, Na Zhao, Dan Xian, Fuzheng Zhang, Qingzhi Meng, Bian Tian und Zhuangde Jiang. „Design of High-Precision Parallel AWG Demodulation System“. Micromachines 14, Nr. 9 (25.08.2023): 1662. http://dx.doi.org/10.3390/mi14091662.
Der volle Inhalt der QuelleDvorak, Nolan M., Cynthia M. Tapia, Aditya K. Singh, Timothy J. Baumgartner, Pingyuan Wang, Haiying Chen, Paul A. Wadsworth, Jia Zhou und Fernanda Laezza. „Pharmacologically Targeting the Fibroblast Growth Factor 14 Interaction Site on the Voltage-Gated Na+ Channel 1.6 Enables Isoform-Selective Modulation“. International Journal of Molecular Sciences 22, Nr. 24 (17.12.2021): 13541. http://dx.doi.org/10.3390/ijms222413541.
Der volle Inhalt der QuelleSinha, Ashish, Haodong Gu, Namwoon Kim und Renu Emile. „Signaling effects and the role of culture: movies in international auxiliary channels“. European Journal of Marketing 53, Nr. 10 (07.10.2019): 2146–72. http://dx.doi.org/10.1108/ejm-09-2017-0587.
Der volle Inhalt der QuelleBrown, Austin L., Zhiwen Liao und Miriam B. Goodman. „MEC-2 and MEC-6 in the Caenorhabditis elegans Sensory Mechanotransduction Complex: Auxiliary Subunits that Enable Channel Activity“. Journal of General Physiology 131, Nr. 6 (26.05.2008): 605–16. http://dx.doi.org/10.1085/jgp.200709910.
Der volle Inhalt der QuelleWilliams, Brittany, Josue A. Lopez, J. Wesley Maddox und Amy Lee. „Functional impact of a congenital stationary night blindness type 2 mutation depends on subunit composition of Cav1.4 Ca2+ channels“. Journal of Biological Chemistry 295, Nr. 50 (08.10.2020): 17215–26. http://dx.doi.org/10.1074/jbc.ra120.014138.
Der volle Inhalt der QuelleDissertationen zum Thema "Auxiliary channels"
Yasuda, Takahiro. „Modulation of calcium channel function and toxin sensitivity by auxiliary subunits /“. [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18052.pdf.
Der volle Inhalt der QuelleMolinarolo, Steven. „Biochemical techniques for the study of voltage-gated sodium channel auxiliary subunits“. Diss., University of Iowa, 2018. https://ir.uiowa.edu/etd/6217.
Der volle Inhalt der QuelleRobinson, Philip. „Targeting of voltage-gated calcium channels to lipid rafts : the role of auxiliary alpha2/delta-1 subunits“. Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/targeting-of-voltagegated-calcium-channels-to-lipid-rafts-the-role-of-auxiliary-alpha2delta1-subunits(db84049c-1445-4486-9e31-34fa4110daf6).html.
Der volle Inhalt der QuelleZhao, Juan. „Biophysical characterization of neuronal and skeletal muscle sodium channels, and their regulation by auxiliary beta subunits“. Thesis, Université Laval, 2012. http://www.theses.ulaval.ca/2012/28793/28793.pdf.
Der volle Inhalt der QuelleVoltage-gated Na channels are responsible for the rising phase of action potentials, and consist of a pore-forming α subunit and one or more auxiliary β subunits. The α subunit alone is sufficient for the functional expression of Na channels, however, β subunits modulate the location, expression and functional properties of α subunits. My thesis will focus on three neuronal Na channels (Nav1.6, Nav1.7 and Nav1.8) and one skeletal muscle Na channel (Nav1.4). Neuronal Na channel are key players in the impulse propagation along axon. Nav1.7 and Nav1.8 are the main Na channels expressed in DRG neurons, and their altered expression and modulation following injury and inflammation play a major role in nociception and chronic pain. Nav1.6 is highly concentrated at nodes of Ranvier, and has a critical role not only in saltatory conduction but also in high-frequency repetitive firing. Skeletal muscle Na channel Nav1.4 is the initiator of muscle contraction. Mutations in Nav1.4 cause skeletal muscle channelopathies. Guiding questions for our investigations were: 1) How do auxiliary β subunits regulate peripheral nerve Na channel Nav1.6 and Nav1.8? 2) What is the underlying biophysical defect of M1476I, a novel founder SCN4A mutation associated with painful cold-induced myotonia in French Canadians? 3) What is the biophysical characterization of the Nav1.6 persistent current? 4) What is the expression pattern of auxiliary subunits, and how do β subunits regulate Nav1.7 in DRG neurons? We addressed these questions by multiple approaches including patch clamp techniques for whole-cell and single-channel recordings in heterologous expression systems; immunohistochemistry, single-cell RT-PCR and immunoprecipitation in DRG neurons. Firstly, we employed single-cell RT-PCR of acutely dissociated DRG neurons to identify the expression of β1-4 subunits in small-diameter sensory neurons. Our results indicated that small-diameter DRG neurons widely expressed Nav1.6 and Nav1.8 channels and β1-β3 subunits. Co-expression studies were used to assess the regulation of Nav1.6 and Nav1.8 by β subunits. The β1 subunit induced a significant increase in the current density of Nav1.8 when co-expressed in HEK293 cells, but had no effect on that of Nav1.6. In addition, the C-terminal domain of β1 was involved in the modulation of Nav1.8 channel based on the results of experiments with β1/β2 chimeras harboring various regions of the strongly regulating β1 together with the weakly regulating β2 subunit. Secondly, we investigated the biophysical defects of M1476I mutation in Nav1.4 channels using whole-cell patch-clamp technique in tsA201 cells. M1476I mutant channel exhibited similar biophysical defects compared with other PAM-causing mutations, including an increased persistent current of Nav1.4, a slower current decay, a positive shift of fast inactivation, and an accelerated recovery from fast inactivation. Lowering the temperature slowed the kinetics for both wide-type and mutant channels, and worsened the defective fast inactivation of M1476I channels by further increasing the amplitude of the persistent current. Mexiletine helps relieve myotonia in M1476I carriers by effectively suppressing the increased persistent current, except for the use-dependent block. However, mexiletine had a reduced effectiveness on the use-dependent block of M1476I channels, and that was associated with a faster recovery from mexiletine block of mutant channels. Thirdly, we characterized the whole-cell and single-channel properties of Nav1.6 persistent currents expressed in HEK293 cells. We noted that Nav1.6 persistent current was highly sensitive to the composition of the internal solution, and persistent current was rarely detectable when CsF instead of CsCl was used. By substituting CsF for CsCl in the intracellular solution, we showed that Nav1.6 persistent current in the whole-cell configuration was 3–5% of the peak transient current. This amplitude of persistent current was similar to the ratio between peak and persistent open probability observed in the single-channel recording, indicating that the occurrence of late channel reopenings accounts for the persistent macroscopic Na current typical of Nav1.6. Finally, we employed a combination of single-cell RT-PCR, immunocytochemistry and immunoprecipitation to investigate subunit expression in subpopulations of sensory neurons. subunits were differentially expressed in small (2, 3) and large (1, 2) DRG neurons. Nav1.7 mRNA was significantly co-expressed with the 2 and 3 subunits in the same population of small-diameter DRG neurons. They formed stable protein-protein interactions and co-localized within the plasma membranes of neurons.When co-expressed in HEK293 cells, 3 and 1 subunits shifted activation and inactivation curves respectively and induced a marked increase in Nav1.7 window current. Our data indicated a preferential expression of subunits in small and large DRG neurons and a subunit-specific Nav1.7 regulation in these subpopulations of sensory neurons.
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Saponaro, A. C. „THE AUXILIARY SUBUNIT TRIP8B ANTAGONIZES THE BINDING OF CAMP TO HCN2 CHANNELS THROUGH AN ALLOSTERIC MECHANISM“. Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/229903.
Der volle Inhalt der QuelleZhou, Wei. „Structural and functional studies on the regulation of K+ channels by local anesthetics and intracellular auxiliary subunits /“. Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2004. http://wwwlib.umi.com/cr/ucsd/fullcit?p3142460.
Der volle Inhalt der QuelleSoubrane, Camille Hélène. „Identification of CACHD1 as a novel [alpha]2[delta]-like auxiliary subunit of Cav3 voltage-gated calcium channels“. Thesis, University of Reading, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658003.
Der volle Inhalt der QuelleTerhag, Jan [Verfasser], Michael [Gutachter] Hollmann und Guiscard Friedrich Aldous [Gutachter] Seebohm. „Auxiliary subunits of voltage-gated Calcium channels as modulators of ionotropic glutamate receptors / Jan Terhag ; Gutachter: Michael Hollmann, Guiscard Friedrich Aldous Seebohm“. Bochum : Ruhr-Universität Bochum, 2016. http://d-nb.info/111670952X/34.
Der volle Inhalt der QuelleDespang, Patrick Sebastian [Verfasser], Dirk [Akademischer Betreuer] Isbrandt und Markus [Akademischer Betreuer] Plomann. „Autism-related mutations of auxiliary Cavβ-subunits : electrophysiological characterization of effects on the function of voltage-gated calcium channels / Patrick Sebastian Despang ; Akademische Betreuer: Dirk Isbrandt, Markus Plomann“. Köln : Deutsche Zentralbibliothek für Medizin, 2020. http://d-nb.info/1215226047/34.
Der volle Inhalt der QuelleStephani, Friederike Lene Brigitte [Verfasser]. „α2δ3 is the preferred auxiliary α2δ subunit of Cav2.1 channels in spiral ganglion neurons and is required for development of auditory nerve fiber synapses / Friederike Lene Brigitte Stephani“. Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2020. http://d-nb.info/1216877823/34.
Der volle Inhalt der QuelleBuchteile zum Thema "Auxiliary channels"
Borowik, Sergej, und Henry M. Colecraft. „Voltage-Gated Calcium Channel Auxiliary β Subunits“. In Voltage-Gated Calcium Channels, 73–92. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-08881-0_4.
Der volle Inhalt der QuelleObermair, Gerald J., und Bernhard E. Flucher. „Neuronal Functions of Auxiliary Calcium Channel Subunits“. In Modulation of Presynaptic Calcium Channels, 29–59. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6334-0_2.
Der volle Inhalt der QuelleMatthes, Jan, und Stefan Herzig. „Auxiliary β-Subunits of L-Type Ca2+ Channels in Heart Failure“. In Pathologies of Calcium Channels, 255–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-40282-1_14.
Der volle Inhalt der QuelleDolphin, Annette C., und Gerald J. Obermair. „Regulation of Calcium Channels and Synaptic Function by Auxiliary α2δ Subunits“. In Voltage-Gated Calcium Channels, 93–114. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-08881-0_5.
Der volle Inhalt der QuelleMolinarolo, Steven, Daniele Granata, Vincenzo Carnevale und Christopher A. Ahern. „Mining Protein Evolution for Insights into Mechanisms of Voltage-Dependent Sodium Channel Auxiliary Subunits“. In Voltage-gated Sodium Channels: Structure, Function and Channelopathies, 33–49. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/164_2017_75.
Der volle Inhalt der QuelleWeik, Martin H. „auxiliary channel“. In Computer Science and Communications Dictionary, 90. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/1-4020-0613-6_1173.
Der volle Inhalt der QuelleRamachandran, Nitya, und P. Yogesh. „Modified Auxiliary Channel Diffie Hellman Encrypted Key Exchange Authentication Protocol“. In Information Technology and Mobile Communication, 417–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-20573-6_74.
Der volle Inhalt der QuellePietrobon, D. „Introduction“. In Guidebook to Protein Toxins and Their Use in Cell Biology, 167–69. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780198599555.003.0058.
Der volle Inhalt der QuelleLevitan, Irwin B., und Leonard K. Kaczmarek. „Diversity in the Structure and Function of Ion Channels“. In The Neuron, 127–50. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199773893.003.0007.
Der volle Inhalt der QuelleLeidenheimer, N. J., J. E. Dildy-Mayfield und R. A. Harris. „Effect of Ethanol on Voltage-Gated Ion Channels“. In The Pharmacology of Alcohol and Alcohol Dependence, 335–55. Oxford University PressNew York, NY, 1996. http://dx.doi.org/10.1093/oso/9780195100945.003.0010.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Auxiliary channels"
Fertonani, D., und T. M. Duman. „Upper Bounding the Deletion Channel Capacity by Auxiliary Memoryless Channels“. In ICC 2009 - 2009 IEEE International Conference on Communications. IEEE, 2009. http://dx.doi.org/10.1109/icc.2009.5199553.
Der volle Inhalt der QuelleWu, Yiting, Chuansheng Yang, Chao Wang, Hongming Chen und Qihong Ye. „Low-light image enhancement combining dehazing and auxiliary channels“. In 3rd International Conference on Advanced Algorithms and Signal Image Processing (AASIP 2023), herausgegeben von Kannimuthu Subramaniam und Pavel Loskot. SPIE, 2023. http://dx.doi.org/10.1117/12.3005867.
Der volle Inhalt der QuelleWang, Shengde, Zhenqiang Yao, Hong Shen und Guohu Luo. „Numerical Analysis on the Hydraulic Performance of the Auxiliary Impeller in Large Capacity Canned-Motor Pump“. In ASME 2018 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/imece2018-87285.
Der volle Inhalt der QuelleLi, Wenming, Fanghao Yang, Tamanna Alam, Benli Peng, Xiaopeng Qu und Chen Li. „Enhanced Flow Boiling in Microchannels Using Auxiliary Channels and Multiple Micronozzles“. In ASME 2016 5th International Conference on Micro/Nanoscale Heat and Mass Transfer. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/mnhmt2016-6712.
Der volle Inhalt der QuellePhung, Cao Vien, Anna Engelmann, Thomas Kuerner und Admela Jukan. „Improving THz Quality-of-Transmission with Systematic RLNC and Auxiliary Channels“. In 2020 IEEE International Conference on Communications Workshops (ICC Workshops). IEEE, 2020. http://dx.doi.org/10.1109/iccworkshops49005.2020.9145148.
Der volle Inhalt der QuelleHalevi, Tzipora, und Nitesh Saxena. „On pairing constrained wireless devices based on secrecy of auxiliary channels“. In the 17th ACM conference. New York, New York, USA: ACM Press, 2010. http://dx.doi.org/10.1145/1866307.1866319.
Der volle Inhalt der QuelleShinmoto, Yasuhisa, Shinichi Miura, Koichi Suzuki, Yoshiyuki Abe und Haruhiko Ohta. „Development of Advanced High Heat Flux Cooling System for Power Electronics“. In ASME 2009 InterPACK Conference collocated with the ASME 2009 Summer Heat Transfer Conference and the ASME 2009 3rd International Conference on Energy Sustainability. ASMEDC, 2009. http://dx.doi.org/10.1115/interpack2009-89082.
Der volle Inhalt der QuellePetrov, Nikolay V., Azat O. Ismagilov, Egor N. Oparin, Vladimir S. Shumigai, Anastasiia K. Lappo-Danilevskaia, Boris A. Nasedkin und Anton N. Tsypkin. „Ghost imaging with auxiliary multiplex channels: a review of the latest results“. In Speckle 2023: VIII International Conference on Speckle Metrology, herausgegeben von Yechuan Zhu, Wei Wang und Weiguo Liu. SPIE, 2024. http://dx.doi.org/10.1117/12.3021804.
Der volle Inhalt der QuelleHan, Yafei, und Guolong Liang. „Blind Multi-User Detection Based on Auxiliary Particle Filter in Fading CDMA Channels“. In 2009 International Conference on E-Business and Information System Security (EBISS). IEEE, 2009. http://dx.doi.org/10.1109/ebiss.2009.5137947.
Der volle Inhalt der QuelleMiura, Shinichi, Yukihiro Inada, Yasuhisa Shinmoto und Haruhiko Ohta. „Development of Cooling System for a Large Area at High Heat Flux by Using Flow Boiling in Narrow Channels“. In ASME 2009 7th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2009. http://dx.doi.org/10.1115/icnmm2009-82279.
Der volle Inhalt der QuelleBerichte der Organisationen zum Thema "Auxiliary channels"
Little, Charles, und David Biedenharn. Technical assessment of the Old, Mississippi, Atchafalaya, and Red (OMAR) Rivers : channel geometry analysis. Engineer Research and Development Center (U.S.), August 2022. http://dx.doi.org/10.21079/11681/45147.
Der volle Inhalt der QuellePokryshen, Dmytro A., Evgeniy H. Prokofiev und Albert A. Azaryan. Blogger and YouTube services at a distant course “Database management system Microsoft Access”. [б. в.], September 2019. http://dx.doi.org/10.31812/123456789/3272.
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