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Auswahl der wissenschaftlichen Literatur zum Thema „Auditive fibers“
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Zeitschriftenartikel zum Thema "Auditive fibers"
Hamdan, Sinin, Khairul Anwar Mohamad Said, Ahmad Faudzi Musib, Md Rezaur Rahman, Marini Sawawi und Aaliyawani Ezzerin Sinin. „Pratuokng: The Borneo bamboo zither of Bidayuh Sarawak“. BioResources 19, Nr. 1 (08.01.2024): 1305–15. http://dx.doi.org/10.15376/biores.19.1.1305-1315.
Der volle Inhalt der QuelleWong, Kylie, Qumrul Ahsan, Azma Putra, Sivarao Subramonian und Mohd Jailani Mohd Nor. „PRELIMINARY STUDY ON THE SOUND ABSORPTION BEHAVIOR OF SPENT TEA LEAVES FILLED WITH NATURAL RUBBER LATEX BINDER“. Jurnal Teknologi 79, Nr. 5-2 (19.07.2017). http://dx.doi.org/10.11113/jt.v79.11284.
Der volle Inhalt der QuelleColla, Marina de Figueiredo, Pamela Papile Lunardelo und Fernanda Abalen Martins Dias. „Cochlear synaptopathy and hidden hearing loss: a scoping review“. CoDAS 36, Nr. 2 (2024). http://dx.doi.org/10.1590/2317-1782/20232023032en.
Der volle Inhalt der QuelleDissertationen zum Thema "Auditive fibers"
Gagliardini, Mathilde. „Les crises audiogènes réflexes : une voie d’entrée dans la compréhension des mécanismes de développement et de fonctionnement des voies rétro-cochléaires“. Electronic Thesis or Diss., Sorbonne université, 2021. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2021SORUS122.pdf.
Der volle Inhalt der QuelleThe genetic approach of inherited forms of deafness has been particularly effective for deciphering the molecular physiology of the cochlea. In contrast, this genetic dissection has so far provided little information about the central auditory system. The hypothesis of my laboratory is that part of the central hearing defects in genetic forms of deafness would be concealed due to cochlear damages, which deprive the auditory brain of all or part of the acoustic information it receives. The objective of my thesis was to characterize these masked central and peripheric deficits in a new mutant mouse model for Otogl, a genetic form of deafness. Mutations in Otogl cause peripheral deafness but are also associated to a susceptibility to audiogenic seizures, reflex seizures induced by a loud sound. In the cochlea, otogl is involved in the cohesion of the hair bundle, the mechanical antenna to the sound of sensory cells and in the tectorial membrane. My results showed that Otogl is expressed in the fibers of the primary auditory nerve and in additional structures of the cochlea, unveiling new roles for Otogl in the cochlea and the central auditory pathways. I also uncovered functional deficits in sound processing in heterozygous Otogl+/- mice that did not have peripheral hearing loss. Otogl therefore has a retrocochlear role in addition to its role in the cochlea. These results involve a particular category of auditory nerve fibers, the high-threshold fibers. Otogl mutant mice are a new model for studying high-threshold fibers and their role in the occurrence of audiogenic seizures in mice
Hasselmann, Florian. „Codage de l'enveloppe temporelle dans le nerf auditif“. Thesis, Montpellier, 2017. http://www.theses.fr/2017MONTT049/document.
Der volle Inhalt der QuelleBackground: Speech intelligibility in quiet is critically dependent on the temporal envelope of a sound signal. An abnormal coding of this temporal cue (due to infectious, immune, genetic, tumoral or environmental of origin) implies a decrease of speech recognition scores. The current proxy to probe deafness in clinical framework (Compound Action Potential of the auditory nerve, auditory brainstem responses) uses simplistic stimuli (clicks, tone bursts) to detect a such abnormal coding of the temporal cues. The aim of this study was to develop a new electrophysiology method in murins able to measure the auditory nerve responses to amplitude-modulation stimuli.Material and methods: The electrophysiology response of the auditory nerve was recorded using an electrode implanted onto the round window niche on normal-hearing gerbil cochlea and aging rat cochlea. The acoustical stimuli consisted of 20 seconds sinusoidally amplitude-modulated noise-band centered on 4, 8 and 16 kHz. We have studied varying sound level, the modulation depth, the modulation frequency and the carrier frequency.Results: Our study on the selective fiber loss ouabain model show the mass potentials recorded at the round window enable the detection of low spontaneous rate fibers in gerbil auditory nerve. This result is important because the current clinical used tests aren’t enough sensitive to detect a such coding impairment (CAP, ABR) (Batrel, Huet, Hasselmann et al., 2017). Then we combined the stimulus of this previous study with a sinusoidal function to develop a new method to assess the envelope coding by the auditory nerve. We validated this new method. Last, we used our method on an aging model (Sprague-Dawley rat). Our results suggest aging leads to a phenotype change of auditory nerve fibers without associated fiber loss (article Occelli, Hasselmann et al, submitted to eNeuro).Conclusion: Our study shows it’s indispensable to expand the number of tools to probe the cochlea because the current clinical used tests aren’t enough sensitive to detect subtle deficits of encoding in the auditory nerve. The recording of the fiber sustained activity enable to detect the selective loss of low-spontaneous rate neurons. A such loss is undetectable with classical clinical tools. The phenotype change of fibers we observed in aging Sprague-Dawley rats is detectable with our method whereas it’s not using the compound action potential of the auditory nerve
Ruel, Jérôme. „Inhibition tonique de l'activité des fibres du nerf auditif par la dopamine : approche pharmacologique de la première synapse auditive et sa modulation par les efférences latérales“. Montpellier 1, 2000. http://www.theses.fr/2000MON1T026.
Der volle Inhalt der QuelleVasconcelos, Carlos Augusto Carvalho de. „Aspectos descritivos e quantitativos da anatomia macroscópica e microscópica do nervo vestíbulo-coclear de cobaias“. Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/17/17140/tde-28092006-091016/.
Der volle Inhalt der QuelleThe vestibulocochlear (VIIIth cranial nerve) nerve of guinea pigs have special features not common for peripheral nerves in general. There are no detailed reports on the morphometric characteristics of the VIII nerve in adult guinea pigs. This is a descriptive and qualitative study that shows normal parameters that are useful in experimental models of the VIIIth nerve neuropathy. Eight adult guinea pigs were perfused with 2.5% glutaraldehyde in isotonic cacodilate buffer (0.025M). The right (N=4) and left (N=2) nerves were dissected in the median region, post-fixed in 1% osmuin tetroxide and embedded in epoxy resin. Semithin (0.5 µm) serial transverse sections were stained with 1% toluidine blue for light microscopy study. The descriptive histology and the topographical and histometric aspects of the median region of the nerve were analyzed, including myelinated fiber density, myelinated fiber diameter distribution, g ratio and number of endoneural capillary vessels. The cochlear nerve is enveloped by the vestibular nerve when they join together. Both, the cochlear and the vestibular nerves join to form the vestibulocochlear nerve with a Y shape, with approximately 5 mm in length. The nerve is widely vascularized and presents a complete mixture of the vestibular and cochlear myelinated fibers when the VIII nerve fascicle is constituted. The myelinated fibers are longitudinally oriented and present few endoneural connective tissue in between. No unmyelinated fibers were evidenced in the VIII nerve. The myelinated fiber diameter distribution was unimodal, with a peak at 3.5, and intervals between 1,5 and 6 m. The average number of fibers was 5.390 ± 1.504. The myelinated axon diameter distribution was also unimodal, with a peak at 2.5 µm of diameter were predominant quantitatively: 6.757 ± 1.922, and intervals between 1 and 4.5 µm. The average myelinated fiber density was 41.474 ± 4384 mm2, spread in a fascicular diameter of 0.77 ± 0.04 mm. The g ratio values varied from 0.2 to 0.9 and most of the myelinated fibers showed g ratio values of 0.7. Our results are original and only few parameters studied here were described in the literature. The vestibulocochlear nerve of the guinea pigs is a special afferent nerve, differing from other peripheral nerves (sensitive and motor) in its main anatomic and physiological characteristics, compared to other nerves from mammals. The adult guinea pig is an excellent model for the vestibulocochlear nerve neuropathy studies once it is similar to the human nerve.
Batrel, Charlène. „Nouvelle méthode d'exploration fonctionnelle du nerf auditif“. Thesis, Montpellier 1, 2014. http://www.theses.fr/2014MON13520/document.
Der volle Inhalt der QuelleBackground: The synchronous activation of the auditory nerve fibers (ANFs), is commonly studied through the compound action potential (CAP), or the auditory brainstem responses (ABR), to probe deafness in experimental and clinical settings. Recent studies have shown that substantial ANF loss can coexist with normal hearing threshold, and even unchanged CAP amplitude, making the detection of auditory neuropathies difficult. In this study, we took advantage of the round window neural noise (RWNN) to probe ANF loss in a physiologically-relevant model of neuropathy.Material and methods: ANF loss was induced by the application of ouabain onto the round window niche. CAP and RWNN of the gerbil's cochlea were recorded through an electrode placed onto the round window niche, 6 days after the ouabain application. Afferent synapse counts and single-unit recordings were carried-out to determine the degree and the nature of ANF loss, respectively. Results: Application of a low ouabain-dose into the gerbil RW niche elicits a specific degeneration of low spontaneous rate (SR) fibers, as shown by single-unit recordings. Simultaneous recordings (CAP/single-unit) demonstrate that low-SR fibers have a weak contribution to the CAP amplitude because of their delayed and broad first spike latency distribution. However, the RWNN amplitude decreases with the degree of synaptic loss. The RWNN method is therefore more sensitive than CAP to detect low-SR fiber loss, most probably because it reflects the sustained discharge rate of ANFs. Based on these data, we proposed a far-field method (Peri-stimulus time response-PSTR) to assess the fast, slow, and sustain vesicular release at the first auditory synapse.Conclusion: The round window neural noise is a faithful proxy to probe the degree and the SR-based nature of fiber loss. This method could be translated into the clinic to probe hidden hearing loss and orient the practitioner toward synaptopathy and/or neuropathy.Key words: Cochlea, auditory nerve, compound action potential, round window neural noise, single fiber recording, ouabain-induced neuropathy
Bücher zum Thema "Auditive fibers"
Assembly, Canada Legislature Legislative. Bill: An act to extend to this province letters patent, granted to William Edward Newton for certain improvements in the method of effecting the separation of the fibres of wood for the manufacture of paper therefrom, and for the separation of other substances for similar or other purposes. Quebec: Thompson, Hunter, 2003.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Auditive fibers"
Al-Azzawi, Abdul. „Internal, External, and ISO Auditing“. In Advanced Manufacturing for Optical Fibers and Integrated Photonic Devices, 209–18. CRC Press, 2017. http://dx.doi.org/10.1201/b18749-16.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Auditive fibers"
„PSICOSIS, CONSUMO Y PERSONAS MIGRANTES. A PROPÓSITO DE UN CASO“. In 23° Congreso de la Sociedad Española de Patología Dual (SEPD) 2021. SEPD, 2021. http://dx.doi.org/10.17579/sepd2021p139v.
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