Auswahl der wissenschaftlichen Literatur zum Thema „Allo-reactivity“
Geben Sie eine Quelle nach APA, MLA, Chicago, Harvard und anderen Zitierweisen an
Inhaltsverzeichnis
Machen Sie sich mit den Listen der aktuellen Artikel, Bücher, Dissertationen, Berichten und anderer wissenschaftlichen Quellen zum Thema "Allo-reactivity" bekannt.
Neben jedem Werk im Literaturverzeichnis ist die Option "Zur Bibliographie hinzufügen" verfügbar. Nutzen Sie sie, wird Ihre bibliographische Angabe des gewählten Werkes nach der nötigen Zitierweise (APA, MLA, Harvard, Chicago, Vancouver usw.) automatisch gestaltet.
Sie können auch den vollen Text der wissenschaftlichen Publikation im PDF-Format herunterladen und eine Online-Annotation der Arbeit lesen, wenn die relevanten Parameter in den Metadaten verfügbar sind.
Zeitschriftenartikel zum Thema "Allo-reactivity"
Huisman, Wesley, Didier A. T. Leboux, Lieve E. van der Maarel, Lois Hageman, Derk Amsen, J. H. Frederik Falkenburg und Inge Jedema. „The Scope of Allo-HLA Cross-Reactivity By (Third Party) Virus Specific T Cells Is Surprisingly Affected By HLA Restriction Rather Than Virus Specificity“. Blood 132, Supplement 1 (29.11.2018): 2048. http://dx.doi.org/10.1182/blood-2018-99-116028.
Der volle Inhalt der QuelleWebb, S. R., J. H. Li, I. MacNeil, P. Marrack, J. Sprent und D. B. Wilson. „T cell receptors for responses to Mls determinants and allo-H-2 determinants appear to be encoded on different chromosomes.“ Journal of Experimental Medicine 161, Nr. 1 (01.01.1985): 269–74. http://dx.doi.org/10.1084/jem.161.1.269.
Der volle Inhalt der QuelleAmir, Avital L., Lloyd J. A. D'Orsogna, Dave L. Roelen, Marleen M. van Loenen, Renate S. Hagedoorn, Renate de Boer, Menno A. W. G. van der Hoorn et al. „Allo-HLA reactivity of virus-specific memory T cells is common“. Blood 115, Nr. 15 (15.04.2010): 3146–57. http://dx.doi.org/10.1182/blood-2009-07-234906.
Der volle Inhalt der QuelleHuisman, Wesley, Didier A. T. Leboux, Lieve E. van der Maarel, Lois Hageman, Derk Amsen, Fred Falkenburg und Inge Jedema. „Off-Target HLA Cross-Reactivity By (Third Party) Virus-Specific T Cells Is Surprisingly Affected By HLA Restriction and HLA Background but Not By Virus Specificity“. Blood 134, Supplement_1 (13.11.2019): 4440. http://dx.doi.org/10.1182/blood-2019-124785.
Der volle Inhalt der QuelleHasenkamp, Justin, Andrea Borgerding, Bjoern Chapuy, Gerald Wulf, Inga Missal, Wolfram Jung, Lorenz Truemper und Bertram Glass. „Allo-Reactive NK Cells after HLA-Matched Allogeneic Hematopoietic Stem Cell Transplantation.“ Blood 108, Nr. 11 (16.11.2006): 2904. http://dx.doi.org/10.1182/blood.v108.11.2904.2904.
Der volle Inhalt der QuelleWebb, S. R., A. Okamoto und J. Sprent. „Analysis of T hybridomas prepared from a T cell clone with three specificities. Recognition of self + X and allo-H-2 determinants segregates from recognition of Mlsa determinants.“ Journal of Immunology 141, Nr. 6 (15.09.1988): 1828–34. http://dx.doi.org/10.4049/jimmunol.141.6.1828.
Der volle Inhalt der QuelleElkon, Keith B., und Dalit Ash Any. „Autoimmunity Versus Allo- and Xeno-Reactivity in SCID Mice“. International Reviews of Immunology 11, Nr. 4 (Januar 1994): 283–93. http://dx.doi.org/10.3109/08830189409051175.
Der volle Inhalt der QuelleLaghmouchi, Aicha, Conny Hoogstraten, Peter Van Balen, Rick van de Water, Marian van de Meent, J. H. Frederik Falkenburg und Inge Jedema. „The Allo-HLA-DP Restricted T Cell Repertoire Contains a Variety of Tissue-Restricted Specificities with Therapeutic Value“. Blood 128, Nr. 22 (02.12.2016): 3356. http://dx.doi.org/10.1182/blood.v128.22.3356.3356.
Der volle Inhalt der QuelleNoonan, Kimberly, Leo Luznik und Ivan M. Borrello. „Enrichment of Allogeneic Tumor Antigen-Specific T Cells From Bone Marrow (BM) of Patients Treated with High-Dose Post-Transplant Cyclophoshamide (Cy) – A Novel Approach to Adoptive Immunotherapy“. Blood 118, Nr. 21 (18.11.2011): 647. http://dx.doi.org/10.1182/blood.v118.21.647.647.
Der volle Inhalt der QuelleD’Orsogna, Lloyd, Heleen van den Heuvel, Cees van Kooten, Sebastiaan Heidt und Frans H. J. Claas. „Infectious pathogens may trigger specific allo-HLA reactivity via multiple mechanisms“. Immunogenetics 69, Nr. 8-9 (17.07.2017): 631–41. http://dx.doi.org/10.1007/s00251-017-0989-3.
Der volle Inhalt der QuelleDissertationen zum Thema "Allo-reactivity"
Nattes, Tristan de. „Rejet humoral d'allogreffe rénale et allo-immunisation HLA“. Electronic Thesis or Diss., Normandie, 2023. http://www.theses.fr/2023NORMR053.
Der volle Inhalt der QuelleKidney transplantation is the best treatment of end-stage renal disease, improving life quality and quantity. Despite advances in pathophysiological knowledge of kidney transplant immunology, kidney transplant rejection remains the major cause of allograft dysfunction, especially antibody-mediated rejection.Antibody-mediated rejection risk assessment is based on the evaluation of donor-specific anti-HLA antibodies. However, these antibodies have a poor predictive value for incidence and prognosis of rejection. This could be explained by the heterogeneity of their intrinsic characteristics. These characteristics depend on cells responsible for their secretion, which include short- and long- lived plasma cells. Consequently, they indirectly depend on the cells responsible for maintaining the pool of these antibody-secreting cells, such as memory B cells. In infectious diseases, it is known that memory B cells are heterogeneous in terms of phenotype, function, degree of clonality, and diversification of their B-cell receptor (BCR). However, this heterogeneity has not been examined in the context of kidney transplantation.The aim of the first part of this thesis was to study the heterogeneity of HLA-specific memory B cells in sensitised patients on kidney transplant waiting list. To this end, single-cell analysis of HLA-specific memory B cells from patients with various aetiologies and degrees of immunisation was performed. This led to their phenotypic and transcriptomic characterisation and to the assessment of their BCR repertoire.The second part of this thesis was dedicated to the diagnosis of kidney transplant rejection.In recent years, biopsy-based transcriptomics has emerged, enabling the assessment of hundreds of transcripts in kidney biopsy tissue. These tools provide the opportunity to elucidate new physiopathological pathways and potentially enhance the diagnosis of rejection, especially humoral rejection. However, their application in clinical practice is still limited due to their restricted availability, required expertise for data processing and interpretation, and cost. Furthermore, their exact impact on patient management remains undetermined. Here, a molecular diagnostic tool with characteristics suitable for clinical use was developed. This tool enables the diagnosis of rejection and its classification between antibody-mediated and T-cell mediated rejection. Subsequently, this tool was assessed in ambiguous clinical situations to evaluate its impact in clinical practice.Through these studies, this thesis focused on enhancing our understanding of the humoral response in renal transplantation, which could help improving immunological risk stratification in transplantation. Additionally, it aimed to improve biopsy-based transcriptomics in the diagnosis of kidney transplant rejection
Buchteile zum Thema "Allo-reactivity"
D’Orsogna, Lloyd J., Ellen M. W. van der Meer-Prins, Yvonne M. Zoet, Dave L. Roelen, Ilias I. N. Doxiadis und Frans H. J. Claas. „Detection of Allo-HLA Cross-Reactivity by Virus-specific Memory T-Cell Clones Using Single HLA-Transfected K562 Cells“. In Methods in Molecular Biology, 339–49. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-842-9_19.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Allo-reactivity"
Howie, Duncan, Terri V. Cornforth, Nathifa Moyo, Suzanne Cole, Emily Lam, Tatiana Lobry, Ron Wolchinsky et al. „362 Allelic variants of MR1 drive cancer and allo-reactivity by MR1-restricted T cells“. In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.0362.
Der volle Inhalt der Quelle