Thematische Bibliographien / AIDS (Disease) / Zeitschriftenartikel
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Zeitschriftenartikel zum Thema „AIDS (Disease)“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 29. Juli 2024

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1

Thomsen, Hauke, Xinjun Li, Kristina Sundquist, Jan Sundquist, Asta Försti und Kari Hemminki. „Familial associations for Addison’s disease and between Addison’s disease and other autoimmune diseases“. Endocrine Connections 9, Nr. 11 (November 2020): 1114–20. http://dx.doi.org/10.1530/ec-20-0328.

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Design Addison’s disease (AD) is a rare autoimmune disease (AID) of the adrenal cortex, present as an isolated AD or part of autoimmune polyendocrine syndromes (APSs) 1 and 2. Although AD patients present with a number of AID co-morbidities, population-based family studies are scarce, and we aimed to carry out an unbiased study on AD and related AIDs. Methods We collected data on patients diagnosed with AIDs in Swedish hospitals and calculated standardized incidence ratios (SIRs) in families for concordant AD and for other AIDs, the latter as discordant relative risks. Results The number of AD patients was 2852, which accounted for 0.4% of all hospitalized AIDs. A total of 62 persons (3.6%) were diagnosed with familial AD. The SIR for siblings was remarkably high, reaching 909 for singleton siblings diagnosed before age 10 years. It was 32 in those diagnosed past age 29 years and the risk for twins was 323. SIR was 9.44 for offspring of affected parents. AD was associated with 11 other AIDs, including thyroid AIDs and type 1 diabetes and some rarer AIDs such as Guillain–Barre syndrome, myasthenia gravis, polymyalgia rheumatica and Sjögren’s syndrome. Conclusions The familial risk for AD was very high implicating genetic etiology, which for juvenile siblings may be ascribed to APS-1. The adult part of sibling risk was probably contributed by recessive polygenic inheritance. AD was associated with many common AIDs; some of these were known co-morbidities in AD patients while some other appeared to more specific for a familial setting.
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2

Kamradt, T., D. Niese und F. Vogel. „SLIM DISEASE (AIDS)“. Lancet 326, Nr. 8469-8470 (Dezember 1985): 1425. http://dx.doi.org/10.1016/s0140-6736(85)92588-7.

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3

Marquart, K. H., H. A. G. Müller, J. Sailer und R. Moser. „SLIM DISEASE (AIDS)“. Lancet 326, Nr. 8465 (November 1985): 1186–87. http://dx.doi.org/10.1016/s0140-6736(85)92707-2.

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4

Chen, Yahong, Jinjin Yuan, Xianlin Han, Xiaolong Liu, Xiao Han und Hanhui Ye. „Coexpression Analysis of Transcriptome on AIDS and Other Human Disease Pathways by Canonical Correlation Analysis“. International Journal of Genomics 2017 (2017): 1–10. http://dx.doi.org/10.1155/2017/9163719.

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Acquired immune deficiency syndrome is a severe disease in humans caused by human immunodeficiency virus. Several human genes were characterized as host genetic factors that impact the processes of AIDS disease. Recent studies on AIDS patients revealed a series disease is complicating with AIDS. To resolve gene interaction between AIDS and complicating diseases, a canonical correlation analysis was used to identify the global correlation between AIDS and other disease pathway genes expression. The results showed that HLA-B, HLA-A, MH9, ZNED1, IRF1, TLR8, TSG101, NCOR2, and GML are the key AIDS-restricted genes highly correlated with other disease pathway genes. Furthermore, pathway genes in several diseases such as asthma, autoimmune thyroid disease, and malaria were globally correlated with ARGs. It suggests that these diseases are a high risk in AIDS patients as complicating diseases.
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5

Simon, Douglas, und Lawrence J. Brandt. „Biliary tract disease in aids: Aids vs. nonaids“. Hepatology 12, Nr. 3 (September 1990): 618–19. http://dx.doi.org/10.1002/hep.1840120330.

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6

McGuinness, G., J. F. Gruden, M. Bhalla, T. J. Harkin, J. S. Jagirdar und D. P. Naidich. „AIDS-related airway disease.“ American Journal of Roentgenology 168, Nr. 1 (Januar 1997): 67–77. http://dx.doi.org/10.2214/ajr.168.1.8976923.

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7

Golbus, Joseph. „Rheumatic disease and AIDS“. Postgraduate Medicine 92, Nr. 4 (15.09.1992): 99–110. http://dx.doi.org/10.1080/00325481.1992.11701468.

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8

McGowan, Ian, und Peter Anton. „AIDS and intestinal disease“. Current Opinion in Gastroenterology 13, Nr. 1 (Januar 1997): 18–23. http://dx.doi.org/10.1097/00001574-199701000-00004.

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9

Metze, K., und J. A. Maciel. „AIDS and Chagas' disease“. Neurology 43, Nr. 2 (01.02.1993): 447. http://dx.doi.org/10.1212/wnl.43.2.447-a.

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10

Rieve, Julia A. „AIDS Disease Management Programs“. Case Manager 11, Nr. 5 (September 2000): 38–39. http://dx.doi.org/10.1067/mcm.2000.110320.

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11

Goodman, Philip C. „Mycobacterial disease in AIDS“. Journal of Thoracic Imaging 6, Nr. 4 (September 1991): 22–27. http://dx.doi.org/10.1097/00005382-199109000-00007.

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12

Navarro, Willis H., und Lawrence D. Kaplan. „AIDS-related lymphoproliferative disease“. Blood 107, Nr. 1 (01.01.2006): 13–20. http://dx.doi.org/10.1182/blood-2004-11-4278.

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Abstract Not long after the recognition of HIV as the causative agent of AIDS, it was evident that individuals infected with HIV developed lymphoma at a greater rate than the population at large. Approximately two thirds of AIDS-related lymphoma (ARL) cases are categorized as diffuse large B-cell type, with Burkitt lymphomas comprising 25% and other histologies a much smaller proportion. Typically, these individuals have presented with advanced extranodal disease and CD4+ lymphocyte counts of less than 200/mm3. Recent clinical trials have demonstrated a better outcome with chemotherapy for ARL since the introduction of combination antiretroviral treatment, termed highly active antiretroviral therapy (HAART). For patients with relapses, solid evidence points to the safety and utility of hematopoietic-cell transplantation as a salvage modality. Coinfection with other viruses such as Epstein-Barr virus and Kaposi sarcoma-associated herpesvirus have led to the genesis of previously rare or unrecognized lymphoma subtypes such as plasmablastic and primary effusion lymphomas. The immunosuppressive impact of treatment for patients with ARL receiving chemotherapy with HAART appears transient and opportunistic infections have become less problematic than prior to HAART. Significant progress has been made in the understanding and management of ARL but outcomes still remain inferior compared to those achieved in HIV- individuals.
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13

Pinto, Amélia Nogueira. „AIDS and Cerebrovascular Disease“. Stroke 27, Nr. 3 (März 1996): 538–43. http://dx.doi.org/10.1161/01.str.27.3.538.

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14

Williams, Jennet, und Mark Bower. „AIDS-related malignant disease“. Medicine 41, Nr. 8 (August 2013): 430–34. http://dx.doi.org/10.1016/j.mpmed.2013.05.013.

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15

Dalla Pria, Alessia, und Mark Bower. „AIDS-related malignant disease“. Medicine 46, Nr. 6 (Juni 2018): 365–69. http://dx.doi.org/10.1016/j.mpmed.2018.03.002.

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16

Mönkemüller, Klaus E., und Martín Olmos. „Gastric disease in AIDS“. Techniques in Gastrointestinal Endoscopy 4, Nr. 2 (April 2002): 66–70. http://dx.doi.org/10.1053/tgie.2002.33013.

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17

Ames, Elizabeth D., Michael S. Conjalka, Arthur F. Goldberg, Richard Hirschman, Sushil Jain, Ariel Distenfeld und Craig E. Metroka. „Hodgkin's Disease and AIDS“. Hematology/Oncology Clinics of North America 5, Nr. 2 (April 1991): 343–56. http://dx.doi.org/10.1016/s0889-8588(18)30446-5.

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18

Lopez-Herce Cid, JoseAndres, Jesus Lopez-Herce Cid, EduardoFlores Sañudo, Javier Menarguez und JaimeCosin Ochaita. „AIDS AND HODGKIN'S DISEASE“. Lancet 328, Nr. 8515 (November 1986): 1104–5. http://dx.doi.org/10.1016/s0140-6736(86)90510-6.

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19

Temple, J. J., und W. Abe Andes. „AIDS AND HODGKIN'S DISEASE“. Lancet 328, Nr. 8504 (August 1986): 454–55. http://dx.doi.org/10.1016/s0140-6736(86)92155-0.

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20

Kuo, Irene, und Narsing A. Rao. „Ocular disease in AIDS“. Springer Seminars in Immunopathology 21, Nr. 2 (Juni 1999): 161–77. http://dx.doi.org/10.1007/bf00810248.

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21

Kuo, Irene, und Narsing A. Rao. „Ocular disease in AIDS“. Springer Seminars in Immunopathology 21, Nr. 2 (26.07.1999): 161–77. http://dx.doi.org/10.1007/s002810050060.

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22

Peppercorn, Mark A. „Anorectal disease in AIDS“. Gastroenterology 104, Nr. 6 (Juni 1993): 1883. http://dx.doi.org/10.1016/0016-5085(93)90684-5.

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23

Collins, Frank M. „AIDS-related mycobacterial disease“. Springer Seminars in Immunopathology 10, Nr. 4 (Dezember 1988): 375–91. http://dx.doi.org/10.1007/bf02053847.

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24

Corti, Marcelo. „AIDS and Chagas' Disease“. AIDS Patient Care and STDs 14, Nr. 11 (November 2000): 581–88. http://dx.doi.org/10.1089/10872910050193752.

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25

Ives, David V. „Cytomegalovirus disease in AIDS“. AIDS 11, Nr. 15 (Dezember 1997): 1791–97. http://dx.doi.org/10.1097/00002030-199715000-00002.

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26

Berger, Joseph R., Jonathan O. Harris, Jocelyn Gregoriost und Michael Norenbergt. „Cerebrovascular disease in AIDS“. AIDS 4, Nr. 3 (März 1990): 239–44. http://dx.doi.org/10.1097/00002030-199003000-00010.

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27

Nyamathi, Adeline. „AIDS-related heart disease“. Journal of Cardiovascular Nursing 3, Nr. 4 (August 1989): 65–76. http://dx.doi.org/10.1097/00005082-198908000-00009.

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28

Brettman, Lee R. „Liver disease in aids“. Hepatology 7, Nr. 2 (März 1987): 403–5. http://dx.doi.org/10.1002/hep.1840070238.

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29

WEBER, JONATHAN. „Gastrointestinal Disease in AIDS“. Clinics in Immunology and Allergy 6, Nr. 3 (Oktober 1986): 519–41. http://dx.doi.org/10.1016/s0260-4639(22)00095-0.

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30

Sunpaweravong, S. „Gastrointestinal disease in AIDS“. Chulalongkorn Medical Journal 41, Nr. 10 (Oktober 1997): 711–15. http://dx.doi.org/10.58837/chula.cmj.41.10.2.

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31

Valenti, William M. „Update on AIDS“. Infection Control 6, Nr. 2 (Februar 1985): 85–86. http://dx.doi.org/10.1017/s0195941700062664.

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Recent advances in our knowledge of the Acquired Immune Deficiency Syndrome (AIDS) could have a major impact on practitioners in both infection control and employee health. The discovery that a retrovirus, the human T-cell leukemia virus (HTLV-III) is the cause of AIDS could be used to help health care workers better understand this disease. Those of us who are trying to provide our employees with up-to-date information on AIDS should use this new information to help employees understand the nature of the disease, its transmissibility and non-transmissibility. Now that the disease can be linked to a specific agent, employees may find AIDS to be much less mysterious. The retrovirus link is a major step forward in understanding this disease and may eventually lead to more effective treatment and possibly a vaccine. Our knowledge continues to evolve in much the same way as our understanding of Legionnaires' Disease and Toxic Shock Syndrome.The Acquired Immune Deficiency Syndrome (AIDS) was discussed extensively at recent infectious diseases meetings in Washington, D.C. The results of original research conducted by investigators at the National Institutes of Health (NIH), Centers for Disease Control (CDC), Pasteur Institute in Paris, and medical centers throughout the world were presented at the meetings. A number of issues have been clarified and the pieces of the puzzle are starting to come together.
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32

Wilcox, C. Mel, und Klaus E. Mönkemüller. „Hepatobiliary Diseases in Patients with AIDS: Focus on AIDS Cholangiopathy and Gallbladder Disease“. Digestive Diseases 16, Nr. 4 (1998): 205–13. http://dx.doi.org/10.1159/000016868.

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33

Samuels, Hadas, Malki Malov, Trishna Saha Detroja, Karin Ben Zaken, Naamah Bloch, Meital Gal-Tanamy, Orly Avni, Baruh Polis und Abraham O. Samson. „Autoimmune Disease Classification Based on PubMed Text Mining“. Journal of Clinical Medicine 11, Nr. 15 (26.07.2022): 4345. http://dx.doi.org/10.3390/jcm11154345.

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Autoimmune diseases (AIDs) are often co-associated, and about 25% of patients with one AID tend to develop other comorbid AIDs. Here, we employ the power of datamining to predict the comorbidity of AIDs based on their normalized co-citation in PubMed. First, we validate our technique in a test dataset using earlier-reported comorbidities of seven knowns AIDs. Notably, the prediction correlates well with comorbidity (R = 0.91) and validates our methodology. Then, we predict the association of 100 AIDs and classify them using principal component analysis. Our results are helpful in classifying AIDs into one of the following systems: (1) gastrointestinal, (2) neuronal, (3) eye, (4) cutaneous, (5) musculoskeletal, (6) kidneys and lungs, (7) cardiovascular, (8) hematopoietic, (9) endocrine, and (10) multiple. Our classification agrees with experimentally based taxonomy and ranks AID according to affected systems and gender. Some AIDs are unclassified and do not associate well with other AIDs. Interestingly, Alzheimer’s disease correlates well with other AIDs such as multiple sclerosis. Finally, our results generate a network classification of autoimmune diseases based on PubMed text mining and help map this medical universe. Our results are expected to assist healthcare workers in diagnosing comorbidity in patients with an autoimmune disease, and to help researchers in identifying common genetic, environmental, and autoimmune mechanisms.
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34

Janaki, CR, GS Selvi, B. Parveen, N. Gomathy und Anupama Roshan. „AIDS defining disease: Disseminated cryptococcosis“. Indian Journal of Dermatology 51, Nr. 3 (2006): 202. http://dx.doi.org/10.4103/0019-5154.27988.

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35

Northup, George W. „AIDS: Disease of the 80s“. Journal of the American Osteopathic Association 85, Nr. 11 (01.11.1985): 46–47. http://dx.doi.org/10.1515/jom-1985-851110.

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36

Silberner, J. „AIDS: Disease, Research Efforts Advance“. Science News 127, Nr. 17 (27.04.1985): 260. http://dx.doi.org/10.2307/3969504.

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37

Ryder, Mark I., Wipawee Nittayananta, Maeve Coogan, Deborah Greenspan und John S. Greenspan. „Periodontal disease in HIV/AIDS“. Periodontology 2000 60, Nr. 1 (22.08.2012): 78–97. http://dx.doi.org/10.1111/j.1600-0757.2012.00445.x.

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38

Lewis, Laurie. „AIDS as a Chronic Disease“. AIDS Patient Care 3, Nr. 3 (Juni 1989): 28–30. http://dx.doi.org/10.1089/apc.1989.3.28.

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39

Faria, PR, PA Vargas, PHN Saldiva, GM Böhm, T. Mauad und OP Almeida. „Tongue disease in advanced AIDS“. Oral Diseases 11, Nr. 2 (März 2005): 72–80. http://dx.doi.org/10.1111/j.1601-0825.2004.01070.x.

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40

Cello, John P. „AIDS-Related Biliary Tract Disease“. Gastrointestinal Endoscopy Clinics of North America 8, Nr. 4 (Oktober 1998): 963–73. http://dx.doi.org/10.1016/s1052-5157(18)30242-3.

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41

Yu, Qianli, Douglas F. Larson und Ronald R. Watson. „Heart disease, methamphetamine and AIDS“. Life Sciences 73, Nr. 2 (Mai 2003): 129–40. http://dx.doi.org/10.1016/s0024-3205(03)00260-1.

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42

Yeager, Barbara. „AIDS not an infectious disease?“ Geriatric Nursing 12, Nr. 1 (Januar 1991): 4. http://dx.doi.org/10.1016/s0197-4572(06)80299-5.

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43

Freeman, William R. „Retinal disease associated with AIDS*“. Australian and New Zealand Journal of Ophthalmology 21, Nr. 2 (Mai 1993): 71–78. http://dx.doi.org/10.1111/j.1442-9071.1993.tb00757.x.

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44

Kohan, Darius, Paul E. Hammerschlag und Roy A. Holliday. „Otologic Disease in AIDS Patients“. Laryngoscope 100, Nr. 12 (Dezember 1990): 1326???1330. http://dx.doi.org/10.1288/00005537-199012000-00016.

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45

Enns, R. „AIDS cholangiopathy: “an endangered disease”“. American Journal of Gastroenterology 98, Nr. 10 (Oktober 2003): 2111–12. http://dx.doi.org/10.1016/s0002-9270(03)00757-3.

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46

Kelly, R. D. „AIDS: Therapeutics in HIV Disease“. Biochemical Education 16, Nr. 4 (Oktober 1988): 238. http://dx.doi.org/10.1016/0307-4412(88)90146-x.

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47

Pratt, Robert J. „AIDS—Therapeutics in HIV disease“. International Journal of Nursing Studies 26, Nr. 1 (Januar 1989): 84. http://dx.doi.org/10.1016/0020-7489(89)90052-7.

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48

Weissler, Jonathan C., und Ann R. Mootz. „Pulmonary Disease in AIDS Patients“. American Journal of the Medical Sciences 300, Nr. 5 (November 1990): 330–43. http://dx.doi.org/10.1097/00000441-199011000-00010.

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49

Lowe, G. D. O. „AIDS—therapeutics of HIV disease“. Fibrinolysis 2, Nr. 4 (Oktober 1988): 259. http://dx.doi.org/10.1016/0268-9499(88)90022-7.

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50

Enns, Robert. „AIDS cholangiopathy: "an endangered disease"“. American Journal of Gastroenterology 98, Nr. 10 (Oktober 2003): 2111–12. http://dx.doi.org/10.1111/j.1572-0241.2003.07724.x.

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