Auswahl der wissenschaftlichen Literatur zum Thema „Aging cardiac“
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Zeitschriftenartikel zum Thema "Aging cardiac"
Wessells, Robert J., und Rolf Bodmer. „Cardiac aging“. Seminars in Cell & Developmental Biology 18, Nr. 1 (Februar 2007): 111–16. http://dx.doi.org/10.1016/j.semcdb.2006.12.011.
Der volle Inhalt der QuelleLi, Zhen, und David J. Lefer. „Demystifying Cardiac Aging“. Circulation Research 128, Nr. 4 (19.02.2021): 508–10. http://dx.doi.org/10.1161/circresaha.121.318741.
Der volle Inhalt der QuelleLIMACHER, MARIAN C. „Aging and Cardiac Function“. Southern Medical Journal 87, Nr. 5 (Mai 1994): S17. http://dx.doi.org/10.1097/00007611-199405000-00003.
Der volle Inhalt der QuelleLIMACHER, MARIAN C. „Aging and Cardiac Function“. Southern Medical Journal 87, Supplement (Mai 1994): S17. http://dx.doi.org/10.1097/00007611-199405001-00003.
Der volle Inhalt der QuelleMiyamoto, Shigeki. „Autophagy and cardiac aging“. Cell Death & Differentiation 26, Nr. 4 (28.01.2019): 653–64. http://dx.doi.org/10.1038/s41418-019-0286-9.
Der volle Inhalt der QuelleRenlund, Dale G., und Gary Gerstenblith. „Aging and cardiac function“. International Journal of Cardiology 10, Nr. 3 (März 1986): 193–96. http://dx.doi.org/10.1016/0167-5273(86)90001-x.
Der volle Inhalt der QuelleCzuriga, D., Z. Papp, I. Czuriga und Á. Balogh. „Cardiac aging – a review“. European Surgery 43, Nr. 2 (April 2011): 69–77. http://dx.doi.org/10.1007/s10353-011-0600-3.
Der volle Inhalt der QuelleObas, Vanessa, und Ramachandran S. Vasan. „The aging heart“. Clinical Science 132, Nr. 13 (09.07.2018): 1367–82. http://dx.doi.org/10.1042/cs20171156.
Der volle Inhalt der QuelleChen, Bijun, Shuaibo Huang und Nikolaos G. Frangogiannis. „Aging, cardiac repair and Smad3“. Aging 10, Nr. 9 (20.09.2018): 2230–32. http://dx.doi.org/10.18632/aging.101567.
Der volle Inhalt der QuelleWestin, Oscar, Finn Gustafsson und Emil Fosbøl. „Occult cardiac amyloidosis?“ Aging 11, Nr. 20 (19.10.2019): 8739–40. http://dx.doi.org/10.18632/aging.102383.
Der volle Inhalt der QuelleDissertationen zum Thema "Aging cardiac"
Inuzuka, Yasutaka. „Suppression of phosphoinositide 3-kinase prevents cardiac aging in mice“. Kyoto University, 2009. http://hdl.handle.net/2433/126462.
Der volle Inhalt der QuelleHaynes, Premi. „TRANSMURAL HETEROGENEITY OF CELLULAR LEVEL CARDIAC CONTRACTILE PROPERTIES IN AGING AND HEART FAILURE“. UKnowledge, 2014. http://uknowledge.uky.edu/physiology_etds/16.
Der volle Inhalt der QuelleYang, Bo, und 杨波. „Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity“. Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47869641.
Der volle Inhalt der Quellepublished_or_final_version
Pharmacology and Pharmacy
Doctoral
Doctor of Philosophy
Ueno, Linda Massako. „Effects of aging and regular exercise upon baroreflex sensitivity and cardiac autonomic activities“. Kyoto University, 2003. http://hdl.handle.net/2433/148937.
Der volle Inhalt der Quelle0048
新制・課程博士
博士(人間・環境学)
甲第10300号
人博第187号
14||151(吉田南総合図書館)
新制||人||46(附属図書館)
UT51-2003-H721
京都大学大学院人間・環境学研究科文化・地域環境学専攻
(主査)教授 森谷 敏夫, 教授 中村 榮太郎, 教授 津田 謹輔
学位規則第4条第1項該当
Amano, Masari. „Effects of exercise training, aging, gender and mental stress upon cardiac autonomic nervous function“. Kyoto University, 2003. http://hdl.handle.net/2433/148936.
Der volle Inhalt der Quelle0048
新制・課程博士
博士(人間・環境学)
甲第10299号
人博第186号
14||150(吉田南総合図書館)
新制||人||45(附属図書館)
UT51-2003-H720
京都大学大学院人間・環境学研究科文化・地域環境学専攻
(主査)教授 森谷 敏夫, 教授 津田 謹輔, 教授 田口 貞善
学位規則第4条第1項該当
Ramsey, Michael Wiechmann. „Effects of head-up tilt on mean arterial pressure, heart rate, and regional cardiac output distribution in aging rats“. Texas A&M University, 2005. http://hdl.handle.net/1969.1/3094.
Der volle Inhalt der QuelleAnstine, Lindsey J. „Valve cell dynamics in developing, mature, and aging heart valves“. The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1478692972995079.
Der volle Inhalt der QuelleLODRINI, ALESSANDRA MARIA. „Cellular senescence and failure in human and animal cardiac myocytes“. Doctoral thesis, Università degli Studi di Milano-Bicocca, 2021. http://hdl.handle.net/10281/301783.
Der volle Inhalt der QuelleDuring my PhD I was involved mainly in two research projects aimed to study myocardial dysfunction induced by aging or chemotherapy. The first study aimed to reproduce and characterize mechanisms involved in aging using cardiomyocytes (CMs) from human induced pluripotent stem cells (hiPSCs), and to test cardioprotective therapies, like cardiac progenitor cell (CPC)-derived exosomes (Exo). Aging of the heart involves adverse remodeling in CMs which results in heart failure with incidence that increases with age. Interestingly, till now we lacked a human model of cardiac aging. We reprogrammed CPCs into hiPSCs and subsequently differentiated in hiPSC-derived CMs. A senescence-like phenotype (SenCMs) was induced by short exposure (3 hours) to doxorubicin (Dox) at sub-lethal concentration (0.2 µM). 24h following DOX treatment, SenCMs were exposed to Exo (~2·103 particles/cell) collected from culture media of CPCs by ultracentrifugation. Dox treatment induced senescence, as confirmed by activation of p21 and increased SA-β-gal positivity compared to control CMs (cCMs). Biochemical analysis revealed presence of oxidative stress and a depolarized mitochondrial membrane potential due to the treatment, which resulted in decreased ATP production by mitochondria. SenCMs also showed impaired calcium handling and prolonged QTc vs. cCMs due to upregulation of INaL. These effects were mitigated by exposure to Exo. Overall, SenCMs recapitulate the phenotype of aged CMs in terms of senescence markers and electrical and metabolic properties. Additionally, exposure to CPC-derived Exo limited age-related cardiac anomalies. The second study aimed to study the cardiac dysfunction dependent on the combined administration of Dox and trastuzumab (Trz) through evaluation of cardiac performance, T-tubule organization, and electrophysiological changes in cardiac myocytes from an in-vivo rat model. Combined treatment with Dox and Trz in patients with HER2-positive cancer is limited by cardiotoxicity, as manifested by contractile dysfunction and arrhythmia. The respective roles of the two agents in the cardiotoxicity of the combined therapy are incompletely understood. Adult rats received 6 doses of either Dox or Trz, or the two agents sequentially. Dox-mediated left ventricular (LV) dysfunction was aggravated by Trz administration. Dox treatment, but not Trz, induced T-tubule disarray. Moreover, Dox, but not Trz monotherapy, induced prolonged action potential duration (APD), increased incidence of delayed afterdepolarizations (DADs) and beat-to-beat variability of repolarization (BVR), and slower Ca2+ transient decay. Although APD, DADs, BVR and Ca2+ transient decay recovered over time after the cessation of Dox treatment, subsequent Trz administration exacerbated these abnormalities. Trz, but not Dox, reduced Ca2+ transient amplitude and SR Ca2+ content. Both agents increased Ca2+ waves and downregulated SERCA. Finally, Dox increased resting Ca2+ waves, Ca2+ spark frequency, spark-mediated sarcoplasmic reticulum (SR) leak, and long-lasting Ca2+ release events (so-called Ca2+ “embers”). These results suggest that Dox, but not Trz, may cause T-tubule disarray in cardiac myocytes in vivo while also inducing overall larger changes in electrical parameters and intracellular Ca2+ handling. While Dox-induced changes in electrical parameters are reversible, subsequent Trz administration prevents their recovery. These findings illustrate the specific roles of Dox and Trz, and their interactions in cardiotoxicity and arrhythmogenicity.
ZENI, FILIPPO. „Circulating levels of soluble Receptor for Advanced Glycation End-products (sRAGE) decrease with aging and may predict age-related cardiac remodeling“. Doctoral thesis, Università degli Studi di Milano-Bicocca, 2017. http://hdl.handle.net/10281/170797.
Der volle Inhalt der QuelleBackground: Aging is an unavoidable risk factor in later life that can influence the onset and progression of many diseases. In fact, the high incidence of cardiovascular diseases in the elderly is mainly attributable to cardiac remodelling associated to physiological intrinsic aging. RAGE is a multi-ligand receptor involved in many age-related disorders. Its soluble isoform (sRAGE) acts as a decoy receptor being able to block the activation of the membrane-bound receptor, and its circulation levels have been found altered in several chronic and acute pathologies. The role of RAGE isoforms in aging and, in particular, cardiac senescence has never been investigated. Moreover, the finding of reliable biomarkers able to assess individual health status of subjects has important applications in prevention, diagnosis, and disease management. In this context, the aim of this study was to ascertain whether sRAGE is a biomarker of aging and age-related cardiac remodelling, and evaluate the contribution of RAGE isoforms to cardiac aging. Results: Serum of male and female from 20 to 92 years old healthy subjects was collected and sRAGE levels were evaluated by ELISA. We found a significant decrease of circulating sRAGE in males while only a trend in females. Accordingly, we observed a strong correlation of sRAGE with chronological age in male but not in female subjects. Male and female mice at different age (2.5-12-22-months, Young, Middle Age (MA) and Old, respectively) undergone 2D-echocardiography to determine the left ventricle (LV) dimensions and function during aging. Serum sRAGE similarly declines from the Young to the MA group in both sexes, and inversely correlate with LV dimensions and function, preferentially in males. No detectable amount of RAGE protein was found in LV at all ages. Rage-/- mice displayed a significant increase of LV volumes and diameters in diastole and systole, and a concomitant decrease in ejection fraction (EF) and fractional shortening (FS), compared to age-matched wt animals during aging with the strongest differences present between the MA groups. Moreover, MA Rage-/- mice exhibited higher deposition of collagen and expression of heart failure marker genes (BNP and Ankrd1) in respect to the wt counterpart. Conversely, no differences in cardiomyocytes size were observed at any age between the two genotypes. Finally, microarray functional annotation analysis based on the interaction between age-genotype revealed that the chronic lack of RAGE affected the expression of genes associated to contractile fibre function, antigen presenting process and adaptive immunity, insulin pathway, cell death and apoptosis. We also found a correlation between LV volumes and diameters in diastole and systole and differentially expressed genes involved in several processes like muscle contraction, fibrosis, wound healing and regulation of apoptosis. Conclusions: Our results indicate that sRAGE is a serum biomarker of healthy aging and age-related cardiac remodeling, preferentially in males. The absence of RAGE in mice exacerbates adverse cardiac remodeling with age. We propose that, among RAGE isoforms, sRAGE may play a pivotal role in cardiac senescence.
Sheikh, Hajer Nisar. „Tropomyosin Phosphorylation in Cardiac Health and Disease“. University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1242913472.
Der volle Inhalt der QuelleBücher zum Thema "Aging cardiac"
United States. Congress. Senate. Special Committee on Aging. Pacemakers revisited: A saga of benign neglect : hearing before the Special Committee on Aging, United States Senate, Ninety-ninth Congres, first session, Washington, DC, May 10, 1985. Washington: U.S. G.P.O., 1986.
Den vollen Inhalt der Quelle findenOffice, General Accounting. Medicare's policies and prospective payment rates for cardiac pacemaker surgeries need review and revision: Report to the Chairman, Special Committee on Aging, United States Senate. Washington, D.C: U.S. General Accounting Office, 1985.
Den vollen Inhalt der Quelle findenFried, Robert, und Richard Carlton. Omega-Factor: Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Den vollen Inhalt der Quelle findenFried, Robert, und Richard Carlton. Omega-Factor: Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Den vollen Inhalt der Quelle findenFried, Robert, und Richard Carlton. Omega-Factor: Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Den vollen Inhalt der Quelle findenFried, Robert, und Richard Carlton. Omega-Factor: Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Den vollen Inhalt der Quelle findenFried, Robert, und Richard Carlton. Omega-Factor: Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Den vollen Inhalt der Quelle findenCardiothoracic surgery in the elderly. New York: Springer, 2011.
Den vollen Inhalt der Quelle findenCardiothoracic surgery in the elderly. New York: Springer, 2011.
Den vollen Inhalt der Quelle findenKatlic, Mark R. Cardiothoracic Surgery in the Elderly. Springer New York, 2014.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Aging cardiac"
Dai, Dao-Fu, Robert J. Wessells, Rolf Bodmer und Peter S. Rabinovitch. „Cardiac Aging“. In The Comparative Biology of Aging, 259–86. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-3465-6_12.
Der volle Inhalt der QuelleChantler, Paul D., und Edward G. Lakatta. „Heart and Arterial Aging“. In Cardiac Adaptations, 111–44. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5203-4_7.
Der volle Inhalt der QuelleFenton, Richard A., Mojca Lorbar und James G. Dobson. „Adenosine and Cardiac Aging“. In Developments in Cardiovascular Medicine, 143–58. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5603-9_9.
Der volle Inhalt der QuelleCesselli, Daniela, Federica D’Aurizio, Patrizia Marcon, Natascha Bergamin, Carlo Alberto Beltrami und Antonio Paolo Beltrami. „Cardiac Stem Cell Senescence“. In Stem Cells and Aging, 81–97. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-317-6_7.
Der volle Inhalt der QuelleJugdutt, Bodh I., und Anwar Jelani. „Aging and Markers of Adverse Remodeling After Myocardial Infarction“. In Cardiac Remodeling, 487–512. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-5930-9_27.
Der volle Inhalt der QuelleKnowlton, Anne A. „Estrogen, Cardiac Protection and Aging“. In Integrative Biology of Women’s Health, 157–74. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8630-5_9.
Der volle Inhalt der QuelleBolli, Roberto, und Piero Anversa. „Stem Cells and Cardiac Aging“. In Cardiovascular Regeneration and Stem Cell Therapy, 171–81. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470988909.ch18.
Der volle Inhalt der QuelleSeara, Fernando A. C., Leonardo Maciel, Tais Hanae Kasai-Brunswick, Jose H. M. Nascimento und Antonio C. Campos-de-Carvalho. „Extracellular Vesicles and Cardiac Aging“. In Advances in Experimental Medicine and Biology, 33–56. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-1443-2_3.
Der volle Inhalt der QuelleAdams, Kent J. „Cardiac Rehabilitation and Exercise“. In Handbook of Clinical Nutrition and Aging, 419–36. Totowa, NJ: Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-391-0_17.
Der volle Inhalt der QuelleZhao, Cuimei, Guoping Li und Jin Li. „Non-coding RNAs and Cardiac Aging“. In Advances in Experimental Medicine and Biology, 247–58. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1671-9_14.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Aging cardiac"
Lai, Yi-Homg, Yi Yuan, Yu-Xian Liang und Jia-Han Yu. „Comparison of aging effect between cardiac complexity and baroreceptor sensitivity“. In 2021 International Conference on Information Technology and Biomedical Engineering (ICITBE). IEEE, 2021. http://dx.doi.org/10.1109/icitbe54178.2021.00068.
Der volle Inhalt der QuelleDimitrakopoulos, Georgios N., Konstantina Dimitrakopoulou, Ioannis A. Maraziotis, Kyriakos Sgarbas und Anastasios Bezerianos. „Supervised method for construction of microRNA-mRNA networks: Application in cardiac tissue aging dataset“. In 2014 36th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2014. http://dx.doi.org/10.1109/embc.2014.6943593.
Der volle Inhalt der QuelleStarc, V., und T. T. Schlegel. „The effect of aging and cardiac disease on that portion of QT interval variability that is independent of Heart Rate Variability“. In 2008 35th Annual Computers in Cardiology Conference. IEEE, 2008. http://dx.doi.org/10.1109/cic.2008.4749041.
Der volle Inhalt der QuelleHeyne, E., R. Musleh, L. G. Koch, S. L. Britton, T. Doenst und M. Schwarzer. „Aging Is Not Related to Increased Cardiac Mitochondrial ROS Production in a Rat Model of Genetically Determined High or Low Exercise Capacity“. In 51st Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1742786.
Der volle Inhalt der QuelleZhingre Sanchez, Jorge D., Emma A. Schinstock, Michael G. Bateman und Paul A. Iaizzo. „The Development and Testing of a Fixation Apparatus for Inducing the Coaptation of the Cardiac Atrioventricular Valves“. In 2019 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/dmd2019-3298.
Der volle Inhalt der QuelleMen, Jing, Airong Li, Zhiwen Yang, Jixu Yu, Rudolph Tanzi und Chao Zhou. „Heartbeat recovery after cardiac arrest and association of maximum pace-able heart rate with aging via optogenetic pacing and OCM imaging of Drosophila (Conference Presentation)“. In Optical Coherence Tomography and Coherence Domain Optical Methods in Biomedicine XXIV, herausgegeben von Joseph A. Izatt und James G. Fujimoto. SPIE, 2020. http://dx.doi.org/10.1117/12.2549151.
Der volle Inhalt der QuelleCoelho, Patricia, Ana Rodrigues, Maria Vieira, Joana Liberal und Francisco Rodrigues. „The influence of Monfortinho thermal waters on human health“. In III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-241.
Der volle Inhalt der QuelleDuvall, Julia, Rachael Granberry, Lucy E. Dunne, Brad Holschuh, Christopher Johnson, Kevin Kelly, Bruce Johnson und Michael Joyner. „The Design and Development of Active Compression Garments for Orthostatic Intolerance“. In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3480.
Der volle Inhalt der QuelleFira, Monica, Liviu Goras, Victor-Andrei Maiorescu und Mihaela Catalina Luca. „Compressed Sensing and Classification of Cardiac Beats using Patient Specific Dictionaries“. In 2nd International Conference on Information and Communication Technologies for Ageing Well and e-Health. SCITEPRESS - Science and and Technology Publications, 2016. http://dx.doi.org/10.5220/0005793401730179.
Der volle Inhalt der QuelleSeong, Jaehoon, Ajay K. Wakhloo und Baruch B. Lieber. „Morphological Age-Depended Development of the Human Carotid Bifurcation“. In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2545.
Der volle Inhalt der QuelleBerichte der Organisationen zum Thema "Aging cardiac"
Slaughter-Acey, Jaime, Kathryn Behrens, Amy M. Claussen, Timothy Usset, Carrie Neerland, Sameerah Bilal-Roby, Huda Bashir et al. Social and Structural Determinants of Maternal Morbidity and Mortality: An Evidence Map. Agency for Healthcare Research and Quality (AHRQ), Dezember 2023. http://dx.doi.org/10.23970/ahrqepccer264.
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