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Thematische Bibliographien / (3) Rome I Regulation

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Auswahl der wissenschaftlichen Literatur zum Thema „(3) Rome I Regulation“

Autor: Grafiati

Veröffentlicht am 1. Juli 2021

Zuletzt aktualisiert am 1. Februar 2022

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Zeitschriftenartikel zum Thema "(3) Rome I Regulation"

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Rammeloo, Stephan. „‘From Rome to Rome’ – Cross-border employment contract. European Private International Law: Intertemporal law and foreign overriding mandatory laws“. Maastricht Journal of European and Comparative Law 24, Nr. 2 (April 2017): 298–322. http://dx.doi.org/10.1177/1023263x17709754.

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To what extent are Greek saving laws, resulting in payment cuts in the public sector (that is employment conditions), capable of overriding the applicable (German) law? A dispute arising from an employment relationship between the Greek Republic and an employee habitually carrying out work in Germany, gave rise to preliminary questions having regard to the temporal scope of EU Regulation No. 593/2008 (the ‘Rome I Regulation’)1 and, closely related thereto, the functional reach of Article 9(3) of that Regulation in respect of ‘foreign’ mandatory laws, in light of the principle of sincere cooperation enshrined in Article 4(3) TEU. An analysis of the Advocate General’s Opinion and the Court of Justice of the European Union’s (CJEU) ruling is followed by critical commentary and suggestions for future EU legislative amendments to the Rome I regime.

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Kronenberg, Alexander. „Foreign overriding mandatory provisions under the regulation (EC) No 593/2008 (Rome I Regulation). Judgment of the European Court of Justice of 18 october 2016, case c-135/15 = Leyes de policía de terceros estados en el ámbito del reglamento (CE) No 593/2008 (Reglamento Roma I). Comentario a la STJUE de 18 de octubre de 2016, asunto c-135/15“. CUADERNOS DE DERECHO TRANSNACIONAL 10, Nr. 2 (05.10.2018): 873. http://dx.doi.org/10.20318/cdt.2018.4409.

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Abstract: The role and treatment of foreign overriding mandatory provisions in international contract law have been subject to academic discussions for a long time. This has not changed with the introduction of Article 9 of the Rome I Regulation. In the judgment discussed in this case note, the European Court of Justice addressed some of the contentious issues in relation to Article 9(3) of the Rome I Regulation. This note examines and evaluates the solutions found by the ECJ and puts them into context. It also points out some questions the ECJ did not discuss; these questions remain open for now but will need to be addressed in the future.Keywords: Article 9(3) Rome I Regulation, foreign overriding mandatory provisions, conflict-of-law level consideration, substantive law level consideration, principle of sincere cooperation.Resumen: El tratamiento de las leyes de policía de terceros estados en derecho de contratos internacionales ha sido objeto de la polémica desde hace tiempo. Esto no ha cambiado con la entrada en vigor del artículo 9 del Reglamento Roma I. Con la sentencia comentada el Tribunal de Justicia de la Unión Europea ha tratado algunas de la cuestiones debatidas respecto al artículo 9.3 del Reglamento Roma I. Este comentario analiza, evalúa y pone en contexto las soluciones encontradas por el TJUE. También aborda las cuestiones que no han sido comentadas por el TJUE; estas cuestiones permanecen abiertas por el momento pero deberán ser examinadas en el futuro.Palabras clave: leyes de policía de terceros estados, consideración en nivel conflictual, consideración en nivel sustantivo, principio de cooperación leal.

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Folkard, Joshua. „THE EFFECT OF ROME II ON NATIONAL PROCEDURAL LAW“. Cambridge Law Journal 74, Nr. 1 (März 2015): 37–40. http://dx.doi.org/10.1017/s0008197315000215.

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AT common law, in cases where the substantive claim is governed by foreign law, questions of procedure are nonetheless governed by the lex fori. In the context of damages, although the existence of damage is a question for the lex causae, its quantification and assessment is determined according to the law of the forum (Boys v Chaplin [1971] A.C. 356). The distinction between substance and procedure is preserved by Article 1(3) of Council Regulation (EC) No 864/2007 (“Rome II”) which provides, with certain exceptions, that Rome II “shall not apply to evidence and procedure”. That rule is, however, qualified by Article 15, which requires the law applicable under the Regulation (i.e. “the law applicable to non-contractual obligations”) to govern, inter alia, “the existence, the nature and the assessment of damage or the remedy claimed” (Article 15(c)). The decision of the Court of Appeal in Wall v Mutuelle De Poitiers Assurances [2014] EWCA Civ 138; [2014] 3 All E.R. 340 concerns the definition of “procedure” in Article 1(3) and the meaning of “applicable law” in Article 15(c). It raises the important question of how far Rome II has encroached on the traditional view of national procedural autonomy in the conflict of laws.

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Lozano-Durán, Rosa, und Silke Robatzek. „14-3-3 Proteins in Plant-Pathogen Interactions“. Molecular Plant-Microbe Interactions® 28, Nr. 5 (Mai 2015): 511–18. http://dx.doi.org/10.1094/mpmi-10-14-0322-cr.

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14-3-3 proteins define a eukaryotic-specific protein family with a general role in signal transduction. Primarily, 14-3-3 proteins act as phosphosensors, binding phosphorylated client proteins and modulating their functions. Since phosphorylation regulates a plethora of different physiological responses in plants, 14-3-3 proteins play roles in multiple signaling pathways, including those controlling metabolism, hormone signaling, cell division, and responses to abiotic and biotic stimuli. Increasing evidence supports a prominent role of 14-3-3 proteins in regulating plant immunity against pathogens at various levels. In this review, potential links between 14-3-3 function and the regulation of plant-pathogen interactions are discussed, with a special focus on the regulation of 14-3-3 proteins in response to pathogen perception, interactions between 14-3-3 proteins and defense-related proteins, and 14-3-3 proteins as targets of pathogen effectors.

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Khudhair, Nagam, Yu Cuiping, Ahmed Khalid und Xuejun Gao. „Role 14-3-3 Protein in Regulation Some Cellular Processes“. Current Research Journal of Biological Sciences 6, Nr. 5 (20.11.2014): 197–204. http://dx.doi.org/10.19026/crjbs.6.5193.

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Banerjee, Hridesh, und Lawrence P. Kane. „Immune regulation by Tim-3“. F1000Research 7 (14.03.2018): 316. http://dx.doi.org/10.12688/f1000research.13446.1.

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T-cell immunoglobulin and mucin domain 3 (Tim-3) is a transmembrane protein that in both mice and humans has been shown to possess various functions in a context-dependent manner. Thus, Tim-3 has been associated with both inhibitory and co-stimulatory function, depending in part on the specific cell type and immune response course. Though originally described on T cells, Tim-3 is now known to be expressed by both lymphoid and non-lymphoid cells within the immune system and even by non-immune cells. In addition, though widely thought of as a negative regulator of immunity, Tim-3 has been shown in more recent studies to have a positive function on both myeloid and lymphoid cells, including T cells. Tim-3 is often expressed at a high level on exhausted T cells in tumors and chronic infection and may engage in crosstalk with other so-called “checkpoint” molecules such as PD-1. Thus, Tim-3 has emerged as a possible therapeutic target, which is being actively explored both pre-clinically and clinically. However, recent research suggests a more complex in vivo role for this protein, compared with other targets in this area.

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Jackson, S. P., C. L. Yap und K. E. Anderson. „Phosphoinositide 3-kinases and the regulation of platelet function“. Biochemical Society Transactions 32, Nr. 2 (01.04.2004): 387–92. http://dx.doi.org/10.1042/bst0320387.

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A clear understanding of the role of PI (phosphoinositide) 3-kinases in supporting the haemostatic function of platelets has been slow to evolve. In fact, insight into the roles of individual PI 3-kinase isoforms in platelet function remains rudimentary. However, based on in vitro studies using wortmannin and LY294002, there is evidence for an important role for PI 3-kinases in regulating a broad range of functional platelet responses, including primary platelet adhesion, cytoskeletal remodelling and platelet aggregation. One of the critical platelet responses involves affinity regulation of the major platelet integrin αIIbβ3, the primary receptor mediating platelet aggregation and thrombus growth. The input signals regulating integrin αIIbβ3 can be divided into three main groups: (1) Gq-coupled receptors linked to the activation of PLCβ (phospholipase Cβ); (2) Gi-coupled receptors linked to the regulation of adenylate cyclase and Rap1b; and (3) adhesion receptor signalling involving Src kinase-dependent activation of PLCγ isoforms. PI 3-kinases have not been demonstrated to play a critical role in Gq-dependent platelet activation; however, one or more PI 3-kinase isoforms appears to be important for Gi-dependent activation of Rap1b and adhesion receptor activation of PLCγ isoforms. Thus distinct co-operative PI 3-kinase signalling mechanisms appear to play an important role in regulating the adhesive function of integrin αIIbβ3.

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Jaroszek, Agata. „European Online Marketplace – New Measures for Consumer Protection against “Old Conflict of Laws Rules”“. Masaryk University Journal of Law and Technology 9, Nr. 1 (30.06.2015): 21–41. http://dx.doi.org/10.5817/mujlt2015-1-3.

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The paper aims at discussing the rationale for protecting consumers under the new directive on consumer rights (CRD) and its relation to conflict of law rules under the Regulation on the law applicable to contractual obligations (Rome I).The author is of the opinion the newly adopted legal framework for consumer protection under the directive on consumer rights seems to be more predictable especially in terms of supporting consumers with more mandatory information before the conclusion of a contract with a professional as well as a single 14 day withdrawal period for all Member States. However, the level of consumer protection in the purchase of digital content is insufficient and from the perspective of conflict of laws rules for consumer contracts under Rome I, a consumer who actively makes a purchase from a professional from another Member State or a third country cannot expect the special protective rules envisaged in the regime under CRD and Rome I to be applied by default; rather, the general rules come into play.

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Dulloo, A. G., S. Samec und J. Seydoux. „Uncoupling protein 3 and fatty acid metabolism“. Biochemical Society Transactions 29, Nr. 6 (01.11.2001): 785–91. http://dx.doi.org/10.1042/bst0290785.

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A role for uncoupling protein (UCP) 3 in fatty acid metabolism is reviewed within the context of our proposal, first put forward in 1998, that this homologue of UCP1 may be involved in the regulation of lipids as fuel substrate rather than in the mediation of thermogenesis. Since then, the demonstrations of muscle-type differences in UCP3 gene regulation in response to dietary manipulations (starvation, high-fat feeding) or to pharmacological interferences with the flux of lipid substrates between adipose-tissue stores and skeletal-muscle mitochondrial oxidation are all in accord with this proposed role for UCP3 in regulating lipids as fuel substrate. However, given the current limitations of gene-knockout technology for evaluating/interpreting the functional importance of genes encoding mitochondrial membrane proteins, the transition from ‘associative’ to ‘cause-and-effect’ evidence for a physiological role of UCP3 in regulating fatty acid metabolism will have to await the development of assays that are sensitive to changes in UCP3 activity. Furthermore, in evaluating the physiological regulators of UCP3, the available evidence points to the existence of adipose-derived factor(s) which, independently of circulating levels of free fatty acids, initiates events leading to the transcription of genes encoding UCP3 and key enzymes of lipid oxidation in the fast glycolytic or fast oxidative-glycolytic muscles, i.e. in the bulk of the skeletal-muscle mass. It is proposed that in tissues where UCP3 co-exists with UCP2 (skeletal muscle, brown adipose tissue, heart) they may act in concert in the overall regulation of lipid oxidation, concomitant to the prevention of lipid-induced oxidative damage.

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Trapani, Laura, und Valentina Pallottini. „Hypercholesterolemia and 3-Hydroxy 3-Methylglutaryl Coenzyme A Reductase Regulation during Ageing“. Scientific World JOURNAL 9 (2009): 564–74. http://dx.doi.org/10.1100/tsw.2009.81.

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We present here a brief description of the path that cholesterol covers from its intestinal absorption to its effect exerted on some enzyme regulation. Some mechanisms underlying hypercholesterolemia onset and, in particular, the role and the regulation of 3-hydroxy 3-methylglutaryl Coenzyme A reductase (HMGR) during adult life and during aging, have been described. In addition some pharmacological interventions to control proper HMGR regulation and, in turn, cholesterol homeostasis maintenance will be introduced.

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Dissertationen zum Thema "(3) Rome I Regulation"

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Lascelles, Carol Veronica. „Regulation of human ketogenesis : role of mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase“. Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627391.

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White, Nicholas Edward. „The role of Relaxin-3 and 3-Iodothyronamine in the regulation of energy homeostasis“. Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479156.

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Palacios, Arnold Raul. „Role of GSK-3 alpha beta in B cell proliferation during germinal center information“. Thesis, Boston University, 2013. https://hdl.handle.net/2144/21229.

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Thesis (M.A.) PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.
Glycogen Synthase Kinase-3αß is an enzyme that is involved in cell cycle regulation by promoting the degradation of cyclin D1 and cycling D3 in cells. Special emphasis is placed in its regulatory role in B cells, as there it is evidence that suggests that this protein is inhibited during germinal center formation, where B cells undergo proliferation, somatic hypermutation and class switch recombination. By inducing DNA recombination via the Cre/lLxP recombination system and utilizing tamoxifen as a Cre activity inducer, B cells were culture in 40LB cells to form induced germinal center in vitro. Flow cytometry analysis suggests that in the absence of GSK-3 αß B cells proliferate extensively in germinal centers and being the process of class switch recombination. Although the results of this study are in accord with current theory, more experiments and research need to be made to validate the conclusions set forth in this study.
2031-01-01

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Lindskog, Maria. „DARPP-32 in the striatum : multiple regulation and physiological role /“. Stockholm : Karolinska Univ. Press, 2001. http://diss.kib.ki.se/2001/91-7349-011-3/.

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Cooke, Frank T. „The regulation of phosphoinositide 3-OH kinase and its role in 3-phosphorylated inositol lipid metabolism in U937 cells“. Thesis, University of Cambridge, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263009.

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Lindgren, Niklas. „Regulation and physiological role of tyrosine hydroxylase phosphorylation in the striatum /“. Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-463-1/.

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Frantz, Aubrey Leigh. „THE ROLE OF INTESTINAL EPITHELIAL CELLS AND THE REGULATION OF THE POLYMERIC IMMUNOGLOBULIN RECEPTOR IN HOMEOSTASIS AND INFLAMMATION“. UKnowledge, 2012. http://uknowledge.uky.edu/microbio_etds/3.

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The mammalian intestine harbors an estimated 100 trillion microorganisms, which normally maintain a mutually beneficial relationship with the host. The intestinal epithelium consists of a single layer of intestinal epithelial cells (IECs) that provides a physical barrier as well as innate immune defense, preventing this vast community of microbes from entering host tissues. Secretory immunoglobulin A (SIgA) acts as the first line of antigen-specific immunity at the interface between the gut microbiota and the intestinal epithelium. Polymeric IgA secreted by plasma cells in the intestinal lamina propria is transported across IECs by the polymeric immunoglobulin receptor (pIgR). Defects in epithelial barrier and immune functions can lead to infections with opportunistic and pathogenic microbes and contribute to the etiology of inflammatory bowel disease (IBD). Here we investigate the ability of IEC biomarkers to define the mechanism and severity of intestinal inflammation, as well as provide insight into the function of IEC in regulating intestinal homeostasis and inflammation. Importantly, down-regulation of pIgR expression was a common feature in human IBD and mouse models of experimental colitis. One molecule of pIgR is consumed for every molecule of SIgA transported, thus high expression of pIgR is required to maintain sufficient supply of SIgA. Accordingly, we investigate the mechanisms by which IECs regulate pIgR expression in response to colonic bacteria. Cross-talk between the microbiota and IECs is mediated by pattern recognition receptors, including Toll-like receptors (TLR), leading to expression of gene products that enhance epithelial barrier function and innate immunity. The cytoplasmic adaptor protein MyD88 transduces signals from TLRs that recognize bacterial products. We show that pIgR induction by colonic bacteria is dependent on TLR4-MyD88 activation of NF-κB signaling. We examined the role of epithelial-specific MyD88 signaling in antibacterial immunity and epithelial expression of key gene products that participate in innate immunity in the gut by generating mice with an IEC-targeted deletion of the Myd88 gene (MyD88ΔIEC). MyD88ΔIEC mice display immunological and antimicrobial defects resulting in increased susceptibility to experimental colitis. We conclude that cross-talk between bacteria and IECs via MyD88-dependent signaling is crucial for maintenance of gut homeostasis.

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Deshpande, Anagha. „Role of 3’UTR Elements in the Regulation of the Cyclin D1 Proto-oncogene“. Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-103346.

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Opel, Daniela. „Role of phosphoinositide-3-kinase (PI3K)/Akt signaling in apoptosis regulation of neuroectodermal tumors“. [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-63916.

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Seccareccia, Erica. „Studies on the regulation of the matrix metalloproteinase-3 and its role in metastasis“. Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=117079.

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Cancer metastasis is the major cause of death from malignant disease. The biological factors that dictate the secondary site(s) of a primary tumor remain largely unknown, yet their understanding would greatly facilitate the management of malignancy. Matrix metalloproteinases (MMPs) are responsible for the degradation of the extracellular matrix and regulate the tumor microenvironment. The expression of specific MMPs in primary tumors has been correlated with the extent and site of metastatic disease and may therefore potentially be predictive of the preferred site of metastasis. In this study, a subline of the Lewis lung carcinoma (M27 cells) was used to study the role of MMP-3 in lung metastasis as well as elucidate the signaling pathways that regulate its expression in a cellular context where expression of the insulin like growth factor-I receptor (IGF-IR) is altered. Previous work demonstrated that following ectopic expression of the IGF-IR in M27 cells (M27R cells) MMP-3 expression was downregulated and this coincided with a loss of lung metastasis. Here it is shown that the ectopic expression of the IGF-IR downregulates the expression of IκB kinase ε (IKKε). Reconstitution of IKKε expression (M27R/IKKε cells), partly restored MMP-3 expression in M27R cells and furthermore, it sensitized MMP-3 expression levels to induction by phorbol 12-myristate 13-acetate (PMA). This induction was, in turn, dependent on protein kinase C (PKC) α and the transcription factor p65. Finally, reconstituting MMP-3 expression in M27R cells enabled their metastasis to the lungs. Collectively, our results implicate IKKε as a regulator of MMP-3 expression and implicate MMP-3 in lung metastasis.
Les métastases sont responsables de la majorité des décès causé par les cancers. Les facteurs biologiques qui déterminent le site des tumeurs secondaires ne sont pas bien compris, mais pourraient aider au traitement de cette maladie. Les métalloproteinases de la matrice (MMPs) sont responsables de la dégradation de la matrice extracellulaire et en plus, elles sont des régulateurs du microenvironnement. Une corrélation a déjà été montrée entre le profil des MMPs chez les tumeurs primaires et les sites où vont s'implanter les métastases. Dans cette étude, on a examiné le rôle de la MMP-3 dans la capacité à former des métastases d'une lignée cellulaire dérivée de carcinome des poumons de Lewis (M27). De plus, on a examiné les signaux de transduction responsables de la régulation de la MMP-3 dans un environnement où l'expression du récepteur au facteur de croissance à l'insuline (IGF-I) était altérée. Précédemment, on a demontré que l'expression de l'IGF-IR (M27R) régulait négativement l'expression de la MMP-3 et ceci a abrogé les métastases aux poumons. Ici, on démontre que l'expression d'IGF-IR régule négativement l'expression de la kinase IκB ε (IKKε). La surexpression d'IKKε dans les cellules M27R (M27R/IKKε), reconstitue partiellement l'expression de MMP-3 dans les cellules M27R. En addition, l'expression de MMP-3 dans ces cellules peuvent être stimulées suite à l'ajout de phorbol 12-myristate 13-acetate (PMA). Cette stimulation dépendait sur la kinase des protéines C (PKC) α et sur le facteur de transcription, p65. Finalement, la reconstitution de l'expression de la MMP-3, dans les cellules M27R, a permis à celles-ci de former des métastases dans les poumons. L'ensemble de nos résultats démontre qu'IKKε participe dans la régulation de l'expression de la MMP-3 et de plus que la MMP-3 serait impliquée dans la formation des métastases aux niveaux des poumons.

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Bücher zum Thema "(3) Rome I Regulation"

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Congress, International Neuromodulation Society International. First International Congress: Rome-Italy, May 3/6/1992. Bologna: Monduzzi Editore, 1993.

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New York University. Center for Transnational Litigation, Arbitration and Commercial Law und European Parliament, Hrsg. Rome I Regulation. München: Sellier European Law Publishers, 2015.

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Ferrari, Franco, Hrsg. Rome I Regulation. Berlin, München, Boston: DE GRUYTER, 2014. http://dx.doi.org/10.1515/9783866539785.

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Parliament, European, Hrsg. Rome II Regulation: Pocket commentary. Munich: Sellier European Law Publishers, 2011.

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Great Britain. Parliament. House of Lords. European Union Committee. The Rome II regulation: Report with evidence. London: Stationery Office, 2004.

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Wright, Maurice. Build up your exports 3. 3. Aufl. Milton Keynes: Tate, 1995.

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J, Woodman A., und Martin Ronald 1915-, Hrsg. The annals of Tacitus: Book 3. Cambridge: Cambridge University Press, 1996.

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Rome Convention-Rome I Regulation: Commentary : new EU conflict-of-laws rules for contractual obligations. Huntington, N.Y: Juris, 2010.

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Vasara, Tuija. Functional analysis of the RHOIII and 14-3-3 proteins of Trichoderma reesei. Espoo [Finland]: Technical Research Centre of Finland, 2002.

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Milton, A. S., Hrsg. Temperature Regulation. Basel: Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-8491-4.

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Buchteile zum Thema "(3) Rome I Regulation"

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Boulant, Jack A. „Neurophysiology of Thermoregulation: Role of Hypothalamic Neuronal Networks“. In Temperature Regulation, 93–101. Basel: Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-8491-4_16.

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Yahata, Takehiro, und Akihiro Kuroshima. „Role of Prolactin in Brown Adipose Tissue Thermogenic Activity“. In Temperature Regulation, 327–31. Basel: Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-8491-4_53.

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Dickinson, Andrew. „Territory in the Rome I and Rome II Regulations“. In The Hamburg Lectures on Maritime Affairs 2011-2013, 69–125. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-55104-8_4.

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Kunda, Ivana. „Law Applicable to Employment Contracts Under the Rome I Regulation“. In Transnational, European, and National Labour Relations, 73–96. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-02219-2_5.

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Campo Comba, María. „Other Possible Weaker Contracting Parties and the Rome I Regulation“. In The Law Applicable to Cross-border Contracts involving Weaker Parties in EU Private International Law, 311–38. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-61481-2_6.

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Lemma, Valerio. „European Approaches to Fintech: The Role of Regulation and the Evolution of Supervision“. In FinTech Regulation, 143–243. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42347-6_4.

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Timan, Tjerk, Charlotte van Oirsouw und Marissa Hoekstra. „The Role of Data Regulation in Shaping AI: An Overview of Challenges and Recommendations for SMEs“. In The Elements of Big Data Value, 355–76. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68176-0_15.

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AbstractIn recent debates around the regulation of artificial intelligence, its foundations, being data, are often overlooked. In order for AI to have any success but also for it to become transparent, explainable and auditable where needed, we need to make sure the data regulation and data governance around it is of the highest quality standards in relation to the application domain. One of the challenges is that AI regulation might – and needs to – rely heavily on data regulation, yet data regulation is highly complex. This is both a strategic problem for Europe and a practical problematic: people, institutions, governments and companies might increasingly need and want data for AI, and both will affect each other technically, socially but also regulatory. At the moment, there is an enormous disconnect between regulating AI, because this happens mainly through ethical frameworks, and concrete data regulation. The role of data regulation seems to be largely ignored in the AI ethics debate, Article 22 GDPR being perhaps the only exception. In this chapter, we will provide an overview of current data regulations that serve as inroads to fill this gap.

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Kosaka, M., J. M. Lee, G. J. Yang, T. Matsumoto, K. Tsuchiya, N. Ohwatari und M. Shimazu. „The Role of Heat Shock Proteins (HSPs) and Interleukin-1 Interaction in Suppression of Fever“. In Temperature Regulation, 35–39. Basel: Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-8491-4_6.

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Tarasiuk, Anna. „European Private Law (Regulation Rome I) and On-Line Insurance Contracts“. In The "Dematerialized" Insurance, 361–91. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-28410-1_14.

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O’Shea, Peter. „The Role of Transgovernmental and Transnational Dialogue in Financial Markets Cooperation“. In Transatlantic Financial Regulation, 103–32. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-74855-5_5.

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Konferenzberichte zum Thema "(3) Rome I Regulation"

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Minten, Elizabeth, Hui Zhang, Chunyang Li, PamelaSara Head und David S. Yu. „Abstract 1751: The role of SIRT2 in BRCA1 regulation“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1751.

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Minten, Elizabeth, Hui Zhang, Chunyang Li, PamelaSara Head und David S. Yu. „Abstract 1751: The role of SIRT2 in BRCA1 regulation“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1751.

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Paul, Manash K., Arkaprabha Basu, Bharti Bisht, Paul Pagano, Yari Fontebasso, Kostyantyn Krysan, Linh Tran et al. „Abstract 1030: Regulation and role of actin dynamics in promoting premalignant cell migration“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1030.

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Paul, Manash K., Arkaprabha Basu, Bharti Bisht, Paul Pagano, Yari Fontebasso, Kostyantyn Krysan, Linh Tran et al. „Abstract 1030: Regulation and role of actin dynamics in promoting premalignant cell migration“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1030.

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Stovall, Daniel B., Meimei Wan, Lance D. Miller, Paul Cao, Qiang Zhang, Martha Stampfer, Wennuan Liu, Jianfeng Xu und Guangchao Sui. „Abstract B025: The role and regulation of SOX7 in breast cancer“. In Abstracts: AACR Special Conference on Advances in Breast Cancer Research: Genetics, Biology, and Clinical Applications - October 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1557-3125.advbc-b025.

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Jenkins, Brittany D., Rachel Martini, Kevin Gardner, Michele Monteil, Dorrah Deeb, Lisa Newman und Melissa Davis. „Abstract 4565: The functional role of atypical chemokine receptor 1 in immune cell regulation of breast cancer“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4565.

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Daulagala, Amanda C., Catherine Bridges, Lauren Rutledge, Joyce Nair-Menon, Micheal Yost und Antonis Kourtidis. „Abstract 64: Regulation and functional role of the cell-cell junction-associated RNAi machinery in colon cancer“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-64.

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Deliard, Sandra, Yasuyuki Okamoto, Jozef Madzo, Somnath Pandey, Jaroslav Jelinek und Jean-Pierre Issa. „Abstract 4331: Potential role of the splicing factor SF3B1 in epigenetic regulation and activation of p53 signaling“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4331.

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Deliard, Sandra, Yasuyuki Okamoto, Jozef Madzo, Somnath Pandey, Jaroslav Jelinek und Jean-Pierre Issa. „Abstract 4331: Potential role of the splicing factor SF3B1 in epigenetic regulation and activation of p53 signaling“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4331.

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Pramanik, Suravi, Shrabasti Roychoudhury, Hannah Harris, Heyu Song und Kishor K. Bhakat. „Abstract 4495: Role of base excision repair (BER) pathway in regulation of KRAS expression in pancreatic cancer“. In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4495.

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Berichte der Organisationen zum Thema "(3) Rome I Regulation"

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Toby, Garabet G., und Pamela Silver. Chemical Genetics of 14-3-3 Regulation and Role in Tumor Development. Fort Belvoir, VA: Defense Technical Information Center, November 2006. http://dx.doi.org/10.21236/ada463902.

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Moore, Gary. Summer Research Program -- 1995, Summer Research Extension Program. Volume 3, Rome Laboratory. Fort Belvoir, VA: Defense Technical Information Center, Mai 1996. http://dx.doi.org/10.21236/ada387106.

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Moore, Gary. Summer Research Program - 1996 Summer Research Extension Program Volume 3 Rome Laboratory. Fort Belvoir, VA: Defense Technical Information Center, Dezember 1996. http://dx.doi.org/10.21236/ada386929.

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Moore, Gary. Summer Research Program - 1999. Summer Research Extension Program. Final Reports. Volume 3, Rome Laboratory. Fort Belvoir, VA: Defense Technical Information Center, Dezember 1999. http://dx.doi.org/10.21236/ada387259.

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Moore, Gary. Summer Research Program - 1998. Summer Research Extension Program, Final Reports. Volume 3, Rome Laboratory. Fort Belvoir, VA: Defense Technical Information Center, Dezember 1998. http://dx.doi.org/10.21236/ada387299.

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Moore, Gary. 1997 Summer Research Program (SRP), Summer Research Extension Program (SREP), Final Report, Volume 3. Rome Laboratory. Fort Belvoir, VA: Defense Technical Information Center, Dezember 1997. http://dx.doi.org/10.21236/ada386997.

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Jerry D. Cohen. Metabolic regulation of the plant hormone indole-3-acetic acid. Office of Scientific and Technical Information (OSTI), November 2009. http://dx.doi.org/10.2172/966706.

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Kim, Hyeong-Reh C. Galectin-3 in the Regulation of Apoptosis Induced by Loss of Cell-Matrix Interactions. Fort Belvoir, VA: Defense Technical Information Center, August 2002. http://dx.doi.org/10.21236/ada411400.

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Yi, Ping. The Regulation of Nuclear Receptor Coactivator SRC-3 Activity Through Membrane Receptor Mediated Signaling Pathways. Fort Belvoir, VA: Defense Technical Information Center, Mai 2005. http://dx.doi.org/10.21236/ada460836.

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von Arnim, Albrecht G. Eukaryotic initiation factor 3 (eIF3) and 5’ mRNA leader sequences as agents of translational regulation in Arabidopsis. Final report. Office of Scientific and Technical Information (OSTI), Februar 2015. http://dx.doi.org/10.2172/1169186.

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